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1 nganese detoxification and manganese-induced thyroid dysfunction.
2 atment of screen-detected, undiagnosed overt thyroid dysfunction.
3 with systemic reactions, renal disease, and thyroid dysfunction.
4 relationships between BDE-209 exposures and thyroid dysfunction.
5 compared to children born to mothers with no thyroid dysfunction.
6 ical entities that encompass mild degrees of thyroid dysfunction.
7 vide a candidate target for the treatment of thyroid dysfunction.
8 ce, and rhythm disturbances that result from thyroid dysfunction.
9 s determined the proportion of patients with thyroid dysfunction.
10 d to have laboratory evidence of subclinical thyroid dysfunction.
11 r than 60 years, and others at high risk for thyroid dysfunction.
12 ease and have few or no signs or symptoms of thyroid dysfunction.
13 Vigilance is required to detect and treat thyroid dysfunction.
14 patient's thyroid cells as the cause of the thyroid dysfunction.
16 95 patients met the criteria for subclinical thyroid dysfunction (127 were hyperthyroid, and 168 were
17 years; range, 18 to 60 years), 36% developed thyroid dysfunction: 19 became hyperthyroid and 14 hypot
21 ine Society in late 2007, and a symposium on thyroid dysfunction and pregnancy was hosted by the Amer
22 f necessary, to avoid increasing the risk of thyroid dysfunction and related adverse health effects i
23 should be aimed at detection of subclinical thyroid dysfunction and whether persons with mildly abno
25 st media include hypersensitivity reactions, thyroid dysfunction, and contrast-induced nephropathy.
26 rction, obesity, smoking, diabetes mellitus, thyroid dysfunction, and endurance exercise training all
27 thyroiditis, iodine-induced and drug-induced thyroid dysfunction, and factitious ingestion of excess
29 of cancer and its treatment such as anemia, thyroid dysfunction, and the neurotoxicity of cancer che
33 lines seemed to successfully compensate for thyroid dysfunction, as serum thyroxine/triiodothyronine
34 nvestigating exacerbating conditions such as thyroid dysfunction, but without specifying the impact o
38 , including weight gain, insulin resistance, thyroid dysfunction, endothelial dysfunction, and oxidat
39 e (TSH) has increased identification of mild thyroid dysfunction, especially in elderly patients.
42 amine the frequency of amiodarone-associated thyroid dysfunction in adults with congenital heart dise
43 Our goal was to assess the prevalence of thyroid dysfunction in association with 131I exposure du
44 lance of benefits and harms of screening for thyroid dysfunction in nonpregnant, asymptomatic adults.
45 ts the importance of determining the role of thyroid dysfunction in the onset and progression of mang
56 sial, office-based screening to detect overt thyroid dysfunction may be indicated in women older than
57 P2 =.0049; vitiligo, P2 < 10(-6)), although thyroid dysfunction may have been related more to the le
59 were significant risk factors for developing thyroid dysfunction (odds ratios, 3.0 and 7.00; P=0.04 a
61 ases such as diabetes nephrotic syndrome and thyroid dysfunction on the metabolism of apoB-containing
62 -infectious pneumonitis, had no uncontrolled thyroid dysfunction or diabetes, had no active brain met
64 nadequate to determine whether screening for thyroid dysfunction reduced cardiovascular disease or re
65 s indicate a direct role of IFN-gamma in the thyroid dysfunction that occurs in autoimmune thyroiditi
66 conditions represent the earliest stages of thyroid dysfunction, the benefits of detecting and treat
68 us children born to mothers with no recorded thyroid dysfunction using Poisson regression models.
69 apsulated hESC-derived progenitor cells, and thyroid dysfunction was maintained for the duration of t
71 medication were controlled for, subclinical thyroid dysfunction was not associated with depression,
72 he local production of IFN-gamma can lead to thyroid dysfunction, we have generated transgenic mice t
74 rms of screening for subclinical and "overt" thyroid dysfunction without clinically obvious symptoms,
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