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1 ry, and chemokine expression, and normalized tissue survival.
2 tration resulted in the greatest decrease in tissue survival.
3 le factor 1alpha stabilization and increased tissue survival.
4 lar networks in vertebrates are essential to tissue survival.
5 pathological TNF-alpha reaction and promote tissue survival.
6 ry progression, suggesting an active role in tissue survival.
7 al activation of these receptors may promote tissue survival.
8 adversely affect microvascular function and tissue survival after an ischemic episode, and previous
9 l mitogenesis, a repair process important to tissue survival after ischemic damage, was not disrupted
10 e results highlight a pathway that preserves tissue survival and muscle function in the setting of is
11 wth, differentiation, homing to their target tissues, survival and activation are all controlled, to
13 playing roles in signaling, cell migration, tissue survival, anti-inflammation, and T-cell-mediated
15 vity (firing rate and thermosensitivity) and tissue survival as a function of time and slice thicknes
19 1%, P<0.005) that resulted in impaired gross tissue survival compared with young mice (2 to 6 months)
20 diated vascular smooth muscle relaxation and tissue survival following ischemic injury in skin flaps
27 myocutaneous flap model for ischemic injury, tissue survival was significantly enhanced in thrombospo
28 rin loss may be incompatible with epithelial tissue survival, whereas partial compensation can result
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