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   1 times to first seizure of any type and first tonic-clonic seizure.                                   
     2 ly occurring during, or immediately after, a tonic-clonic seizure.                                   
     3 ment of eclampsia, as defined by a witnessed tonic-clonic seizure.                                   
     4 ed to our institution, she had a generalized tonic-clonic seizure.                                   
     5 s and approximately 30 suspected generalized tonic-clonic seizures.                                  
     6 P occurred immediately following generalized tonic-clonic seizures.                                  
     7 ociated with the tonic phases of generalized tonic-clonic seizures.                                  
     8 with progressively severe myoclonus and rare tonic-clonic seizures.                                  
     9 ractable childhood epilepsy with generalized tonic-clonic seizures.                                  
    10 istently involved in secondarily generalized tonic-clonic seizures.                                  
    11 festations and for impaired consciousness in tonic-clonic seizures.                                  
    12 hibit seizures, required ASIC1a to interrupt tonic-clonic seizures.                                  
    13 age caused by kainic acid, in the absence of tonic-clonic seizures.                                  
    14 ted behavioral endpoints of epileptogenesis, tonic-clonic seizures.                                  
    15 lity at week 3 to 4 of life from generalized tonic-clonic seizures.                                  
    16 of a unique capacity to initiate generalized tonic-clonic seizures.                                  
    17 nd die at a mean age of 42 days from massive tonic-clonic seizures.                                  
    18 sy phenotype consisting of 3/sec absence and tonic-clonic seizures.                                  
  
  
    21 ythmic myoclonus (all patients), generalized tonic-clonic seizures (all patients) and complex partial
  
    23 ding childhood absence epilepsy, generalized tonic clonic seizures and the epileptic encephalopathy, 
  
    25  seizures, and 1 had 4 witnessed generalized tonic-clonic seizures and approximately 30 suspected gen
  
    27 evere epilepsy with early onset, generalized tonic-clonic seizures and juvenile lethality by 3 weeks 
  
    29  exhibits acoustically triggered generalized tonic-clonic seizures, and often times the EEG, recorded
    30 etion died prematurely following generalized tonic-clonic seizures, and they were equally susceptible
  
  
  
    34 sorders of GABA metabolism, and one in which tonic-clonic seizures associate with increased central n
    35 osition to sound stimuli-induced generalized tonic-clonic seizures (audiogenic reflex seizures) and i
    36 ilepsy, neurological insult, total number of tonic-clonic seizures before randomisation, seizure type
    37 f350, caused low seizure threshold, sporadic tonic-clonic seizures, brain enlargement and ectopic neu
    38 tanding the networks involved in generalized tonic-clonic seizures can provide insights into mechanis
    39 xhibited early onset spontaneous generalized tonic-clonic seizures concurrent with a significant redu
  
  
    42 f those with EEG abnormality were at risk of tonic-clonic seizures during follow-up compared with 16%
  
    44 presented with afebrile focal or generalized tonic-clonic seizures during the first to second year of
  
    46 pendent paradigm, pentylenetetrazole-induced tonic-clonic seizures, exclude the possibility that the 
    47 lepsy (JME), one predisposing to generalized tonic-clonic seizures (GTCS) and a second to myoclonic s
    48  analyzed with the algorithm: 25 generalized tonic-clonic seizures (GTCSs) from 11 patients, and 19 e
    49 mice increased latencies to forelimb clonus, tonic clonic seizures, hindlimb extension, and death com
    50 ractable childhood epilepsy with generalized tonic-clonic seizures (ICEGTC), which bears similarity t
    51 pathetic hyperactivity immediately following tonic-clonic seizures in DS mice, which leads to lethal 
  
    53 rtial seizures are at low risk of subsequent tonic-clonic seizures in the period of time to which the
    54 the effects of treatment on the frequency of tonic-clonic seizures in those patients with such pre-ra
    55 had a greater latency and lower incidence of tonic-clonic seizures induced by pentylenetetrazol (PTZ;
    56  ictal SPECT from 59 secondarily generalized tonic-clonic seizures obtained during epilepsy surgery e
    57 ilepsy (JME), epilepsy with only generalized tonic-clonic seizures occurring either randomly during t
    58 uvenile absence epilepsy, five patients with tonic-clonic seizures on awakening and 30 control subjec
    59 e epilepsy and two out of five patients with tonic-clonic seizures on awakening, but in none of the 3
    60 juvenile absence epilepsy, and epilepsy with tonic clonic seizures only under the sole heading of idi
    61 eters can provide notification of an ongoing tonic-clonic seizure, or peace of mind in the absence of
  
  
    64 drugs used in the MESS study are greater for tonic-clonic seizures than they are for partial seizures
    65 rs indicate that the higher the frequency of tonic-clonic seizures, the higher the risk of SUDEP; fur
    66  E injection alone while there was a reduced tonic--clonic seizure threshold in the presence of both 
  
    68 e strain is unusually sensitive to recurrent tonic-clonic seizures upon routine handling and to seizu
  
  
  
    72 ut 25% of aged apoE4 TR mice had more severe tonic-clonic seizures which occasionally progressed to t
    73 474S mice) exhibited spontaneous generalized tonic-clonic seizures (which occurred in the absence of 
  
    75 zed by normal early development, generalized tonic-clonic seizures with onset at 5-10 years of age, a
  
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