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1 blockers/inhibitors (edaravone and lazaroids/tirilazad).
2                                    Desmethyl tirilazad (20 mg/kg) and 100 mg/kg deferoxamine inhibit
3                                    Desmethyl tirilazad (20 mg/kg) improves neurologic exam, but 3 mg/
4 nisms of cytoprotective action may exist for tirilazad and the antioxidant gossypol.
5  unlikely that the antioxidant properties of tirilazad are involved in the suppression of motor nerve
6 ssigned to receive either 20 mg/kg desmethyl tirilazad at -15 mins followed by 8 mg/kg/hr for 90 mins
7                          High-dose desmethyl tirilazad improves neurologic function after hypoxic isc
8 a significant increase in r, suggesting that tirilazad intercalates into the membrane bilayer and pro
9                                    Desmethyl tirilazad is a lipid-soluble free radical quencher.
10                                              Tirilazad itself caused a significant increase in r, sug
11              Enhanced relative survival with tirilazad mesylate also was seen in cultures containing
12                                     However, tirilazad mesylate attenuated sepsis-induced acute lung
13                        Pretreatment with the tirilazad mesylate did not change P. aeruginosa sepsis-i
14                                              Tirilazad mesylate enhanced relative ganglion cell survi
15 ith the 21-aminosteroid antioxidant compound tirilazad mesylate has been previously shown to retard t
16               The lipid peroxidation blocker tirilazad mesylate inhibits retinal ganglion cell death
17                               Treatment with tirilazad mesylate significantly attenuated hypoxemia an
18 cantly greater when it was co-incubated with tirilazad mesylate than with diluent control (91.8% +/-
19 hibition, and the effect on cell survival of tirilazad mesylate was assessed.
20                                          The tirilazad mesylate-treated group received a 5-mg/kg bolu
21                            Neither desmethyl tirilazad nor deferoxamine improves pathologic results.
22 roves neurologic exam, but 3 mg/kg Desmethyl tirilazad or 100 mg/kg deferoxamine does not.
23 f cells with lipophilic antioxidants such as tirilazad or gossypol.
24 assigned to receive either 3 mg/kg desmethyl tirilazad or vehicle at -15 and 90 mins.
25                                              Tirilazad pretreatment caused a dose-related suppression
26            In the present study, we examined tirilazad's effects (7.7, 13.0 or 30.0 mg/kg twice daily
27 y because of its antioxidant action, whereas tirilazad's protection is mediated not only via its anti
28                  Rather, it is proposed that tirilazad suppresses delayed motor nerve terminal Ca2+ c

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