ライフサイエンスコーパス: thermosensitivity

1 with Ser322 phosphomutants displayed altered thermosensitivity.

2 so identifying portable modules that specify thermosensitivity.

3 e region that robustly reduces the channel's thermosensitivity.

4 at of wild-type levels and further increases thermosensitivity.

5 en firing rate thermosensitivity and current thermosensitivity.

6 stitute the underlying mechanism of neuronal thermosensitivity.

7 m-induced depolarization determines neuronal thermosensitivity.

8 spore viability, slow growth, and increased thermosensitivity.

9 ive phenotype but partially rescued ydj1-151 thermosensitivity.

10 , suggesting a likely cause of the increased thermosensitivity.

11 of Akt or ERK1 and -2 kinases increased cell thermosensitivity.

12 here were reductions in both firing rate and thermosensitivity.

13 bicuculline had no change in firing rate or thermosensitivity.

14 exhibited reductions in both firing rate and thermosensitivity.

15 ns, although these neurons had a higher mean thermosensitivity.

16 t exhibit unique substrate specificities and thermosensitivities.

17 there was no correlation between firing rate thermosensitivity and current thermosensitivity.

18 drogel to investigate effects on the polymer thermosensitivity and gelation properties.

19 ivation by amylopectin, response to citrate, thermosensitivity and the processivity of glucan chain e

20 s between neuronal activity (firing rate and thermosensitivity) and tissue survival as a function of

21 major differences in sequence specificities, thermosensitivities, and structural features, all orthol

22 es: spontaneous DNA damage, chromosome loss, thermosensitivity, and acute sensitivity to genotoxic ag

23 There were no differences in firing rate or thermosensitivity between 350 and 450 microm slices; but

24 These findings indicate that reducing thermosensitivity can be critical for TRP channel functi

25 specialized class of neurons, the degree of thermosensitivity can be strongly modulated by synaptic

26 iciency and near elimination of the splicing thermosensitivity characteristic of MuSVts110 were obser

27 osensory-specific TRPA1 isoform with reduced thermosensitivity compared to the previously described i

28 Changes in POAH neuronal thermosensitivity could be a component of the mechanism

29 he present study compared discharge rate and thermosensitivity (discharge rate change/degree C) of WS

30 discharge in NREM sleep exhibited increased thermosensitivity during NREM sleep compared to wakefuln

31 he dorsal HDB, 65% of the neurons maintained thermosensitivity during SB.

32 ignificant differences in membrane potential thermosensitivity for the different neuronal types.

33 The change in thermosensitivity from wakefulness to NREM sleep was cor

34 that gene deletions resulting in the highest thermosensitivity generally are not the same as those tr

35 TTX decreased the current thermosensitivity in most neurones, but there were no re

36 charge during NREM sleep exhibited decreased thermosensitivity in NREM sleep.

37 P amplitude contributed to an enhancement of thermosensitivity in some neurones.

38 f TOC1 and its close homologue PRR5 restores thermosensitivity in the evening, whereas TOC1 overexpre

39 died, 37 (25%) neurons met the criterion for thermosensitivity including 17 warm-sensitive (WSNs) and

40 ed9-1 double mutant by restoring germination thermosensitivity, indicating that both NCED4 genes enco

41 is supports studies suggesting that neuronal thermosensitivity is controlled, not by resting currents

42 The thermosensitivity of 24 WSNs and 31 CSNs from the medial

43 d to determine the mechanisms underlying the thermosensitivity of alpha2C-ARs.

44 have identified mutations that suppress the thermosensitivity of an ipl1-2 mutant.

45 f those mutant groES alleles to suppress the thermosensitivity of bacteria bearing the dnaA46 mutatio

46 minCDE might at least partially suppress the thermosensitivity of ftsZ84, which can form colonies bel

47 and 1.0 microM doses) on the firing rate and thermosensitivity of HDB neurons.

48 We studied the thermosensitivity of neurons in the rat horizontal limb

49 the Q(A)/Q(A)(-) redox couple and increased thermosensitivity of photosystem II (PSII), suggesting s

50 pable of either enhancing or suppressing the thermosensitivity of SCN neurones.

51 dosage of the division gene zipA suppresses thermosensitivity of the ftsZ84 mutant by stabilizing th

52 The thermosensitivity of the neurons in the diagonal band of

53 The ability to tune the thermosensitivity of the polymer in order to compensate

54 d here indicate that the previously reported thermosensitivity of the respiratory complexes of abc1/c

55 orylation of Orp2 suppress or exacerbate the thermosensitivity of the spb70 mutants, respectively, in

56 iciency in coenzyme Q had a much more severe thermosensitivity phenotype for these oxidative endpoint

57 To study the basic mechanisms of neuronal thermosensitivity, rat hypothalamic tissue slices were u

58 SSE1-dependent suppression of ydj1-151 thermosensitivity required the wild-type ATP-binding dom

59 c efficiency at low temperatures and greater thermosensitivity than their mesophilic counterparts.

60 nal mutations cause basal, but not acquired, thermosensitivity that occurs in conjunction with hypera

61 Of 34 neurons tested for thermosensitivity under SB, 11 were WS, 4 were CS and 19

62 ecular level, GR28B(D) misexpression confers thermosensitivity upon diverse cell types, suggesting th

63 We found that AFD thermosensitivity, which exceeds that of most biological

64 PIF4 target genes show circadian rhythms of thermosensitivity, with minimum responsiveness in the ev