1 dase inhibitor, epidermal growth factor, and tick anticoagulant peptide.

2  naturally occurring FXa inhibitors, such as tick anticoagulant peptide and antistasin, has helped su

3 anemone neurotoxin, and the homology between tick anticoagulant peptide and bovine trypsin inhibitor.

4 ence of hirudin and is completely blocked by tick-anticoagulant-peptide and D-Glu-Gly-Arg-chloromethy

5 sue factor pathway inhibitor and recombinant tick anticoagulant peptide, but does not compete for bin

6 or Xa chromogenic substrate, and recombinant tick anticoagulant peptide did not suppress activity of

7 din but abolished by active site inhibitors, tick anticoagulant peptide, or Glu-Gly-Arg-chloromethyl

8                                              Tick anticoagulant peptide reduced the occurrence of reo

9                 The structure of recombinant tick anticoagulant peptide (rTAP) complexed to bovine fa

10                                  Recombinant tick anticoagulant peptide (rTAP) is a competitive slow-

11 short-term administration of the recombinant tick anticoagulant peptide (rTAP), a factor Xa inhibitor

12 specific inhibitor of factor Xa, recombinant tick anticoagulant peptide (rTAP), alone or in combinati

13 actor XII), heparin, enoxaparin, recombinant tick anticoagulant peptide (rTAP), or recombinant hirudi

14 parin (100 U/kg per h, n = 6) or recombinant tick anticoagulant peptide (rTAP, 1-mg/kg bolus followed

15                              In this report, tick anticoagulant peptide (TAP) (60 amino acids and 3 d

16 FPI), and the selective factor Xa inhibitor, tick anticoagulant peptide (TAP), after thrombolytic the

17 actions with prothrombin and the recombinant tick anticoagulant peptide that is known to interact wit

18 een probed using a recombinant derivative of tick anticoagulant peptide where Arg3 has been replaced