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1 nd relationship in both benign and malignant thyroid neoplasms.
2 yroid cancers develop de novo or from benign thyroid neoplasms.
3 and the same mutation was also seen in some thyroid neoplasms.
4 ne the PTEN status in a series of epithelial thyroid neoplasms.
5 on or maintenance of cell differentiation in thyroid neoplasms.
7 ulation may be useful to objectively subtype thyroid neoplasms and more accurately predict outcomes.
8 nign thyroid neoplasms progress to malignant thyroid neoplasms and that most occult thyroid cancers d
9 ation of the chromosome 2p21 locus in 28% of thyroid neoplasms, and in the WRO thyroid carcinoma cell
13 of primary hypothyroidism, hyperthyroidism, thyroid neoplasms, hypopituitarism, obesity, diabetes me
15 contrast, no difference was seen with benign thyroid neoplasms in which the prevalence of rs17849071G
22 of the RAS proto-oncogene occurs in various thyroid neoplasms such as papillary thyroid carcinomas (
23 lly, follicular adenoma, a benign subtype of thyroid neoplasm, was also found to harbor mutations (12
24 higher or lower) in malignant versus benign thyroid neoplasms were identified by extracellular matri
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