コーパス検索結果 (1語後でソート)
通し番号をクリックするとPubMedの該当ページを表示します
1 ically involved in the reinforcing effect of tobacco smoking.
2 models adjusted for age, sex, and cumulative tobacco smoking.
3 increased with each additional pack-year of tobacco smoking.
4 Risk factors for periodontal disease include tobacco smoking.
5 r levels by 6 to 12 weeks of abstinence from tobacco smoking.
6 xplore the mutational burden associated with tobacco smoking.
7 is a major cause for starting or increasing tobacco smoking.
8 ntribute to the perpetuation of addiction to tobacco smoking.
9 on between the level of fasting glycemia and tobacco smoking.
10 ich despite some success have not eliminated tobacco smoking.
11 use of death, and almost all of it is due to tobacco smoking.
12 the youngest infants and in those exposed to tobacco smoking.
13 exposure of the upper aerodigestive tract to tobacco smoking.
14 on with the effect of nonsmoking and regular tobacco smoking.
15 ion treatments and reduce the harm caused by tobacco smoking.
16 association with risk for IBD is modified by tobacco smoking.
17 t disease, diseases strongly associated with tobacco smoking.
18 , but this does not seem to be made worse by tobacco smoking.
19 ical tumors and that LOY could be induced by tobacco smoking.
20 e, the principal addictive component driving tobacco smoking.
21 geting prevention and cessation of waterpipe tobacco smoking.
22 ntified strong epigenetic signals related to tobacco smoking.
23 x disease, Barrett's esophagus, obesity, and tobacco smoking.
24 hrenia is associated with very high rates of tobacco smoking.
25 , for example, the "pack-years" variable for tobacco smoking.
26 diseases, respectively, were attributable to tobacco smoking.
27 (beta +/- SE 0.07 +/- 0.01; P < 0.0001) and tobacco smoking (0.64 +/- 0.26; P = 0.016), as well as w
28 risk ratio [RR] 1.41 [95% CI 1.26-1.57]) and tobacco smoking (1.46 [1.30-1.65]) than in HIV-negative
29 use (RR 1.36 [95% CI 1.10-1.69]; p=0.0050), tobacco smoking (1.90 [1.38-2.62]; p<0.0001), and smokel
30 positive women was 1.3% (95% CI 0.8-1.9) for tobacco smoking, 2.1% (1.1-3.4) for smokeless tobacco us
32 ositive men was 24.4% (95% CI 21.1-27.8) for tobacco smoking, 3.4% (1.8-5.6) for smokeless tobacco us
35 e no significant differences in gender, age, tobacco smoking, Acute Physiology and Chronic Health Eva
36 st associations, with the exception of daily tobacco smoking (adjusted OR 1.74, 95% CI 1.08-2.81), an
37 rders in general), including breast-feeding, tobacco smoking, alcohol consumption, and exposure to do
38 extensive questionnaire containing items on tobacco smoking, alcohol consumption, and occupational e
39 sion models that were adjusted for age, sex, tobacco smoking, alcohol drinking, and hypertension.
40 terms for age, sex, study center, education, tobacco smoking, alcohol drinking, hepatitis B surface a
42 from the general population, in relation to tobacco smoking, an exposure with well-characterised hea
43 o specific risk factors, such as alcohol and tobacco (smoking and smokeless), but dietary factors and
44 ted with the widely accepted risk factors of tobacco smoking and alcohol use and is the end result of
45 mokers to assess whether baseline water pipe tobacco smoking and baseline snus use were associated wi
46 models to assess whether baseline water pipe tobacco smoking and baseline snus use were associated wi
49 population density areas, and pack-years of tobacco smoking and decreases in subjects with history o
53 anged by at least 2 percentage points (e.g., tobacco smoking and mental health conditions) were assoc
54 s to investigate the differential effects of tobacco smoking and nicotine on changes in GABAA recepto
57 childhood behavior might reduce early onset tobacco smoking and risk of tobacco dependence among smo
59 emographic risk factors, baseline water pipe tobacco smoking and snus use were independently associat
70 tablished risk factors, that is, obesity and tobacco smoking, are overrepresented among patients who
71 in Nha Trang, Vietnam, to evaluate paternal tobacco smoking as a risk factor for infectious and non-
73 are hypothesized to contribute to relapse to tobacco smoking but mechanisms underlying and linking su
74 5 million deaths a year are attributable to tobacco smoking, but attempts to help people either quit
75 Modeled age-standardized prevalence of daily tobacco smoking by age, sex, country, and year; cigarett
76 nicotine consumption and prevent relapse to tobacco smoking by modulating glutamate transmission.
81 F reviewed the evidence on interventions for tobacco smoking cessation that are relevant to primary c
85 ibutable and unattributable to hypertension, tobacco smoking, diabetes mellitus, and obesity using an
88 has been reported that pre-conditioning with tobacco smoking during adolescence predisposes those you
90 ChRs) may underlie differential nicotine and tobacco smoking effects and related behaviors in women v
92 nd chemotherapy-responsive tumor, related to tobacco smoking, environmental arsenic exposure, industr
93 t risk reduction, followed by a reduction in tobacco smoking for men and obesity for women, but these
94 ine is clearly recognized by the tenacity of tobacco smoking for most users, and has prompted extensi
95 variety of psychiatric disorders, including tobacco smoking, for which there is strong evidence supp
96 % confidence interval [CI], 1.7-49.3), heavy tobacco smoking (>20 pack-years vs none; OR, 9.2; 95% CI
97 ught to be the key substance responsible for tobacco-smoking habits and appears to trigger reinforcem
103 (reflux), higher body mass index (BMI), and tobacco smoking have been individually associated with e
104 factors related to DALYs were dietary risks, tobacco smoking, high body mass index, high blood pressu
106 A 62-year-old man with a 45 pack per year tobacco-smoking history presented with painless gross he
107 ars (wave 10), mental health problems, daily tobacco smoking, illicit drug use, and dependence were a
109 ne, a noradrenergic alpha2a agonist, reduced tobacco smoking in a 4-week trial and in animal models h
110 elated neuroadaptations of cAMP signaling to tobacco smoking in human subjects and suggest that smoki
113 modeled age-standardized prevalence of daily tobacco smoking in the population older than 15 years de
114 lysis of covariance with age (in months) and tobacco smoking (in pack-years) as covariates showed tha
115 from solid fuels (HAP; 7.0% [5.6-8.3]), and tobacco smoking including second-hand smoke (6.1% [5.4-6
116 ertainty interval 6.2-7.7] of global DALYs), tobacco smoking including second-hand smoke (6.3% [5.5-7
120 ]PHNO PET may be more sensitive to measuring tobacco smoking-induced DA release in human tobacco smok
121 latory effects of nicotine in the brain (ie, tobacco smoking-induced upregulation of beta(2)*-nAChRs)
122 ttributable risk (PAR) of bladder cancer for tobacco smoking is 50% to 65% in men and 20% to 30% in w
145 ing that there are various pathways by which tobacco smoking might contribute to development of Barre
148 lcohol withdrawal with or without concurrent tobacco smoking/nicotine consumption resulted in signifi
151 risk, we sought to investigate the impact of tobacco smoking on the risk of premature death and its c
152 ere we report on the sex-specific effects of tobacco smoking on transcriptomic and epigenetic feature
155 This blunting may contribute to relapse to tobacco smoking, particularly in depression-vulnerable i
156 moking status, demographics, alcohol intake, tobacco smoking, physical activity, and included a food-
158 ver, this excess risk may be attributable to tobacco smoking rather than the three-fold higher preval
162 0.9-4.0) for 29 to 39 years and >39 years of tobacco smoking, respectively, as compared with those th
164 significantly increase risk for SP included tobacco smoking (RR, 2.47; 95% CI, 2.12-2.87), alcohol i
165 her odds of lung cancer after adjustment for tobacco smoking, serum cotinine levels, educational atta
166 , and overall relative prevalence ratios for tobacco smoking, smokeless tobacco use, and any tobacco
168 gender, age at time of initial examination, tobacco smoking status at time of initial examination, p
169 e likely to smoke, and, after adjustment for tobacco-smoking status and other potential confounders,
170 y factors, including childhood maltreatment, tobacco smoking, substance dependence, psychiatric medic
171 a substantial decrease in the prevalence of tobacco smoking, the adverse health effects of tobacco s
174 and cause-specific mortality associated with tobacco smoking using adjusted hazard ratios and their 9
175 between ages 40-60 after adjusting for age, tobacco smoking, viral load, and traditional risk factor
176 studies established for the first time that tobacco smoking was a risk factor for the development of
177 r, prevalence of psychiatric comorbidity and tobacco smoking was alarmingly high in severe patients w
178 healthy infants whether exposure to parental tobacco smoking was associated with airway hyperreactivi
184 eristics, especially alcohol consumption and tobacco smoking, we examined promoter methylation of the
188 gh concentrations of nicotine resulting from tobacco smoking will cause an abnormal activation, a des
WebLSDに未収録の専門用語(用法)は "新規対訳" から投稿できます。