戻る
「早戻しボタン」を押すと検索画面に戻ります。 [閉じる]

コーパス検索結果 ( left1)

通し番号をクリックするとPubMedの該当ページを表示します
1                                                             This mechanism enables Prim-PolC to couple primer extension w
2                                                             This mechanism generates a metabolic homeostatic circuit that
3                                                             This mechanism has a crucial role in Birt-Hogg-Dube syndrome,
4                                                             This mechanism has been validated in human liver specimens fr
5                                                             This mechanism involves the silencing of H3K27me3 writers, ac
6                                                             This mechanism is regulated by the organization and dynamics
7                                                             This mechanism of substrate-assisted product release explains
8                                                             This mechanism of suppressing regeneration by silencing damag
9                                                             This mechanism pushes CMG beyond a '5-ubiquitin threshold' th
10                                                             This mechanism relies on the restriction of pericentrin-like
11                                                             This mechanism underlies an interplay between microglia, vasc
12                                                             This mechanism was supported by several experimental findings
13                                                    Although this mechanism contributes to peripheral metabolic dysfunctio
14  of antibodies conjugated to drugs, which presumably act by this mechanism as well, but with the antibody replaced by an
15                                    It is possible to defeat this mechanism and activate T cells with solution ligands by
16                                  While we have demonstrated this mechanism in human adipocytes in vitro, and correlations
17                                  We quantitatively describe this mechanism with respect to the size of liposomes, lipid c
18                            We show preliminary evidence for this mechanism by demonstrating that the enhanced viability i
19 cription factors; however, direct experimental evidence for this mechanism is lacking.
20                                                    However, this mechanism has not been directly tested in experiments.
21 n the maintenance of calcium homeostasis and alterations in this mechanism are responsible for several pathological condi
22 oducing HONO which can in turn oxidize SO(2) to yield N(2)O.This mechanism could provide an explanation for sulfate forma
23               In particular, we describe the application of this mechanism to a dual nickel/photoredox catalytic protocol
24                                 Further characterization of this mechanism may provide information for developing strateg
25                       However, the relative contribution of this mechanism is still not fully clear in humans.
26                  As such, it should motivate exploration of this mechanism in studies in vivo, in wound healing or angiog
27                                   Despite the importance of this mechanism of glycinergic dis-inhibition associated with
28 A bending by APE1, providing evidence for the importance of this mechanism.
29         Quantitative modelling confirms the plausibility of this mechanism.
30                                 Given the prominent role of this mechanism in both herpesvirus biology and cancer, our sc
31                                                    Overall, this mechanism allows access to both traditional C-N adducts
32                                            Physiologically, this mechanism may serve to direct glucose into the muscles i
33                                                    As such, this mechanism is an example of dynamic allostery.
34 r development in a Wnt-dependent manner and further suggest this mechanism is important in attenuating hypoxic injury.
35                                                   We tested this mechanism for inactivation using a KcsA mutant (H25R/E11
36  through activation of endogenous opioid signaling and that this mechanism mediates the addictive effects of AAS.
37 n phosphorylation regulates vascular tone in vitro and that this mechanism plays an important role in modulation of vascu
38                                            We conclude that this mechanism for retinal oxygenation played a vital role in
39                                         We demonstrate that this mechanism further supports efficient and unprecedented m
40 RNA processing in roosters and green lizards, implying that this mechanism is evolutionarily conserved in amniotes.
41                                           It is likely that this mechanism of PIP2 activation is conserved among Kv7 chan
42 travacuolar pathogens such as Salmonella Here, we show that this mechanism requires aconitate decarboxylase 1 (IRG1), whi
43                                                     Through this mechanism, elesclomol prevented detrimental neurodegener
44                                                     Through this mechanism, sodium channels effectively measure the frequ
45                                                  Central to this mechanism is the direct effect of IL-33-ST2 signaling on
46          It is unknown if other ultra-black butterflies use this mechanism.
47 pport this, further studies are needed to determine whether this mechanism explains RBP4-induced insulin resistance in hu
48                                             Consistent with this mechanism, benzamides impaired growth of microtubules fo
49                                             Consistent with this mechanism, pharmacological inhibition of NER using spiro
50                      Study 4 found evidence consistent with this mechanism.