ライフサイエンスコーパス: thymidine incorporation

1 ted beta-TCP or CaSO(4) was assessed by (3)H thymidine incorporation.

2 ered DNA synthesis block, as measured by [3H]thymidine incorporation.

3 T-cell proliferation was assessed by [3H] thymidine incorporation.

4 lation, p70S6K kinase activity, or ASM [(3)H]thymidine incorporation.

5 s cell line FHL124, it was assessed by [(3)H]thymidine incorporation.

6 n D1, cyclin A, and cdk4, and rescued [(3)H]-thymidine incorporation.

7 ability of elevated glucose to increase [3H]thymidine incorporation.

8 Lymphocyte proliferation was tested by H(3)-thymidine incorporation.

9 ogen, and proliferation was measured as (3)H-thymidine incorporation.

10 bility by the dye-exclusion assay or in [3H] thymidine incorporation.

11 tly isolated IOMM-Lee clones, as assessed by thymidine incorporation.

12 in D(1) caused a threefold increase in [(3)H]thymidine incorporation.

13 acid-induced increase in PGE2 production and thymidine incorporation.

14 p21 showed increased basal and IGF-I-induced thymidine incorporation.

15 nduced MC proliferation as measured by [(3)H]thymidine incorporation.

16 n FSH-induced progesterone production and GC thymidine incorporation.

17 ic T cell proliferation was measured by (3)H-thymidine incorporation.

18 ls with INSL3 stimulated cAMP production and thymidine incorporation.

19 3) and Thr(308)), but did not increase [(3)H]thymidine incorporation.

20 vented the hypoxia-induced increase in [(3)H]thymidine incorporation.

21 o acid completely reversed the inhibition of thymidine incorporation.

22 1-methyl-tryptophan prevented inhibition of thymidine incorporation.

23 ogenesis was measured as a function of [(3)H]thymidine incorporation.

24 DNA synthesis in VSMCs was examined by [(3)H]thymidine incorporation.

25 entration-dependent manner as well as [(3)H] thymidine incorporation.

26 ect on epidermal growth factor-induced [(3)H]thymidine incorporation.

27 so blocked the PMA-induced increase in [(3)H]thymidine incorporation.

28 Cell proliferation was measured by 3H-thymidine incorporation.

29 in the presence of CLT was determined by [3H]thymidine incorporation.

30 ins on RPE proliferation was investigated by thymidine incorporation.

31 Jkt cell proliferation was measured by [(3)H]thymidine incorporation.

32 ffects on DNA synthesis were assayed by [3H]-thymidine incorporation.

33 ellular proliferation, as measured by [(3)H]-thymidine incorporation.

34 uction of gene expression, and inhibition of thymidine incorporation.

35 d DNA synthesis of RASMCs as measured by [3H]thymidine incorporation.

36 quantified by Ki67 immunostaining and [(3)H]thymidine incorporation.

37 ial cells using capillary tube formation and thymidine incorporation.

38 mM peptides for 7 days) was assessed by (3)H-thymidine incorporation.

39 eas suppression of Nkx6.1 expression reduces thymidine incorporation.

40 ction, COX2 expression, PGE2 production, and thymidine incorporation.

41 T cell proliferation was determined using 3H-thymidine incorporation.

42 was evaluated using siRNA knockdown and [3H] thymidine incorporation.

43 CD4+ T-cell responses were measured by [3H]-thymidine incorporation.

44 cell nuclear antigen (PCNA) and radiolabeled thymidine incorporation.

45 ation was assessed for proliferation by [3H]-thymidine incorporation.

46 t also induced CVC proliferation based on 3H-thymidine incorporation.

47 s that stimulates Ca2+ influx, modulates [3H]thymidine incorporation.

48 doxically potentiated glucose-stimulated [3H]thymidine incorporation 2-4-fold above elevated glucose

49 In contrast, TGF-beta(1) stimulated (3)H-thymidine incorporation (200%; P<0.001) and cell prolife

50 ) induced a dose-responsive increase in (3)H-thymidine incorporation (25 ng/ml VEGF(165) : 2.3-fold i

51 nd cytokine secretion were measured by [(3)H]thymidine incorporation, (51)Cr release, and enzyme-link

52 o matrix prior to scrape-wounding induced 3H-thymidine incorporation 8-fold and shortened the time to

53 ine in the absence of serum stimulated [(3)H]thymidine incorporation 8-fold.

54 As measured by [(3)H]thymidine incorporation, 95% of 168 T cell clones from s

55 A dramatic reduction in 14C-thymidine incorporation after gamma irradiation was obse

56 There was only a 35% increase in [(3)H]-thymidine incorporation, after 24 hours of E(2) treatmen

57 ncrease in DNA synthesis assessed with [(3)H]thymidine incorporation, an increase in G(2) and M cell

58 ell proliferation was evaluated using [(3)H]-thymidine incorporation and 3,(4,5-dimethylthiazol-2-yl)

59 Cell proliferation was measured by thymidine incorporation and 5,6-carboxyfluorescein succi

60 ed leukocyte reaction were determined by [3H]thymidine incorporation and 51Cr release assays, respect

61 eatic cancer cell lines was evaluated by [3H]thymidine incorporation and a TGFbeta-responsive reporte

62 luid shear of 20 dynes/cm(2) increased [(3)H]thymidine incorporation and alkaline phosphatase activit

63 Moreover, (3)H-thymidine incorporation and an in vivo angiogenic assay

64 a concentration-dependent increase in [(3)H]thymidine incorporation and an increase in IGF-I secreti

65 Thymidine incorporation and apoptosis analyses further d

66 e cAMP pools that modulate effects of PKB on thymidine incorporation and BAD phosphorylation in FDCP2

67 6-fold increases, respectively, in tritiated thymidine incorporation and bromodeoxyuridine incorporat

68 Keratocyte proliferation was measured by [3H]thymidine incorporation and by DNA content of the cultur

69 iability, or apoptosis, as detected by [(3)H]thymidine incorporation and by propidium iodide staining

70 heral blood were measured by using tritiated thymidine incorporation and carboxyfluorescein succinimi

71 ntigens using proliferation assays (based on thymidine incorporation and carboxyfluorescein succinimi

72 d growth suppressor function, as measured by thymidine incorporation and caspase-3 activation.

73 roliferation of the cells was assessed by 3H-thymidine incorporation and cell attachment measured by

74 Cell growth was determined using [3H]thymidine incorporation and cell counting.

75 eration and cell growth as measured by [(3)H]thymidine incorporation and cell counts and a 2.3-fold i

76 cle cells, and measuring its effect on (3)[H]thymidine incorporation and cell number.

77 table prostacyclin analogues on [methyl-(3)H]thymidine incorporation and cell proliferation were inve

78 ely 25-30%) inhibition of EGF-stimulated ASM thymidine incorporation and cell proliferation, whereas

79 10 ng/ml GH stimulated chondrocyte thymidine incorporation and collagen X mRNA expression,

80 GF21 (5 and 10 mug/ml) inhibited chondrocyte thymidine incorporation and collagen X mRNA expression.

81 vation was normal, as demonstrated by normal thymidine incorporation and CSFE dilution of T cells sti

82 isolated hepatoma 7288CTC in vivo and on [3H]thymidine incorporation and DNA content during perfusion

83 situ with melatonin also decreased tumor [3H]thymidine incorporation and DNA content; these effects o

84 responder T cells were determined by [(3)H]-thymidine incorporation and ELISA, respectively.

85 o assess B lymphocyte activity, we used (3)H-thymidine incorporation and enzyme-linked immunosorbent

86 a 30-min steady shear stress increased [(3)H]thymidine incorporation and Erk1/2 phosphorylation in ob

87 Concordantly, thymidine incorporation and extracellular signal-regulat

88 re lymphocyte proliferation (tritium labeled thymidine incorporation and flow cytometric bivariate pr

89 lockade on cell growth, then we performed 3H-thymidine incorporation and flow cytometry assays to inv

90 tion was assessed for proliferation by [(3)H]thymidine incorporation and for IL-2, IFN-gamma, IL-4, a

91 knock-out (P4KO) mice showed impaired [(3)H]thymidine incorporation and G1 phase arrest as compared

92 P cells caused inhibition of cell growth and thymidine incorporation and halted cells at the G(1) pha

93 IGFBP-5-stimulated [(3)H]thymidine incorporation and IGF-I secretion were partly

94 na tabacum) BY-2 cells caused an increase in thymidine incorporation and in the mitotic index of aphi

95 h a decrease in mitogenicity, as measured by thymidine incorporation and increased sensitivity to sta

96 Both [(3)H]thymidine incorporation and Ki67 expression were inhibit

97 [(3)H]Thymidine incorporation and Ki67 immunohistochemistry we

98 division rates were determined by tritiated thymidine incorporation and Ki67 immunoreactivity.

99 d not induce a significant increase in [(3)H]thymidine incorporation and PAR2-AP, PAR3-AP, and PAR4-A

100 ckade of Tcf-4 resulted in inhibition of [3H]thymidine incorporation and partial blockade of the G1-S

101 division and tissue repair, assessed by [3H]thymidine incorporation and proliferative cell nuclear a

102 These findings suggest that increased [(3)H]thymidine incorporation and reducing power indicate incr

103 pathway was associated with increased [(3)H]thymidine incorporation and resistance to apoptosis.

104 nd migration rates were determined by [(3)H]-thymidine incorporation and scratch wound healing assay,

105 Proliferation was measured by (3)H-thymidine incorporation and TGF-beta by RT-PCR and ELISA

106 lian target of rapamycin (mTOR) leads to [3H]thymidine incorporation and that mTOR activation is medi

107 portionally increased both the rate of [(3)H]thymidine incorporation and the rate of reduction of MTS

108 or determining T cell responses (i.e., [(3)H]thymidine incorporation and the use of cell proliferatio

109 d cytokine secretion of T cells using [(3)H]-thymidine incorporation and TNF-alpha assays, respective

110 [(3)H]-Thymidine incorporation and total CD8(+) cell numbers, a

111 rimed ex vivo autologous proliferation ((3)H-thymidine incorporation) and cytotoxicity (chromium-51 r

112 cytometry), T-cell proliferative responses (thymidine incorporation), and cytokine expression (Fluor

113 Cell proliferation was detected by H-thymidine incorporation, and apoptosis by Annexin V and

114 Cell numbers, 3H-thymidine incorporation, and Boyden chamber assays were

115 sponse to DC stimulation was assessed by [3H]thymidine incorporation, and by multicolor flow cytometr

116 Intracellular staining, [(3)H]-thymidine incorporation, and carboxy-fluorescein succini

117 Trypan blue exclusion, [(3)H]thymidine incorporation, and cell cycle analyses, couple

118 d the effects of glutamine on MAPK activity, thymidine incorporation, and cell number in glutamine-st

119 Cell proliferation was determined by [(3)H]thymidine incorporation, and cell viability was measured

120 ts by flow cytometry, DNA synthesis by [(3)H]thymidine incorporation, and collagen synthesis by [(3)H

121 , non-protein thiol, ATP, transport systems, thymidine incorporation, and DNA cleavage.

122 5-bromo-2'-deoxyuridine incorporation, [(3)H]thymidine incorporation, and fluorescence-activated cell

123 cytokine enzyme-linked immunosorbent assay, thymidine incorporation, and gene array studies.

124 ll proliferation was determined by tritiated thymidine incorporation, and IL-2 and -15 receptor expre

125 DNA synthesis was determined by (3)H-thymidine incorporation, and maximal cell density was de

126 FRbeta-promoted phosphoinositide hydrolysis, thymidine incorporation, and overall PDGFRbeta tyrosyl p

127 lastoma (pRB) phosphorylation; and (c) [(3)H]thymidine incorporation, and partially reversed TGF-beta

128 acid-induced increase in mPGES1 expression, thymidine incorporation, and PGE(2) production.

129 acid-induced increase in mPGES1 expression, thymidine incorporation, and PGE2 production.

130 ited the TGF-beta-mediated decrease in [(3)H]thymidine incorporation, and repressed TGF-beta-activate

131 L) inhibited basal and serum-stimulated (3)H-thymidine incorporation, and TGF-beta(1) and BMPs inhibi

132 e of which was found to partially inhibit 3H-thymidine incorporation as a result of increased cell de

133 ymphocyte proliferation was assessed by (3)H-thymidine incorporation as well as by the carboxyfluores

134 and U46619 all enhanced EGF-stimulated [3H]-thymidine incorporation as well as late-phase Akt and p7

135 ocyte proliferation was demonstrated by (3)H thymidine incorporation assay and carboxyfluorescein suc

136 Tritiated thymidine incorporation assay revealed that Cu(GTSC) and

137 re with WT MSCs as evaluated by CFU-F assay, thymidine incorporation assay, and beta-galactosidase st

138 cytokine responses were assessed using (3)H-thymidine incorporation assay, enzyme-linked immunosorbe

139 f gemcitabine was retained as tested using a thymidine incorporation assay.

140 ion of IRBP-specific CD4(+) T cells by [(3)H]thymidine incorporation assay.

141 T cell proliferation was assessed by thymidine incorporation assay.

142 ls in response to mitogens, we perform a [3H]thymidine incorporation assay.

143 staining and caspase-3 activation, and [(3)H]thymidine incorporation assay.

144 Thymidine incorporation assays also showed a peripheral

145 d KLF6 mutants was characterized by in vitro thymidine incorporation assays and Western blotting.

146 Lastly, thymidine incorporation assays show that cells seeded on

147 (3)H-Thymidine incorporation assays showed that epidermal gro

148 within 30 min, which preceded a decrease in thymidine incorporation at 6 and 24 h.

149 es upon culture exhibit a 2-fold increase in thymidine incorporation at day 5 (D5) when compared to h

150 A linear increase in [3H]thymidine incorporation began approximately 16 hours aft

151 rterial blood reversed the inhibition of [3H]thymidine incorporation but had no effect on FA uptake.

152 lb MK, exhibits growth arrest as measured by thymidine incorporation, but does not prevent cells that

153 t starved of glutamine, glutamine stimulated thymidine incorporation by 3-fold, and 8-CPT-cAMP comple

154 PACAP markedly inhibited Shh-induced thymidine incorporation by 50 and 85% in rat and mouse G

155 um- or PDGF-induced SMC cell count and [(3)H]thymidine incorporation by 62% and 81%, respectively.

156 Occludin depletion also increased thymidine incorporation by 90% and Ki67-positive cells b

157 athway is associated with induction of [(3)H]thymidine incorporation by a molecule activated downstre

158 it significantly enhanced insulin-stimulated thymidine incorporation by about 2-fold.

159 e Ki for the inhibition of LA uptake and [3H]thymidine incorporation by alpha-linolenic acid was 0.18

160 nce KHGK) exhibited a biphasic effect on [3H]thymidine incorporation by cultured endothelial cells an

161 erodimer also stimulated cAMP production and thymidine incorporation by cultured thyroid cells and in

162 MEHP inhibited [(3)H]thymidine incorporation by primary murine bone marrow B

163 th [3H]thymidine and examining levels of [3H]thymidine incorporation by scintillation counting.

164 After treatment with VEGF, [(3)H]thymidine incorporation, c-fos induction, and the number

165 oration in primary fetal cardiomyocytes, and thymidine incorporation, cell cycle progression, and ind

166 Proliferation ([(3)H]-thymidine incorporation, cell number, cell cycle) and hy

167 luid obtained from normal subjects increased thymidine incorporation, cell number, ERK phosphorylatio

168 bited serum (2.5%)-induced cell growth ((3)H-thymidine incorporation), collagen synthesis ((3)H-proli

169 ing as measured by reporter gene expression, thymidine incorporation, collagen production, and phosph

170 id not prevent the GH stimulatory effects on thymidine incorporation, collagen X, and IGF-1 expressio

171 ected with control siRNA, GH increased [(3)H]thymidine incorporation, collagen X, and IGF-1 mRNA expr

172 Cancer growth was assessed in vitro by [(3)H]thymidine incorporation, colony formation assays, and in

173 pressing iex-1-infected cells had lower 3[H]-thymidine incorporation compared with AdGFP-infected con

174 This increased thymidine incorporation could be suppressed by the thiol

175 eus-specific T-cells were quantified by (3)H-thymidine incorporation; cytokine release was measured b

176 On the contrary, [(3)H]-thymidine incorporation decreased at high TMr-GAD concen

177 3H-thymidine incorporation demonstrated that the recombinan

178 Proliferation (cell number and [3H]-thymidine incorporation), differentiation (alkaline phos

179 As measured by [3H]thymidine incorporation, DNA synthesis in all seven MTAP

180 [3H]Thymidine incorporation during perfusion in situ was als

181 nd the rates of tumor growth in vivo and [3H]thymidine incorporation during perfusion were decreased.

182 ent levels, correlating with the increase in thymidine incorporation during the same time period.

183 y a formazan-based colorimetric assay, [(3)H]thymidine incorporation, fluorescent nuclear staining, a

184 h exhibited a significant increase in [(3)H]thymidine incorporation followed by a significant increa

185 as a mitogen showed 9202 +/- 2107 cpm [(3)H]thymidine incorporation for BALMs from IL-10(-/-) mice a

186 culture, exhibited markedly diminished [(3)H]thymidine incorporation (>90% decreased), and died spont

187 ymphocyte proliferation was assessed by (3)H-thymidine incorporation in 12 severe AAH patients and ag

188 eratinocytes to 8-Cl-adenosine inhibited [3H]thymidine incorporation in a dose-dependent manner with

189 th EGF and FBS significantly increased [(3)H]thymidine incorporation in CSK cells.

190 Thymidine incorporation in early-passage RPE cells showe

191 Exogenous ATP stimulated [(3)H]thymidine incorporation in fibroblasts as did UTP, ADPbe

192 ith anti-CD3 mAb induced transiently greater thymidine incorporation in IL-16-deficient CD4(+) T cell

193 b inhibited both Annexin V(FITC) binding and thymidine incorporation in mast cells maintained with IL

194 = 7, P = 0.0257, t test) increase in [(3)H]-thymidine incorporation in mucosal biopsy specimens, abo

195 ge of OCL precursors and that AXII increased thymidine incorporation in OCL precursors.

196 n = 6, t test, P = 0.001) increase in [(3)H]-thymidine incorporation in OE33(E)(GR) cells, abolished

197 actors into conditioned media that stimulate thymidine incorporation in primary fetal cardiomyocytes,

198 ivative was determined by inhibition of (3)H-thymidine incorporation in primary normal human bladder

199 ctivating peptides were unable to induce [3H]thymidine incorporation in pulmonary artery fibroblasts.

200 CSK cells failed to increase [(3)H]thymidine incorporation in response to exogenous 14,15-E

201 V(FITC) binding and only partially inhibited thymidine incorporation in sPLA(2)-supplemented mast cel

202 -12.5% to 27.9+/-13.6% of cells; P<0.01) and thymidine incorporation in successfully transfected cell

203 th plate proliferative zone and of the total thymidine incorporation in the metatarsal bone.

204 te of a profound T-cell expansion and higher thymidine incorporation in unstimulated Nf1-deficient T

205 ed Ki-67 mRNA expression levels and enhanced thymidine incorporation in Wnt6-treated macrophage cultu

206 increased DNA synthesis (determined by [(3)H]thymidine incorporation) in fetal and adult cells by 211

207 Functional assays using [3H]thymidine incorporation indicated that granulin may be a

208 also decreased EGF- and serum-mediated [(3)H]thymidine incorporation, indicating that alterations in

209 nd that these compounds potently inhibit [3H]thymidine incorporation into a murine microglial cell li

210 ours resulted in a 1.8-fold increase of (3)H-thymidine incorporation into DNA and a significant incre

211 tion with EGF, however, stimulation of [(3)H]thymidine incorporation into DNA and an increase in hist

212 ed by a caffeine-inhibitable decrease in [3H]thymidine incorporation into DNA and Cdc25A down-regulat

213 monitored, indirectly by measuring tritiated thymidine incorporation into DNA and directly by the qua

214 In proliferating SMCs, apoE inhibited [(3)H]thymidine incorporation into DNA by 50%; however, despit

215 S-CAAX desensitized insulin-stimulated [(3)H]thymidine incorporation into DNA by about an order of ma

216 approaches, we show here that GCR1-enhanced thymidine incorporation into DNA depends on an increase

217 r cell proliferation determined by measuring thymidine incorporation into DNA following culture in th

218 rom its inhibitory effect on the increase in thymidine incorporation into DNA induced by recombinant

219 However, blockade of PKC diminished thymidine incorporation into DNA induced by VEGF but not

220 eration, as determined by inhibition of (3)H-thymidine incorporation into DNA of human peripheral blo

221 in vitro in 2% (but not 10%) FCS, [methyl-3H]thymidine incorporation into DNA was significantly incre

222 liferation, assessed as [3H-methyl]thymidine-thymidine incorporation into DNA, 2 h after irradiation.

223 ocalization, MAP and PI 3-kinase activation, thymidine incorporation into DNA, and cell viability, st

224 revious study, we showed that LA uptake, [3H]thymidine incorporation into DNA, and total DNA content

225 on, as its expression has no effect on [(3)H]thymidine incorporation into DNA.

226 cycle, assessed by flow cytometry and [(3)H]thymidine incorporation into DNA.

227 in mitogenic response, as indicated by [(3)H]thymidine incorporation into DNA.

228 tifying cell number, cell protein, and [(3)H]thymidine incorporation into DNA.

229 ion diminished VEGF-, FGF-, and EGF-promoted thymidine incorporation into DNA.

230 ctivated protein kinase (MAPK), and promoted thymidine incorporation into DNA.

231 ith IC50 values that match inhibition of [3H]thymidine incorporation into DNA.

232 n the capsular bag model and increased [(3)H]thymidine incorporation into FHL124 cells.

233 Cell proliferation was measured by 3H-thymidine incorporation into growing HGF and HUVEC.

234 rtial hepatectomy and peaked at 32 h ([(3)H]-thymidine incorporation into hepatic DNA and mitotic ind

235 Liver regeneration peaked at 24 h ([(3)H]-thymidine incorporation into hepatic DNA and mitotic ind

236 Liver regeneration was evaluated by [(3)H]-thymidine incorporation into hepatic DNA, the mitotic in

237 retains IGF-1 activity as indicated by [(3)H]thymidine incorporation into L6 myoblasts.

238 Three days after L4/5 ventral rhizotomy, [3H]thymidine incorporation into Remak Schwann cells increas

239 Nicotine also stimulates [(3)H]thymidine incorporation into the genome of these cells a

240 Although using radiolabeled [3H]thymidine incorporation is a limitation, the greatest be

241 By measuring [(3)H]thymidine incorporation, it was found that expression of

242 promoted cell death as shown by reduction of thymidine incorporation, loss of mitochondrial membrane

243 0 ng/ml) as determined by dye uptake and [3H]thymidine incorporation methods.

244 The increase in thymidine incorporation occurring in NOX5-S-overexpressi

245 s in suppression of mitogen-stimulated [(3)H]thymidine incorporation of preactivated lymphocytes with

246 3H-thymidine incorporation of the human lymphoid cell line

247 oM), an ErbB2-neutralizing antibody, blocked thymidine incorporation only in NE-treated cells.

248 Thymidine incorporation opposite C4'-AP is distinct from

249 ted by addition of DHT, no increase of [(3)H]thymidine incorporation or apoptosis resistance was demo

250 For studies of (3)H-thymidine incorporation or cell proliferation, PASMCs (p

251 ular DNA synthesis as detected by methyl-[3H]thymidine incorporation (P<0.001).

252 Increased [3H]thymidine incorporation persisted at least 10 d after ve

253 transgenic OT-II) was measured via a [(3)H]-thymidine incorporation proliferation assay.

254 in hydrochloride produced increases in [(3)H]thymidine incorporation, (R)-(+)-2-dipropylamino-7-hydro

255 Whereas [3H]thymidine incorporation remained negligible in PARP-/- c

256 rial dehydrogenase assay, and tritiated [3H] thymidine incorporation, respectively.

257 emonstrating a 50, 100, and 300% increase in thymidine incorporation, respectively.

258 ell line, respectively), inhibition of [(3)H]thymidine incorporation resulted primarily from apoptosi

259 gnificantly enhanced the inhibition of [(3)H]thymidine incorporation seen with MEHP alone, potentiall

260 [3H]Thymidine incorporation showed that complexing the ODN w

261 s determined by the changes in the levels of thymidine incorporation (significant at P = 0.003), apop

262 GRK2 also inhibited SMC [(3)H]thymidine incorporation stimulated not only by these ago

263 Tritiated thymidine incorporation studies revealed cardiomyocyte c

264 [(3)H]-thymidine incorporation studies showed that Beclin 1-ove

265 ligand, 9-cis-retinoic acid, decreased [(3)H]thymidine incorporation synergistically, thereby implica

266 ghtly but consistently higher value of [(3)H]thymidine incorporation than the controls.

267 ect, as shown by a much lower value of [(3)H]thymidine incorporation than the vector controls, and a

268 We confirmed by [3H]thymidine incorporation that active DNA replication occu

269 ets caused an increase in the level of [(3)H]thymidine incorporation that was twice the control level

270 G0-G1, S, and G2 phases nor the rate of [3H]thymidine incorporation, thus ruling out a proliferative

271 ls (MDDC) and CD4 T cells and measured [(3)H]thymidine incorporation to determine the factors respons

272 12-LO overexpression also caused cell [(3)H]thymidine incorporation to increase by 3.4+/-0.3-fold (P

273 Using bromodeoxyuridine and [(3)H]thymidine incorporation to label cells in S phase and ce

274 IL-8 caused a dose-dependent increase of [3H]thymidine incorporation to maximal levels of 46.3 +/- 3.

275 (10-100 nm) at basal glucose stimulated [3H]thymidine incorporation to the same magnitude as elevate

276 apoptosis were examined by western blotting, thymidine incorporation, transwell assay and TUNEL assay

277 ration and also dramatically inhibited [(3)H]thymidine incorporation upon stimulation with PDGF-BB or

278 3H-thymidine incorporation was also initially depressed but

279 of DAB(389)EGF, the percentage reduction in thymidine incorporation was determined.

280 Thymidine incorporation was greater in F/B* cells than i

281 hermore, E(m) was more depolarized and [(3)H]thymidine incorporation was greater in PASMCs infected w

282 imulation by HRG, a strong increase in [(3)H]thymidine incorporation was observed in human T47D breas

283 Thymidine incorporation was significantly inhibited in t

284 [(3)H]Thymidine incorporation was used to evaluate EGF-in-duce

285 [3H]-Thymidine incorporation was used to measure RCE cell pro

286 ositive (DNA-PKcs(+))), as measured by [(3)H]thymidine incorporation, was significantly arrested by 4

287 mbin on Akt/p70S6K phosphorylation and [(3)H]thymidine incorporation were all attenuated by heterolog

288 strictures, Ki67 immunoreactivity and [(3)H]thymidine incorporation were increased and apoptosis was

289 lymph node mass, cell number, and lymphocyte thymidine incorporation were lowered by 70, 69, and 72%,

290 ation as assessed by CFSE labeling and [(3)H]thymidine incorporation were suppressed in fat-1 cells.

291 mmunosorbent assay, flow cytometry, and (3)H-thymidine incorporation were used to determine immunolog

292 n wild-type fibroblasts as assessed by [(3)H]thymidine incorporation, whereas the growth of S2KO or S

293 ly active AMPK attenuated PDGF-induced [(3)H]thymidine incorporation, which was abolished by either a

294 ckdown of NOX5 significantly decreased [(3)H]thymidine incorporation, which was restored by 10(-13) M

295 gnificantly increased cell number and [(3)H]-thymidine incorporation, while (Pre)miR-519 reduced thes

296 a significant decrease in DNA synthesis upon thymidine incorporation with a cell cycle arrest in the

297 ed a concentration-dependent increase in [3H]thymidine incorporation with a maximal effect observed a

298 The enhanced [3H]thymidine incorporation with chronic elevated glucose or

299 Since diazoxide significantly enhanced [3H]thymidine incorporation without altering S phase accumul

300 Moreover, mp110*ER induced modest levels of thymidine incorporation without subsequent cell division