ライフサイエンスコーパス: tiopronin

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1 Here we demonstrate that tiopronin (1), a thiol-substituted N-propanoylglycine de

2 e-coated gold MPCs with aqueous solutions of tiopronin-coated gold MPCs yields toluene-phase MPCs tha

3 te-coated gold MPCs and aqueous solutions of tiopronin-coated silver MPCs, in which tiopronin ligands

4 Synthesis and screening of analogues of tiopronin demonstrated that the thiol functional group w

5 despite functional assays demonstrating that tiopronin does not interact with P-gp.

6 The tiopronin/ethidium MPC binding to DNA was imaged by AFM.

7 were either N-(2-mercaptopropionyl)glycine (tiopronin/ethidium MPC) or trimethyl(mercaptoundecyl)amm

8 The negatively charged tiopronin/ethidium MPC, in contrast, exhibits slow inter

9 The drug tiopronin has been previously shown to elicit CS.

10 culty in exploiting this hypersensitivity to tiopronin in the clinic.

11 We have determined that the CS activity of tiopronin is mediated by the generation of reactive oxyg

12 (GPx), and the mode of inhibition of GPx1 by tiopronin is shown in this report.

13 Treatment of MRP1-overexpressing cells with tiopronin led to a significant reduction in MRP1 protein

14 PCs yields toluene-phase MPCs that have some tiopronin ligands and aqueous-phase MPCs that have some

15 ns of tiopronin-coated silver MPCs, in which tiopronin ligands are transferred to the former and gold

16 by monolayers of N-acetyl-l-cysteine and of tiopronin ligands.

17 e used to study the synthesis of a series of tiopronin monolayer-protected gold nanoclusters (MPCs) a

18 tionation of N-(2-mercaptopropionyl)glycine (tiopronin) monolayer protected gold clusters into monodi

19 N-(2-mercaptopropionyl)glycine (tiopronin) monolayer-protected silver particles were par

20 trophotometry verified the separation of the tiopronin MPCs through the inspection of surface plasmon

21 CFE separation of water-soluble tiopronin MPCs yielded fractions that varied in color, U

22 abundance (with respect to disulfide bridged tiopronin species) before dramatically increasing in abu

23 Specifically, selective toxicity of tiopronin toward MDR cells is achieved by inhibition of

24 of tri(ethylene glycol) thiol (EG(3)-SH) and tiopronin (Tp), which was prepared by a one-step synthes

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