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1 ighting the spread to epithelia as an attractive target for therapeutic strategies.
2 g pathogenic mechanisms underlying AS and identifying novel therapeutic strategies.
3 nding of natural gene circuits and the design of cell-based therapeutic strategies.
4 for both IST and BMT, and must be considered when designing therapeutic strategies.
5 ical processes and has a crucial impact in developing novel therapeutic strategies.
6 in part a driving force in developing future diagnostic and therapeutic strategies.
7 hs, underscoring the importance of developing effective new therapeutic strategies.
8 AC) remains a highly lethal disease in critical need of new therapeutic strategies.
9 vidence-based examinations that are available to help guide therapeutic strategies.
10 These findings may guide the development of new therapeutic strategies.
11 ks of existing therapies emphasise the necessity to improve therapeutic strategies.
12 tant implications regarding the design of novel miRNA-based therapeutic strategies against angiogenesis.
13 for caspase-3 in the neurobiology of tau, and suggest that therapeutic strategies aimed at inhibiting this protease-depe
14 tcomes has yielded progress toward the development of novel therapeutic strategies and are summarized.
15 otential in inhibiting AXL for the development of different therapeutic strategies and inhibitors.
17 erefore present a significant challenge for preventative or therapeutic strategies based on broadly neutralizing antibodi
18 theranostics, the ultimate vision of synchronising tailored therapeutic strategies based on specific diagnostic data, dem
19 Defining pathways for amyloid assembly could impact therapeutic strategies for as many as 50 disease states.
21 d suggest it could be a target for the development of novel therapeutic strategies for GVHD treatment.
22 rate together demonstrate that the development of efficient therapeutic strategies for IPF is an important future endeavo
23 rotective potential of HSJ1, and deserve future interest as therapeutic strategies for neurodegenerative disease.
24 ing has led to the development of effective mechanism-based therapeutic strategies for patients with histiocytic diseases
25 ptional modules may open new avenues for the development of therapeutic strategies for progressive MS.
27 egulates JAK2 in normal and malignant HSPCs and suggest new therapeutic strategies for treating CBL(mut) myeloid malignan
29 portance of subtypes and genotypes in designing nonsurgical therapeutic strategies for uterine leiomyoma.
31 provement of endothelial NO production represent reasonable therapeutic strategies in addition to the treatment of establ
33 drug carriers at the 'nano'-scale is providing exciting new therapeutic strategies in clinical management of cancer beyon
34 and RNA-interference approaches are emerging as attractive therapeutic strategies in neurological diseases.
37 stem cells (MSCs) are regularly utilized for translational therapeutic strategies including cell therapy, tissue enginee
38 Vs may be efficient tools for in utero cord transduction in therapeutic strategies such as for treatment of inherited ear
39 the pathogenetic basis of L-ORD has implications for future therapeutic strategies such as gene augmentation therapy.
40 eath in maintenance of T-cell homeostasis and outline novel therapeutic strategies tailored to manipulate cell death to l
42 (LSC) niche dependencies might be exploited to devise novel therapeutic strategies that aim at disrupting essential niche
45 bacterial infections is critical for the development of new therapeutic strategies that resolve infectious inflammation w
46 neity in patient outcome and a need for rationally designed therapeutic strategies that target disease biology.
48 These observations have encouraged the development of therapeutic strategies to treat and prevent telomere-associat
49 ma cells have translated to only limited success; effective therapeutic strategies will need also to target elements of t
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