ライフサイエンスコーパス: tick bite

1 haped and unable to infect mice by needle or tick bite.

2 uitment of leukocytes to the location of the tick bite.

3 C3H/HeN mice via either needle injection or tick bite.

4 nd imply host exposure to alpha-Gal during a tick bite.

5 ere non-infectious in mice when delivered by tick bite.

6 nvasion and erythema migrans formation after tick bite.

7 g with the host inflammatory response to the tick bite.

8 Many patients do not recall a tick bite.

9 ially by syringe inoculation or naturally by tick bite.

10 h N40 by syringe than in those inoculated by tick bite.

11 tral North Carolina with a recent history of tick bite.

12 an infectious dose of B. burgdorferi B31 by tick bite.

13 eri B31 via the natural transmission mode of tick bite.

14 susceptible to B. burgdorferi infection via tick bite.

15 associated with total IgE levels and recent tick bites.

16 e virus is transmitted to humans by infected tick bites.

17 rom homologous substances transferred during tick bites.

18 ed associations between IgE to alpha-Gal and tick bites.

19 he Flaviviridae family and is transmitted by tick bites.

20 k bites has been shown effective in reducing tick bites.

21 Most cases (69%) were attributed to tick bites.

22 led to establish a new infection in mice via tick bites.

23 d in ng/ micro l, was used as a biomarker of tick bites.

24 in wooded environments that are conducive to tick bites.

25 o of the four infected persons were aware of tick bites.

26 se tick bites, as measured by a biomarker of tick bites.

27 and their persistence in the blood prior to tick bites.

28 gdorferi, is introduced into human hosts via tick bites.

29 B) are acquired after Ixodes ricinus-complex tick bites.

30 ick typhus even in the absence of recognised tick-bites.

31 ified 142 patients (61.2%) with diagnoses of tick bites, 40 patients (17.2%) with LD, and 50 patients

32 32% of direct charges were for patients with tick bites, 48% were for patients with LD, and 20% were

33 Of the 142 patients seen for tick bites, 95 (67%) underwent serologic testing for LD.

34 with Ixodes scapularis acquire resistance to tick bites, a phenomenon, known as tick immunity, that i

35 sympatric isolates of A. phagocytophilum via tick bite and challenged 16 weeks later by Ixodes scapul

36 ice were infected with E. phagocytophila via tick bite and challenged either 12 or 16 weeks later by

37 The pathogenic relationship between tick bites and sensitization to red meat, alpha-Gal, and

38 icomponent vaccine that will protect against tick bites and the pathogens they transmit.

39 estimate the frequency of Ixodes scapularis tick bites and the resulting incidence of Lyme disease i

40 collected from all animals for 14 days after tick bite, and spirochete densities were assessed by qua

41 th strain N40 by syringe, with strain N40 by tick bite, and with strain Pbi by syringe).

42 thorough exposure history, particularly for tick bites, and awareness of clinical features may promp

43 d LD, presumptive cases of LD, patients with tick bites, and diagnostic tests ordered for LD.

44 esumptive LD, seen and given prophylaxis for tick bites, and having diagnostic tests indicate that re

45 es and those of others strongly suggest that tick bites are a cause, if not the only significant caus

46 This provides further evidence that tick bites are associated with IgE responses to alpha-Ga

47 ck bite prevention and consequently decrease tick bites, as measured by a biomarker of tick bites.

48 istatin is a RAGE antagonist that suppresses tick bite-associated inflammation, allowing successful b

49 otic therapy for prophylaxis of LD following tick bites, but the extent to which physicians in endemi

50 given within 72 hours after an I. scapularis tick bite can prevent the development of Lyme disease.

51 Tick bites constitute the primary sensitization source,

52 No patients with tick bite developed clinical LD.

53 product was incapable of infecting mice via tick bite even though the mutant was (i) infectious in m

54 nt failed to establish infection in mice via tick bite, exogenous supplementation of GlcNAc into unfe

55 tive to doxycycline for treatment of African tick bite fever in some selective situations.

56 African tick bite fever is the most commonly encountered travel-

57 n-impregnated clothing for the prevention of tick bites has been shown effective in reducing tick bit

58 rythema migrans developed at the site of the tick bite in a significantly smaller proportion of the s

59 lood samples from patients with a history of tick bite in the preceding 2 months at Mudanjiang Forest

60 index patient acquired the disease through a tick bite in the province of Avila - 300 km away from th

61 informed decisions concerning management of tick bites in HGE-endemic areas.

62 nse in Lyme disease patients, and in mice by tick bite inoculation, provides evidence that the Rev pr

63 ination of B. burgdorferi from the site of a tick bite likely occurs, in part, via attachment to host

64 ch are likely to be present at the site of a tick bite, may play a role in preparing the organism for

65 lmost exclusively in the limb closest to the tick bite (median incubation period, 66 days).

66 were made to infect immunodeficient mice by tick bite or inoculation of tick contents.

67 were completely protected against a natural tick bite or subcutaneous (s.c.) inoculation of 297.

68 . burgdorferi sensu strictu by syringe or by tick bite or with strain Pbi of B. burgdorferi genospeci

69 effectiveness of these pesticides to prevent tick bites or human tick-borne diseases is unknown.

70 ted dogs, and four had a reported history of tick bite preceding the illness.

71 5-year-old female with a history of multiple tick bites presented with fever and pancytopenia.

72 attitudes, and reported behaviors related to tick bite prevention and consequently decrease tick bite

73 Mice infected with B31 by tick bite produced antibodies that recognized each of th

74 Lyme disease begins at the site of a tick bite, producing a primary infection with spread of

75 nty-eight patients (55%) with a diagnosis of tick bite received antibiotic therapy.

76 ing, most patients consulting physicians for tick bites received prophylactic antibiotic therapy of u

77 develop an influenzalike illness following a tick bite should include HGE.

78 f severe nocturnal anaphylaxis due to pigeon tick bite showing the diagnostic value of the extract an

79 on of topical antibiotics to the skin at the tick bite site could eradicate Borrelia burgdorferi infe

80 is article, we describe NET formation at the tick bite site in vivo in mice.

81 ctive in eliminating B. burgdorferi from the tick bite site or in preventing dissemination to other t

82 eliosis, disseminate hematogenously from the tick bite site to the joints, the heart, and the central

83 igrans (EM) skin lesion that develops at the tick bite site typically between 7 and 14 days after inf

84 cond that causes only local infection at the tick bite site, and a third that causes systemic disease

85 s within neutrophil granulocytes that invade tick bite sites are likely ingested by the vector, to be

86 h a transient expression in the host only at tick-bite sites.

87 ssues of the somatic quadrant closest to the tick bite than in tissues further from the site of infec

88 Rash, history of tick bite, thrombocytopenia, and hyponatremia were often

89 sease, spirochetes spread from the site of a tick bite to colonize multiple tissue sites, causing mul

90 After tick-bite transmission back to mice, the process was rev

91 Importantly, tick bite upregulated RAGE ligands in skin, and endogeno

92 m is often abundant and aggressive, and many tick bite victims receive multiple bites.

93 Nymphs remain attached to adult tick-bite victims longer than they remain attached to ch

94 albeta1-(3)4GlcNAc-R (alpha-gal) following a tick bite was associated with allergies to red meat, cet

95 , disease caused by Borrelia turicatae after tick bite was compared in 2 rhesus macaques in which rad

96 nted with a definite Ixodes scapularis (Say) tick bite were measured to determine the utility of the

97 clinical symptoms, antibiotic treatments, or tick bites were not more frequent in E. equi- or B. burg

98 Persons with tick bites were prospectively studied to determine if th

99 crobial treatment after an Ixodes scapularis tick bite will prevent Lyme disease.

100 observed with serum from a mouse infected by tick bite, with the recognition of two additional bands

101 olina presenting with fever and a history of tick bite within the preceding 14 days.

102 n order to determine whether transmission by tick bite would alter the dissemination dynamics and dis