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1 imal mechanical ventilation and injection of thrombin receptor-activating peptide.
2 r LYN following the exposure of platelets to thrombin receptor-activating peptide.
3 hibitor, and was mimicked by a 14-amino acid thrombin receptor-activating peptide.
4 denosine diphosphate, thrombin, and the PAR4 thrombin receptor-activating peptide.
5 of cell death after exposure to thrombin or thrombin receptor activating peptides.
6 mobility shift assay, thrombin (10 U/ml) or thrombin receptor-activating peptide (100 microM) stimul
8 G(i) signaling pathways and (2) thrombin and thrombin receptor-activating peptides cause platelet agg
9 Platelet stimulation using either ADP or the thrombin receptor-activating peptide enhanced the access
11 U2-OS osteosarcoma cells with thrombin and a thrombin receptor-activating peptide induced pro-MMP-9 s
12 on of agonists including epinephrine and the thrombin receptor-activating peptide induced the alphaII
14 tathione have additive inhibitory effects on thrombin receptor-activating peptide-induced platelet ag
15 n-ADP-induced (arachidonic acid-, collagen-, thrombin receptor-activating, peptide-induced) platelet
17 because addition of calcium, fibrinogen, and thrombin receptor-activating peptide led to aggregation.
18 d a collagen-related peptide-induced but not thrombin receptor-activating peptide- or ADP-induced agg
20 TR42-55, SFLLRNPNDKYEPF, also known as TRAP (thrombin receptor-activating peptide)], previously have
21 ed neuronal cultures exposed to thrombin and thrombin receptor activating peptides revealed rapid act
23 n receptors responded to thrombin, the human thrombin receptor-activating peptide SFLLRNP-NH2 (TRAP)
24 Treatment of the cultures with a synthetic thrombin receptor-activating peptide (SFLLRNP) mimicked
25 let aggregation induced by ADP, collagen, or thrombin receptor-activating peptide, suggesting that su
26 let aggregation induced by a TXA2 analog and thrombin receptor-activating peptides that were rescued
27 low and high adenosine diphosphate (ADP) and thrombin receptor activating peptide (TRAP) concentratio
28 endent morphologic changes when activated by thrombin receptor activating peptide (TRAP) or when spre
29 se GPV, we show that neutrophil cathepsin G, thrombin receptor activating peptide (TRAP), and a combi
30 were observed after platelet activation with thrombin receptor activating peptide (TRAP), collagen, a
31 n between unactivated neutrophils and either thrombin receptor activating peptide (TRAP)-stimulated p
32 ustment for platelet count, higher levels of thrombin receptor activating peptide (TRAP)-stimulated p
36 rmation in human whole blood stimulated with thrombin receptor-activating peptide (TRAP) using ex viv
37 (ChA)(hR)Y-NH2 ([3H]haTRAP), a high affinity thrombin receptor-activating peptide (TRAP), and human p
38 thrombin receptor stimulation by >/=5 microM thrombin receptor-activating peptide (TRAP), which cause
40 rachidonic acid, ADP, collagen, epinephrine, Thrombin receptor activating-peptide, U46619, and ristoc
41 ide that imitates the fully active receptor, thrombin receptor activating peptide, was also found to
42 r ligands acetylcholine, serotonin, ATP, and thrombin receptor-activating peptide were similarly unaf
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