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1 the lateral corticospinal tract (CST) in the thoracic spinal cord.
2 G graft in the moderately contused adult rat thoracic spinal cord.
3 period, prior to virus isolation from lower thoracic spinal cord.
4 pathetic preganglionic neurons (SPNs) in the thoracic spinal cord.
5 ation of sympathetic afferents in the caudal thoracic spinal cord.
6 d and in the intermediolateral column of the thoracic spinal cord.
7 ted peripheral nerve graft and back into the thoracic spinal cord.
8 trolateral reticular formation (rvlm) to the thoracic spinal cord.
9 investigated in the lateral horn of the rat thoracic spinal cord.
10 us, raphe magnus, and in the ventral horn of thoracic spinal cord.
13 the interomedial lateral cell column of the thoracic spinal cord also contained CNTFR alpha-ir neuro
14 This included the descending projections to thoracic spinal cord and a network in brainstem, midbrai
15 ral arcuate nucleus (Arc) that innervate the thoracic spinal cord and also contain cocaine- and amphe
16 to preganglionic, sympathetic neurons in the thoracic spinal cord and increase energy expenditure upo
17 urosemide-induced neuronal activation in the thoracic spinal cord and NTS is due to activation of sec
18 PFC that project to autonomic regions of the thoracic spinal cord and probably to other additional su
19 ferent fibers from the heart enter the upper thoracic spinal cord and synapse on cells of origin of a
20 ived identical contusion injuries to the mid-thoracic spinal cord and were allowed to recover for 6 h
21 and the intermediolateral cell column of the thoracic spinal cord, as well as the dorsomedial medulla
24 xogenous L1, detectable already 1 week after thoracic spinal cord compression and immediate vector in
26 estigated whether activated microglia in the thoracic spinal cord contribute to chronic pain in a rat
28 We used a clinically relevant rat model of thoracic spinal cord contusion injury to examine the eff
29 l medulla (RVL)) and in three regions of the thoracic spinal cord (dorsal horn, intermediate zone and
30 n, GABAergic neurons with projections to the thoracic spinal cord exist in a restricted number of med
32 l administration of NEP1 40 to rats with mid-thoracic spinal cord hemisection results in significant
33 l subregions of the grey matter of the upper thoracic spinal cord in male and female lean and obese-d
35 e dorsal horn and intermediate layers of the thoracic spinal cord in the clonidine-atipamezole group
38 locomotor function seen in the rat following thoracic spinal cord injuries that spare as little as 20
39 postural stability and propulsion after low thoracic spinal cord injury (SCI) in animals and humans.
40 either subcortical lesion sites or sites of thoracic spinal cord injury failed to elicit corticospin
43 ide-induced activation in the NTS and caudal thoracic spinal cord is abolished by prior bilateral ren
45 emistry we find that the AT1 receptor in the thoracic spinal cord is located on neurones and ependyma
46 decreased on both sides by 50%, while in the thoracic spinal cord, it was increased by 18% on the con
48 Following spinal transection of the upper thoracic spinal cord, male Sprague-Dawley rats given leg
49 injection of Fluoro-Gold (FG) into the upper thoracic spinal cord, neurons with PPE mRNA (PPE(+) neur
52 sidase (beta-gal) reporter gene into the mid-thoracic spinal cord of adult rats, transgene expression
54 corded with spike-triggered averaging in the thoracic spinal cord of anaesthetized, paralysed cats.
58 of motor-evoked potentials recorded from the thoracic spinal cord of symptomatic SOD1(G93A) rats show
61 SCI, we performed contusion injuries in the thoracic spinal cord of transgenic (Tg) mice in which >9
64 mic sympathetic preganglionic neurons in the thoracic spinal cord produces pupillary dilatation, we p
65 ks following an acute compression of the mid-thoracic spinal cord, rats displayed markedly decreased
67 l of respiratory-related motoneurones in the thoracic spinal cord remains the subject of some debate.
68 ridging the transected dorsal columns in the thoracic spinal cord resulted in an 81% decrease in XT-1
73 in humans, stimulus parameters during upper thoracic spinal cord stimulation were adjusted to provid
74 penetrations and cell injections in the mid-thoracic spinal cord; subsequent effects on local axons
75 ing sympathetic preganglionic neurons in the thoracic spinal cord suggests that this pathway may cont
76 nsport of cholera toxin B from medullary and thoracic spinal cord sympathetic outflow areas showed la
78 ransported cholera toxin B injected into the thoracic spinal cord (T1) revealed that bulbospinal PPG(
80 itioned on the epidural surface of the lower thoracic spinal cord to activate the expiratory muscles.
81 itioned on the epidural surface of the upper thoracic spinal cord to activate the inspiratory interco
83 approximately 3 months after a complete mid-thoracic spinal cord transection at P5 in non-trained an
84 ed (step-trained 6 min/day) spinal rats (mid-thoracic spinal cord transection at post-natal day 5) at
85 MNs) and Renshaw cells (RCs) is disrupted by thoracic spinal cord transection at postnatal day 5 (P5T
87 nd standing can be reacquired after complete thoracic spinal cord transection in adult cats with appr
89 nts with LAR-targeting peptides in mice with thoracic spinal cord transection injuries induce signifi
90 tion of axons across and beyond an extensive thoracic spinal cord transection lesion in adult rats.
92 ding a clinical dose of lithium to rats with thoracic spinal cord transection or contusion injuries i
93 ns of ibuprofen via minipumps to rats with a thoracic spinal cord transection or contusion injury res
95 either partial midcervical or complete upper thoracic spinal cord transections and examined whether c
98 of GABAergic neurons with projection to the thoracic spinal cord was studied by using in situ hybrid
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