ライフサイエンスコーパス: tobacco

1 sture gradients, was cloned and expressed in tobacco.

2 is understood about the effect of smokeless tobacco.

3 ay have more profound effects than smokeless tobacco.

4 aine, cannabis, methamphetamine, alcohol, or tobacco.

5 elatively young users who did not also smoke tobacco.

6 he 2016 Society for Research on Nicotine and Tobacco 22nd Annual Meeting abstracts, the 2016 Society

7 idual risk factors assessed were as follows: tobacco 92.9%; blood pressure 51.2%; body mass index 33.

8 atric disorders and other vulnerabilities to tobacco addiction respond to cigarettes with reduced nic

9 PsycINFO, MEDLINE, PubMed, and the Cochrane Tobacco Addiction Specialized Register were searched for

10 Varenicline, a pharmacotherapy for tobacco addiction, reduces alcohol consumption in humans

11 trong association between mood disorders and tobacco addiction.

12 in populations that are highly vulnerable to tobacco addiction.

13 squamous cell carcinoma with risk factors of tobacco and alcohol is more frequent among blacks.

14 to have a significantly higher prevalence of tobacco and alcohol use than did high socioeconomic grou

15 ension, diabetes mellitus, and dyslipidemia; tobacco and alcohol use; and APOE epsilon4 on the rates

16 k time for initiation of substance use, with tobacco and alcohol usually preceding the use of illicit

17 weetening and flavouring agents in the food, tobacco and beer industries.

18 9 to 13.0x the median amount spent on smoked tobacco and between 8% and 52% of nonsubsistence income.

19 acebo, and similar effects were observed for tobacco and cocaine craving.

20 nthropometric Traits (GIANT) Consortium, the Tobacco and Genetics (TAG) Consortium, and the Social Sc

21 y level genomewide association data from the Tobacco And Genetics Consortium and Psychiatric Genomics

22 ibe the association between use of smokeless tobacco and head and neck cancer in 11 US case-control s

23 cember 2014) of the Population Assessment of Tobacco and Health (PATH) Study, a large, nationally rep

24 associations between each type of smokeless tobacco and HNC were observed among ever cigarette smoke

25 e selectable marker gene as demonstrated for tobacco and maize.

26 of HaCHI gene-specific siRNAs in HaCHI-RNAi tobacco and tomato lines.

27 and was used to generate several HaCHI-RNAi tobacco and tomato plants.

28 The flavour notes of 'sour', 'tobacco' and 'sweet' were mostly associated with the cof

29 ledonous plants, such as Nicotinana tabacum (tobacco) and Solanum lycopersicum (tomato), greater than

30 dy, 28% of U.S. adults were current users of tobacco, and 9% of youths had used tobacco in the previo

31 Only methamphetamine/tobacco- and tobacco-exposed girls showed persistently l

32 cigarettes (0.4, 2.3, 5.2, and 15.8 mg/g of tobacco) at 3 academic sites included 169 daily smokers

33 animal rRNA species, and is toxic to wheat, tobacco, bacterial and yeast cells, but not to Z. tritic

34 ouches, other smokeless tobacco, dissolvable tobacco, bidis, and kreteks.

35 erry localized to the cytosol and nucleus of tobacco bright yellow-2 cells, but colocalized with mito

36 ers silenced in NaJAZi are more resistant to tobacco budworm attack, a florivore.

37 agous parasitoid of the larval stages of the tobacco budworm, Heliothis virescens (Lepidoptera, Noctu

38 have been explored to produce monoterpene in tobacco but only trace amounts of monoterpenes have been

39 ia is the world's second largest consumer of tobacco, but tobacco cessation remains uncommon due, at

40 growth, the Te-Rubisco was transformed into tobacco by chloroplast transformation.

41 pinene/beta-pinene or myrcene, in transgenic tobacco by elevating geranyl diphosphate synthase (GPS)

42 Synchronized tobacco BY-2 cell cultures were used to study the role o

43 Inducing the expression of FUS3 in tobacco BY2 cells also increased TAG accumulation, and c

44 Like alcohol and tobacco, cannabis can have serious adverse effects on he

45 In addition, overexpression of MpGPS.SSU in tobacco caused early flowering phenotype and increased s

46 gan with the need to address the epidemic of tobacco-caused disease, an imperative for action documen

47 ts over the past several decades directed at tobacco cessation and control.

48 romotion is critical, particularly regarding tobacco cessation and dental care.

49 cy, practice, and research action to promote tobacco cessation and to improve the health outcomes in

50 Tobacco cessation drugs increased the odds of continued

51 Addition of tobacco cessation medication onto national and state ess

52 Tobacco cessation medications are unavailable in the Ker

53 ld's second largest consumer of tobacco, but tobacco cessation remains uncommon due, at least in part

54 on of I-SceI, and showed experimentally that tobacco chloroplast DNAs insert into nuclear genomes thr

55 xA effector, which we show translocates into tobacco chloroplasts.

56 xamples of how environmental exposures, like tobacco, chronic stress, or dietary factors, induce an a

57 meeting study criteria, including no current tobacco cigarette smoking and no known health problems o

58 e-cigarette users and 19 self-identified non-tobacco cigarette, non-e-cigarette user control particip

59 (ECs) are a much less harmful alternative to tobacco cigarettes, there is concern as to whether long-

60 s, and provide a close simulation of smoking tobacco cigarettes.

61 Transnational tobacco companies have sought to undermine tobacco tax p

62 The interest of tobacco companies in genetic information as well as the

63 the development and progression of HNC, how tobacco components activate STAT3 is unclear.

64 uals aged 40 years or older, with cumulative tobacco consumption of ten pack-years or higher, and wit

65 This historic shift reflects the increase in tobacco consumption worldwide, spurring public health ef

66 The WHO Framework Convention on Tobacco Control (WHO FCTC) has mobilised efforts among 1

67 between highest-level implementation of key tobacco control demand-reduction measures of the WHO FCT

68 whether implementation of WHO's recommended tobacco control policies (MPOWER) was of benefit to peri

69 o, for which the WHO Framework Convention on Tobacco Control provides evidence-based policies for red

70 acco Control, and Director of the Center for Tobacco Control Research and Education at the University

71 ion of evidence-based policies (particularly tobacco control) and integrated health systems strategie

72 Truth Initiative Distinguished Professor of Tobacco Control, and Director of the Center for Tobacco

73 lobal health law-the Framework Convention on Tobacco Control, International Health Regulations (2005)

74 Compared with males who never used any tobacco daily, pure ever tobacco smokers, pure ever snus

75 multistakeholder committee with expertise in tobacco dependence treatment and/or LDCT screening.

76 vity to which may influence vulnerability to tobacco dependence, yet mechanisms of nicotine avoidance

77 bacco, hookah, snus pouches, other smokeless tobacco, dissolvable tobacco, bidis, and kreteks.

78 The most challenging to purify, tobacco epi-aristolochene synthase, was purified in only

79 forts among 180 parties to combat the global tobacco epidemic.

80 sion system based on a light-inducible split tobacco etch virus protease.

81 based on the exogenous enzymatic activity of Tobacco Etch Virus Protease.

82 neonates were evaluated (36 methamphetamine/tobacco exposed, 32 tobacco exposed, and 71 unexposed co

83 ated (36 methamphetamine/tobacco exposed, 32 tobacco exposed, and 71 unexposed controls).

84 Only methamphetamine/tobacco- and tobacco-exposed girls showed persistently lower FA in an

85 Tobacco-exposed infants showed persistently lower axial

86 ion of factors that increase PAI-1 including tobacco exposure and obesity.

87 ce of pleiotropic effects and accounting for tobacco exposure did not alter the association (OR of sc

88 While all forms of tobacco exposure have negative health effects, the signi

89 Parent and child's passive tobacco exposure was assessed using interview-assisted q

90 Exposures: Prenatal methamphetamine and/or tobacco exposure.

91 Understanding the relationship between tobacco exposures and specific mutations may yield etiol

92 on solid or liquid media, the transplastomic tobacco expressing nfsI were significantly more tolerant

93 cigarette smoke extract (CSE) and smokeless tobacco extract (STE) on cell survival and motility of p

94 d basal and induced STAT3 phosphorylation by tobacco extract or interleukin 6.

95 A new tobacco filler Standard Reference Material (SRM) has bee

96 Unlike tobacco, for which the WHO Framework Convention on Tobac

97 A 72-year-old man with a 40-pack-year tobacco history developed a cough and decreased exercise

98 al cigars, cigarillos, filtered cigars, pipe tobacco, hookah, snus pouches, other smokeless tobacco,

99 ed for activation of moricin promoter in the tobacco hornworm, Manduca sexta, and a 140-bp region in

100 , diamondback moth, Plutella xylostella, and tobacco hornworm, Manduca sexta, as well as the spotted

101 Strikingly, overexpression of Hsp31 in tobacco imparts robust stress tolerance against diverse

102 users of tobacco, and 9% of youths had used tobacco in the previous 30 days.

103 evidence of higher female susceptibility to tobacco-induced cardiovascular disease and underline the

104 included in two systematic reviews examining tobacco industry influence on taxation and marketing of

105 this allelic variation in people who smoked tobacco (interaction P value=8.7x10(-5)).

106 Nicotine, the main addictive substance in tobacco, is known to play a role in the development and/

107 y in Arabidopsis and transiently in rice and tobacco, it localises to the Golgi.

108 The real sample prepared using cigarette tobacco leaves and analyzed using the fabricated sensor

109 latory mechanisms in control and AHO-treated tobacco leaves.

110 r study suggests a novel role of exosomes in tobacco-mediated HIV-1 pathogenesis.

111 hin 2min; and cross-selectivity studies with tobacco mosaic virus (TMV) showed an excellent specifici

112 Tobacco mosaic virus is used as a probe to measure surfa

113 Furthermore, tobacco mosaic virus was recently shown to replicate in

114 RNA by transiently delivering sgRNAs using a Tobacco mosaic virus-derived vector (TRBO) designed with

115 viruses: potato virus Y, potato virus X, and tobacco mosaic virus.

116 ome', might also imply cross-reactivity with tobacco, natural latex and plant-food-derived alcoholic

117 Satellite tobacco necrosis virus (STNV) is one of the smallest vir

118 model 1) as well as those who had never used tobacco (never-users) (model 2).

119 rect defenses against herbivores of the wild tobacco Nicotiana attenuata; whether they also function

120 to trigger cell death when overexpressed in tobacco (Nicotiana benthamiana) leaves and does so in a

121 gnin and reduces recalcitrance in transgenic tobacco (Nicotiana benthamiana).

122 this, we have developed nfsI transplastomic tobacco (Nicotiana tabacum L.) to reduce pollen-borne tr

123 stinct phases during cell plate expansion in tobacco (Nicotiana tabacum) 'Bright Yellow-2' cells: mas

124 terminus of KAT1 and cocrystallized it with tobacco (Nicotiana tabacum) 14-3-3 proteins to describe

125 this strategy to the Clp protease complex of tobacco (Nicotiana tabacum) and identified a set of chlo

126 Plastid transformation is routine in tobacco (Nicotiana tabacum) but 100-fold less frequent i

127 mitrella patens for transient expression and tobacco (Nicotiana tabacum) BY2 cells for stable transfo

128 on, localization, and functional analysis of tobacco (Nicotiana tabacum) EXO70 isoforms.

129 Here, by generating transplastomic tobacco (Nicotiana tabacum) mutants with point mutations

130 id infestation was explored using transgenic tobacco (Nicotiana tabacum) plants that have either high

131 Here, we show that tobacco (Nicotiana tabacum) trichomes contain a specific

132 validated in tomato (Solanum lycopersicum), tobacco (Nicotiana tabacum), Medicago truncatula, wheat

133 ing are highlighted, with a special focus on tobacco (Nicotiana tabacum).

134 ory-induced transcriptomic responses in wild tobacco, Nicotiana attenuata, using a phylotranscriptomi

135 Over-expression and knocked down transgenic tobacco plant analysis confirmed the role of XET gene in

136 our laboratory we use either Arabidopsis or tobacco plant seedlings): a Petri dish containing two co

137 biochemical route, the shikimate pathway of Tobacco plant was engineered to accumulate protocatechua

138 elated genes also allowed the engineering of tobacco plants and cell cultures to produce a palette of

139 research reveals the genetic basis by which tobacco plants control the temporal emission of volatile

140 Furthermore, betalain-producing tobacco plants exhibited significantly increased resista

141 ment of monoterpene production in transgenic tobacco plants.

142 the bacterial nitroreductase gene, nfsI, in tobacco plastids conferred the ability to detoxify TNT.

143 aracterized as the site of exocytosis in the tobacco pollen tube, while NtEXO70B1 surprisingly coloca

144 s were current users of at least one type of tobacco product in 2013 and 2014, although the prevalenc

145 A total of 8.9% of youths had used a tobacco product in the previous 30 days; 1.6% of youths

146 e format on prevalence estimates of emerging tobacco products among youth and suggest that estimates

147 Noncigarette tobacco products are evolving rapidly, with increasing p

148 for monitoring the epidemiology of emerging tobacco products in the United States, and survey scienc

149 Nicotine is the major stimulant in tobacco products including e-cigarettes.

150 Many of the underlying effects of tobacco products on periodontal tissues may be due to di

151 present prevalence estimates for 12 types of tobacco products, using data from 45,971 adult and youth

152 ithin-person changes over time in the use of tobacco products.

153 sers, adults and youths alike, used multiple tobacco products; cigarettes plus e-cigarettes was the m

154 a, San Francisco Comprehensive Cancer Center Tobacco Program.

155 d by the application of the most widely used tobacco rattle virus (TRV)-based vectors, using comprehe

156 , and the 2016 National Institutes of Health Tobacco Regulatory Science Program Conference were searc

157 edure was developed and validated to measure tobacco-related alkaloids, carcinogens, and their metabo

158 To compare exposure to nicotine, tobacco-related carcinogens, and toxins among smokers of

159 lence, with anticipated future reductions in tobacco-related morbidity and mortality.

160 Assessment of exposure to tobacco-related toxicants and carcinogens at the populat

161 methods for the simultaneous measurement of tobacco-related toxicants and carcinogens in wastewater

162 ular evidence for the mechanisms involved in tobacco SAR, which are likely to be present in other pla

163 Overexpression of PsnSHN2 in tobacco significantly altered the expression of both tra

164 prevalence have reported that environmental tobacco smoke (ETS) exposure of non-smokers is associate

165 Exposure to environmental tobacco smoke (ETS) is an important modifiable risk fact

166 The association between tobacco smoke and acute myeloid leukemia (AML) is well e

167 titis, family asthma, early-life exposure to tobacco smoke and rhinovirus aetiology were early-life r

168 The association between tobacco smoke exposure and childhood rhinitis has not be

169 Prenatal tobacco smoke exposure and higher maternal stress and de

170 Tobacco smoke exposure has been associated with risk of

171 results suggest that prenatal and early-life tobacco smoke exposure increase the frequency of somatic

172 s to explore whether prenatal and early-life tobacco smoke exposure influences the formation of leuke

173 h awareness about the detrimental effects of tobacco smoke exposure on children's respiratory health.

174 portive evidence for positive association of tobacco smoke exposure with rhinitis, while the effect i

175 ions per case was positively associated with tobacco smoke exposure, in particular for maternal ever-

176 Tobacco smoke exposure, lung function at 6 weeks, infant

177 abolites that are associated with secondhand tobacco smoke exposure.

178 tabolite of nicotine that is proportional to tobacco smoke exposure.

179 epithelial cultures (HBECs) to air or whole tobacco smoke from cigarettes vs. LCs.

180 Exposure to tobacco smoke has been associated with harmful effects o

181 nearly half of the patients were exposed to tobacco smoke in early life.

182 BACKGROUND & AIMS: The role of tobacco smoke in the etiology of inflammatory bowel dise

183 ed with non-smokers and that constituents of tobacco smoke other than nicotine affect inflammatory pr

184 allergic sensitization and an environmental tobacco smoke pathway.

185 leep surfaces (including sofas), exposure to tobacco smoke, and prematurity; protective factors inclu

186 at illness onset, exposure to environmental tobacco smoke, bacterial colonization, and breastfeeding

187 S. population are primarily from exposure to tobacco smoke, where smokers have blood levels on averag

188 on but fails to prevent or reverse the major tobacco smoke-induced oxidative lung injury.

189 inogens have also been implicated, including tobacco smoke.

190 ed for exposures to secondhand and thirdhand tobacco smoke.

191 Sixteen tobacco smokers had one set of [(11)C]FLB457 PET scans o

192 Tobacco smokers titrate their nicotine intake to avoid i

193 th loss, with smaller alterations in current tobacco smokers, increased production of antioxidants in

194 who never used any tobacco daily, pure ever tobacco smokers, pure ever snus users, and combined user

195 cine treatment reduced [(11)C]FLB457 BPND in tobacco smokers, suggesting an increase in dopaminergic

196 risk ratio [RR] 1.41 [95% CI 1.26-1.57]) and tobacco smoking (1.46 [1.30-1.65]) than in HIV-negative

197 use (RR 1.36 [95% CI 1.10-1.69]; p=0.0050), tobacco smoking (1.90 [1.38-2.62]; p<0.0001), and smokel

198 st associations, with the exception of daily tobacco smoking (adjusted OR 1.74, 95% CI 1.08-2.81), an

199 significantly increase risk for SP included tobacco smoking (RR, 2.47; 95% CI, 2.12-2.87), alcohol i

200 We studied the association between tobacco smoking and epigenome-wide methylation in non-tu

201 and 95% confidence intervals, adjusting for tobacco smoking and other potential confounders.

202 Tobacco smoking and smoke exposure during pregnancy and

203 HIV-infected men ages 40-60, independent of tobacco smoking and traditional risk factors.

204 in Nha Trang, Vietnam, to evaluate paternal tobacco smoking as a risk factor for infectious and non-

205 nicotine consumption and prevent relapse to tobacco smoking by modulating glutamate transmission.

206 hanism contributing to its ability to aid in tobacco smoking cessation.

207 Tobacco smoking enhances peri-implant soft tissue inflam

208 ioural risk factors were unhealthy diets and tobacco smoking in 2015.

209 ne, a noradrenergic alpha2a agonist, reduced tobacco smoking in a 4-week trial and in animal models h

210 Paternal tobacco smoking independently increased the risk of LRTI

211 ented in countries where ETS due to maternal tobacco smoking is negligible.

212 Our results suggest that tobacco smoking may alter the genetic susceptibility to

213 ere we report on the sex-specific effects of tobacco smoking on transcriptomic and epigenetic feature

214 ome variable (the percentage point change in tobacco smoking prevalence between 2005 and 2015).

215 Tobacco smoking remains an important public health issue

216 To assess how tobacco smoking status affects baseline dopamine D2/D3 (

217 r, prevalence of psychiatric comorbidity and tobacco smoking was alarmingly high in severe patients w

218 positive women was 1.3% (95% CI 0.8-1.9) for tobacco smoking, 2.1% (1.1-3.4) for smokeless tobacco us

219 ositive men was 24.4% (95% CI 21.1-27.8) for tobacco smoking, 3.4% (1.8-5.6) for smokeless tobacco us

220 had higher body mass index, a higher rate of tobacco smoking, and lower lung function.

221 obiome were observed with poor oral hygiene, tobacco smoking, and oral cancer.

222 Tobacco smoking, driven by the addictive properties of n

223 ars (wave 10), mental health problems, daily tobacco smoking, illicit drug use, and dependence were a

224 her odds of lung cancer after adjustment for tobacco smoking, serum cotinine levels, educational atta

225 , and overall relative prevalence ratios for tobacco smoking, smokeless tobacco use, and any tobacco

226 between ages 40-60 after adjusting for age, tobacco smoking, viral load, and traditional risk factor

227 ion treatments and reduce the harm caused by tobacco smoking.

228 association with risk for IBD is modified by tobacco smoking.

229 ically involved in the reinforcing effect of tobacco smoking.

230 x disease, Barrett's esophagus, obesity, and tobacco smoking.

231 A 62-year-old man with a 45 pack per year tobacco-smoking history presented with painless gross he

232 rmation as well as the success of a few wild tobacco species as experimental model organisms have res

233 nd were analyzed for biomarkers of nicotine, tobacco-specific N-nitrosamines (TSNAs), and volatile or

234 Tobacco-specific nitrosamines 4-(methylnitrosamino)-1-(3

235 e first SRM for reduced nicotine and reduced tobacco-specific nitrosamines with certified values for

236 utilized data from the 2014 New Jersey Youth Tobacco Survey (n = 3,909) to compare estimates of the p

237 rcise [E], weight [W], alimentation [A], and tobacco [T]) score (FBS), are also available.

238 h suggests that legislators should implement tobacco tax and price control measures that eliminate bu

239 l tobacco companies have sought to undermine tobacco tax policy by adopting pricing strategies that m

240 WER policies: smoke-free legislation (n=35), tobacco taxation (n=11), and smoking cessation services

241 not possible; all four studies on increasing tobacco taxation and one of two on offering disadvantage

242 On a population level, we recommend tobacco taxation, bans on trans fats, and compulsory red

243 exual minorities generally had higher use of tobacco than their counterparts.

244 We describe a system in tobacco that adds additional decorations to the basic an

245 after its development for Chlamydomonas and tobacco, the transformation of the chloroplast genome st

246 randed DNA breaks into the nuclear genome of tobacco through inducible expression of I-SceI, and show

247 major cause of morbidity and mortality, and tobacco usage, alcohol consumption, and poor oral hygien

248 (1.90 [1.38-2.62]; p<0.0001), and smokeless tobacco use (1.32 [1.03-1.69]; p=0.030) than in HIV-nega

249 persistent opioid use included preoperative tobacco use (adjusted odds ratio [aOR], 1.35; 95% CI, 1.

250 r point, 1.92; 95% CI, 1.55-2.37; P < .001), tobacco use (OR, 3.64; 95% CI, 1.36-9.73; P = .01), ence

251 higher prevalence in HIV-positive men of any tobacco use (risk ratio [RR] 1.41 [95% CI 1.26-1.57]) an

252 gher prevalence in HIV-positive women of any tobacco use (RR 1.36 [95% CI 1.10-1.69]; p=0.0050), toba

253 regional, and overall prevalence of current tobacco use (smoked, smokeless, and any tobacco use) amo

254 We assessed the prevalence of tobacco use among people living with HIV in LMICs.

255 er, very little is known about the extent of tobacco use among people living with HIV in low-income a

256 Tobacco use among people living with HIV results in exce

257 Previous studies on smokeless tobacco use and head and neck cancer (HNC) have found in

258 onally representative, longitudinal study of tobacco use and health in the United States.

259 tween 2003 and 2014 from 28 LMICs where both tobacco use and HIV test data were made publicly availab

260 They also had increased tobacco use and more comorbidities.

261 ncluded studies presented clear patterns for tobacco use and physical activity, heterogeneity between

262 Goal 3, setting a global target of reducing tobacco use and premature mortality from non-communicabl

263 archers should use caution when interpreting tobacco use data obtained from check-all-that-apply form

264 Tobacco use disorder is associated with dysregulated neu

265 ission and enhancing IC during the course of tobacco use disorder treatment.

266 tudy of Nicotine Dependence (ascertained for tobacco use disorder; n = 918 cases; 988 control subject

267 INTERPRETATION: The high prevalence of tobacco use in people living with HIV in LMICs mandates

268 vernments can create an environment in which tobacco use is reduced and citizens maintain good levels

269 The difference in smokeless tobacco use prevalence between HIV-positive and HIV-nega

270 acco smoking, smokeless tobacco use, and any tobacco use separately for males and females to study di

271 We found that tobacco use was associated with a lower risk of PD in ma

272 Marijuana and tobacco use were each independently associated with high

273 rent tobacco use (smoked, smokeless, and any tobacco use) among 6729 HIV-positive men from 27 LMICs (

274 ; lipid disorders, 12%-21%; diabetes, 4%-7%; tobacco use, 5%-16%; and obesity, 4%-9%).

275 ultiple measures of socioeconomic status and tobacco use, alcohol use, diet, and physical activity.

276 obacco smoking, 3.4% (1.8-5.6) for smokeless tobacco use, and 27.1% (22.8-31.7) for any tobacco use.

277 obacco smoking, 2.1% (1.1-3.4) for smokeless tobacco use, and 3.6% (95% CI 2.3-5.2) for any tobacco u

278 alence ratios for tobacco smoking, smokeless tobacco use, and any tobacco use separately for males an

279 ace/ethnicity, education, employment status, tobacco use, and scanner technology.

280 ng for sex, race, education, income, region, tobacco use, and select chronic medical conditions (0-1

281 ce/ethnicity, and income level), alcohol and tobacco use, diabetes mellitus, and past periodontal tre

282 tional individual level risk factors (mainly tobacco use, lipids, and elevated blood pressure) and so

283 rovides evidence-based policies for reducing tobacco use, no global policy exists for the control of

284 dence intervals for the associations between tobacco use, oral health indicators, and PD risk.

285 , more years on dialysis before waitlisting, tobacco use, panel-reactive antibody greater than 0, pub

286 hypertension, diabetes, hyperlipidemia, and tobacco use, risk differences comparing participants wit

287 oeconomic status and harmful use of alcohol, tobacco use, unhealthy diets, and physical inactivity wi

288 Reductions in tobacco use, uptake of preventive measures, adoption of

289 oth toxicants and carcinogens resulting from tobacco use.

290 bacco use, and 3.6% (95% CI 2.3-5.2) for any tobacco use.

291 male sex, white race, abdominal obesity, and tobacco use.

292 s tobacco use, and 27.1% (22.8-31.7) for any tobacco use.

293 increased age, prior radiation therapy, and tobacco use.

294 Approximately 40% of tobacco users, adults and youths alike, used multiple to

295 Use of multiple products was common among tobacco users.

296 The PCA extracted from the engineered Tobacco was further converted into ccMA using the engine

297 ustry influence on taxation and marketing of tobacco; we included 45 of 46 papers in the former categ

298 Tobacco withdrawal is associated with deficits in cognit

299 one variation and nicotine on suppression of tobacco withdrawal symptoms and smoking behavior.

300 completed repeated self-report measures (ie, tobacco withdrawal symptoms, smoking urge, and negative