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1 EF fractionation were further separated in a third stage.
2 lood loss compared with an intervention-free third stage.
3 ion of large-input neurons is relayed to the third stage.
4            Initial larval infection with 100 third-stage A. ceylanicum larvae resulted in predominant
5              Vaccination of mice with either third-stage Ancylostoma caninum infective hookworm larva
6 delayed growth following oral infection with third-stage Ancylostoma ceylanicum hookworm larvae.
7 layer of the cuticle of third-stage, molting third-stage, and fourth-stage larvae, the body channels
8                                          The third stage, based on the inferior temporal and frontal
9 y second or third layer, creating second- or third-stage controlled graphite intercolation compounds,
10 modules separately for the first, second and third stage, each containing both a group of co-expresse
11 ses, host-parasite interactions during early third-stage filarial larva (L3) migration are poorly und
12 so show that lin-46 is required prior to the third stage for normal adult cell fates, suggesting that
13                                          The third stage generates, for the major docking modes, inte
14 f laboratory animals with living irradiated, third-stage hookworm larvae (L3), we examined the antibo
15 ted Fontan procedure has been performed as a third stage in 32 patients whose median age was 28.7 mon
16        There is now compelling evidence of a third stage in the immune response, in 'tertiary lymphoi
17 ncer cases and 4,316 controls, followed by a third stage in which 30 single nucleotide polymorphisms
18                 The developmentally arrested third stage infective larva of hookworms resumes develop
19 3,650 PrCa cases and 3,940 controls and in a third stage involving an additional 16,229 cases and 14,
20                                          The third stage is myotome boundary formation, where the bou
21  chitinase gene, expressed in the infective, third-stage (L3) larvae of the human filarial parasite O
22 omparing transcriptomes from linker cells of third-stage (L3) larvae, fourth-stage (L4) larvae, and n
23 s in 3311 clusters from first- and infective third-stage larva (L1, L3i) of the parasitic nematode St
24  As-p18 is not present in the post-infective third-stage larva or adult worm tissues, it appears to b
25 teine protease inhibitors on the survival of third stage larvae (L3), and the molting of L3 to L4 in
26  recover and resume development as postdauer third stage larvae, with the same VPC spatial-patterning
27 terning events as in continuously developing third stage larvae.
28 cts of adult hookworms but not the infective third stage larvae.
29 shment of Onchocerca volvulus infection, the third-stage larvae (L3) and the molting L3.
30 l infection is initiated by mosquito-derived third-stage larvae (L3) deposited on the skin that trans
31 Protective immunity in mice to the infective third-stage larvae (L3) of Strongyloides stercoralis was
32 n their exposure to mosquito-borne infective third-stage larvae (L3) of these parasitic helminths.
33 t antigens prepared from Onchocerca volvulus third-stage larvae (L3), molting L3 (mL3), and crude ext
34 ary and challenge infections with B. pahangi third-stage larvae (L3).
35  the ability of H. aspersa to shed infective third-stage larvae (L3s) of A. abstrusus and T. brevior
36 hey were inoculated with H. bakeri infective third-stage larvae (L3s).
37  and antibody responses to infective larval (third-stage larvae [L3] and molting L3 [mL3]), adult fem
38 e body channels and multivesicular bodies of third-stage larvae and the processed material found betw
39             NSG mice were then infected with third-stage larvae and treated for 6 wk with methylpredn
40 enorhabditis elegans, developmental fates of third-stage larvae are determined in part by environment
41 mmunized with irradiated Onchocerca volvulus third-stage larvae developed protective immunity.
42 identified through the analysis of a molting third-stage larvae expressed sequence tag dataset.
43  (FEC) and immunoglobulin A activity against third-stage larvae from Teladorsagia circumcincta, as in
44 and is expressed in microfilariae but not in third-stage larvae or adult worms.
45 expression increased following activation of third-stage larvae with serum and that the highest level
46 mmunized against O. volvulus with irradiated third-stage larvae, and it was observed that Th2 respons
47 rval stages, but also the unembryonated egg, third-stage larvae, and ovaries of adult worms, even tho
48  oral infection of Heligmosomoides polygyrus third-stage larvae, were given intrarectal saline or tri
49                   In addition, autoinfective third-stage larvae, which initiate hyperinfection, were
50 kfly vector Simulium sp. carrying infectious third-stage larvae.
51 cts pertaining to morphogenesis of infective third-stage larvae.
52 t reduction in both molting and viability of third-stage larvae.
53 n), followed by infection with A. ceylanicum third-stage larvae.
54 equence primers to amplify a fragment from a third stage larval cDNA library by polymerase chain reac
55                The generation of second- and third-stage mass spectra in an ESI-ion trap-MS showed si
56                                              Third-stage mass spectrometry of the PS derivative of E2
57 calized to the basal layer of the cuticle of third-stage, molting third-stage, and fourth-stage larva
58 TECs has recently been extended to include a third stage, namely the post-Aire MHCII(lo) subset as id
59        Ascarosides secreted by the dispersal third-stage nematode LIII larvae promote beetle pupation
60                                       In the third stage, new epistatic QTL are searched in pairs.
61 adult molt by application of precocene II to third-stage nymphs caused a loss of br mRNA at the preco
62                                          The third stage of adhesion occurs as additional cells are a
63 t least rostral and caudal subdivisions as a third stage of cortical processing.
64 the hypotheses that active management of the third stage of labour lowers the rates of primary postpa
65 eceived oxytocin prophylactically during the third stage of labour.
66                                          The third stage of pregnancy was estimated from fundal heigh
67                 Stalling may also occur at a third stage of translocation (III), just before release
68 TL, the statistical power for the second and third stages of analysis for mapping epistatic QTL can b
69 peared and diversified during the second and third stages of oxygenation.
70 e randomly assigned active management of the third stage (prophylactic oxytocic within 2 min of baby'
71                     Active management of the third stage reduces the risk of PPH, whatever the woman'
72 ion amount of newly selected yeast F-78 from third-stage selection) on the level of monomeric anthocy
73                                       In the third stage (stage of refractory status epilepticus), a
74    Therefore, with multicomponent systems, a third stage that involves elemental redistribution withi
75        This selected population provides the third stage (the user) with an odor label that can be us
76 in the percentage of larvae molting from the third stage to the fourth stage was observed with mice i
77                            Surprisingly, the third stage, which appears to be critical for neural fat

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