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1 estphal nucleus was identified as containing thyrotropin-releasing hormone.
2 ted responses to leptin and a suppression of thyrotropin-releasing hormone.
3 thyroid-stimulating hormone and hypothalamic thyrotropin-releasing hormone.
4 in, corticotropin-releasing hormone (CRH) or thyrotropin-releasing hormone.
5 lex and includes suppression of hypothalamic thyrotropin-releasing hormone, accounting for persistent
6 hormone, corticotropin-releasing factor, and thyrotropin-releasing hormone also stimulated calcium si
8 omatostatin, corticotropin-releasing factor, thyrotropin-releasing hormone and calcitonin gene-relate
9 express corticotrophin-releasing hormone or thyrotropin-releasing hormone and do not express arginin
10 or lung disease, antenatal administration of thyrotropin-releasing hormone and glucocorticoid is no m
11 expression was found in PVH cells producing thyrotropin-releasing hormone and in cholinergic DMV cel
12 that CG5911 is evolutionarily related to the thyrotropin-releasing hormone and neuromedin U receptors
14 r neurotransmitters such as glutamate, GABA, thyrotropin releasing hormone, and substance P encoded b
15 d by serotonin, norepinephrine, substance P, thyrotropin-releasing hormone, and 3,5-dihydroxyphenylgl
17 than another inducer of prolactin secretion, thyrotropin-releasing hormone, both in vitro and in vivo
18 by the expression of oxytocin, vasopressin, thyrotropin-releasing hormone, corticotropin-releasing h
19 onadotropin-releasing hormone, somatostatin, thyrotropin-releasing hormone, corticotropin-releasing h
20 sed patients, supporting the hypothesis that thyrotropin-releasing hormone could be a positive modula
21 ng juvenile chum salmon (Oncorhynchus keta), thyrotropin-releasing hormone gene expression increased
23 bo-controlled, randomized trial of antenatal thyrotropin-releasing hormone, given intravenously in fo
24 peutic effects of the tripeptide protirelin (thyrotropin-releasing hormone) have been postulated in t
25 ytocin, corticotropin-releasing hormone, and thyrotropin-releasing hormone in an appropriate spatial
26 regulating the effect of glucocorticoids on thyrotropin-releasing hormone in fetal rat diencephalic
27 pattern was clarified including the role of thyrotropin-releasing hormone in synthesis of thyrotropi
28 le metabolism, regulation of food intake, or thyrotropin-releasing hormone levels in the hypothalamus
29 eport of diurnal variations in the levels of thyrotropin-releasing hormone-like peptides (pGlu-X-Pro-
30 with chronic ECS such as neuropeptide Y and thyrotropin-releasing hormone, may provide novel ways to
34 ith the calcium ionophore ionomycin, or with thyrotropin releasing hormone or vasoactive intestinal p
35 he nonhydrolysable cAMP analog 8-bromo-cAMP, thyrotropin releasing hormone, or cholecystokinin reveal
36 modulation of RTN activity by Substance P or thyrotropin-releasing hormone, previously identified neu
37 ral preoptic nucleus, and a mainly glutamate-thyrotropin-releasing hormone projection to the wake-pro
38 ysis showed that proteolytic cleavage of pro-thyrotropin-releasing hormone (proTRH) at known PC cleav
39 ovirus vector expressing the mouse pituitary thyrotropin releasing hormone receptor (TRHR) cDNA ectop
41 rmore, IGSF1 stimulates transcription of the thyrotropin-releasing hormone receptor (TRHR) by negativ
42 ling are required for internalization of the thyrotropin-releasing hormone receptor (TRHR), we compar
44 93 cells co-expressing TREK-1 and either the thyrotropin-releasing hormone receptor (TRHR1) or the Or
46 udy phosphorylation of the endogenous type I thyrotropin-releasing hormone receptor in the anterior p
47 the C-terminal tail of either the mammalian thyrotropin-releasing hormone receptor or the catfish Gn
49 tion-selective ganglion cells (dsGCs): TRHR (thyrotropin-releasing hormone receptor) and Drd4 (dopami
50 otensin receptor 1, vasopressin V2 receptor, thyrotropin-releasing hormone receptor, and substance P
51 five structurally diverse antagonists of the thyrotropin-releasing hormone receptors (TRH-R1 and TRH-
52 rvicellular corticotropin-releasing hormone, thyrotropin-releasing hormone, somatostatin, and dopamin
56 ated by food availability via leptin-induced thyrotropin-releasing hormone/thyroid-stimulating hormon
58 y cooperative interaction between leptin and thyrotropin releasing hormone (TRH) in the hindbrain to
59 istry and RT-PCR demonstrated a reduction of Thyrotropin Releasing Hormone (TRH) in the hypothalamus
61 gene expression of Sim1, oxytocin (OXT) and thyrotropin releasing hormone (TRH) was reduced by about
62 of 5-hydroxytryptamine (serotonin, 5-HT) and thyrotropin-releasing hormone (TRH) act synergistically
63 tility following bilateral microinjection of thyrotropin-releasing hormone (TRH) analog, RX 77368, in
64 n-Leu-Pro-Gly, a progenitor sequence for the thyrotropin-releasing hormone (TRH) analogue [Leu(2)]TRH
65 Metabolically stable and centrally acting thyrotropin-releasing hormone (TRH) analogues were desig
66 f these two Ca2+ signaling pathways mediates thyrotropin-releasing hormone (TRH) and angiotensin II (
67 greater ligand-independent activation on the thyrotropin-releasing hormone (TRH) and common glycoprot
68 has an important action on hypophysiotropic thyrotropin-releasing hormone (TRH) and corticotropin-re
69 cifically from subsets of neurons expressing thyrotropin-releasing hormone (TRH) and pituitary adenyl
70 nism of trans-repression of the hypothalamic thyrotropin-releasing hormone (TRH) and pituitary thyroi
71 y and pontine raphe and receives inputs from thyrotropin-releasing hormone (TRH) and substance P-expr
72 pituitary-thyroid (HPT) axis hormones, i.e., thyrotropin-releasing hormone (TRH) and thyrotropin (TSH
73 al cells were found to express receptors for thyrotropin-releasing hormone (TRH) and to be a primary
77 calization of an epitope-tagged receptor for thyrotropin-releasing hormone (TRH) expressed in differe
78 Hypothalamic T3 content is decreased while thyrotropin-releasing hormone (TRH) expression is elevat
79 daptive response is caused by a reduction in thyrotropin-releasing hormone (TRH) expression that can
80 y marginally elevated in transgenic mice and thyrotropin-releasing hormone (TRH) gene expression in t
83 and prolonged increases in the neuropeptide thyrotropin-releasing hormone (TRH) in epileptogenic sit
85 ve feedback loop that inhibits production of thyrotropin-releasing hormone (TRH) in the mediobasal hy
87 nin) enhanced GIRK channel currents, whereas thyrotropin-releasing hormone (TRH) inhibited both basal
95 ed in fibers that innervate hypophysiotropic thyrotropin-releasing hormone (TRH) neurons and modulate
96 nal and cellular effects of the neuropeptide thyrotropin-releasing hormone (TRH) on the spontaneously
98 jection of peptide YY (PYY) and low doses of thyrotropin-releasing hormone (TRH) or TRH analog, RX 77
100 ly that left ventricular gene expression for thyrotropin-releasing hormone (TRH) precursor was increa
101 ected to express the long isoform of the rat thyrotropin-releasing hormone (TRH) receptor (clone E2)
102 ryptophan and other aromatic residues of the thyrotropin-releasing hormone (TRH) receptor (TRH-R) tha
103 k, a model of the extracellular loops of the thyrotropin-releasing hormone (TRH) receptor (TRHR) was
104 ced Cl- currents in oocytes co-injected with thyrotropin-releasing hormone (TRH) receptor cRNA 48 h p
105 mutational and computational studies of the thyrotropin-releasing hormone (TRH) receptor identified
107 smembrane helices 5 and 6 (TMH5 and TMH6) of thyrotropin-releasing hormone (TRH) receptor type I (TRH
115 feedback inhibition of thyrotropin (TSH) and thyrotropin-releasing hormone (TRH) synthesis in the pit
117 Galanin, gamma-aminobutyric acid (GABA), and thyrotropin-releasing hormone (TRH) were colocalized wit
118 calcium concentration ([Ca2+]i) responses to thyrotropin-releasing hormone (TRH) were measured in HEK
120 the activity of orexin cells is modulated by thyrotropin-releasing hormone (TRH), an endogenous stimu
121 cretin, melanin-concentrating hormone (MCH), thyrotropin-releasing hormone (TRH), gonadotropin-releas
123 t neurotransmitter substances, glutamate and thyrotropin-releasing hormone (TRH), upon craniofacial g
124 ear ER-alpha ir was found in a population of thyrotropin-releasing hormone (TRH)-expressing neurons i
125 In rat pituitary lactotrophs, a component of thyrotropin-releasing hormone (TRH)-induced prolactin se
127 center for the central actions of leptin on thyrotropin-releasing hormone (TRH)-synthesizing neurons
133 dose: 100 nM), compared to that observed for thyrotropin-releasing hormone (TRH, minimum effective do
134 We and others have previously reported that thyrotropin-releasing hormone (TRH, pGlu-His-Pro-NH(2))
138 unit [TSH alpha], TSH beta, and hypothalamic thyrotropin-releasing hormone [TRH] genes), stimulation
139 y cooperative interaction between leptin and thyrotropin-releasing hormone [TRH] to activate hindbrai
140 y cooperative interaction between leptin and thyrotropin-releasing hormone [TRH] to activate hindbrai
141 f these channels also disrupted transmitter (thyrotropin-releasing hormone, TRH) inhibition and did s
142 including corticotropin-releasing hormone-, thyrotropin-releasing hormone-, vasopressin-, and oxytoc
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