ライフサイエンスコーパス: tobacco etch virus

1 to the heterologous nuclear NIa protein from tobacco etch virus.

2 IYV; a crinivirus), and HC-Pro proteinase of Tobacco etch virus (a potyvirus).

3 iple pathogens: tobacco vein mottling virus, tobacco etch virus, black shank fungus Phytophthora para

4 nd competes with structured RNA derived from tobacco etch virus for PAP binding.

5 te that the P1/HC-Pro polyprotein encoded by tobacco etch virus functions as a suppressor of PTGS.

6 for restriction of long-distance movement of tobacco etch virus in Arabidopsis thaliana without causi

7 n the inoculum, high, but not low, levels of tobacco etch virus inoculum resulted in escape of virus

8 ity; and 4) in contrast to cap-dependent and tobacco etch virus internal ribosome entry site interact

9 higher affinity than for either m(7)G cap or tobacco etch virus internal ribosome entry site, suggest

10 Using this system, the Tobacco Etch Virus protease (TEV-P), which strongly pref

11 t expression of the highly sequence-specific tobacco etch virus protease (TEVP) is harmless.

12 sions, we have used a novel method using the tobacco etch virus protease and confirm that Mgm1p is pr

13 taining a double FLAG epitope, followed by a tobacco etch virus protease cleavage site and calmodulin

14 We first show, by introduction of tobacco etch virus protease cleavage site in the middle

15 Engineering NCX1-Met(369) into a tobacco etch virus protease cleavage site revealed that

16 tilizing an N-terminal six-histidine tag and tobacco etch virus protease cleavage site upstream of th

17 nsertion of N-linked glycosylation sites and tobacco etch virus protease cleavage sites provides evid

18 By placing a cleavage site for the tobacco etch virus protease prior to the CTD, we have cr

19 ite-specific protein cleavage of YFP-TRF1 by tobacco etch virus protease resolves telomere aggregates

20 By examining the accessibility of tobacco etch virus protease sites and single-cysteine re

21 domain cleavage of caspase-8 by adapting the tobacco etch virus protease to create a system in which

22 sion system based on a light-inducible split tobacco etch virus protease.

23 based on the exogenous enzymatic activity of Tobacco Etch Virus Protease.

24 that is susceptible to inducible cleavage by tobacco etch virus protease.

25 of the fusion protein at a designed site by tobacco etch virus protease.

26 Using Protein C Epitope -Tobacco Etch Virus-Protein A Epitope (PTP)-tagged Tb11.0

27 The NH(2)-terminal tag is followed by a tobacco etch virus proteinase cleavage site to ensure th

28 alfa [correction of alfafa] mosaic virus, or tobacco etch virus resulted in necrotic lesions.

29 dsGFP inducer was partially inhibited by the tobacco etch virus silencing suppressor, P1/HC-Pro.

30 he addition of a structured RNA derived from tobacco etch virus suggests that translation initiation

31 eIFiso4F, and a structured RNA derived from tobacco etch virus (TEV RNA).

32 The tobacco etch virus (TEV) 5'-leader promotes cap-independ

33 The tobacco etch virus (TEV) 6 kDa protein associated with m

34 ence specificity, the 3C-like proteases from tobacco etch virus (TEV) and human rhinovirus are often

35 by which untagged PRMTs can be made using a tobacco etch virus (TEV) cleavage site at the N-terminal

36 nto the CI protein coding region of modified tobacco etch virus (TEV) genomes expressing either beta-

37 The 5' leader of tobacco etch virus (TEV) genomic RNA directs efficient t

38 The 5'-leader of tobacco etch virus (TEV) genomic RNA directs the efficie

39 with the use of a viral suppressor of PTGS, tobacco etch virus (TEV) helper component proteinase (HC

40 Restriction of long-distance movement of tobacco etch virus (TEV) in Arabidopsis ecotype Col-0 pl

41 uence specificity, the 3C-type protease from tobacco etch virus (TEV) is frequently used to remove af

42 This method uses tobacco etch virus (TEV) NIa protease that recognizes a

43 ed an expression cassette that relies on the tobacco etch virus (TEV) nuclear inclusion a (NIa) prote

44 pecificity as the PVY enzyme, but not by the tobacco etch virus (TEV) or the potato virus A (PVA) pro

45 ired for the viral infection in the Capsicum-Tobacco etch virus (TEV) pathosystem.

46 ewly generated TAPa tag we have replaced the tobacco etch virus (TEV) protease cleavage site with the

47 Tobacco etch virus (TEV) protease is a cysteine protease

48 We constructed mutant hCaRs with a unique tobacco etch virus (TEV) protease recognition site inser

49 ardiac troponin T (hcTnT) and an intervening tobacco etch virus (TEV) protease site that allows purif

50 e revealed the presence of a single putative tobacco etch virus (TEV) protease site within gD, while

51 engineered mammalian cells with an inducible tobacco etch virus (TEV) protease that cleaves TDP-43 co

52 Our approach uses a split-tobacco etch virus (TEV) protease under small-molecule c

53 f Arabidopsis thaliana for susceptibility to tobacco etch virus (TEV) revealed that each of 10 ecotyp

54 Two selectable tobacco etch virus (TEV) strains were developed for iden

55 lleles and VPg from two different strains of Tobacco etch virus (TEV) that differentially infected Ca

56 mRNA internal ribosome entry site (IRES) of tobacco etch virus (TEV) to promote translation initiati

57 The genomic RNA of tobacco etch virus (TEV), a potyvirus that belongs to th

58 ll supported systemic spread of an unrelated tobacco etch virus (TEV), suggesting multiple pathways f

59 DMFE of Vesicular stomatitis virus (VSV) and Tobacco etch virus (TEV), we found that increasing mutat

60 latable versions of the coat protein gene of Tobacco etch virus (TEV).

61 fer a highly resistant state to infection by tobacco etch virus (TEV).

62 heless, there was a substantial reduction of tobacco etch virus yield as measured by ELISA assay in t