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1 ine derivative, may be a safe alternative to ticlopidine.
2 clofenac, imipramine, meclofenamic acid, and ticlopidine.
3 r side effects make it a safe alternative to ticlopidine.
4 lopidogrel exhibits better tolerability than ticlopidine.
5 er clopidogrel is at least as efficacious as ticlopidine.
6 ng acetylsalicylic acid, clopidogrel, and/or ticlopidine.
7 utine antiplatelet therapy with aspirin plus ticlopidine.
8 All patients received aspirin and ticlopidine.
9 d chemical structure are similar to those of ticlopidine.
10 angiopathy, be aware of its association with ticlopidine.
13 tive stent patients treated with aspirin and ticlopidine (500 mg loading dose and 250 mg twice daily
17 emilofiban (5, 10, 15, or 20 mg) or placebo (ticlopidine) administered 8 to 18 hours after terminatio
18 360 patients who discontinued clopidogrel or ticlopidine after 6 months, stent thrombosis subsequentl
20 common, and affected patients must switch to ticlopidine after drug-eluting stent placement, despite
23 n, clopidogrel confers similar protection as ticlopidine against subacute stent thrombosis and major
25 d by 133 (14.7%) of 902 patients assigned to ticlopidine and 112 (12.3%) of 907 patients assigned to
28 went coronary stenting and were treated with ticlopidine and aspirin (TA group, n=1406), and patients
29 acy of clopidogrel and aspirin with those of ticlopidine and aspirin in patients undergoing coronary
30 statistically significant difference between ticlopidine and aspirin in the prevention of recurrent s
35 yridamole (Persantine), and thienopyridines (ticlopidine and clopidogrel) are all examples of agents
38 linical settings include the thienopyridines ticlopidine and clopidogrel, specific inhibitors of ADP-
40 curred in 3.64% of the patients treated with ticlopidine and in 1.62% of the patients treated with cl
41 vent occurred in 4.60% of patients receiving ticlopidine and in 3.85% of patients receiving clopidogr
42 veloping thrombotic microangiopathy while on Ticlopidine and the possible mechanisms underlying this
46 ed antiplatelet drugs, including aspirin and ticlopidine, are effective against certain but not all o
47 2 antagonists (separate from clopidogrel and ticlopidine) as an approach to the treatment of thromboe
52 h clopidogrel-associated TTP cases (n = 35), ticlopidine-associated TTP cases (n = 93) were more like
54 cedure, but 58% of these patients were given ticlopidine before stenting at the discretion of the inv
55 ng patients randomized to stenting, starting ticlopidine before the percutaneous coronary interventio
56 lyses revealed a less than 1% probability of ticlopidine being shown superior to aspirin in the preve
57 idogrel has a similar mechanism of action as ticlopidine, but both its efficacy and its safety as a p
58 1 in 1600 to 1 in 5000 patients who receive ticlopidine, but little is known about the pathogenesis
60 Y(12) is the target of antithrombotic drugs (ticlopidine, clopidogrel), whereas the role of P2Y(1) in
62 rapy with aspirin and a P2Y12 inhibitor (eg, ticlopidine, clopidogrel, prasugrel, ticagrelor) reduces
66 teristics and for the propensity of being on ticlopidine, Cox proportional hazards regression identif
67 were closely monitored for side effects from ticlopidine developed side effects requiring its discont
70 hout thrombocytopenia who had been receiving ticlopidine for 3 to 5 weeks and 10 randomly selected ho
74 ent occurred in 1.92% of the patients in the ticlopidine group and in 2.02% of the clopidogrel group
75 r in the clopidogrel group compared with the ticlopidine group-0.48% versus 1.09% (OR 0.55, 95% CI 0.
82 ety and efficacy have not been compared with ticlopidine in a randomized manner in the United States.
85 nd registries that compared clopidogrel with ticlopidine in patients receiving coronary stents were p
87 nt with the platelet ADP-receptor antagonist ticlopidine in preventing both the early and late compli
90 in is routinely used with stenting, although ticlopidine is commonly not begun until the day of the p
92 and little is known about the duration that ticlopidine needs be administered to prevent stent throm
93 84), and postoperative use of clopidogrel or ticlopidine (odds ratio, 1.35; 95% confidence interval,
99 tinuation of oral antiplatelet therapy (with ticlopidine or clopidogrel) and only in patients who had
101 s were randomized to receive placebo (250 mg ticlopidine P.O. BID) or xemilofiban in doses of 5, 10,
103 nd 30 (5.5 percent) who received aspirin and ticlopidine (P<0.001 for the comparison of all three gro
104 0.5 percent) assigned to receive aspirin and ticlopidine (P=0.001 for the comparison of all three gro
105 0.4% in clopidogrel patients versus 1.1% in ticlopidine patients; nonfatal myocardial infarction occ
106 ore, clopidogrel plus aspirin should replace ticlopidine plus aspirin as the standard antiplatelet re
108 et inhibitors: thienopyridines (clopidogrel, ticlopidine, prasugrel) or IIb/IIIa receptor antagonists
109 x proportional hazards regression identified ticlopidine pretreatment as an independent predictor of
114 he adenosine diphosphate receptor antagonist ticlopidine prior to intracoronary stenting is associate
115 mes in 175 consecutive patients treated with ticlopidine prior to stenting at the Cleveland Clinic Fo
117 ing coronary artery stenting have shown that ticlopidine reduces the risk for subacute stent thrombos
118 timal" stenting combined with an aspirin and ticlopidine regimen carries a low (0.5%) incidence of su
119 rin and warfarin, treatment with aspirin and ticlopidine resulted in a lower rate of stent thrombosis
121 the observed 30-day event rate of 1.6% with ticlopidine, the statistical power of the study was 43%
122 intracoronary stents, the discontinuation of ticlopidine therapy 14 days after stent placement is ass
126 study was to determine if combined abciximab/ticlopidine therapy inhibits arterial thrombosis more ef
128 ary stents, stent thrombosis is reduced when ticlopidine therapy is combined with aspirin after the p
130 Of the 3 treatments, combined abciximab/ticlopidine therapy produced the most consistent reducti
131 undergoing intracoronary stenting, beginning ticlopidine therapy several days prior to the procedure
133 ergoing PTCA who received abciximab therapy, ticlopidine therapy, or both treatments were evaluated u
135 conformation allows smaller ligands such as ticlopidine to bind to the 2B6 active site in the expect
139 of a TTP incidence of 0.02% in our cohort of ticlopidine-treated patients following coronary stenting
140 hrombosis for only 1 day after PTCA, whereas ticlopidine treatment alone had no significant effect.
146 eeks of ticlopidine were exclusion criteria; ticlopidine was discontinued after 14 days in all remain
148 d NMR relaxation of drug protons showed that ticlopidine was preferentially oriented with the chlorop
150 mic event with clopidogrel, as compared with ticlopidine, was 0.72 (95% confidence interval [CI] 0.59
151 and the risk of serious adverse events with ticlopidine, we regard aspirin as a better treatment for
152 t in research protocols requiring 4 weeks of ticlopidine were exclusion criteria; ticlopidine was dis
153 ports of TTP associated with clopidogrel and ticlopidine were identified from medical records, publis
155 with the antiplatelet drugs clopidogrel and ticlopidine, which were expected to have greater freedom
156 he study compared the safety and efficacy of ticlopidine with clopidogrel in patients receiving coron
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