1 rategies span from killing (new antibiotics) to disarming (antivirulence) the pathogen.

2             Thus, EBV coopts the lipid rafts to disarm both the signaling and antigen-processing func

3 to deregulate lymphocyte receptor expression to disarm host immune control.

4 el mechanism to deregulate T cell activation to disarm host immune surveillance.

5 l a previously unrecognized capacity of SIgA to "disarm" microbial pathogens on mucosal surfaces and

6 most versatile evolutionary innovators, able to disarm multiple chemical plant defenses.

7 ionally, developing new treatments that seek to disarm pathogens without killing them, or that modula

8 athogenesis, primarily by enabling the virus to disarm the host cell type IFN defense system.

9                                 This ability to disarm the IFN-beta response offers an explanation fo

10 entry: to induce expression of VZV genes and to disarm the IFN-dependent antiviral defense through a

11 crobe targets MAP kinases in dendritic cells to disarm the immune response.

12             Thus, gp120 may be used by HIV-1 to disarm the monocyte response to inflammatory stimulat

13 requires the appropriate sequence of touches to disarm the system.

14 RF3 protein levels is important with respect to disarming the IFN response during productive infectio