ライフサイエンスコーパス: thiobarbituric acid

1 and TMA), oxidative (peroxides value, K230, thiobarbituric acid and K270) and sensory analyses were

2 We used in situ chemical trapping with 2-thiobarbituric acid and mass spectrometry with a deutera

3 ptible to lipid peroxidation (as measured by thiobarbituric acid) and to cell kllling within a 90-min

4 al production as determined with a cell-free thiobarbituric acid assay.

5 We identified a thiobarbituric acid derivative, 5376753, that allosteric

6 Merbarone (5-[N-phenyl carboxamido]-2-thiobarbituric acid) is an anticancer drug that inhibits

7 (2)-isoprostanes, malondialdehyde (MDA), and thiobarbituric acid reacting substances (TBARS), in the

8 ilting, loss of chlorophyll, and increase in thiobarbituric acid reacting substances.

9 amounts of glucose, urea, triglycerides, and thiobarbituric acid-reacting substances in diabetic rats

10 y weight ratio, urinary albumin, and urinary thiobarbituric acid-reacting substances.

11 thin-layer chromatography and the periodate-thiobarbituric acid reaction, we found that the hepatopa

12 Thiobarbituric acid reactive material (TBARM) in tissue

13 roxidation in retinal homogenates was by the thiobarbituric acid reactive species (TBARS) formation

14 in liver homogenates from Wistar rats by the thiobarbituric acid reactive species test.

15 ioxidant activity, as assessed indirectly by thiobarbituric acid reactive substance (TBARS) formation

16 Spectrofluorometry was used to analyze thiobarbituric acid reactive substance (TBARS); ferric-r

17 Heat caused increased thiobarbituric acid reactive substance levels (an indica

18 MDA was measured as thiobarbituric acid reactive substance.

19 ia and the high K+-mediated increase in lung thiobarbituric acid reactive substance.

20 cellular adhesion molecule-1 (p = 0.001) and thiobarbituric acid reactive substances (p = 0.001) as w

21 We measured diaphragmatic concentrations of thiobarbituric acid reactive substances (TBAR), a marker

22 creased the hexanal content (0.21mug/ml) and thiobarbituric acid reactive substances (TBARS) (2.56mug

23 ent and composition, free fatty acids (FFA), thiobarbituric acid reactive substances (TBARS) and fluo

24 from W-MR-Al had lower peroxide value (PV), thiobarbituric acid reactive substances (TBARS) and non-

25 tent, total volatile basic nitrogen (TVB-N), thiobarbituric acid reactive substances (TBARS) and pero

26 Results from thiobarbituric acid reactive substances (TBARS) and sens

27 The thiobarbituric acid reactive substances (TBARS) assay is

28 of fish oil oxidation was studied using the thiobarbituric acid reactive substances (TBARS) assay.

29 on via oxidative stress was also detected by thiobarbituric acid reactive substances (TBARS) assay.

30 glycated albumin content, by measurement of thiobarbituric acid reactive substances (TBARs) for lipi

31 oxy-9,11-octadecadienoic acid (13-HODE), and thiobarbituric acid reactive substances (TBARS) in 252 w

32 Serum MDA levels were measured as thiobarbituric acid reactive substances (TBARS) in 634 p

33 However, thiobarbituric acid reactive substances (TBARS) increase

34 Oxidative stability was evaluated by thiobarbituric acid reactive substances (TBARS) on day (

35 xidation was monitored in parallel using the thiobarbituric acid reactive substances (TBARS) test.

36 Lipid hydroperoxides and thiobarbituric acid reactive substances (TBARS) were ana

37 ity (ORAC) and lipid peroxidation assayed as thiobarbituric acid reactive substances (TBARS) were mea

38 9-hydroxyoctadecadieneoic acid (9-HODE), and thiobarbituric acid reactive substances (TBARS) were mea

39 Thiobarbituric acid reactive substances (TBARS) were red

40 Serum ALT value, and hepatic thiobarbituric acid reactive substances (TBARS), and hep

41 profiles, formation of hydroperoxides (PV), thiobarbituric acid reactive substances (TBARS), fluores

42 Hydroperoxide, thiobarbituric acid reactive substances (TBARS), malondi

43 The increases in thiobarbituric acid reactive substances (TBARS), p-anisi

44 nally analyzed by two Raman spectroscopy and thiobarbituric acid reactive substances (TBARS).

45 (PFP, 1.18-1.32 mmol peroxides/kg mince) and thiobarbituric acid reactive substances (TBARS, 0.30-0.3

46 as determined by conjugated dienes (CDs) and thiobarbituric acid reactive substances (TBARSs) product

47 cells were treated with ox-LDL (50 microg/mL thiobarbituric acid reactive substances 12 to 16 nmol/mg

48 ed by measuring lipid peroxides (measured as thiobarbituric acid reactive substances [TBARS]) in reti

49 Thiobarbituric acid reactive substances activity indicat

50 was assessed by histology and measurement of thiobarbituric acid reactive substances and NOX-related

51 Peroxide value, concentration of thiobarbituric acid reactive substances and oxygen uptak

52 eptibility to lipid peroxidation by both the thiobarbituric acid reactive substances assay and the fl

53 oxidants in preventing lipoperoxidation in a thiobarbituric acid reactive substances assay.

54 y measuring malondialdehyde levels using the thiobarbituric acid reactive substances assay.

55 of skeletal muscle carbonylated proteins and thiobarbituric acid reactive substances during hyperammo

56 edium and high oxidative groups according to thiobarbituric acid reactive substances values after 9da

57 Thiobarbituric acid reactive substances values and lipox

58 The increased trypan blue uptake and thiobarbituric acid reactive substances were inhibited b

59 hydroxy--deoxyguanosine, and H2O2 and plasma thiobarbituric acid reactive substances were significant

60 TBARS (thiobarbituric acid reactive substances) test was used t

61 es of oxidative stress, urinary excretion of thiobarbituric acid reactive substances, 8-hydroxy--deox

62 lactate dehydrogenase, laccase) and damage (thiobarbituric acid reactive substances, acetylcholinest

63 tile base nitrogen, trimethylamine nitrogen, thiobarbituric acid reactive substances, ATP catabolism

64 Quantitative measurements of thiobarbituric acid reactive substances, conjugated dien

65 , evidenced by an 80% reduction (P<0.001) in thiobarbituric acid reactive substances, effective inhib

66 and alterations in the heat shock response, thiobarbituric acid reactive substances, heat shock prot

67 ed bacterial cells show an enhanced level of thiobarbituric acid reactive substances, indicating the

68 Lipid oxidation, assessed as thiobarbituric acid reactive substances, was significant

69 ed with decreased urinary levels of H2O2 and thiobarbituric acid reactive substances.

70 by 2-hydroxyethidine, and lipid peroxides by thiobarbituric acid reactive substances.

71 ght ratio (W/D), tissue albumin index (TAI), thiobarbituric acid-reactive material content (TBARM), a

72 rotein and lactate dehydrogenase (LDH), lung thiobarbituric acid-reactive species, and lung histology

73 ues, was subject to analysis by conventional thiobarbituric acid-reactive substance (TBARS) and ferro

74 e, anserine, homocarnosine, pentosidine, and thiobarbituric acid-reactive substance (TBARS) contents.

75 The peroxide value (PV) and thiobarbituric acid-reactive substance (TBARS) value of

76 Thiobarbituric acid-reactive substance and oxidized and

77 s compared with H441 showed less increase in thiobarbituric acid-reactive substance and phosphatidylc

78 of low density lipoprotein was measured by a thiobarbituric acid-reactive substance, which was confir

79 throcytes glutathione peroxidase (FB400) and thiobarbituric acid-reactive substances (FB100, FA400, F

80 lement increased the plasma concentration of thiobarbituric acid-reactive substances (TBARS) (P: = 0.

81 esulted in a fivefold increased formation of thiobarbituric acid-reactive substances (TBARS) and acti

82 Lipid peroxidation was measured as thiobarbituric acid-reactive substances (TBARS) and alph

83 after the heat treatment, there were higher thiobarbituric acid-reactive substances (TBARs) and chol

84 de value (PV) was found and large amount of, thiobarbituric acid-reactive substances (TBARS) and hexa

85 The increases in thiobarbituric acid-reactive substances (TBARS) and p-an

86 significantly reduced (p<0.05) the levels of thiobarbituric acid-reactive substances (TBARS) and prot

87 d symptoms, exercise, ejection fraction, and thiobarbituric acid-reactive substances (TBARS) as an in

88 n a lipid system was determined by using the thiobarbituric acid-reactive substances (TBARS) assay.

89 Glucose, insulin, and thiobarbituric acid-reactive substances (TBARS), a marke

90 Furthermore, the content of thiobarbituric acid-reactive substances (TBARS), and cat

91 els of lipid peroxidation products including thiobarbituric acid-reactive substances (TBARS), glutath

92 e authors analyzed the association of plasma thiobarbituric acid-reactive substances (TBARS), glutath

93 dation, breath ethane (an in vivo assay) and thiobarbituric acid-reactive substances (TBARS, an in vi

94 a lipid antioxidant, suppressed increases in thiobarbituric acid-reactive substances (TBARSs; a measu

95 olvents exhibited the lowest peroxide value, thiobarbituric acid-reactive substances and beany odour

96 Both proteins block accumulation of thiobarbituric acid-reactive substances and conjugated d

97 n of 8-epi-PGF2 alpha coincided with that of thiobarbituric acid-reactive substances and lipid hydrop

98 O-deethylase activity), or oxidative stress (thiobarbituric acid-reactive substances and ratios of re

99 develop normally, and their plasma levels of thiobarbituric acid-reactive substances do not differ fr

100 volume in 1 second (FEV1) with 1) levels of thiobarbituric acid-reactive substances in plasma (p-TBA

101 d deletions in kidney mitochondrial DNA, and thiobarbituric acid-reactive substances in plasma, toget

102 d prevented the diabetes-induced increase in thiobarbituric acid-reactive substances in serum and sig

103 low density lipoprotein (measured as either thiobarbituric acid-reactive substances or the oxidant s

104 Thiobarbituric acid-reactive substances were higher prec

105 se, hair copper, urinary copper, and urinary thiobarbituric acid-reactive substances were measured du

106 one-binding globulin, F(2)-isoprostanes, and thiobarbituric acid-reactive substances were measured up

107 ion, hair copper concentrations, and urinary thiobarbituric acid-reactive substances were significant

108 incubated with 10 microgram/ml oxLDL (10-15 thiobarbituric acid-reactive substances) and blocking an

109 Lipid peroxidation (thiobarbituric acid-reactive substances) increased after

110 otype in which oxidative damage (measured as thiobarbituric acid-reactive substances) was significant

111 of lipid peroxidation (conjugated dienes and thiobarbituric acid-reactive substances) were also great

112 xposure to IL-1alpha, TNF-alpha, and ox-LDL (thiobarbituric acid-reactive substances, 13.4 nmol/mg LD

113 Thiobarbituric acid-reactive substances, a less specific

114 37, suppressed formation of 8-epi-PGF2alpha, thiobarbituric acid-reactive substances, and lipid hydro

115 f several markers of oxidative status (i.e., thiobarbituric acid-reactive substances, erythrocyte glu

116 stress, as exemplified by the generation of thiobarbituric acid-reactive substances, expression of h

117 the diabetes-associated increases in plasma thiobarbituric acid-reactive substances, mitochondrial D

118 s of headspace aldehydes, malonaldehyde, and thiobarbituric acid-reactive substances.

119 ipid peroxidation was determined by assaying thiobarbituric acid-reactive substances.

120 The commonly used thiobarbituric-acid-reactive-substances (TBARS) assay to

121 by free radical-mediated lipid peroxidation (thiobarbituric acid reactivity), which could be suppress

122 s of lipid peroxidation (lipid peroxides and thiobarbituric acid-reactivity), plasma chelatable iron,

123 re widely followed, including measurement of thiobarbituric acid substances and the sole use of fluor

124 , pH, total volatile basic-nitrogen (TVB-N), thiobarbituric acid (TBA) as well as free amino acid (FA

125 etermined by assay of snap frozen tissue for thiobarbituric acid (TBA) concentrations (nmol/g tissue

126 PLs showed a proportionate relationship with thiobarbituric acid (TBA) number, an indicator of lipid

127 ts, total volatile basic nitrogen (TVBN) and thiobarbituric acid (TBA) of the dried milkfish samples

128 de value, iodine value specification number, thiobarbituric acid (TBA) value and colour of pistachio

129 cing systems, according to peroxide (PV) and thiobarbituric acid (TBA) values.

130 3) products of reactive oxygen species (ROS; thiobarbituric acid [TBA]-reacting material, and dichlor

131 lipid peroxidation further confirmed by the thiobarbituric acid (TBAR) assay.

132 n in mitochondrial membrane fragments by the thiobarbituric acid test and by measurement of nonrespir

133 measuring Free Fatty Acids, peroxide value, Thiobarbituric Acid value and color value during 12 mont

134 Anisidine and thiobarbituric acid values indicated the formation of se