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1 nless thyroiditis in patients with untreated thyrotoxicosis.
2 mones that produce the symptoms and signs of thyrotoxicosis.
3 poorly with the mouse TSHR and do not cause thyrotoxicosis.
4 art disease, size of goiter, and severity of thyrotoxicosis.
5 among 37 consecutive patients with untreated thyrotoxicosis (25 female and 12 male; mean age of 44 ye
6 ery, infection, acute myocardial infarction, thyrotoxicosis, acute alcohol consumption, acute pericar
12 urs, choriocarcinoma, and amiodarone-induced thyrotoxicosis are, more often than not, a challenge to
13 y peripheral vasodilatation during sepsis or thyrotoxicosis, blood shunting, or reduced blood oxygen
16 ts include age > or =75 years; hypertension; thyrotoxicosis; diabetes; cardiovascular disease; conges
17 the lack of D3 function results in neonatal thyrotoxicosis followed later by central hypothyroidism
20 sm during pregnancy, severe life-threatening thyrotoxicosis in pregnancy, neonatal thyrotoxicosis, an
26 , and might be the only treatment needed for thyrotoxicosis not caused by excessive production and re
29 appeared to be a risk factor for developing thyrotoxicosis (odds ratio, 4.0; P=0.17), and amiodarone
31 r (OR = 0.69, p = 3.9 x 10(-5)) goiters, and thyrotoxicosis (OR = 0.76, p = 1.5 x 10(-3)), but not Gr
33 nd secretion from the thyroid gland, whereas thyrotoxicosis refers to the clinical syndrome of excess
35 en 1946 and 1964 in the original Cooperative Thyrotoxicosis Therapy Follow-up Study; 91 % had Graves
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