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1 nical diagnosis, magnitude of early and late thyroidal 131I uptake values, and the 4- to 24-hr 131I u
2 plications, such as aberrations of tracheal, thyroidal, and vascular structures, were determined with
3 IL-6 during EAGD induction in the setting of thyroidal CD40 overexpression and showed decreased level
4 g a murine model of GD, we demonstrated that thyroidal CD40 overexpression augmented the production o
6 the effective half-lives of 131I in both the thyroidal component and the extrathyroidal component dur
7 is unknown and the presence of an intrinsic thyroidal defect has not been conclusively demonstrated.
8 of symptoms before diagnosis and presence of thyroidal disease, non-coeliac food intolerance or gastr
9 ases (8 % vs 40 % and 25 %, P = 0.028), more thyroidal diseases (18 % vs 16 % and 13 %, P = 0.043) an
11 g export, affected individuals suffer from a thyroidal ER storage disease characterized by a distende
12 at iodine and IFN-gamma cooperate to promote thyroidal expression of ICAM-1 in this mouse model of th
13 ender, tumor size, histologic variant, extra-thyroidal extension (ETE), and central and lateral nodal
18 roid function test abnormalities seen in non-thyroidal illness usually resolve spontaneously, treatme
19 l illness." In psychiatric patients with non-thyroidal illness, patterns of thyroid function test abn
20 non is now more commonly referred to as "non-thyroidal illness." In psychiatric patients with non-thy
21 during sepsis often occur in the absence of thyroidal illness; however, the mechanisms involved in t
22 he initial ICAM-1 kinetics was paralleled by thyroidal infiltration of CD45(+) hemopoietic cells, whi
24 1I uptake ratio was evaluated as an index of thyroidal iodide retention in hyperthyroid patients.
31 due to incomplete thyrotropin suppression, a thyroidal radioiodide uptake of < 1%, and a low saliva t
35 r suppressor activity of THRB, the effect of thyroidal status on longevity, and the occurrence of tis
36 te significantly to the relative increase in thyroidal T3 production in patients with Graves' disease
37 TG) at residues Tyr(5) and Tyr(130), whereas thyroidal T3 production may originate in several differe
41 nduction of gross chromosomal aberrations in thyroidal TPC-1 cells following overexpression of PBF an
42 Of the 71 masses, 41 were thymic, 16 were thyroidal, two were esophageal cancers, six were trachea
43 so in many other normal tissues; conversely, thyroidal uptake of [131I]FIBG was 2-3-fold lower than t
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