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1 nd relationship in both benign and malignant thyroid neoplasms.
2 yroid cancers develop de novo or from benign thyroid neoplasms.
3  and the same mutation was also seen in some thyroid neoplasms.
4 ne the PTEN status in a series of epithelial thyroid neoplasms.
5 on or maintenance of cell differentiation in thyroid neoplasms.
6  time to development of benign and malignant thyroid neoplasms after radiation exposure.
7 ulation may be useful to objectively subtype thyroid neoplasms and more accurately predict outcomes.
8 nign thyroid neoplasms progress to malignant thyroid neoplasms and that most occult thyroid cancers d
9 ation of the chromosome 2p21 locus in 28% of thyroid neoplasms, and in the WRO thyroid carcinoma cell
10           The majority of familial medullary thyroid neoplasms are associated with germ-line mutation
11 rnia, San Francisco-affiliated hospitals for thyroid neoplasms between 1960 and 1999.
12                 Hurthle cell tumors are rare thyroid neoplasms for which disease biology is poorly un
13  of primary hypothyroidism, hyperthyroidism, thyroid neoplasms, hypopituitarism, obesity, diabetes me
14     Therapeutic radiation is associated with thyroid neoplasms in humans and animals.
15 contrast, no difference was seen with benign thyroid neoplasms in which the prevalence of rs17849071G
16                       Clinical management of thyroid neoplasms is based on light microscopic diagnosi
17 s were independent predictors of a malignant thyroid neoplasm (P < 0.003).
18 n malignant (n = 57) than in benign (n = 38) thyroid neoplasms (P< 0.002).
19         Our findings question whether benign thyroid neoplasms progress to malignant thyroid neoplasm
20 plying these biomarkers in the management of thyroid neoplasm should be considered.
21           While the genomic landscape of all thyroid neoplasm subtypes will inevitably be elucidated,
22  of the RAS proto-oncogene occurs in various thyroid neoplasms such as papillary thyroid carcinomas (
23 lly, follicular adenoma, a benign subtype of thyroid neoplasm, was also found to harbor mutations (12
24  higher or lower) in malignant versus benign thyroid neoplasms were identified by extracellular matri

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