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1 synergism, e.g., human platelet thrombin and thromboxane A2 receptors.
2 d with a net increase in Galphaq coupling to thromboxane A2 receptors.
3 ors caused an increase in ligand affinity of thromboxane A2 receptors.
4 5 kDa) indistinguishable from human platelet thromboxane A2 receptors.
5 ation of human platelets by thrombin and the thromboxane A2 receptor agonist U46619 lead to phosphory
6 e to inhibit vasoconstriction induced by the thromboxane A2 receptor agonist U46619, which suggest a
10 f cyclooxygenase inhibition with aspirin and thromboxane A2 receptor blockade with ifetroban on the c
11 in E2 (EP)1, EP4, prostaglandin F2alpha, and thromboxane A2 receptors but not anti-inflammatory EP2,
12 lts demonstrate the presence of a functional thromboxane A2 receptor in oligodendrocytes and are cons
13 us observations indicating a high density of thromboxane A2 receptors in myelinated brain and spinal
16 he same position as the second intron of the thromboxane A2 receptor, prostaglandin D2 receptor, pros
17 receptors increases Galphaq association with thromboxane A2 receptors thereby shifting them to a high
18 rostaglandin F2alpha receptor (FP) (61), and thromboxane A2 receptor (TP) (11) while sparing EP2, EP3
19 rs include the PGF2 alpha receptor (FP), the thromboxane A2 receptor (TP) and the prostacyclin recept
20 structural flexibility of the purified human thromboxane A2 receptor (TP) was characterized by spectr
22 ted to elucidate the molecular mechanisms of thromboxane A2 receptor (TP)-induced insulin resistance
25 oupled receptors; two splice variants of the thromboxane A2 receptor (TPalpha and TPbeta) have been c
26 racterization of the signaling properties of thromboxane A2 receptor (TPalpha) -Galpha12 and -Galpha1
27 n interacting partner of the beta-isoform of thromboxane A2 receptor (TPbeta) by yeast two-hybrid scr
29 d created by oxidative stress, activates the thromboxane A2 receptor (TXAR) and the Rho-associated ki
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