ライフサイエンスコーパス: thyroid hormone

1 Iodine is an essential component of thyroid hormone.

2 the fetus is entirely dependent on maternal thyroid hormone.

3 es 3,5,3'-triiodothyronine (T3 ), the active thyroid hormone.

4 yroid-stimulating hormone despite low plasma thyroid hormone.

5 irmed the GAT1 inhibiting properties of this thyroid hormone.

6 ns are still too low to enable production of thyroid hormone.

7 thyroid follicular cells and biosynthesis of thyroid hormone.

8 with the TR, in Pax8-KO mice, which make no thyroid hormone.

9 nd positively associated with free and total thyroid hormones.

10 d by excessive production and release of the thyroid hormones.

11 function, and factitious ingestion of excess thyroid hormones.

12 that the skin is an important target for the thyroid hormones.

13 le is known about the modulating role of the thyroid hormones.

14 to their structural similarity to endogenous thyroid hormones.

15 despite normal concentrations of circulating thyroid hormones.

16 sure was not associated with levels of other thyroid hormones.

17 m lipid) basis, were assessed in relation to thyroid hormones.

18 omycin administration is also reduced by the thyroid hormones.

19 Local levels of active thyroid hormone (3,3',5-triiodothyronine) are controlled

20 mediate most of the effects elicited by the thyroid hormone, 3,5,3'-L-triiodothyronine (T3).

21 e supplementation, we examined the timing of thyroid hormone action on specific brain systems.

22 on, physiological and biochemical markers of thyroid hormone action, and THRA sequence.

23 show that food availability also stimulates thyroid hormone activation by accelerating the conversio

24 (D2-D3) provides cell-specific regulation of thyroid hormone activity.

25 ced by carbon tetrachloride is attenuated by thyroid hormone administration to mice, whereas aged TR

26 atients with TRalpha1-mediated resistance to thyroid hormone, although our patients had a heterozygou

27 ke of iodine without prolonged withdrawal of thyroid hormone, although the impact of reducing lesiona

28 not demonstrate an associated disruption of thyroid hormone, although this association may have been

29 Initial screening revealed 1a (KB2115), a thyroid hormone analog, as a lead compound with low micr

30 Thyroid hormone and antibody levels were assessed.

31 e shortest larval period, highest whole-body thyroid hormone and corticosterone content, and highest

32 ses during metamorphosis point to a role for thyroid hormone and retinoic acid signaling, as well as

33 determined that these genes are regulated by thyroid hormone and that TRbeta1 is recruited to their r

34 tors for steroids, retinoids, vitamin D, and thyroid hormone and their structural and functional anal

35 Serum thyroid hormones and anti-thyroid antibodies and urinary

36 Thyroid hormones and moderate exposure to perchlorate du

37 Previous studies on the association between thyroid hormones and prognosis of acute ischemic stroke

38 The novel homeostatic link between thyroid hormones and the pathophysiological role of macr

39 Proton pump inhibitors, thyroid hormones, and dihydropyridine derivatives were f

40 Serum estradiol, thyroid hormones, and urinary free cortisol levels were

41 and upregulation of prestin expression after thyroid hormone application.

42 Thyroid hormones are also important regulators of fetal

43 KEY POINTS: Thyroid hormones are important regulators of growth and

44 This may bias our view of how thyroid hormones are produced in other organisms.

45 onclude, our data define a critical role for thyroid hormones as potent alphavbeta3-ligands, driving

46 Hypothalamic thyroid hormone availability is controlled by deiodinase

47 Thyroid hormones binding to alphavbeta3 integrin produce

48 lear receptors TRalpha1 and TRbeta (the main thyroid hormone-binding isoforms) results in impaired ep

49 Thyroid hormone binds to nuclear receptors and regulates

50 LIS3 as a key regulator of TSH/TSHR-mediated thyroid hormone biosynthesis and proliferation of thyroi

51 nal activation of several genes required for thyroid hormone biosynthesis, including the iodide trans

52 ) transport in the thyroid-the first step in thyroid hormone biosynthesis-with a 2 Na(+): 1 I(-) stoi

53 genes that are involved in various steps of thyroid hormone biosynthesis.

54 tyrosine derivatives formed as byproducts of thyroid hormone biosynthesis.

55 several studies have shown that PBDEs affect thyroid hormones, cause oxidative stress, and disrupt Ca

56 We found that increased intracellular thyroid hormone concentration through D3 depletion induc

57 aternal PBDE levels were not associated with thyroid hormone concentrations in cord serum.

58 elationship between maternal PBDE levels and thyroid hormone concentrations in maternal and cord sera

59 ations between urinary perchlorate and serum thyroid hormone concentrations in models adjusted for ur

60 Thyroid hormone concentrations in the blood are stable,

61 Thyroid hormone concentrations were measured in maternal

62 lesterol and other lipids, liver parameters, thyroid hormone concentrations, and adverse effects of t

63 REE, plasma thyroid hormone concentrations, and insulin resistance w

64 been masked by impacts of prey abundance on thyroid hormone concentrations.

65 from animal and human studies indicates that thyroid hormone deficiency during early gestation alters

66 together, these data suggest that long-term thyroid hormone deficiency may drive the differentiation

67 Hypothyroidism is a common condition of thyroid hormone deficiency, which is readily diagnosed a

68 -tetrabromoethylcyclohexane (beta-TBECH), on thyroid hormone deiodinase (DIO) and sulfotransferase (S

69 e, T3), through spatiotemporal expression of thyroid hormone deiodinase (dio) genes.

70 cts on the fetal PT to control expression of thyroid hormone deiodinases in ependymal cells (tanycyte

71 us nonthyroid tissues was-in part-induced by thyroid (hormone)-dependent signaling.

72 Associated disruption of thyroid hormone-dependent protein synthesis in the hippo

73 rian cancer and we hypothesized that natural thyroid hormone derivatives may antagonize these actions

74 To conclude, the cytotoxic potential of thyroid hormone derivatives, tetrac, triac and T1AM, in

75 ecules besides trace amines (TAs), including thyroid hormone-derivatives like 3-iodothyronamine (T1AM

76 Thyroid hormone disrupting chemicals (THDCs) interfere w

77 One possible molecular target of thyroid hormone disrupting chemicals (THDCs) is transthy

78 d the potential windows of susceptibility to thyroid hormone disturbances related to study visit of s

79 ndent mechanisms, including glucocorticoids, thyroid hormone, dopamine, and growth factors.

80 t of infant visual attention is sensitive to thyroid hormone during the early prenatal period, when t

81 gs of this study highlight the importance of thyroid hormones during pregnancy for normal development

82 Small changes in thyroid hormones during pregnancy, including changes wit

83 hird trimester, and also measured cord serum thyroid hormones for 116 neonates.

84 serum concentrations of nine PFASs and four thyroid hormones for 285 pregnant women in their third t

85 RATIONALE: Thyroid hormones have been linked with various proathero

86 thetic mammalian gene circuit that maintains thyroid hormone homeostasis by monitoring thyroid hormon

87 The physiological control of thyroid hormone homeostasis by the feedback loops involv

88 me PFASs during pregnancy may interfere with thyroid hormone homeostasis in pregnant women and fetuse

89 Polybrominated diphenyl ethers (PBDEs) alter thyroid hormone homeostasis, but their relationship with

90 ats and their pups, PFASs can interfere with thyroid hormone homeostasis.

91 ence that administration of supraphysiologic thyroid hormone improves depressive symptoms in patients

92 d perchlorate exposure and the importance of thyroid hormone in fetal neurodevelopment.

93 mical measurements showed elevated levels of thyroid hormone in plasma from naive and CF pups.

94 n of type 2 deiodinase (D2), which activates thyroid hormone in skeletal muscle is upregulated by acu

95 a-analysis to assess the prognostic value of thyroid hormones in AIS.

96 thers (PBDEs) reduce blood concentrations of thyroid hormones in laboratory animals, but it is unclea

97 als, but it is unclear whether PBDEs disrupt thyroid hormones in pregnant women or newborn infants.

98 late exposure to alter circulating levels of thyroid hormones in pregnant women.

99 Consequently, production of thyroid hormones in the thyroid gland and circulating th

100 ganoids that provide functional secretion of thyroid hormones in vivo and rescue hypothyroid mice aft

101 metric pigmentation and, via cross-talk with thyroid hormones, in modulating eye migration.

102 The thyroid hormone-inactivating (TH-inactivating) enzyme ty

103 ed the hippocampal expression changes in the thyroid hormone-inactivating enzyme, deiodinase-III (Dio

104 nscriptomic responses included alteration of thyroid hormone induced bZip protein (thibz), deiodinase

105 solated fetal sheep islets studied in vitro, thyroid hormones inhibited beta cell proliferation in a

106 comes of children exposed prenatally to mild thyroid hormone insufficiency are understudied.

107 Maternal thyroid hormone insufficiency during pregnancy can affec

108 ncreatic beta cell is therefore sensitive to thyroid hormone, insulin and leptin before birth, with p

109 kers confirmed the requirement for an intact thyroid hormone-integrin interaction in ERK activation.

110 the clinical syndrome of excess circulating thyroid hormones, irrespective of the source.

111 Thyroid hormone is a pleiotropic factor that controls ma

112 Thyroid hormone is critical for normal brain development

113 integrin, a plasma membrane receptor, binds thyroid hormones (L-thyroxine, T4; 3,5,3'-triiodo-L-thyr

114 ost often, followed by gonadal, adrenal, and thyroid hormones, leading to abnormal growth and puberty

115 ormones in the thyroid gland and circulating thyroid hormone level were increased.

116 Maternal thyroid hormone levels (thyrotropin, free thyroxine, thy

117 ns thyroid hormone homeostasis by monitoring thyroid hormone levels and coordinating the expression o

118 , this synthetic circuit sensed pathological thyroid hormone levels and restored the thyrotrophic fee

119 exposure has been associated with decreased thyroid hormone levels in animals, but human studies are

120 ed the degree to which phthalates may affect thyroid hormone levels in particularly susceptible popul

121 atistical evidence supporting the utility of thyroid hormone levels in prognosis of acute stroke.

122 erimental evidence suggests that TCDD alters thyroid hormone levels in rodents, human data are incons

123 of urinary phthalate metabolites and plasma thyroid hormone levels in samples collected at up to fou

124 s that intrauterine exposure to insufficient thyroid hormone levels influences neurodevelopment in of

125 ssociation between maternal TCS exposure and thyroid hormone levels of mothers and newborns.

126 ome or standardized mean difference (SMD) of thyroid hormone levels with 95% confidence intervals (95

127 ver, little is known about how variations in thyroid hormone levels within the normal range affect el

128 study investigated the effects of different thyroid hormone levels, ranging from hypothyroidism to h

129 d hormone synthesis, might explain these low thyroid hormone levels.

130 traditionally been attributed solely to high thyroid hormone levels.

131 ns of PBBs and PCBs with thyroid disease and thyroid hormone levels.

132 elationship between maternal urinary TCS and thyroid hormone levels.

133 generation inhibitors and have no detectable thyroid hormone-like activity.

134 In skeletal muscle, physical exercise and thyroid hormone mediate the peroxisome proliferator-acti

135 nimals demonstrated subsequent disruption of thyroid hormone-mediated reprogramming in the liver tran

136 d biosynthesis, cholesterol biosynthesis and thyroid hormone metabolic processes.

137 Therefore, we investigated the role of thyroid hormone metabolism and signaling in colorectal C

138 eading to seasonal changes in the control of thyroid hormone metabolism in the hypothalamus.

139 hat several antiepileptic drugs can increase thyroid hormone metabolism.

140 n dogs, raise serious doubts about selective thyroid hormone mimetics as a therapeutic approach to lo

141 Thyroid hormones modulate not only multiple functions in

142 (T3), like many other ligands of the steroid/thyroid hormone nuclear receptor superfamily, is a stron

143 eviously showed that deletion in mice of the thyroid hormone nuclear receptors TRalpha1 and TRbeta (t

144 in view of the confounding effect of excess thyroid hormone on immune responses, spontaneously arisi

145 ass, fat, and moulting and decreasing plasma thyroid hormones over time.

146 re them with patients who have resistance to thyroid hormone owing to a mutation affecting only TRalp

147 f maternal urinary phthalate metabolites and thyroid hormone parameters during pregnancy.

148 thways, including phase I/II metabolism, the thyroid hormone pathway, lipid/cholesterol metabolism, o

149 ding xenobiotic and lipid metabolism and the thyroid hormone pathway.

150 potential effects in estrogen, androgen, and thyroid hormone pathways, and it is one of the only regu

151 s supply the fetus throughout pregnancy, and thyroid hormones play a critical role in fetal growth an

152 ite their obvious biological importance, the thyroid hormone precursor protein, thyroglobulin (Tg), h

153 vironmental perchlorate exposures may affect thyroid hormone production during pregnancy.

154 Iodine is required throughout pregnancy for thyroid hormone production, which is essential for fetal

155 iodide uptake into the thyroid and decrease thyroid hormone production.

156 the present study tested the hypotheses that thyroid hormones promote beta cell proliferation in the

157 Thyroid hormone promotes expression of peroxisome prolif

158 g the final exposure, and males had a higher thyroid hormone ratio (T3/T4).

159 ning silencing mediator of retinoic acid and thyroid hormone receptor (SMRT) and nuclear receptor cor

160 and silencing mediator of retinoic acid and thyroid hormone receptor (SMRT) corepressors and is larg

161 restingly, EBI was found to be a very potent thyroid hormone receptor (THR) agonist, while NH-3 is an

162 to elevated expression of genes regulated by thyroid hormone receptor (TR) and liver X receptor (LXR)

163 We used a thyroid hormone receptor (TR) beta mutant mouse (TRbetaP

164 t mediates ligand-independent actions of the thyroid hormone receptor (TR) during development and in

165 gene through this interaction, and enhances thyroid hormone receptor (TR)-driven transcription in a

166 o describe transcriptional regulation by the thyroid hormone receptor (TR).

167 is alternatively spliced to generate either thyroid hormone receptor (TR)alpha1 or a non-hormone-bin

168 We found that the thyroid hormone receptor (TRalpha 3) has a differential

169 ASO-T3 (NAT3) and ApoB-ASO-T3 (AAT3) enhance thyroid hormone receptor activity.

170 Eprotirome is a liver-selective thyroid hormone receptor agonist that has been shown to

171 , including cardiac effects, are mediated by thyroid hormone receptor alpha (THR-alpha).

172 Thyroid hormone receptor alpha (THRA) gene mutations, vi

173 Mutations of the thyroid hormone receptor alpha gene (THRA) cause hypothy

174 The thyroid hormone receptor alpha gene (THRA) transcript is

175 identified patients that have a mutation in thyroid hormone receptor alpha1.

176 expression analysis and the use of different thyroid hormone receptor antagonists suggest thyroid hor

177 evels are primarily due to its action at the thyroid hormone receptor beta (THR-beta) in the liver, w

178 Mice with mutations in the thyroid hormone receptor beta (TRbeta) gene that cannot

179 ction with the transcription factors CRX and thyroid hormone receptor beta 2, it enhances M-opsin exp

180 X6 (Sex Determining Region Y-Box 6) and cTR (Thyroid hormone receptor beta).

181 Down-regulation of the thyroid hormone receptor beta1 (TRbeta) appears to be as

182 thyroid hormone receptor antagonists suggest thyroid hormone receptor beta1 as the major player media

183 methyltransferase Dot1L is a coactivator for thyroid hormone receptor during Xenopus development.

184 Thyroid hormone receptor expression analysis and the use

185 The AAA-ATPase thyroid hormone receptor interacting protein 13 (TRIP13)

186 Inhibition of thyroid hormone receptor locally in the retina is a ther

187 metabolic processes in the liver through the thyroid hormone receptor, TRbeta1.

188 T3 binds to thyroid hormone receptor, which heterodimerizes with ret

189 We have provided evidence that thyroid hormone receptor-alpha (TR-alpha), a transcripti

190 We show here that Thrap3 (thyroid hormone receptor-associated protein 3) can direc

191 xploiting the specificity of the coactivator thyroid hormone receptor-associated protein 80 (TRAP80).

192 We identified TRIP12 (thyroid hormone receptor-interacting protein 12), an E3

193 The silencing mediator of retinoic acid and thyroid hormone receptors (SMRT) is an established histo

194 H expression, presumably by interacting with thyroid hormone receptors (THRs) bound to TSH subunit ge

195 Thyroid hormone receptors (TRs) are ligand-dependent tra

196 findings define an important function of the thyroid hormone receptors and suggest TR ligands could h

197 otein and silencing mediator of retinoid and thyroid hormone receptors to a newly identified putative

198 and SMRT (silencing mediator of retinoid and thyroid hormone receptors; NCoR2) are well-recognized co

199 1 (NCoR1)/silencing mediator for retinoid or thyroid-hormone receptors (SMRT) corepressors in skin ke

200 1 (NCoR1)/silencing mediator for retinoid or thyroid-hormone receptors (SMRT) corepressors or histone

201 Thyroid hormones regulate mitochondrial function.

202 ptor (TSHR) on the thyroid gland, triggering thyroid hormone release.

203 reated, resulting in a dramatic reduction in thyroid hormone replacement dosage, and 2) the identific

204 Thyroid hormone replacement has been used for more than

205 The standard treatment is thyroid hormone replacement therapy with levothyroxine.

206 concentrations of T3 restored expression of thyroid hormone-responsive target genes in patient-deriv

207 t a fetal/adult expression predominance, are thyroid hormone-responsive, and are regulated by beta1-a

208 eatment of other patients with resistance to thyroid hormone resulting from mutations in THRA.

209 Resistance to thyroid hormone (RTH), a human syndrome, is characterize

210 expenditure, which were not due to impaired thyroid hormone secretion.

211 ylestradiol, including steroid biosynthesis, thyroid hormone signaling and metabolism, testicular dev

212 We conclude that thyroid hormone signaling is an important determinant of

213 metabolic mechanism by which CR-CSCs exploit thyroid hormone signaling to facilitate their self-renew

214 autoregulatory feedback loop that modulates thyroid hormone signaling.

215 key elements of the response to photoperiod, thyroid hormone signalling components were assessed in t

216 Thyroid-hormone signalling, particularly through inducti

217 Deregulation of deiodinase function and thyroid hormone status has been implicated in tumorigene

218 sociation between maternal PFAS exposure and thyroid hormone status in pregnant women and neonates.

219 The thyroid hormone status, insulin sensitivity, glucose upt

220 In humans, maternal thyroid hormones supply the fetus throughout pregnancy,

221 Here, we show that a thyroid hormone surge activates the IGF-1/IGF-1-R/Akt pa

222 iferation during preadolescence, driven by a thyroid hormone surge, with therapeutic implications for

223 yperthyroidism is characterised by increased thyroid hormone synthesis and secretion from the thyroid

224 Because thyroid hormone synthesis is affected by iodine deficien

225 que mutation in the DUOX2 gene, critical for thyroid hormone synthesis, might explain these low thyro

226 rupting chemicals (THDCs) interfere with the thyroid hormone system and may induce multiple severe ph

227 esis of antisense-oligonucleotides (ASO) and thyroid hormone T3 conjugates for obesity treatment.

228 tive peptide that combines glucagon with the thyroid hormone T3 lowers lipid levels, improves glucose

229 However, the thyroid hormone T3 up-regulated mitoIK, ATP, conferring

230 in the biosynthesis of the iodine-containing thyroid hormones T3 and T4.

231 Thyroid hormone (T3) affects development and metabolism

232 Glucagon and thyroid hormone (T3) exhibit therapeutic potential for m

233 with both processes taking place when plasma thyroid hormone (T3) levels are high.

234 Thyroid hormone (T3), like many other ligands of the ste

235 We have been studying thyroid hormone (T3)-dependent amphibian metamorphosis i

236 Here we report that a thyroid hormone (T3)-free window, with or without a demy

237 and rT3 separately) and measuring changes in thyroid hormone (T4, T3, rT3, and 3,3'-T2) concentration

238 ctive human liver subcellular fractions with thyroid hormones (T4 and rT3 separately) and measuring c

239 ermore, we demonstrate that BMP4 is a direct thyroid hormone target and is involved in a positive aut

240 sm, a metabolic disease characterized by low thyroid hormone (TH) and high thyroid-stimulating hormon

241 Thyroid hormone (TH) and retinoic acid (RA) are powerful

242 Thyroid hormone (TH) and retinoic acid (RA) within the t

243 Thyroid hormone (TH) and TH receptors (TRs) alpha and be

244 , a human syndrome, is characterized by high thyroid hormone (TH) and thyroid-stimulating hormone (TS

245 mediated I(-) uptake plays a pivotal role in thyroid hormone (TH) biosynthesis.

246 cumulation in the thyroid, the first step in thyroid hormone (TH) biosynthesis.

247 ltiple animal studies have shown PBDEs to be thyroid hormone (TH) disruptors.

248 3) is considered to be the primary bioactive thyroid hormone (TH) due to its high affinity for TH nuc

249 Interestingly, both FOXO1 and thyroid hormone (TH) have similar effects on carbohydrat

250 els of thyroid stimulating hormone (TSH) and thyroid hormone (TH) in an inverse relationship.

251 d a related species, we identified roles for thyroid hormone (TH) in pigment cell development and pat

252 dism during pregnancy, suggesting a role for thyroid hormone (TH) in the development of central thyro

253 Thyroid hormone (TH) is a critical regulator of perinata

254 Thyroid hormone (TH) is an ancestral signal linked to se

255 Thyroid hormone (TH) is critical for the maintenance of

256 An effect of thyroid hormone (TH) on erythropoiesis has been known fo

257 The beneficial effects of thyroid hormone (TH) on lipid levels are primarily due t

258 orm of psychomotor retardation with abnormal thyroid hormone (TH) parameters, is linked to mutations

259 There is increasing evidence that the thyroid hormone (TH) receptors (THRs) can play a role in

260 Thyroid hormone (TH) regulates many cellular events unde

261 effects can occur in tissues that depend on thyroid hormone (TH) regulation for normal physiologic f

262 ing postnatal day 7-10, when serum levels of thyroid hormone (TH) rise.

263 Their DNT effects are suspected to involve thyroid hormone (TH) signaling disruption.

264 Recent studies have implicated thyroid hormone (TH) signaling in cone photoreceptor via

265 ver, their unique roles in the regulation of thyroid hormone (TH) signaling in specific cell types ha

266 Thyroid hormone (TH) signaling promotes tissue maturatio

267 Thyroid hormone (TH) signaling regulates cell proliferat

268 Thyroid hormone (TH) signaling, which regulates cell pro

269 Inactivating mutations in the thyroid hormone (TH) transporter Monocarboxylate transpo

270 A major regulator is thyroid hormone (TH), which acts through its nuclear rec

271 Thyroid hormones (TH) are critical for development, grow

272 gh the production and subsequent function of thyroid hormones (TH).

273 Here, we investigate whether thyroid-hormones (TH) and their receptors (TR) coordinat

274 Calcitonin is a 32-amino acid thyroid hormone that can form amyloid fibrils.

275 ate (SMR), and elevate whole-body content of thyroid hormone (the primary morphogen controlling metam

276 Thyroid hormones (THs) are essential for establishing la

277 ley syndrome patients.SIGNIFICANCE STATEMENT Thyroid hormones (THs) are essential to establish the st

278 demonstrated to be the membrane receptor for thyroid hormones (THs) in several tissues.

279 Since it is unknown whether thyroid hormones (THs) regulate mitochondrial function i

280 ed "grandfather products" like the synthetic thyroid hormone thyroxine, strict regulations enforce a

281 In addition, circulating levels of thyroid hormones thyroxine (T4) and triiodothyronine (T3

282 of the pituitary hormone thyrotropin and the thyroid hormones thyroxine and triiodothyronine) are som

283 of thyroid function, including those for the thyroid hormones thyroxine and triiodothyronine, are amo

284 a stress hormone acting synergistically with thyroid hormone to accelerate metamorphosis).

285 esponsive by a non-permissive dimer partner, thyroid hormone (TR) receptor.

286 chemicals (THDCs) is transthyretin (TTR), a thyroid hormone transporter in vertebrates.

287 Since then, a subgroup of thyroid hormone-treated patients with residual symptoms

288 iagnosed before delivery who did not receive thyroid hormone treatment during pregnancy (IRR=1.37, 95

289 evaluated whether the risk was moderated by thyroid hormone treatment during pregnancy.

290 Thyroid hormone treatment in juveniles enhanced NR1 gene

291 in vivo evidence of a critical role for the thyroid hormone triiodothyronine (T3) in controlling the

292 Here, we show that the thyroid hormone triiodothyronine (T3), through binding t

293 ver, little work on the effects of Hg on the thyroid hormones triiodothyronine (T3) and thyroxine (T4

294 Dysregulation of thyroid hormones triiodothyronine and thyroxine (T3/T4)

295 brain-dead potential organ donor can include thyroid hormone (triiodothyronine [T3] or levothyroxine

296 ns blood plasma levels of estradiol (E2) and thyroid hormones (TSH, T3t, T4t) were also determined.

297 between maternal PFASs and maternal and cord thyroid hormones were examined in multiple linear regres

298 Thyroid hormones were measured in a subset without thyro

299 imulating hormone (TSH) stimulation methods (thyroid hormone withdrawal [THW] and recombinant human T

300 Subsequently, after 4-6 wk of thyroid hormone withdrawal patients were treated with 5.