ライフサイエンスコーパス: this may

1 This may aid clinicians in presurgical evaluation by providin

2 This may alleviate the restriction of N supply for increased

3 is a hallmark of cerebrovascular events, we postulated that this may also be true for transient ischaemic attack, and tha

4 anced survival during apoptosis compared with controls, and this may also contribute to the increased accumulation observ

5 modulated by resource availability and we hypothesized that this may alter biodiversity-invasion relationships.

6 How this may alter the outcome of disease remains to be determine

7 ectrolocation and other active sensory systems suggest that this may be a mechanism shared by (active) sensory systems.

8 This may be a significant contributor to excessive inflammati

9 This may be considered a paradigm of the ground work needed t

10 This may be due to conditions associated with fetal growth re

11 This may be due to the overuse of terminology that stresses r

12 This may be exploited in the future to predict viscosity in n

13 This may be from patients being motivated by, and modelling,

14 This may be important in slowing disease progression and prov

15 This may be important, since the administration of lactate du

16 3, are essential for C. muridarum to induce tubal fibrosis; this may be induced by the gastrointestinal C. muridarum, as

17 This may be related to lower likelihood of retransplantation

18 irmed in larger studies with longer follow-up and controls, this may be the desired treatment modality.

19 Hot zones like this may be the primary reservoirs for arc volcanism, influen

20 This may be used to identify unique chemical signatures of va

21 This may compromise information coding capacity and thereby m

22 This may constitute the basis for a new mouse model of ACPA-p

23 This may contribute to the fine tuning of expression of the d

24 This may explain our findings of increased lymph node invasio

25 This may explain why TBI patients are more vulnerable to cogn

26 This may facilitate better population-level disease subtyping

27 This may facilitate the targeted manipulation of metabolic ro

28 itations of this form of analysis and the implications that this may have for bioinformatics database design.

29 This may indicate a block in the downstream glycerolipid meta

30 ar pattern recognition by the innate immune system, and how this may influence adaptive immunity.

31 This may lead to novel applications using blue light induced

32 layer provide evidence that the material is "high quality", this may not be true for electronic performance.

33 This may not preclude the longer-term effects of fetal exposu

34 ing technique can be demotivating in some contexts and that this may occur through an attention-based mechanism.

35 This may open new avenues for the study of ion pumps and simi

36 This may partly explain the mechanism underlying adipocyte hy

37 KZF1 result in deregulation of mitochondrial integrity, and this may play a pathogenic role in the development of IO IBD.

38 This may pose problems with managing adverse effects and the

39 This may prompt the consideration for pharmacological manipul

40 This may provide insight into disease pathogenesis, diagnosis

41 We suggest this may represent one of the control loops that sense the ra

42 This may result from stronger equatorial westerly ducts and s

43 This may result from the variety of selection pressures from

44 orylation of Akt and mTOR, impairing Akt-mTOR signaling and this may result in an increased risk for developmental neurop

45 This may result in highly differentiated therapeutics with gr

46 senior PIs with research time in exchange for less funding, this may reverse a decades-long trend of more money going to

47 This may seriously compromise the conservation of biodiversit

48 This may suggest alternate pathways are at work in copper-bas

49 tter estimated by using selective fungal culture media, and this may translate to important clinical decisions.

50 This may underlie the mechanisms by which cells sense directi