ライフサイエンスコーパス: tissue-resident

1 one of which recirculates while the other is tissue resident.

2 se revealed that retinal CD8(+) T cells were tissue resident.

3 Tissue-resident adult stem cells (AdSCs) also form organ

4 sequences associated with global monocyte or tissue-resident alveolar macrophage depletion.

5 e showed that DAP12 promotes the survival of tissue-resident alveolar macrophages and contributes to

6 the lung ameliorated lung fibrosis, whereas tissue-resident alveolar macrophages did not contribute

7 ly unrecognized role for DAP12 expression in tissue-resident alveolar macrophages in mediating acute

8 veolar macrophages differ significantly from tissue-resident alveolar macrophages in their expression

9 from pathogens by alveolar epithelial cells, tissue-resident alveolar macrophages, dendritic cells, a

10 s, where they became increasingly similar to tissue-resident alveolar macrophages.

11 Different cellular sources, including tissue-resident and bone marrow-derived fibroblasts, per

12 ed DCs (moDCs), including CD11b(hi)Ly-6C(lo) tissue-resident and CD11b(hi)Ly-6C(hi) inflammatory moDC

13 We conclude that tissue-resident and circulating Fgfbp1-expressing cells

14 is restrained by the concerted activation of tissue-resident and circulating immune cells.

15 on of MAFB and MAFB-target genes in CD163(+) tissue-resident and tumor-associated macrophages.

16 ii) epithelial-associated, or (iii) lymphoid tissue-resident, and we discuss the role and regulation

17 Adipose tissue resident B cells account for more than 20% of str

18 c lineage tracing analysis demonstrates that tissue-resident, but not circulating, Gli1(+) cells prol

19 cate TGF-beta-Smad2/3 signaling in activated tissue-resident cardiac fibroblasts as principal mediato

20 However, lung-tissue-resident CD1c(+) DCs, but not CD141(+) DCs, were

21 umber of circulating CD4 and CD8 T cells and tissue-resident CD3 T cells in the gut mucosa and bone m

22 ather than residing in the lung as bona fide tissue-resident CD69(+) NK cells.

23 resent Ags to CD8(+) T cells, mouse lymphoid tissue-resident CD8(+) dendritic cells (DCs) and their m

24 onse to IL-15 trans presentation by lymphoid tissue-resident CD8alpha(+) dendritic cells in the perip

25 play between the inflammatory infiltrate and tissue resident cell populations invokes fibrogenesis.

26 lations, such as Ag-specific, functional, or tissue-resident cell subsets isolated by sorting, microd

27 Notably, CXCL1 expression was restricted to tissue-resident cell types, but IC-activated neutrophils

28 pansion of blood vessel networks directed by tissue-resident cells and morphogenetic cues.

29 wed us to dissect the importance of DAP12 in tissue-resident cells and those that infiltrate injured

30 associated with inflammation, revealing that tissue-resident cells are poised to sense and respond to

31 he production of interleukin-17A (IL-17A) by tissue-resident cells early during infection, but the me

32 Mobilization of tissue-resident cells into the blood removes CLL cells f

33 Mast cells (MCs), which are granulated tissue-resident cells of hematopoietic lineage, constitu

34 Mast cells (MCs), which are granulated tissue-resident cells of hematopoietic lineage, contribu

35 icular, the influence of mast cells on other tissue-resident cells remains poorly understood.

36 ne homeostasis is maintained by a network of tissue-resident cells that continually monitor the exter

37 VAT-Treg cells) are functionally specialized tissue-resident cells that prevent obesity-associated in

38 dicate that DEJ CD8alphaalpha(+) T cells are tissue-resident cells that seem to have a fundamental ro

39 d nonlymphoid organs of adult mice, ILCs are tissue-resident cells that were maintained and expanded

40 ctate the cytoarchitecture and fate of other tissue-resident cells to suppress their malignant outgro

41 N2-specific memory CD8(+) T cells, including tissue-resident cells, compared with placebo treatment.

42 Tissue-resident cells, such as epithelial cells, are tho

43 ce of inflammatory and infectious stimuli as tissue-resident cells, with enhanced recruitment, activa

44 pathogen-specific memory T cells, including tissue-resident cells.

45 he three groups of macaques were observed in tissue-resident cells.

46 between macrophages, dying cells, and other tissue-resident cells.

47 Therefore, the mobilization of tissue-resident CLL cells into the circulation is a usef

48 In particular, tissue-resident CLL cells show prominent activation of b

49 Here, we demonstrate that lymphoid-tissue-resident commensal bacteria (LRC) colonized murin

50 differentially infect and replicate in these tissue-resident DC and support the hypothesis that these

51 this study, we identify an immunoregulatory tissue-resident dendritic cell (DC) in the dermis of hum

52 sed skin as a model to determine the role of tissue-resident dendritic cells (DCs) in local and syste

53 ies that coordinate mesenchymal migration of tissue-resident dendritic cells.

54 al role for inflammatory monocytes and their tissue-resident derivatives in the first 48 hours postin

55 These data suggest that tissue resident effector cell numbers and low FcgammaR e

56 ute a distinct population of circulating and tissue-resident effector T cells with immune-regulatory

57 Instead of circulating cells, tissue-resident endothelial cells or EPCs may primarily

58 MHCII(+) macrophages into macrophages with a tissue-resident F4/80(hi)CD206(-)PD-L2(-)MHCII(-)UCP1(+)

59 can alter the composition of circulating and tissue-resident fetal immune cells.

60 Mechanistically, loss of Smad2/3 from tissue-resident fibroblasts attenuated injury-induced ce

61 sing myofibroblasts in the heart derive from tissue-resident fibroblasts of the Tcf21 lineage, but no

62 sis in the heart, which arises from Tcf21(+) tissue-resident fibroblasts.

63 Innate lymphoid cells (ILCs) are tissue-resident "first responders" of the immune system

64 achieving sufficient levels of IL-2 to boost tissue-resident Foxp3(+)Tregs while avoiding the potenti

65 , requires interleukin (IL)-13 production by tissue-resident group 2 innate lymphoid cells (ILC2s) an

66 The IL-23-driven tissue-resident group 3 innate lymphoid cells (ILC3s) pl

67 and functional maturation of circulating and tissue-resident human NK and CD8(+) T cells and promoted

68 How ILCPs give rise to mature tissue-resident ILCs remains unclear.

69 s is critically dependent on the function of tissue-resident immune cells and the differentiation cap

70 ss-talk between the bronchial epithelium and tissue-resident immune cells controls the tissue microen

71 obes modulate the maturation and function of tissue-resident immune cells in the CNS.

72 Mast cells are rare tissue-resident immune cells that are involved in allerg

73 Mast cells are unique tissue-resident immune cells that express an array of re

74 a constitute a highly specialized network of tissue-resident immune cells that is important for the c

75 Mast cells are tissue-resident immune cells that play a central role in

76 distinct hematopoietic cell types, including tissue-resident immune cells, distinguishes fetal from a

77 he ability of lifelong HSCs to contribute to tissue-resident immune cells.

78 matic increase in inflammatory mediators and tissue-resident immune cells.

79 indicating that human CD49e(-) NK cells are tissue resident in the liver.

80 dictate ILC phenotype and function for these tissue-resident innate effector cells.

81 8(+) T cells and promoted the development of tissue-resident innate lymphoid cell (ILC) subsets.

82 The discovery of tissue-resident innate lymphoid cell populations effecti

83 The signals that maintain tissue-resident innate lymphoid cells (ILC) in different

84 In this Review, we discuss the hallmarks of tissue-resident innate, innate-like, and adaptive lympho

85 se data identify a physiological function of tissue-resident kidney macrophages and a basic mechanism

86 quence of both the disruption of homeostatic tissue resident leukocytes and the recruitment of antago

87 n parallel with shifts in the composition of tissue-resident leukocytes and with an accumulation of a

88 A putative subset of tissue-resident long-lived stem cells is characterized b

89 ites, such as the skin, gut, and lung, these tissue-resident lymphocyte populations are ideally posit

90 rize recent advances in the understanding of tissue-resident lymphocyte populations, review the avail

91 we show that cell transformation triggers a tissue-resident lymphocyte response in oncogene-induced

92 These specialized tissue-resident lymphocyte subsets span the innate-adapt

93 ells have an integrin profile reminiscent of tissue-resident lymphocytes and express TRAIL for killin

94 Whether tissue-resident lymphocytes confer early antiviral immun

95 r for Kruppel-like factor (KLF)4-a driver of tissue-resident macrophage differentiation.

96 sults provide insight into the regulation of tissue-resident macrophage functional specialization by

97 nd these metabolites are crucially linked to tissue-resident macrophage functions.

98 of both bone marrow-derived inflammatory and tissue-resident macrophage lineage branches is a key fea

99 Like other tissue-resident macrophage populations, microglia expres

100 s of four histone modifications across seven tissue-resident macrophage populations.

101 le of metabolite-driven differentiation of a tissue-resident macrophage subset and provide new insigh

102 veal a mechanism by which lipid oxidation by tissue resident macrophages could inhibit the engulfment

103 s of GFP in both monocyte and embryo-derived tissue resident macrophages in adult animals.

104 The specific function of microglia, the tissue resident macrophages of the brain and spinal cord

105 can be derived either from proliferation of tissue resident macrophages or recruited inflammatory mo

106 een fluorescence protein (GFP) in monocytes, tissue resident macrophages, and inflammatory macrophage

107 lls, or inflammatory monocytes, functions of tissue resident macrophages, including alveolar macropha

108 lexes that activate strategically positioned tissue resident macrophages.

109 F, which also controls homeostatic levels of tissue resident macrophages.

110 atory reactions, besides self-replication of tissue resident macrophages.

111 er within 12-24 hours, whereas the number of tissue-resident macrophages (Kupffer cells) decreased.

112 Although classified as hematopoietic cells, tissue-resident macrophages (MFs) arise from embryonic p

113 Most tissue-resident macrophages (Mphis) are believed to be d

114 How tissue-resident macrophages (TRM) impact adaptive immune

115 in mouse EMPs results in clonal expansion of tissue-resident macrophages and a severe late-onset neur

116 Collectively, these results indicate that tissue-resident macrophages and circulating monocytes sh

117 ese data correspond with previous studies on tissue-resident macrophages and raise important question

118 poptotic cells impaired the proliferation of tissue-resident macrophages and the induction of anti-in

119 tionship of this macrophage subset to CD169+ tissue-resident macrophages and their contribution to sh

120 Tissue-resident macrophages are a heterogeneous populati

121 ophages do not fit the current paradigm that tissue-resident macrophages are derived from embryonic p

122 Some tissue-resident macrophages are derived from yolk sac er

123 The first is the realization that most tissue-resident macrophages are established prenatally a

124 Tissue-resident macrophages are heterogeneous as a conse

125 Tissue-resident macrophages are highly heterogeneous in

126 provide evidence that M2-like TIM-4hiCD169+ tissue-resident macrophages are immunoregulatory and pro

127 Inflammatory monocytes and tissue-resident macrophages are key regulators of tissue

128 ng central nervous system (CNS), but how the tissue-resident macrophages are maintained throughout th

129 We propose that tissue-resident macrophages are metabolically poised in

130 After acute inflammation or infection, tissue-resident macrophages are replaced by inflammatory

131 ese results identify the fetal precursors of tissue-resident macrophages as a potential cell-of-origi

132 e identified both inflammatory monocytes and tissue-resident macrophages as sources of TAMs.

133 ising a long-held view that monocytes become tissue-resident macrophages by default.

134 This demonstrates that tissue-resident macrophages can also develop from fetal

135 Tissue-resident macrophages can develop from circulating

136 In agreement with these findings, adipose-tissue-resident macrophages did not express TH.

137 In ischemic myocardium, we observed that tissue-resident macrophages died locally, whereas some a

138 Tissue-resident macrophages display varying phenotypic a

139 Adult bone marrow monocytes can give rise to tissue-resident macrophages during infection or inflamma

140 ophage-specific enhancers and establish that tissue-resident macrophages have distinct enhancer lands

141 In addition, tissue-resident macrophages have many tissue-specific fu

142 Moreover, tissue-resident macrophages have the ability to self-ren

143 rve both embryo-derived and monocyte-derived tissue-resident macrophages in a G1-like phase at freque

144 function in health and disease, the role of tissue-resident macrophages in human immunodeficiency vi

145 show in mice that the vast majority of adult tissue-resident macrophages in liver (Kupffer cells), br

146 and explaining the observed accumulation of tissue-resident macrophages in some HIV-infected patient

147 bout the relative importance of monocyte and tissue-resident macrophages in the development of lung f

148 Organ-specific functions of tissue-resident macrophages in the steady-state heart ar

149 lating HIV-1 in primary cells, we used human tissue-resident macrophages isolated from tonsil as a tr

150 however, it emerged that, in several organs, tissue-resident macrophages may self-maintain through lo

151 Dysfunction of tissue-resident macrophages might contribute to the syst

152 The tissue-resident macrophages of barrier organs constitute

153 Tissue-resident macrophages support embryonic developmen

154 Here we identified a population of M2-like tissue-resident macrophages that express high levels of

155 Moreover, CD169+ tissue-resident macrophages were resistant to oxidative

156 osis of apoptotic thymocytes was enhanced in tissue-resident macrophages where this process resulted

157 oted the development of mature monocytes and tissue-resident macrophages whereas GM-CSF did not.

158 Microglia and other tissue-resident macrophages within the central nervous s

159 Microglia (tissue-resident macrophages) represent the main cell typ

160 RON signaling in tissue-resident macrophages, both ligand dependent and i

161 eterogeneous group of leukocytes composed of tissue-resident macrophages, dendritic cells, and monocy

162 ed that this population is a major subset of tissue-resident macrophages, homes to draining LNs follo

163 flammatory macrophages can adopt features of tissue-resident macrophages, or what mechanisms might me

164 Expressed on tissue-resident macrophages, the receptor tyrosine kinas

165 Ron receptor tyrosine kinase is expressed on tissue-resident macrophages, where it limits inflammator

166 eneric processes including interactions with tissue-resident macrophages.

167 infected in vivo are memory CD4 T cells and tissue-resident macrophages.

168 now considered to be a specialized subset of tissue-resident macrophages.

169 works controlling chromatin specification in tissue-resident macrophages.

170 studies also demonstrate distinct origins of tissue-resident macrophages.

171 rs provide new insight into efferocytosis by tissue-resident macrophages.

172 In tissues, resident macrophages exhibit distinct phenotype

173 on of the urinary tract that is dependent on tissue-resident mast cells (MCs).

174 , we report that this influx depends on both tissue-resident mast cells and macrophages.

175 antigens, or allergens, binds and sensitizes tissue-resident mast cells expressing the high-affinity

176 Thus, tissue-resident MCs could translate external chemical ch

177 The generation of tissue resident memory (TRM) cells at the body surfaces

178 Here, we show in mice that tissue resident memory CD8 T cells (T(RM) cells), non-re

179 fective strategy to enhance establishment of tissue resident memory CD8 T cells within mucosal tissue

180 was enriched in regulatory T cells (Tregs), tissue resident memory CD8(+) T cells (TRMs), resident n

181 cells in the insulitic lesions to display a tissue resident memory T cell (TRM) (CD8(+)CD69(+)CD103(

182 gammadelta T cells with a tissue resident memory T cell phenotype (CD69(+)CD103(+)

183 tion and find that the majority of cells are tissue resident memory T cells with high levels of CD69

184 These data extend the range of tissue resident memory to SLO.

185 GFAP-specific CD8 TCR-transgenic (BG1) mice, tissue resident memory-like CD8 T cells spontaneously in

186 udies have identified a third subset, called tissue-resident memory (TRM ) cells, based on its migrat

187 Tissue-resident memory (Trm) CD8(+) T cells are function

188 nd that chronic infection drove MNV-specific tissue-resident memory (Trm) CD8(+) T cells to a differe

189 o central memory, effector memory (TEM), and tissue-resident memory (TRM) subsets, which cooperate to

190 We also investigated tissue-resident memory CD8 T cells (TRM), which protect

191 Tissue-resident memory CD8 T cells are a unique subset o

192 ms and signals that control the formation of tissue-resident memory CD8 T cells are poorly understood

193 Tissue-resident memory CD8(+) T (TRM) cells are found at

194 Tissue-resident memory CD8(+) T cells (TRM) constitute a

195 of effector memory CD8(+) T cells (TEM) and tissue-resident memory CD8(+) T cells (TRM), but not of

196 Because tissue-resident memory CD8(+) T cells share this profile

197 d form, induced circulating and intravaginal-tissue-resident memory CD8(+) T cells that were able to

198 Given the discontinuous nature of tissue-resident memory CD8(+) T cells, we hypothesized t

199 Tissue-resident memory CD8+ T (CD8 TRM) cells are an ess

200 showed enrichment for transcripts linked to tissue-resident memory cells (TRM cells), such as CD103,

201 tral memory cells in the lymphoid organs and tissue-resident memory cells in the lung.

202 tially effector-like memory cells, including tissue-resident memory cells.

203 bionts of immunized mice, demonstrating that tissue-resident memory is not required for vaccine-induc

204 ts; ontogeny and defining characteristics of tissue-resident memory lymphocytes; and origins of the r

205 er CD8(+) T cells commit to recirculating or tissue-resident memory populations.

206 These tissue-resident memory T (T(RM)) cells are identified by

207 CD8 tissue-resident memory T (T(RM)) cells provide efficient

208 lta T cells, and CD8alphaalpha(+) IELs), and tissue-resident memory T (T(RM)) cells.

209 Tissue-resident memory T (Trm) cells constitute a recent

210 These nonrecirculating tissue-resident memory T (TRM) cells develop within peri

211 Tissue-resident memory T (TRM) cells have been ascribed

212 Tissue-resident memory T (Trm) cells patrol barrier tiss

213 Tissue-resident memory T (TRM) cells persist indefinitel

214 However, recent evidence indicates that tissue-resident memory T (TRM) cells play an important r

215 Tissue-resident memory T (Trm) cells provide enhanced pr

216 Tissue-resident memory T (Trm) cells represent a populat

217 Tissue-resident memory T (TRM) cells serve as vanguards

218 There is emerging evidence that local tissue-resident memory T (TRM) cells that accumulate in

219 on of Ag-specific IFN-gamma-producing CD8(+) tissue-resident memory T (TRM) cells.

220 et of memory T cells that resides in tissues-tissue-resident memory T (TRM) cells.

221 epidermis and form populations of long-lived tissue-resident memory T (TRM) cells.

222 ions, highlight a relatively new player, the tissue-resident memory T cell (TRM), and emphasize the p

223 s as a platform for the induction of genital-tissue-resident memory T cell responses and the control

224 CD8(+)/CD103(+) tissue-resident memory T cells (TRM cells) accumulate in

225 inct populations of pathogen-specific CD8(+) tissue-resident memory T cells (TRM cells) in the lamina

226 Langerhans cells (LCs) and CD8(+) tissue-resident memory T cells (TRM cells) require activ

227 issues is mediated by a long-lived subset of tissue-resident memory T cells (Trm cells).

228 estines, liver, and skin as non-circulating, tissue-resident memory T cells (Trm cells).

229 These tissue-resident memory T cells (TRM) are preferentially

230 Tissue-resident memory T cells (TRM) have been shown to

231 Tissue-resident memory T cells (TRM) persist at sites of

232 A small number of tissue-resident memory T cells (Trm) provide potent prot

233 iral immunity studies identifying protective tissue-resident memory T cells (Trm) suggest an alternat

234 ells migrate to diverse sites and persist as tissue-resident memory T cells (TRM), which mediate rapi

235 d and non-lymphoid tissues and gives rise to tissue-resident memory T cells (TRM).

236 These include recently identified tissue-resident memory T cells (TRM).

237 In adult mice, lung-localized, tissue-resident memory T cells (TRMs) mediate optimal pr

238 and phenotypic signatures characteristic of tissue-resident memory T cells and frequently express PD

239 inhibitor tofacitinib effectively suppresses tissue-resident memory T cells and inhibits core vasculi

240 r and colleagues find that allergen-specific tissue-resident memory T cells are maintained by IL-2 an

241 indings emphasize the role of CD8(+)CD103(+) tissue-resident memory T cells in promoting intratumoral

242 Together, these data suggest that skin tissue-resident memory T cells persist within a tightly

243 Tissue-resident memory T cells provide local immune prot

244 e knockouts, we uncover a multi-organ web of tissue-resident memory T cells that functionally adapt t

245 e results have implications for manipulating tissue-resident memory T cells through vaccination and o

246 F-beta promotes the development of pulmonary tissue-resident memory T cells via a signaling pathway t

247 onal T cells, including the newly identified tissue-resident memory T cells, and whether such T cells

248 ly recruit CD8(+) T cells and retain them as tissue-resident memory T cells, independently of local i

249 plastic intima, and minimized CD4(+)CD103(+) tissue-resident memory T cells.

250 promotes the accumulation of salivary gland tissue-resident memory T cells.

251 pocytes in adults are typically derived from tissue resident mesenchymal progenitors.

252 Thus, adult tissue-resident MFs established from hematopoietic stem

253 creases in the richness and diversity of the tissue-resident microbiota within the intestine, results

254 ory reaction that includes activation of the tissue-resident microglia and recruitment of blood-deriv

255 Immunity, Kumamoto et al. (2016) identify a tissue-resident mononuclear phagocyte population that pr

256 same lobe was used to identify extravascular tissue-resident mononuclear phagocytes and exclude cells

257 These cells comprise distinct populations of tissue-resident Mphis (resMphis), Ly6c(hi) monocyte-deri

258 scriptome identical to embryonically derived tissue-resident Mphis.

259 n of inflammatory functions of recruited and tissue-resident Mvarphi.

260 The intestinal microbiota and tissue-resident myeloid cells promote immune responses t

261 Microglia belong to a lineage of adult tissue-resident myeloid cells that develop during organo

262 ococcus pneumoniae in this organ mediated by tissue-resident MZ and RP macrophages and a protective r

263 decidual leukocytes detected an elevation in tissue resident neutrophils in the second trimester.

264 described two populations of liver NK cells, tissue-resident NK (trNK) cells and those resembling spl

265 Interestingly, NFIL3-independent SMG tissue-resident NK (trNK) cells are developmentally dist

266 present study, we tested the contribution of tissue-resident NK (trNK) cells to tissue homeostasis by

267 The recent discoveries of tissue-resident NK cell developmental intermediates, non

268 Thus, these studies indicate that tissue-resident NK cells are evolutionarily conserved in

269 he phenotype and function of circulatory and tissue-resident NK cells in a unique cohort of SIV-contr

270 ent studies in mice indicate the presence of tissue-resident NK cells in certain organs, such as the

271 Among the variety of tissue-resident NK-like populations recently distinguish

272 ted in the selective accumulation of adipose-tissue-resident NKT cells.

273 -resident Treg cells in comparison to normal tissue-resident ones.

274 onal plasticity to differentiate into either tissue-resident or inflammatory Mphis, depending on micr

275 Under homeostatic conditions, non-lymphoid tissue-resident pDC play a critical role in the regulati

276 u has revealed a division of labor among the tissue resident phagocytes that sample them.

277 The full spectrum of functions executed by tissue-resident phagocytes in response to homeostatic ap

278 also expressed CXCR3/CCR5/LFA-1 trafficking/tissue-resident phenotypes and consistently trafficked t

279 ls had effector memory (CD44(+)CD62L(-)) and tissue-resident phenotypes and expressed markers associa

280 at have been described to contribute to this tissue-resident population in other organs, including in

281 trated that human dermal CD14(+) cells are a tissue-resident population of monocyte-derived macrophag

282 A tissue-resident population of natural killer cells (NK c

283 The adipose iNKT cells were a tissue-resident population that induced an anti-inflamma

284 This tissue resident predominance of CEACAM1-S expression was

285 Here, we have identified tissue-resident progenitor cells that mediate regenerati

286 view of endogenous pericytes as multipotent tissue-resident progenitors and suggest that the plastic

287 s mesenchymal stem cells (MSCs), multipotent tissue-resident progenitors with great potential for reg

288 nate lymphoid cells (ILC2s), and a subset of tissue-resident regulatory CD4(+) T cells can express AR

289 onal properties, we found that only lymphoid-tissue resident Rorc(fm+) ILCs can suppress tumor growth

290 Ly6C(-) macrophages acted as tissue-resident sentinels that attracted circulating neu

291 ne cells and the differentiation capacity of tissue-resident stem cells (SCs).

292 Here, we consider the varied roles of these tissue-resident stroma-associated cells, synthesize rece

293 Increasing evidence suggests that tissue-resident stromal cells not only provide positiona

294 ble protection to malaria through long-lived tissue-resident T cells and that administration of highe

295 ffector memory T cells and CD103(high)CD8(+) tissue-resident T cells in TG of latently infected HLA-A

296 ount for a major fraction of circulating and tissue-resident T cells.

297 henotype, function and memory of nonlymphoid tissue-resident Treg cells.

298 Compared to tissue-resident tumor-associated macrophages, these newl

299 Here, we show that tissue-resident type 1 innate lymphoid cells (ILC1) serv

300 Swift production of interleukin (IL)-12 by tissue-resident XCR1(+) conventional dendritic cells (cD