ライフサイエンスコーパス: thiamine deficiency

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1 lsive status epilepticus, endocrinopathy, or thiamine deficiency.

2 chanism, which appears to be up-regulated in thiamine deficiency.

3 cofactors, particularly a low-protein diet, thiamine deficiency, alcoholism, and hypothyroidism.

4 occur during the acute and chronic phases of thiamine deficiency and describe how rodent models of We

5 extending the focus from lethal to sublethal thiamine deficiency, and by linking biochemical alterati

6 Undiagnosed and untreated thiamine deficiencies are often fatal or lead to severe

7 mical and cognitive deficits associated with thiamine deficiency as well as proven useful toward grea

8 us on day 10 of PTD treatment, a duration of thiamine deficiency associated with perivascular edema i

9 ley rats were assigned to one of 4 stages of thiamine deficiency based on behavioral symptoms: pre-sy

10 ignificantly, these results demonstrate that thiamine deficiency causes selective cholinergic dysfunc

11 Thiamine deficiency disorders, such as thiamine-responsi

12 rrier (BBB) breakdown in the pathogenesis of thiamine deficiency encephalopathy was investigated in R

13 RBE4 cells may be important early events in thiamine-deficiency encephalopathy.

14 Primary and secondary conditions leading to thiamine deficiency have overlapping features in childre

15 Rodent models of thiamine deficiency have yielded significant insight int

16 ent enzymes, revealed episodically occurring thiamine deficiency in all three animal classes.

17 supplementation may be necessary to correct thiamine deficiency in breastfed infants.

18 defect of TRMA may shed light on the role of thiamine deficiency in such common diseases.

19 iovascular traits previously associated with thiamine deficiency, including elevated cardiac stroke v

20 ces from granulocytes may be responsible for thiamine deficiency-induced vascular breakdown and periv

21 It is unlikely that the thiamine deficiency is caused by impaired phosphorylatio

22 Thiamine deficiency is common in parts of Asia and cause

23 effects, we demonstrate that the problem of thiamine deficiency is considerably more widespread and

24 ative role of the BBB in the pathogenesis of thiamine deficiency is discussed.

25 In those with baseline thiamine deficiency, patients in the thiamine group had

26 The effects of thiamine deficiency produced by addition of pyrithiamine

27 Using the pyrithiamine-induced thiamine deficiency (PTD) animal paradigm of WKS, our la

28 ith the thalamus of the pyrithiamine-induced thiamine deficiency (PTD) rat model of Wernicke's enceph

29 In the pyrithiamine-induced thiamine deficiency (PTD) rat model of WKS, there are si

30 Pyrithiamine-induced thiamine deficiency (PTD) was used to produce a rodent m

31 f diencephalic amnesia, pyrithiamine-induced thiamine deficiency (PTD), was used to investigate dienc

32 iberi, a potentially fatal disease caused by thiamine deficiency, remains a public health concern in

33 Experimental thiamine deficiency (TD) is a classical model of a nutri

34 Experimental thiamine deficiency (TD) is a model of impaired oxidativ

35 Thiamine deficiency (TD) models the cellular and molecul

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