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1 a and hindbrain which express immunoreactive thromboxane synthase.
2 ns, suggesting a direct inhibitory effect on thromboxane synthase.
3 ell motility was attenuated by inhibitors of thromboxane synthase.
4 nhibitor, and is enhanced by an inhibitor of thromboxane synthase.
5 a series of cell specific prostaglandin and thromboxane synthases.
6 xpression of nuclear factor-kappaB-dependent thromboxane synthase and microsomal PGE(2) synthase was
8 ation of the efficiency of prostaglandin and thromboxane synthase-directed endoperoxide isomerization
9 unohistochemical techniques, we demonstrated thromboxane synthase immunostaining in regions important
12 glandin H2 receptor antagonist SQ29548 and a thromboxane synthase inhibitor, furegrelate, either alon
14 hat, consistent with physiological evidence, thromboxane synthase is present in brain regions which a
15 The sequential action of purified COX-2 and thromboxane synthase on AEA and 2-AG provided thromboxan
20 ntly, we reported prognostic significance of thromboxane synthase (TXAS) gene expression in invasive
21 ance Raman (RR) spectra of recombinant human thromboxane synthase (TXAS) in both the presence and the
27 xane A(2) (TxA(2)) is a prostanoid formed by thromboxane synthase using the cyclooxygenase product pr
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