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1 growth factor, insulin-like growth factor 2, thrombospondin 1.
2 ive expression of angiogenic inhibitors like thrombospondin-1.
3 ain, but they do not hetero-oligomerize with thrombospondin-1.
4 r soluble P-selectin, platelet factor 4, and thrombospondin-1.
5  responses to the matricellular glycoprotein thrombospondin-1.
6 IF-1alpha) and VEGF and a down-regulation of thrombospondin-1.
7 DL (oxLDL), cell-derived microparticles, and thrombospondin-1.
8  of both transforming growth factor-beta and thrombospondin-1.
9 o dependent and might have been regulated by thrombospondin-1.
10 rivative of the natural angiogenic inhibitor thrombospondin-1.
11 le guanylyl cyclase-cGMP signaling inhibitor thrombospondin-1.
12 the production of the angiogenesis inhibitor thrombospondin-1.
13 d its receptor on T cells, but also by Ab to thrombospondin-1.
14 the production of the angiogenesis inhibitor thrombospondin-1.
15 otably plasminogen activator inhibitor-1 and thrombospondin-1.
16  CD47 is a receptor for the secreted protein thrombospondin-1.
17 ding activity and structural similarities to thrombospondin-1.
18 tivities of recombinant signature domains of thrombospondin-1, -2, and -4 to interact with CD47 and m
19 ant protein containing functional domains of thrombospondin-1, 3TSR, have been shown to be necessary
20 l fibroblasts through the down-regulation of thrombospondin 1, a latent transforming growth factor-be
21                                Additionally, thrombospondin-1, a naturally occurring inhibitor of ang
22  activity of nitric oxide (NO) is blocked by thrombospondin-1, a potent antagonist of NO/cGMP signali
23 ucose-responsive fragment of the promoter of thrombospondin-1, a potent antiangiogenic and proatherog
24            A SIRP-alpha antibody that blocks thrombospondin-1 activation of SIRP-alpha mitigated the
25 and downregulates the angiogenesis inhibitor thrombospondin-1, along with other members of the thromb
26 AME, the guanylyl cyclase inhibitors ODQ and thrombospondin-1 also abated IR-induced IL10 expression
27                                              Thrombospondin-1 also inhibits cGMP-mediated activation
28                                    Exogenous thrombospondin-1 also reverses the suppression by NO of
29                                              Thrombospondin-1 also stimulated phosphorylation of p47(
30 s related to decreased protein expression of thrombospondin-1, an antiangiogenic regulator.
31        Matricellular proteins such as SPARC, thrombospondin 1 and 2, and tenascin C and X subserve im
32 responsible for binding to LRP1, whereas the thrombospondin 1 and spacer domains are responsible in A
33 hough very similar, the signature domains of thrombospondin-1 and -2 differ in several potentially im
34                    The following candidates, thrombospondin-1 and 5, alpha-1B-glycoprotein, serum amy
35             CD47 is a signaling receptor for thrombospondin-1 and an attractive therapeutic target be
36      Furthermore, the CD36-binding domain of thrombospondin-1 and anti-angiogenic peptides derived fr
37 dostatin (an angiogenesis inhibitor), or for thrombospondin-1 and basic fibroblast growth factor, con
38 ncreased expression levels of antiangiogenic thrombospondin-1 and inhibited S1177 phosphorylation of
39  responses by the matricellular glycoprotein thrombospondin-1 and is therefore assumed to be the rece
40                                   Therefore, thrombospondin-1 and its receptor CD47 may be useful tar
41                               Interaction of thrombospondin-1 and its recombinant NoC1 domain with al
42 evels of two potent angiogenesis inhibitors, thrombospondin-1 and maspin, were lower compared to cont
43  at least in part through down-regulation of thrombospondin-1 and plasminogen activator inhibitor typ
44  Here, we report a novel interaction between thrombospondin-1 and SIRP-alpha on nonphagocytic cells.
45 treatment, rescued the induced expression of thrombospondin-1 and the defect in endothelial cell prol
46 can also be heterotypic-for example, between thrombospondin-1 and thrombospondin-5.
47 y high levels of the angiogenesis inhibitors thrombospondin-1 and TIMP-1.
48 that exogenous decorin induced expression of thrombospondin-1 and TIMP3, two powerful angiostatic age
49          Binding studies with isolated human thrombospondin-1 and various Hic domains suggest that th
50 progeny, platelets, are important sources of thrombospondins 1 and 2 and that these thrombopoietic ce
51                                              Thrombospondins 1 and 2 are members of a family of angio
52 , GFAP, inducible nitric oxide synthase, and thrombospondins 1 and 2 in DS astroglia.
53                                              Thrombospondins 1 and 2 were among the first natural ang
54 entified vascular endothelial growth factor, thrombospondins 1 and 2, and slit as mediators partially
55 al growth factor165, ADAMTS9 neither cleaved thrombospondins 1 and 2, nor bound vascular endothelial
56 vior such as CD44, CCNE2 (cyclin E2), THBS1 (thrombospondin 1), and CSPG2 (chondroitin sulfate proteo
57 nsforming growth factor-beta induced factor, thrombospondin 1, and platelet derived growth factor-C w
58 essel maturation, including PDGFb, TGF-beta, thrombospondin-1, and CXCL10; consistently, they were ch
59 argo, including von Willebrand factor (VWF), thrombospondin-1, and platelet factor 4.
60 crovascular endothelial cells interacts with thrombospondin-1, and this interaction is involved in mo
61 ocrine cancer, administration of endostatin, thrombospondin-1, and tumstatin peptides, as well as del
62 DEB fibroblasts decreased type XII collagen, thrombospondin-1, and Wnt-5A expression, reduced tumor c
63 roblasts led to increased type XII collagen, thrombospondin-1, and Wnt-5A, while reexpression of wild
64                                         Both thrombospondin-1- and CD47-dependent radiosensitization
65                               In contrast to thrombospondin-1- and CD47-null cells, primary vascular
66 he annexin A6/LDL receptor-related protein 1/thrombospondin 1 (ANXA6/LRP1/TSP1) complex in tumor cell
67 al cells returned the antiangiogenic protein thrombospondin-1 as a common target of both miRNAs.
68 ve to wild-type mice, implicating endogenous thrombospondin-1 as a physiologic antagonist of NO-media
69                              We now identify thrombospondin-1 as a potent antagonist of NO for regula
70  only CD47 is necessary for this activity of thrombospondin-1 at physiological concentrations.
71                                              Thrombospondin 1 binding to calreticulin-LRP1 signals re
72          Overall, these results suggest that thrombospondin-1 binding to SIRP-alpha on nonphagocytic
73 nding site but not by a peptide based on the thrombospondin-1-binding site.
74                                              Thrombospondin-1 bound in a heparin-inhibitable manner t
75                    In cell-free experiments, thrombospondin-1 bound SIRP-alpha.
76  for proapoptotic proteins including p53 and thrombospondin-1, but p53 was unchanged, and thrombospon
77 ll type-specific regulation of production of thrombospondin-1 by high glucose.
78 ery of the endogenous angiogenesis inhibitor thrombospondin-1 by platelets may be a critical host res
79 and (3) a stimulatory role for thrombin, the thrombospondin-1/CD36 axis and cyclooxygenase 1 in subse
80 mia-reperfusion injury is exacerbated by the thrombospondin-1-CD47 system through inhibition of nitri
81                                   Therefore, thrombospondin-1/CD47 antagonists may have selective rad
82 on injury (IRI), which is exacerbated by the thrombospondin-1/CD47 pathway through inhibition of nitr
83                                          The thrombospondin-1 concentrations in ST-Elevation Myocardi
84 ncluding vascular endothelial growth factor, thrombospondin 1, connective tissue growth factor, hepat
85  ADAMTS13 constructs, we show that the first thrombospondin-1, Cys-rich, and spacer domains of ADAMTS
86 on of ancillary C-terminal disintegrin-like, thrombospondin-1, cysteine-rich, and spacer domains to b
87    The results demonstrate that noncatalytic thrombospondin-1/cysteine-rich/spacer domains are princi
88                                Replacing the thrombospondin-1/cysteine-rich/spacer domains of ADAMTS5
89 nvolved in the angiogenic process, including thrombospondin 1, delta-like 4, BclII modifying factor,
90    The role of CD47 was determined using the thrombospondin-1-derived agonist peptide 4N1K and the CD
91                                              Thrombospondin-1 did not inhibit NO signaling in CD47-nu
92  2), MMP9 (matrix metallopeptidase 9), TSP1 (thrombospondin 1), etc.
93                  These data demonstrate that thrombospondin-1 exerts CD47-dependent and -independent
94              Only the CD47-binding domain of thrombospondin-1 exhibits this activity.
95 c EPCs have phenotypic differences involving thrombospondin-1 expression compared with nondiabetic EP
96 loproteinase-1, matrix metalloproteinase-10, thrombospondin-1, extracellular matrix matricryptic frag
97 ondin-2 and -4 were less active than that of thrombospondin-1 for inhibiting binding of radiolabeled
98                             Thus, release of thrombospondin-1 from alpha-granules during activation p
99           Recent evidence indicates that the thrombospondin 1 gene is up-regulated in patients with R
100 active form of AhR induced activation of the thrombospondin-1 gene promoter.
101                The anti-angiogenic effect of thrombospondin-1 has been shown to be mediated through b
102                                        Human thrombospondin 1 (hTSP-1) is a matricellular glycoprotei
103         The human matricellular glycoprotein thrombospondin-1 (hTSP-1) is released by activated plate
104 ical PspC molecule, with human matricellular thrombospondin-1 (hTSP-1).
105 te the major rapidly releasable reservoir of thrombospondin-1 in higher animals.
106                    Deletion of tumstatin and thrombospondin-1 in mice lacking the p53 tumor suppresso
107            We have uncovered a novel role of thrombospondin-1 in modulating production and activation
108                                  The role of thrombospondin-1 in radiosensitization is specific becau
109           EBV addition did not induce IDO or thrombospondin-1 in T-DC cocultures, suggesting that the
110 sed the levels of the angiogenesis inhibitor thrombospondin-1 in the xenografts.
111 istic relation between NO/cGMP signaling and thrombospondin-1 in vascular smooth muscle cells to regu
112 enuated in vitro antiangiogenic responses to thrombospondin-1, including blockade of migration, tube
113       In rodent aortic rings, treatment with thrombospondin-1 increased the production of superoxide
114 ion of the powerful angiostatic glycoprotein Thrombospondin 1 independently of Beclin 1 transcription
115  CD47, CD36, and integrin-binding domains of thrombospondin-1 independently enhance the inflammasome-
116 nsplantations, we show that platelet-derived thrombospondin-1 is a critical negative regulator during
117                                              Thrombospondin-1 is a glycoprotein with multiple biologi
118                                              Thrombospondin-1 is a multifunctional protein interactin
119                                              Thrombospondin-1 is a stress protein typically secreted
120                                              Thrombospondin-1 is a trimeric, modular calcium-binding
121 , inhibition of T cell receptor signaling by thrombospondin-1 is mediated by CD47 and requires its mo
122                  We now show that endogenous thrombospondin-1 is necessary for platelet aggregation i
123                                   Therefore, thrombospondin-1 is the dominant regulator of NO/cGMP si
124 ed previously that the matricellular protein thrombospondin-1 is upregulated in IRI.
125  of the most potent angiogenesis inhibitors, thrombospondin-1, is up-regulated in the platelets of tu
126  and retina from enucleated eyes of C57BL/6, thrombospondin-1 knockout (TSP-1KO), and TGF-beta2 recep
127 rotein containing the CD36 binding domain of thrombospondin-1 (known as the TSR domain) induced assoc
128   Finally, crossing the KrP transgene onto a thrombospondin-1 KO background reversed the vascular cha
129 -and-actin bundling when clustered either by thrombospondin-1, laminin, or antibody to the syndecan-1
130 nal tubular epithelial cells, treatment with thrombospondin-1 led to phosphorylation of SIRP-alpha an
131                                        Serum thrombospondin-1 level was significantly increased after
132     Further, this work implicates the use of thrombospondin-1 levels in platelets as an indicator of
133               PEDF overexpression suppressed thrombospondin-1 levels in vitro.
134 te (p < 0.02) but not with changes in plasma thrombospondin-1 levels.
135 is study, we investigate the function of the thrombospondin-1-like glycoprotein, Nel (neural epiderma
136              Physiological concentrations of thrombospondin-1 limit the induction by lipopolysacchari
137 s demonstrated by showing that LPA abrogated thrombospondin-1-mediated inhibition of neovascularizati
138 DAMTS-13 mutant lacking the second to eighth thrombospondin-1 motifs and the complement components C1
139  Disintegrin-like and Metalloproteinase with Thrombospondin-1 motifs) protein family, cleaves large p
140                                     Notably, thrombospondin-1 mRNA expression was significantly upreg
141 is a consequence of both increased levels of thrombospondin-1 mRNA in megakaryocytes, as well as incr
142                                              Thrombospondin-1 mRNA increased in 7- and 12-week-old ra
143 lasma levels of von Willebrand factor (VWF), thrombospondin-1, myeloperoxidase, ADAMTS-13, and active
144  NO/cGMP signaling could be detected only in thrombospondin-1-null cells.
145 responses to NO were preserved in irradiated thrombospondin-1-null hindlimbs.
146            We show here that soft tissues in thrombospondin-1-null mice are remarkably resistant to r
147 y of oxygen levels in the ischemic tissue of thrombospondin-1-null mice as measured by electron param
148 elve hours after 25-Gy hindlimb irradiation, thrombospondin-1-null mice showed significantly less cel
149 cking CD47 showed radioresistance similar to thrombospondin-1-null mice.
150 issue survival was significantly enhanced in thrombospondin-1-null mice.
151                                              Thrombospondin-1-null murine platelets fail to aggregate
152 tions of the NO synthase substrate arginine, thrombospondin-1-null platelets have elevated basal cGMP
153 in skeletal muscle perfusion was enhanced in thrombospondin-1-null relative to wild-type mice, implic
154 ed cGMP accumulation and mimic the effect of thrombospondin-1 on aggregation.
155 sis factor-related apoptosis-induced ligand, thrombospondin-1, optineurin, and palladin.
156 y to promote angiogenesis in the presence of thrombospondin-1 or -2.
157  marrow-derived macrophages that lack either thrombospondin-1 or CD47 exhibit diminished induction of
158                                  Blockade of thrombospondin-1 or CD47 provides local radioprotection
159  binding of radiolabeled signature domain of thrombospondin-1 or SIRPalpha (signal-regulatory protein
160 M organization, we find that accumulation of thrombospondin-1 or thrombospondin-5 puncta within cell-
161 pression levels of either tubulointerstitial thrombospondin-1 or transforming growth factor-beta desp
162 by antibodies, recombinant type 1 repeats of thrombospondin-1, or CD36-binding peptides was sufficien
163  putative latent TGFbeta1 activators include thrombospondin-1, oxidants, and various proteases.
164 nant C-terminal regions of thrombospondin-1, thrombospondin-1 peptides, or CD47 antibodies was also s
165 d messenger RNA expression of interleukin-6, thrombospondin-1, plasminogen activator inhibitor-1, and
166                                              Thrombospondin-1 prevents NO-mediated relaxation of prec
167 sferase 2 (POFUT2) was described for TSRs of thrombospondin-1, properdin, and F-spondin within the se
168 e evolved from cell-SELEX that can recognize thrombospondin-1 protein in human plasma samples.
169  design of an M55-aptasensor for quantifying thrombospondin-1 protein in plasma samples.
170                              CD47 is a known thrombospondin-1 receptor but was not previously reporte
171                              Ligation of the thrombospondin-1 receptor CD47 by recombinant C-terminal
172  of CD47 (an alphavbeta3 integrin coreceptor/thrombospondin-1 receptor) and integrins alphavbeta3 and
173                                              Thrombospondin-1 regulates inflammation by engaging seve
174                                              Thrombospondin-1 regulates nitric oxide (NO) signaling i
175 evels of another key antiangiogenic protein, thrombospondin-1, reinforcing its antitumor and antiangi
176           Furthermore, the endogenous ligand thrombospondin-1, responsible for the synaptogenic prope
177                            In the absence of thrombospondin-1, retinal neovascular membranes are mark
178 h shear and static conditions, and exogenous thrombospondin-1 reverses this delay.
179 ng in ischemic soft tissues, suggesting that thrombospondin-1 signaling via its receptor CD47 could c
180 le cells, picomolar concentrations of native thrombospondin-1 similarly inhibited NO signaling in vas
181                    Renal IRI upregulated the thrombospondin-1-SIRP-alpha signaling axis and was assoc
182 -898 mimics the antiangiogenic properties of thrombospondin-1, so we hypothesized that ABT-898 will p
183 ugh some fragments and peptides derived from thrombospondin-1 stimulate or inhibit platelet aggregati
184 l metalloprotease (M), disintegrin-like (D), thrombospondin-1 (T), Cys-rich (C), and spacer (S) domai
185 ARID domain-dependent, BAF-A associations at THROMBOSPONDIN 1 (THBS1) led to the concomitant upregula
186                                              Thrombospondin 1 (THBS1) was recently shown to promote b
187 on of the TGF-beta-regulated biomarker genes thrombospondin-1 (THBS1) and cartilage oligomeric protei
188                                              Thrombospondin-1 (THBS1) is a large extracellular matrix
189                                  We examined thrombospondin-1 (THBS1, alias TSP-1) expression in huma
190 -regulated the potent angiogenesis inhibitor thrombospondin-1, thereby triggering a negative feedback
191 or CD47 by recombinant C-terminal regions of thrombospondin-1, thrombospondin-1 peptides, or CD47 ant
192 nhibition can be explained by the ability of thrombospondin-1 to disrupt the interaction between CD47
193 jury, possibly due to the stimulation of the thrombospondin-1-transforming growth factor-beta pathway
194 rtilage oligomeric matrix protein (COMP) and thrombospondin 1 (TSP-1) correlated moderately well with
195                                              Thrombospondin 1 (TSP-1) is a matricellular protein that
196 aneous somatic Ca2+ transients, synaptogenic thrombospondin 1 (TSP-1) release, and synapse formation.
197                   Basal muscle expression of thrombospondin 1 (TSP-1) was approximately 900% greater
198 ial growth factor (VEGF), but high levels of thrombospondin 1 (TSP-1), predicted liver dysfunction af
199 n-associated protein, and its natural ligand thrombospondin 1 (TSP-1).
200 cluding Rho-activated kinase II (ROCKII) and thrombospondin 1 (TSP-1).
201 eptidomimetic of the anti-angiogenic protein thrombospondin-1 (TSP-1 PM).
202 sis through binding to the type 1 repeats of thrombospondin-1 (TSP-1) and activating Fyn tyrosine kin
203 grins) and ECM noncellular components [i.e., thrombospondin-1 (TSP-1) and fibronectin (FN)] seem to t
204 uced angiogenesis and elevated expression of thrombospondin-1 (TSP-1) and its receptor CD36, anti-ang
205 he capacity to bind the CD47-binding partner thrombospondin-1 (TSP-1) and that treatment of aged eryt
206 tion, the loss of p53 led to a deficiency in thrombospondin-1 (TSP-1) expression, a potent antiangiog
207 nteractions with proteins and proteoglycans, thrombospondin-1 (TSP-1) functions at the interface of t
208                    The N700S polymorphism of thrombospondin-1 (TSP-1) has been identified as a potent
209 ntact dependent, leading to up-regulation of thrombospondin-1 (TSP-1) in DCs.
210 ine fashion by stimulating the expression of thrombospondin-1 (Tsp-1) in fibroblasts present in both
211                                              Thrombospondin-1 (TSP-1) inhibits angiogenesis and activ
212                                              Thrombospondin-1 (TSP-1) inhibits growth factor signalin
213                                              Thrombospondin-1 (TSP-1) is a glycoprotein considered as
214                                              Thrombospondin-1 (TSP-1) is a large extracellular matrix
215                                              Thrombospondin-1 (TSP-1) is a major activator of latent
216                                              Thrombospondin-1 (TSP-1) is a multifunctional protein wh
217                                              Thrombospondin-1 (TSP-1) is a multimodular glycoprotein
218 cular permeability and angiogenesis, whereas thrombospondin-1 (TSP-1) is a potent angiogenic inhibito
219                                              Thrombospondin-1 (TSP-1) is an endogenous inhibitor of a
220                                              Thrombospondin-1 (TSP-1) is an endogenous inhibitor of a
221          Here we show TGF-beta activation by thrombospondin-1 (TSP-1) is both required and sufficient
222 he expression of an anti-angiogenic protein, thrombospondin-1 (TSP-1) is down-regulated in the prosta
223                                              Thrombospondin-1 (TSP-1) is one of the anti-angiogenic f
224                            Here we show that thrombospondin-1 (TSP-1) prevents cAMP/protein kinase A
225 d decrease in p53 expression, thus enhancing thrombospondin-1 (TSP-1) release from EC and reducing FG
226  of atherogenesis, we considered the role of thrombospondin-1 (TSP-1), a matricellular protein releas
227                                              Thrombospondin-1 (TSP-1), a potent antiangiogenic and pr
228                 Increasing evidence suggests thrombospondin-1 (TSP-1), a potent proatherogenic matric
229 ntiangiogenic factors, including endostatin, thrombospondin-1 (TSP-1), and pigment epithelium-derived
230 nd re-expression of a synaptogenic molecule, thrombospondin-1 (TSP-1), apart from supporting neuronal
231 xtensive in vitro and in vivo validations on thrombospondin-1 (TSP-1), because it has been previously
232 ent derived from the second type-1 repeat of thrombospondin-1 (TSP-1), is known to possess antiangiog
233                   The matricellular protein, thrombospondin-1 (TSP-1), is prominently expressed durin
234  mice deficient in tumstatin, endostatin, or thrombospondin-1 (TSP-1), to address the role that these
235 iptionally down-regulating the production of thrombospondin-1 (TSP-1)--known earlier for both its ant
236  spectrometry helped pinpoint this factor as thrombospondin-1 (TSP-1).
237 y involving the TGF-beta-activating protease thrombospondin-1 (TSP-1).
238  a receptor for the anti-angiogenic molecule thrombospondin-1 (TSP-1).
239 mposed of the three type-1 repeats (3TSR) of thrombospondin-1 (TSP-1).
240 n-8 (IL-8), and on an antiangiogenic factor, thrombospondin-1 (TSP-1).
241  muscle cells that mediates up-regulation of thrombospondin-1 (TSP-1).
242 ic destruction of the antitumorigenic factor thrombospondin-1 (Tsp-1).
243 on of a key secreted anti-angiogenic factor, thrombospondin-1 (Tsp-1).
244 ts high levels of the angiogenesis inhibitor thrombospondin-1 (TSP-1).
245       The Nel protein contains an N-terminal thrombospondin-1 (TSP-N) domain, five cysteine-rich doma
246 ssion of two endogenous vascular regulators, thrombospondins 1 (TSP-1) and 2 (TSP-2).
247 tein expression of anti-angiogenic peptides (thrombospondin-1, TSP-1; and endostatin) as well as pro-
248 le expressed significantly reduced levels of thrombospondin 1 (TSP1) (P = 0.042) and increased levels
249 onic opiate treatment of rats down-regulates thrombospondin 1 (TSP1) expression in the nucleus accumb
250                                              Thrombospondin 1 (TSP1) expression is increased in diabe
251                                              Thrombospondin 1 (TSP1) has been suggested as a counter
252                                              Thrombospondin 1 (TSP1) induces focal adhesion disassemb
253                    The matricellular protein thrombospondin 1 (TSP1) regulates cell adhesion and moti
254 e levels of an extracellular matrix protein, thrombospondin 1 (TSP1), an angiogenesis inhibitor.
255                                              Thrombospondin 1 (TSP1), an oligomeric matrix protein, i
256  variety of endogenous inhibitors, including thrombospondin 1 (TSP1).
257 /-) manifested higher expression of CTGF and thrombospondin 1 (TSP1).
258 s 1, IV and XVIII as well as fibronectin and thrombospondin 1 (TSP1).
259 lated with downregulation of anti-angiogenic thrombospondin-1 (Tsp1) and related proteins, such as co
260 -regulated STAT3 phosphorylation, astrocytic thrombospondin-1 (TSP1) and synaptic low-density lipopro
261 njury via a mechanism mediated by astrocytic thrombospondin-1 (TSP1) and synaptic low-density lipopro
262                                              Thrombospondin-1 (TSP1) and TSP2 are matricellular prote
263                                              Thrombospondin-1 (TSP1) binding to calreticulin (CRT) on
264                                              Thrombospondin-1 (TSP1) binding to calreticulin (CRT) on
265                  Here we report that soluble thrombospondin-1 (TSP1) binds to the extracellular part
266                                              Thrombospondin-1 (TSP1) can inhibit angiogenic responses
267                               The N-terminal thrombospondin-1 (TSP1) domain forms an unexpectedly ext
268 from mice lacking the angiogenesis inhibitor thrombospondin-1 (TSP1) exhibit exaggerated angiogenic r
269                            Plasmin (PLS) and thrombospondin-1 (TSP1) have been studied individually a
270                   We confirm upregulation of thrombospondin-1 (TSP1) in Id cDKO hearts.
271 s calcineurin/NFATc1-dependent expression of thrombospondin-1 (Tsp1) in lung endothelial cells to dri
272                                              Thrombospondin-1 (TSP1) inhibits angiogenesis, in part,
273                                              Thrombospondin-1 (TSP1) is a multifunctional matricellul
274                         The secreted protein thrombospondin-1 (TSP1) limits NO-stimulated blood flow
275                                              Thrombospondin-1 (TSP1) limits the angiogenic and vasodi
276 nvolved in the regulation of angiogenesis is thrombospondin-1 (TSP1), a matricellular glycoprotein kn
277  that, upon CREB activation, HDAC2 represses thrombospondin-1 (TSP1), a potent angiogenesis inhibitor
278 ntiation 1 (Id1), which negatively regulates thrombospondin-1 (TSP1), an inhibitor of angiogenesis.
279 ited tumor angiogenesis via the induction of Thrombospondin-1 (TSP1), and consequently suppressed tum
280  in the levels of the angiogenesis inhibitor thrombospondin-1 (TSP1), but the mechanisms underlying t
281  by JHDM1D-mediated epigenetic regulation of thrombospondin-1 (TSP1), forming a JHDM1D/TSP1/TGF-beta/
282 d to be an inhibitory signaling receptor for thrombospondin-1 (TSP1), the molecular mechanism for tra
283 he hypothesis that the matricellular protein thrombospondin-1 (TSP1), through binding to and activati
284 ently, we reported that the secreted protein thrombospondin-1 (TSP1), through its cell surface recept
285       We recently found that leukocytes from thrombospondin-1 (TSP1)-deficient mice exhibit significa
286 lating erythrocyte microparticles (MPs), and thrombospondin-1 (TSP1).
287 nsin II induce the latent TGF-beta activator thrombospondin-1 (TSP1).
288 ine in the levels of angiogenesis inhibitor, thrombospondin-1 (TSP1).
289                        Combined therapy with thrombospondin-1 type I repeats (3TSR) and chemotherapy
290  EOC cells with a recombinant version of the thrombospondin-1 type I repeats (3TSR) induced more apop
291 TS (A Disintegrin and metalloproteinase with thrombospondin-1 type repeats) family of enzymes have be
292                          The Ser-700 variant thrombospondin-1 was conformationally more labile than t
293   The KD value of the aptamer M55 binding to thrombospondin-1 was determined as 0.5 +/- 0.2 muM with
294   Inhibition of T cell receptor signaling by thrombospondin-1 was lost in CD47-deficient T cells that
295 binding selectivity, the signature domain of thrombospondin-1 was more potent than those of thrombosp
296              Levels of VEGF, E-selectin, and thrombospondin-1 were not associated with clinical outco
297       Plasma levels of VEGF, E-selectin, and thrombospondin-1 were obtained serially.
298 ty group box-1 protein, chemokine CXCL4, and thrombospondin-1 were significantly higher in patients w
299 Here, we found that both CD47 and its ligand thrombospondin-1 were upregulated after renal IRI in mic
300 telet-derived growth factor, collagen I, and thrombospondin-1) were higher in PEDF-null mice at basel

WebLSDに未収録の専門用語(用法)は "新規対訳" から投稿できます。
 
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