ライフサイエンスコーパス: this mechanism

1 This mechanism adds significant insight into our knowledge of

2 This mechanism also decelerates DNA translocation, a desired

3 This mechanism does not explain certain manifestations of HIV

4 This mechanism enables proteasome activity to adapt to the su

5 This mechanism evolved to more rapidly produce RPs on demand

6 This mechanism further predicts that residues are exposed to

7 This mechanism has served as the predominant model of GR-medi

8 This mechanism is consistent with observations of hybrid eart

9 This mechanism is distinct from the BRCA2-dependent fork prot

10 This mechanism is not based on an ordered sarcomeric organiza

11 This mechanism is not restricted to the eye, as similar resul

12 This mechanism of FoxP3 control may allow escape from dominan

13 This mechanism prevents the cancer from being eliminated by t

14 This mechanism results in a higher attachment of particles on

15 This mechanism, combined with frequent homologous recombinati

16 This mechanism, which employs recurrent loops with an angular

17 Inspired by this mechanism, we have explored the possibility of using ant

18 anism of SERMs, even the cationic amphiphilic drugs (CADs), this mechanism led to the endolysosomal calcium as a critical

19 We call this mechanism friction-driven scission (FDS).

20 use regulation of mitotic entry by Cdc25 is well conserved, this mechanism may provide a widespread solution to the probl

21 imilar actin-binding proteins, interact with actin, and how this mechanism can be perturbed to cause disease.

22 However, this mechanism cannot apply to CNAbeta1, an atypical CNA isof

23 However, this mechanism is controversial, and the identity of the natu

24 Importantly, this mechanism of HR is independent of RAD51, which encodes t

25 vitro differentiation promotes only partial development of this mechanism.

26 Dysregulation of this mechanism leads to numerous human neurodegenerative or n

27 e demand and in aging flies, underscoring the generality of this mechanism.

28 c resistance arteries supported the biological relevance of this mechanism.

29 Understanding the role of this mechanism in specific circuits requires identifying the

30 Highlighting the significance of this mechanism, we found that ranolazine and elevated serum d

31 A better understanding of this mechanism may point toward new treatments.

32 nge is a promising new approach but better understanding of this mechanism of action is needed.

33 Remarkably, this mechanism is homoplastic with the evolution of the C4-as

34 itrogen reactive species, and (ii) l-citrulline can reverse this mechanism by recoupling NOS, restoring NO production and

35 that DHHC7 represents the principal PAT for Glut4 and that this mechanism is essential for insulin-regulated glucose hom

36 Furthermore, we demonstrate that this mechanism is disrupted in congenital muscular dystrophy

37 We found that this mechanism allows H pylori to persist in proximity to inf

38 We show here that this mechanism is mediated not via cell-autonomous clock prop

39 d with reduced survival of melanoma patients, implying that this mechanism also drives human disease.

40 Simulations predict that this mechanism may operate independent of the specific mutati

41 Reverse mutations in Kir1.1 suggest that this mechanism is conserved in other Kir channels.

42 of simple yes/no units revealed in this study suggest that this mechanism may be useful in applications across various f

43 Promotion of AD-related signaling pathways through this mechanism may contribute to female vulnerability to AD.

44 We aimed to understand this mechanism, and how it may contribute to thrombus develop

45 not clear whether the viral genome is also transferred via this mechanism and further, whether transfer via this route c

46 s cannot apply to the yeast Saccharomyces cerevisiae, where this mechanism would provoke a never-ending increasing mRNA s

47 Here, we asked whether this mechanism generalizes to OFF-selective T5 cells and to a

48 int in yeast in response to replicative stress, but whether this mechanism functions in multicellular eukaryotes remains

49 While this mechanism has been well documented in monocultures, its

50 While this mechanism is homeostatic we show that it does not achiev