ライフサイエンスコーパス: thyroid peroxidase

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1 ine receptor subtypes 3, 4, and 5 as well as thyroid peroxidase.

2 offspring developed Abs to thyroglobulin and thyroid-peroxidase.

3 ific targets including gastric ATPase (11%), thyroid peroxidase (14%), and anti-IgA autoantibodies ag

4 panels of single substitution analogs of the thyroid peroxidase 632-645Y epitope.

5 ion ratios and high selectivity for MPO over thyroid peroxidase and cytochrome P450 isoforms.

6 sed during its differentiation (for example, thyroid peroxidase and thyroglobulin genes).

7 ability of recombinant antigen [particularly thyroid peroxidase and thyrotropin (TSH) receptor] and o

8 , increased levels of autoantibodies against thyroid peroxidase, and also exhibited the strongest dis

9 imal overlap among anti-gastric ATPase, anti-thyroid peroxidase, and anti-transglutaminase seropositi

10 bulin, thyroid-stimulating hormone receptor, thyroid peroxidase, and sodium iodide transporter.

11 ransglutaminase antibodies (AGTAs), and anti-thyroid peroxidase (anti-TPO) antibodies were measured.

12 unchanged when women with elevated levels of thyroid peroxidase antibodies were excluded from the ana

13 hormone levels (thyrotropin, free thyroxine, thyroid peroxidase antibodies) were measured at a mean (

14 hyroxine (T4)), thyroid-stimulating hormone, thyroid peroxidase antibodies, iodine, selenium, and mer

15 els, thyroid-stimulating hormone levels, and thyroid peroxidase antibodies.

16 ting hormone (TSH), free thyroxine (FT4) and thyroid peroxidase antibody (TPOAb) positivity.

17 FT4), thyroid-stimulating hormone (TSH), and thyroid peroxidase antibody (TPOAb) were obtained from m

18 2(h4) mice associated with thyroglobulin and thyroid-peroxidase, but not TSHR, Abs.

19 ation of iodide within the follicular lumen (thyroid peroxidase, DUOX2, DUOXA2), the substrate for th

20 with thyroiditis and specific for a cryptic thyroid-peroxidase epitope.

21 s), IA2 antigen (IA-2A), zinc transporter 8, thyroid peroxidase, gastric parietal cells (PCAs), tissu

22 uggested that only approximately 2% of human thyroid peroxidase (hTPO(933)) reaches the surface of st

23 h FcepsilonRIalpha, but not thyroglobulin or thyroid peroxidase, resulted in the decreased ability to

24 likely resulted from decreased expression of thyroid peroxidase, sodium iodine symporter, and thyrogl

25 ial genetic architecture with positivity for thyroid-peroxidase-specific antibody, driven generally b

26 g expression of the sodium iodide symporter, thyroid peroxidase, TG, and thyrotropin receptor genes.

27 of mRNA transcripts for thyroglobulin (TG), thyroid peroxidase (TPO) and RET/PTC1 in the peripheral

28 serum thyroid-stimulating hormone (TSH) and thyroid peroxidase (TPO) antibody levels using regressio

29 Levels of IgG against thyroid peroxidase (TPO) are more often elevated in CSU

30 Autoantibodies to thyroid peroxidase (TPO) are the hallmark of the humoral

31 Thyroid peroxidase (TPO) autoantibody epitopes are large

32 lularly, and this might be representative of thyroid peroxidase (TPO) behavior in thyrocytes.

33 For thyroid hormone synthesis, thyroid peroxidase (TPO) molecules must be transported f

34 oantibody epitopes in amino acids 513-633 of thyroid peroxidase (TPO), a region frequently recognized

35 ession of the sodium iodide symporter (NIS), thyroid peroxidase (TPO), and TG genes.

36 tients and specific for the main autoantigen thyroid peroxidase (TPO).

37 ned, and these cells were found to reexpress thyroid peroxidase (TPO).

38 thyrotropin [TSH], free thyroxine [fT4], and thyroid peroxidase [TPO] antibodies) were assessed in 5,

39 h-affinity IgG1, kappa human autoantibody to thyroid peroxidase, was determined crystallographically

40 ccurring autoantibodies to thyroglobulin and thyroid peroxidase were unaffected.

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