ライフサイエンスコーパス: thirst

1 rrent internal motivational state (hunger or thirst).

2 and thalamus after overdrinking, relative to thirst.

3 e signals and oppositely regulate hunger and thirst.

4 ted with increased AVP levels and suppressed thirst.

5 e subfornical organ that trigger or suppress thirst.

6 g overdrinking compared with drinking during thirst.

7 hese responses correlated significantly with thirst.

8 e level of water in a pitcher and quench its thirst.

9 ve, similar to temperature, itch, hunger and thirst.

10 large volumes of dilute urine and persistent thirst.

11 by drinking inherent in the consciousness of thirst.

12 creased in the papilla of mice after 36 h of thirsting.

13 +/- 22% in the papilla of mice after 36 h of thirsting.

14 253% (P < 0.01) in the papilla upon 36 h of thirsting.

15 ncreased further in the papilla upon 36 h of thirsting.

16 PVS have awareness and experience hunger and thirst; 30% believe they experience pain.

17 Thirst also permitted flies to learn olfactory cues pair

18 Older persons fail to recognize thirst and as such have an increased risk of dehydration

19 V and hypothalamic neurons in the control of thirst and AVP-mediated body fluid homeostasis.

20 bjective appetite responses while increasing thirst and core-body temperature.

21 Tolvaptan caused increased thirst and dry mouth, but frequencies of major adverse e

22 the early phase of sepsis features impaired thirst and enhanced vasopressin release, the basis for t

23 onstrate a pivotal role in the regulation of thirst and salt appetite of angiotensin II generated in

24 eostatic and behavioral responses associated thirst and salt appetite, although clearly it may relate

25 ding how motivated behaviors such as hunger, thirst and sexual behaviors arise.

26 active ligands in brain angiotensin-mediated thirst and sodium appetite.

27 n II (AngII) and angiotensin III (AngIII) on thirst and sodium appetite.

28 At maximum thirst and then during irrigation of the mouth with wate

29 hanges in extracellular osmolarity stimulate thirst and vasopressin secretion through a central osmor

30 c NaCl and other osmolytes readily stimulate thirst and vasopressin secretion.

31 ulosum lamina terminalis (OVLT; which drives thirst) and attenuates that of neurosecretory neurons in

32 eral sensations, vasomotor activity, hunger, thirst, and 'air hunger'.

33 nsin II (AngII)-regulated behaviors, such as thirst, and may do so by influencing the central renin-a

34 Participants rated hunger, thirst, and satiety at baseline and at 20-min intervals

35 rinking during meals, the rapid satiation of thirst, and the fact that oral cooling is thirst-quenchi

36 ress competing motivational systems, such as thirst, anxiety-related behavior, innate fear, and socia

37 Hunger and thirst are ancient homeostatic drives for food and water

38 otivational mechanism by which the forebrain thirst circuit drives drinking.

39 These findings reveal a distributed thirst circuit that motivates drinking by the common mec

40 off, and probably functions as a centre for thirst control in the mammalian brain.

41 Here we show that thirst converts water avoidance into water-seeking in na

42 associated with tolvaptan included increased thirst, dry mouth, and increased urination.

43 lated to vegetative and affective aspects of thirst experiences, whereas activity in neocerebellar (p

44 ts attributed to tolvaptan were pollakiuria, thirst, fatigue, dry mouth, polydipsia, and polyuria.

45 in humans that drinking water in response to thirst following fluid loss is a pleasant experience.

46 However, continuing to drink water once thirst has been satiated becomes unpleasant and, eventua

47 Thirst has conventionally been viewed as a homeostatic r

48 While central neural circuits regulating thirst have been well studied, it is still unclear how m

49 c mice display high blood pressure, enhanced thirst, high urine output, proteinuria, and kidney damag

50 across the cerebellum is similar to that for thirst, hunger, and their satiation.

51 rnal state of an individual-as it relates to thirst, hunger, fear, or reproductive drive-can be infer

52 vegetative systems including hunger for air, thirst, hunger, pain, micturition, and sleep, is discuss

53 ma sodium concentration and the emergence of thirst in 10 healthy adults.

54 We induced either moderate or severe thirst in humans using intravenous saline, and examined

55 rgan (SFO) in the anticipatory regulation of thirst in mice.

56 organs of the hypothalamus are activated by thirst-inducing conditions.

57 ncovered, less is known about how hunger and thirst interact.

58 Thirst is the basic instinct to drink water.

59 o cognitive function, dyspnea, constipation, thirst, leg swelling, numbness, dry mouth, and balance p

60 ly, the volume of water drunk in response to thirst matches the deficit.

61 NA species, whereas a behavior as complex as thirst may be influenced by changes in multiple genes.

62 influence of motivational state (hunger and thirst), memory demand, and spatial behavior in 2 tasks:

63 ive internal state that dynamically controls thirst-motivated behavior.

64 Thirst motivates animals to drink in order to maintain f

65 ntaining solutions in mice, independently of thirst or hunger.

66 dictably increased water losses and impaired thirst or restricted free water intake or both.

67 response to overnight fluid deprivation, or thirst or salt appetite in response to an isotonic hypov

68 rimination cued by internal state (hunger or thirst) or on performance of conditional visuospatial ob

69 Despite the induction of even severe thirst, our subjects rejected unfair offers.

70 pparently not involved in the computation of thirst per se but rather is activated during changes in

71 Complementary experiments activating thirst-promoting neurons also conditioned avoidance.

72 ng deep-brain calcium dynamics, we show that thirst-promoting SFO neurons respond to inputs from the

73 We show that thirst-promoting subfornical organ neurons are negativel

74 of thirst, and the fact that oral cooling is thirst-quenching.

75 ppreciated role of GABAergic MnPO neurons in thirst regulation.

76 nervous system (CNS), including promotion of thirst, regulation of vasopressin secretion, and modulat

77 lumn, orofacial motor-related, humorosensory/thirst-related, brainstem autonomic control network, neu

78 Increasing dietary sodium drives the thirst response.

79 compensatory thresholds for antidiuretic and thirst responses.

80 se but rather is activated during changes in thirst/satiation state when the brain is "vigilant" and

81 erebral blood flow with subjects' ratings of thirst showed major activation in the vermal central lob

82 Finally, we demonstrate that the thirst signal exits these regions through at least three

83 hes fluid loss and satiates the sensation of thirst that accompanies dehydration.

84 sleep disturbance, or unsatisfied hunger or thirst that they rated as moderate or severe, whereas de

85 During the development of thirst, the anterior and posterior quadrangular lobule,

86 sent distinct cellular processes to regulate thirst, vasopressin secretion and autonomic function.

87 The instinct of thirst was a cardinal element in the successful coloniza

88 At 90 min, an increase in thirst was associated with a decline in subjective energ

89 In the short-term, thirst was associated with poorer memory.

90 Thirst was moderately intense, and shortness of breath,

91 tate during random foraging, when hunger and thirst were incidental to behavior, and signals derived

92 ass, urine osmolality, body temperature, and thirst were monitored.

93 -OH-DPAT-induced 5-HT hypofunction increases thirst without substantially affecting the palatability