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1 site, SP-D promoter activity did not require thyroid transcription factor 1.
2 -D; Clara cell-associated protein CC-10; and thyroid transcription factor-1.
3 DEF interacted with the C-terminal domain of thyroid transcription factor 1, activating transcription
4 ned the homeobox transcription factor TITF1 (thyroid transcription factor 1; also called NKX2-1), pre
5 were correctly specified as shown by normal thyroid transcription factor 1 and surfactant protein A
6 cells seen in the SP-C/HFH-4 mice expressed thyroid transcription factor-1 and hepatocyte nuclear fa
8 e finding regions of limited homology to the thyroid transcription factor 1-binding site, SP-D promot
11 of transcription factors including Sp1, Sp3, thyroid transcription factor 1, hepatocyte nuclear facto
12 te phenotype characterized by surfactant and thyroid-transcription factor-1 immunoreactivities and la
13 d the expression of surfactant protein C and thyroid transcription factor-1 in cultured normal lungs,
16 tion assays, whereas another related factor, thyroid transcription factor 1, showed a more limited ra
17 emical stains were positive for napsin A and thyroid transcription factor 1, suggestive of adenocarci
23 ated that retinoic acid receptors (RARs) and thyroid transcription factor 1 (TTF-1) stimulated SP-B g
24 by increased phosphorylation and binding of thyroid transcription factor 1 (TTF-1) to an upstream re
25 ial cell-derived cell line, MLE-15, and that thyroid transcription factor 1 (TTF-1) transactivates pr
27 by protein kinase A (PKA) phosphorylation of thyroid transcription factor 1 (TTF-1), expressed select
29 pic thyroid-restricted transcription factor, thyroid transcription factor 1 (TTF-1), in rat FRTL-5 th
32 otably, cAMP increased binding of endogenous thyroid transcription factor 1 (TTF-1/Nkx2.1) to the miR
33 binding sites for nuclear factor I (NFI) and thyroid transcription factor 1 (TTF-1; also called Nkx2.
35 In the present study, we observed that three thyroid transcription factor-1 (TTF-1) binding elements
36 transcriptional activity 7-10-fold, whereas thyroid transcription factor-1 (TTF-1) increased the act
43 increased phosphorylation and DNA binding of thyroid transcription factor-1 (TTF-1/Nkx2.1), a master
47 trate the ability of aptamers that recognize thyroid transcription factor 1 (TTF1) to bind their targ
48 oderm; RAR antagonism disrupts expression of thyroid transcription factor 1 (Ttf1), an early marker o
50 High levels of surfactant protein C (Sp-C), thyroid transcription factor-1 (Ttf1), and Gata6, but no
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