ライフサイエンスコーパス: thermal injury

1 mortality caused by sepsis is high following thermal injury.

2 ed therapeutic options, but without inducing thermal injury.

3 ed in the process of cytoprotection from the thermal injury.

4 ing insulin receptor signaling in rats after thermal injury.

5 responsible for the insulin resistance after thermal injury.

6 ambient temperature on metabolic rate after thermal injury.

7 of morbidity and mortality in patients after thermal injury.

8 ophy, Stevens-Johnson syndrome, and chemical/thermal injury.

9 and also transfer the heat created to avoid thermal injury.

10 e perineurium and epineurium consistent with thermal injury.

11 ut potentially lethal complication of severe thermal injury.

12 tive agent against damage after experimental thermal injury.

13 significant levels between 7 and 24 h after thermal injury.

14 ute to observed cerebral edema in peripheral thermal injury.

15 up was quantified at 3, 7, 24, and 72 h post thermal injury.

16 eg muscle for up to 24 months after a severe thermal injury.

17 10, p < 0.03) and attenuated the sequelae of thermal injury.

18 lycan present on many host cells involved in thermal injury.

19 of morbidity and mortality in the setting of thermal injury.

20 on in primarily cultured retinal cells after thermal injury.

21 n baby syndrome, and pediatric nonaccidental thermal injury.

22 y be triggered by MCP-1 produced early after thermal injury.

23 ss acute bacterial models of sepsis or after thermal injury.

24 P-1 into their culture fluids 12 hours after thermal injury.

25 th a mechanism similar to that reported with thermal injury.

26 mechanical hyperalgesia generated by a mild thermal injury.

27 these difficult cases of severe periorbital thermal injuries.

28 ty to infection in individuals with severely thermal injuries.

29 observed in animals and patients with severe thermal injuries.

30 especially in patients with major trauma or thermal injuries.

31 irless mice were subjected to full-thickness thermal injury (30% of total body surface area), cold st

32 l exudates of mice 2 days after third-degree thermal injuries affecting 15% total body surface area.

33 Propranolol treatment for 12 months after thermal injury, ameliorates the hyperdynamic, hypermetab

34 In a mouse model of thermal injury, an increase in burn size produced a decr

35 Thermal injuries and endotoxemia have been shown to indu

36 eratopathy occurred after severe periorbital thermal injuries and followed a predictable course of sc

37 surface for patients with severe periorbital thermal injuries and resultant exposure keratopathy.

38 c ischemia and reperfusion injury induced by thermal injury and endotoxemia by improving mesenteric b

39 tio, showed a 7- and 10-fold elevation after thermal injury and LPS, respectively.

40 o, showed 6-fold and 12-fold increases after thermal injury and LPS, respectively.

41 attenuates the hypermetabolic response after thermal injury and may improve the clinical outcome.

42 This produced irreversible thermal injury and necrosis.

43 motic stress and helps protect cells against thermal injury and oxygen radicals.

44 resent important modifiable risk factors for thermal injury and poisoning but not fractures in presch

45 the premise that enhanced monocytopoiesis in thermal injury and sepsis results from an imbalance in m

46 pmental hierarchy of bone marrow cells after thermal injury and sepsis was determined by assessing th

47 by norepinephrine and the milieu created by thermal injury and sepsis.

48 e pathogenesis of P. aeruginosa infection of thermal injury and that syndecan 1-neutralizing agents m

49 at MCP-1 is produced in mice within 1 day of thermal injury, and the subsequent development of burn-a

50 Thermal injury at the camera trocar incision was lower i

51 the mechanical sensitivity caused by a mild thermal injury by both GBP and IBG.

52 he liver plays an important role in a severe thermal injury by modulating immune function, inflammato

53 medically recorded injury, comprising 3,649 thermal injury cases, 4,050 fracture cases and 2,193 poi

54 ignificantly increased as early as 3 h after thermal injury compared to controls, remained at 7 h (p<

55 nic effect on hepatocytes 1 and 2 days after thermal injury compared with controls (p<.05).

56 We previously showed that thermal injury depletes plasma vitamin E in pediatric bu

57 The primary outcome variable was thermal injury determined by histology from skin biopsie

58 Temperature measurements indicated that thermal injury did not contribute to tumor response.

59 cells from burned mice (6 h to 3 days after thermal injury) did not produce significant amounts of M

60 s (16 eyes) who sustained severe periorbital thermal injuries during combat missions in Iraq and Afgh

61 oiding this complication, which is caused by thermal injury during ablation.

62 sue may serve to insulate the esophagus from thermal injury, explaining why atrioesophageal fistulas

63 (THIN) database to identify risk factors for thermal injury, fractures and poisoning in pre-school ch

64 ord from the active electrode cord decreases thermal injury from antenna coupling at the camera troca

65 During the initial phase of thermal injury gene expression profiles in the liver may

66 horda tympani section > trigeminal section > thermal injury = glossopharyngeal section > greater supe

67 n (1 to 16 years of age) sustaining a severe thermal injury (> or =40% TBSA) were included into the s

68 Recent research in the metabolic response to thermal injury has identified many potentially beneficia

69 A thermal injury, however, causes hepatic damage by induci

70 were increased between 36 and 48 hours after thermal injury in platelet-deficient mice compared with

71 of granule contents occurred by 2 min after thermal injury in wild-type (WT) C57BL/6 mice and in the

72 ed with increased BBB permeability following thermal injury, indicates that MMP-9 may contribute to o

73 Thermal injury induces a hypermetabolic state, which is

74 Severe thermal injury is associated with extreme and prolonged

75 suggest that the hypermetabolic response to thermal injury is maximal in burns as small as 20% total

76 despite adequate nutritional support, severe thermal injury leads to decreased anabolic hormones over

77 A variety of chemical, physical, and thermal injuries may involve the gingiva.

78 Mortality after serious systemic thermal injury may be linked to significant increases in

79 ously established a mast cell (MC)-dependent thermal injury model in mice with ulceration and scar fo

80 sruption of microvascular integrity in a rat thermal injury model is associated with gelatinase expre

81 Previous findings using a rat thermal injury model suggested that re-epithelialization

82 e active electrode/camera cords would reduce thermal injury occurring at the camera trocar incision i

83 sophageal fistula can develop as a result of thermal injury of the esophagus during ablation along th

84 of this study was to evaluate the effect of thermal injury on novel haematological parameters and to

85 sed the mechanical hyperalgesia induced by a thermal injury or the TRPV4-selective agonist 4alpha-PDD

86 city of the drugs used in protocols to treat thermal injury patients.

87 hologic findings consistent with athermal or thermal injury, respectively, such as axonal swelling, a

88 used in the treatment of acne scarring, with thermal injury resulting in collagen synthesis and remod

89 Thermal injury results in a number of physiologic altera

90 Here we show for the first time that thermal injury results in shedding of syndecan 1 from ho

91 exhibited RF-induced coagulation columns of thermal injury, separately generated around each microne

92 one (rhGH), given to children after a severe thermal injury, successfully improved lean muscle mass,

93 However, thermal injury suppresses many of these factors, and the

94 nt modifiable risk factor for poisonings and thermal injuries (tests for trend p </= 0.001) as were h

95 on blood samples acquired on the day of the thermal injury to 12 months post-injury in 39 patients (

96 group, there was significantly less (P <.05) thermal injury to biliary epithelium in the chilled sali

97 this study, we utilized the murine model of thermal injury to examine the contribution of hepP to th

98 Selective thermal injury to follicles was observed histologically,

99 Tissue transplantation for conditions from thermal injury to Parkinson disease is being investigate

100 ser pulses, microscopy revealed preferential thermal injury to sebaceous follicles and glands, consis

101 n additional group was studied that received thermal injury to the anteroventral tongue.

102 Following severe traumatic or thermal injury to the dermis, hypertrophic scars (HTSs)

103 Following severe traumatic or thermal injury to the dermis, hypertrophic scars (HTSs)

104 hological evaluation revealed no evidence of thermal injury to the myocardium.

105 Paradoxically, we found that thermal injury to the skin induced a robust time-depende

106 activity found in PMNs from individuals with thermal injury was associated with a specific, quantitat

107 but leaflets of residual tissue remained and thermal injury was noted in all samples.

108 f heat-shock proteins can protect cells from thermal injury, we tested whether the proteasome inhibit

109 y type; compared with children under 1 year, thermal injuries were highest in those age 1-2 (OR = 2.4

110 of apoptosis and accidental cell death after thermal injury were evaluated in normal human epidermal

111 Alterations in gene expression unique to a thermal injury were identified.

112 No histologic changes consistent with thermal injury were observed in blood vessels or collage

113 15 years with LSCD secondary to chemical or thermal injury who underwent CLET from April 1, 2001, th

114 In thermal injury with superimposed sepsis, neutropenia and

115 failure and improve clinical outcomes after thermal injury without any detectable adverse side effec