ライフサイエンスコーパス: tobacco exposure

1 d to prenatal cannabis (mostly combined with tobacco exposure).

2 ly, smokers were stratified by pack-years of tobacco exposure.

3 results were largely mirrored with increased tobacco exposure.

4 PCE; a subset of each group additionally had tobacco exposure.

5 data regarding the cardiovascular effects of tobacco exposure.

6 isk of lung cancer increased with cumulative tobacco exposure.

7 s, nor considered other risk factors such as tobacco exposure.

8 suggesting a common airway-wide response to tobacco exposure.

9 Exposures: Prenatal methamphetamine and/or tobacco exposure.

10 e types of DNA damage, including damage from tobacco exposure.

11 havioral anomalies associated with perinatal tobacco exposure.

12 hs but not thereafter, and may reflect heavy tobacco exposure.

13 h weights for women with different levels of tobacco exposure.

14 olic syndrome, and prevalence increased with tobacco exposure: 1.2% for nonexposed, 5.4% for those ex

15 a causal relationship between environmental tobacco exposure and adverse behavioral and cognitive ou

16 Reported associations between gestational tobacco exposure and autism spectrum disorders (ASDs) ha

17 the relations between these two measures of tobacco exposure and birth weight.

18 ion of factors that increase PAI-1 including tobacco exposure and obesity.

19 Associations between tobacco exposure and overall survival (OS) and progressi

20 med a modest (inverse) relation between true tobacco exposure and serum beta-carotene.

21 Understanding the relationship between tobacco exposures and specific mutations may yield etiol

22 errors in reported smoking (relative to true tobacco exposure) and assumed a modest (inverse) relatio

23 its of mm Hg-years (similar to pack-years of tobacco exposure) and related to the presence of coronar

24 ake of retinol and provitamin A carotenoids, tobacco exposure, and asbestos exposure.

25 rds models adjusted for age, black ancestry, tobacco exposure, and socioeconomic position.

26 some individuals with a history of prolonged tobacco exposure, and that expression of the GRP recepto

27 stment for age, race, tumor and nodal stage, tobacco exposure, and treatment assignment, had a 58% re

28 hildren with prenatal methamphetamine and/or tobacco exposure are present at birth before childhood e

29 scular disease the cardiovascular effects of tobacco exposure are substantial.

30 l update recent findings supporting diet and tobacco exposure as etiologic factors in the development

31 ized to develop a non-invasive biomarker for tobacco exposure as well as a non-invasive screening or

32 nd death increases directly as a function of tobacco exposure at diagnosis and during therapy and is

33 we used two assays with mutagens relevant to tobacco exposure (benzo[a]pyrene diol epoxide (BPDE) and

34 data demonstrates that the current burden of tobacco exposure both in the United States and worldwide

35 Tobacco exposure, both current and lifetime, was linearl

36 hylation in certain genes is associated with tobacco exposure but it is unknown whether these methyla

37 Marijuana exposure was nearly as common as tobacco exposure but was mostly light (median, 2-3 episo

38 Environmental tobacco exposure, but not allergic sensitization, also h

39 ed birth weights for women who reduced their tobacco exposure by 50 percent or more and for those who

40 Prenatal cocaine or tobacco exposure can differentially affect structural br

41 Prenatal tobacco exposure compared with nonexposed control subjec

42 Despite reportedly having less tobacco exposure compared with whites, African Americans

43 ates and abroad, the cardiovascular risks of tobacco exposure despite improvements in medical therapy

44 s, only a small fraction of individuals with tobacco exposure develop cancer.

45 ce of pleiotropic effects and accounting for tobacco exposure did not alter the association (OR of sc

46 undertaken to determine 1) whether reducing tobacco exposure during pregnancy increases the birth we

47 on elucidating the complex interaction among tobacco exposure, genetics and environmental factors.

48 In utero tobacco exposure has been associated with fetal growth r

49 While all forms of tobacco exposure have negative health effects, the signi

50 hibited a mutational profile consistent with tobacco exposure; human papillomavirus was detectable by

51 Tobacco exposure in cardiac transplant recipients, befor

52 r experiments reveal that pretransplantation tobacco exposure in donors and/or recipients results in

53 HPV infection may interact with alcohol and tobacco exposure in tumor promotion.

54 activation of signaling pathways relevant to tobacco exposure, including ATM, BCL2, GPX1, NOS2, IKBKB

55 The cardiovascular effects of tobacco exposure, including even low levels from SHS, re

56 Birth weight declined as tobacco exposure increased; however, the relation was no

57 ogether, these new findings demonstrate that tobacco exposure induces the abnormal expression of SNCG

58 year 1 but not with race/ethnicity, income, tobacco exposure, maternal stress, or early introduction

59 Prenatal tobacco exposure may be one suspected cause of bipolar d

60 ollectively, prenatal methamphetamine and/or tobacco exposure may lead to delayed motor development a

61 ions and Relevance: Prenatal methamphetamine/tobacco exposure may lead to delays in motor development

62 As in the case of alcohol use, intensity of tobacco exposure (measured as packs per day) was not ass

63 t (LT) recipients to determine the impact of tobacco exposure on 10-year survival and de novo cancer

64 cioeconomic variables such as race, poverty, tobacco exposure, or general nutritional status.

65 as smaller in adolescents following prenatal tobacco exposure (P = .03).

66 e following either PCE (P = .05) or prenatal tobacco exposure (P = .04).

67 sure and is one potential mechanism by which tobacco exposure predisposes to adverse health outcomes,

68 -ethnicity, education, poverty-income ratio, tobacco exposure, previous diagnosis of diabetes, and bo

69 Prenatal tobacco exposure (PTE) affected speech processing, level

70 This association was related to past tobacco exposure, regardless of whether the subjects wer

71 Tobacco exposure remains the main but not exclusive caus

72 Tobacco exposure results in chronic inflammation, tissue

73 edings from 2007 and 2008 that link in-utero tobacco exposure to neurodevelopmental outcomes in expos

74 Parent and child's passive tobacco exposure was assessed using interview-assisted q

75 ity of studies reviewed were prospective and tobacco exposure was quantified biologically.

76 e potential confounding variables, including tobacco exposure, was 9.4 (95% CI, 2.6 to 33.8).

77 ubstantially larger and to quantify lifetime tobacco exposure with more precision than have past stud