ライフサイエンスコーパス: thyroid epithelial cell

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1 , and FasL and FLIP were mainly expressed by thyroid epithelial cells.

2 egulation of Fas ligand (FasL) expression on thyroid epithelial cells.

3 NE2 beta subunit form a potassium channel in thyroid epithelial cells.

4 lecule to induce proinflammatory pathways in thyroid epithelial cells.

5 Raf-1, to examine Ras-dependent signaling in thyroid epithelial cells.

6 s and is thought to be uniquely expressed by thyroid epithelial cells.

7 the epithelial Na(+) channel in Fischer rat thyroid epithelial cells also stimulates Na(+) current,

8 globulin (Tg), the major protein secreted by thyroid epithelial cells and precursor of thyroid hormon

9 ors for thyroid stimulating hormone (TSH) on thyroid epithelial cells, and represents the prototype f

10 tive response to ras between fibroblasts and thyroid epithelial cells cannot readily be explained by

11 tion of forskolin (0.5 microm) in Fisher rat thyroid epithelial cells co-expressing human CFTR and a

12 Rp72, and grp94 were increased in kidney and thyroid epithelial cell culture models.

13 thelial cells, stably transfected Fisher rat thyroid epithelial cells expressing individual airway/lu

14 Thyroid epithelial cells frequently express one or more

15 y shows that activated CD8(+) T cells induce thyroid epithelial cell hyperplasia and proliferation an

16 , and anti-TNF-alpha inhibits development of thyroid epithelial cell hyperplasia and proliferation in

17 oped thyroid lesions characterized by severe thyroid epithelial cell hyperplasia and proliferation, w

18 es a proliferative phenotype in normal human thyroid epithelial cells in vitro, consistent with its p

19 KAT-50, a well differentiated thyroid epithelial cell line, expresses high levels of P

20 e and in KAT-50, a well differentiated human thyroid epithelial cell line.

21 ormed (Vh1) and non-transformed (FRTL-5) rat thyroid epithelial cell lines as a model, we have examin

22 Neoplastic transformation of rodent thyroid epithelial cell lines by mutant RAS genes has be

23 ing the development of thyroid cancer and in thyroid epithelial cells of patients with Hashimoto's th

24 ncer, whereas it is not detectable in normal thyroid epithelial cells or in papillary and follicular

25 t of L-SAT, and it also functions to inhibit thyroid epithelial cell proliferation.

26 thesis that transgenic expression of FLIP on thyroid epithelial cells promotes earlier resolution of

27 In Smad1/5(dKO) embryonic thyroids, epithelial cells remained associated in large

28 cterized by hyperplasia and proliferation of thyroid epithelial cells (TEC H/P).

29 e site of expression of FLIP and Fas ligand [thyroid epithelial cells (TECs) versus inflammatory cell

30 nduces strong and sustained proliferation of thyroid epithelial cells (TECs), or thyrocytes, in IFN-g

31 iven 0.05% NaI in their water develop severe thyroid epithelial cell (thyrocyte) hyperplasia and prol

32 Using primary human thyroid epithelial cells to model tumour initiation by R

33 ort- and long-term responses of normal human thyroid epithelial cells to mutant RAS introduced by mic

34 Minimal apoptosis of thyroid epithelial cells was observed unless the mice we

35 Primary cultures of human thyroid epithelial cells were infected with a RET/PTC re

36 istinguish these possibilities, normal human thyroid epithelial cells were microinjected with the pap

37 immune thyroiditis (G-EAT) was promoted when thyroid epithelial cells were protected from Fas-mediate

38 more directly relevant model - human primary thyroid epithelial cells - which are a major target of n

39 ion in two normal cell types--fibroblast and thyroid epithelial cell--which give rise to human tumour

40 Treatment of cultured thyroid epithelial cells with antimycin A greatly inhibi

41 -gamma resulted in abnormal proliferation of thyroid epithelial cells with minimal lymphocyte infiltr

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