ライフサイエンスコーパス: tonsil

1 been identified in human blood, spleen, and tonsil.

2 f T cell development occurs within the human tonsil.

3 rge intestine, bursa of Fabricius, and cecal tonsil.

4 ent in the variant Creutzfeldt-Jakob disease tonsil.

5 r germinal centre (GC) B cells compared with tonsil.

6 on T lymphocytes in normal human spleen and tonsil.

7 common site (60.4%), followed by tongue and tonsil.

8 an colon tissue as well as hyperplasia human tonsils.

9 ies can persist in pediatric adenoids and/or tonsils.

10 teraction that probably takes place in human tonsils.

11 vivo in T-cell areas of lingual and palatine tonsils.

12 induction to food and aeroallergens in human tonsils.

13 e identified both in lingual and in palatine tonsils.

14 ic cell-compartments in palatine and lingual tonsils.

15 CL6-expressing CD4(+) T-cell subset in human tonsils.

16 al-associated lymphoid tissue, adenoids, and tonsils.

17 ally those arising in the oropharynx and the tonsils.

18 keratinocytes from the foreskin, cervix, and tonsils.

19 o potent chemokines made abundantly in human tonsils.

20 ted B cell-helping effector T cells in human tonsils.

21 e salivary glands, oral mucosa, and palatine tonsils.

22 increased germinal center reactivity in the tonsils.

23 atal cord blood, adult peripheral blood, and tonsils.

24 source of TGF-beta1 in chronically infected tonsils.

25 ls at a specific stage (CD69(+)CD45RO(+)) in tonsils.

26 T cells in neonatal blood, adult blood, and tonsils.

27 diagnosis and showed prion infection of the tonsils.

28 on subsets of B cells in blood, spleen, and tonsils.

29 gen sampling M cells in the rabbit cecum and tonsils.

30 of epithelium using keratinocytes from human tonsils.

31 ce, a tissue functionally analogous to human tonsils.

32 macrophages) GC B cell fractions from seven tonsils.

33 viral gene expression is detected in TG and tonsils.

34 natural interferon-producing cells (IPCs) in tonsils.

35 close connection with each other in palatine tonsils.

36 largest reservoir in the spleen, followed by tonsils.

37 virus sequences were identified in 19 of 101 tonsils (19%), but not in any other tissues.

38 igher than that of contralateral cancer-free tonsils (2.54 + or - 0.88; P < .0001) and tonsils in con

39 Furthermore, we identify within the tonsil a cNK precursor population that is characterized

40 ased innate antiviral factors may render the tonsil a potential site for oral transmission.

41 Thus, EBV-specific CTL in the tonsil, a crucial site for EBV entry and replication, ar

42 significantly decreased virus replication in tonsils, a transient viremia, limited generalization of

43 CD34) isolated from a human lymphoid organ, (tonsils), adding to our understanding of how L-selectin

44 AAV was detected in 7 of 101 tonsil-adenoid samples and in 2 of 74 other tissue sampl

45 Tonsil allergen-specific FOXP3(+) regulatory T (Treg) ce

46 The spleen and tonsil, although harboring the largest number of overall

47 l and umbilical cord blood (UCB), thymus and tonsil, although mRNA levels were reduced compared with

48 -risk HPV infections were present in 2.1% of tonsil and 6.3% of oral-rinse specimens.

49 n CD19(+) purified B lymphocytes from normal tonsil and 7 cases of histologically confirmed MCL.

50 In this study, human tonsil and adenoid tissues were analyzed to determine th

51 analysis indicates that, in comparison with tonsil and bone marrow (BM) PCs, these PCs distinctively

52 illar memory B cells (MB) and PCs, from both tonsil and bone marrow tissues, express BCMA.

53 Tonsil and hypermutating Ramos B-cells convert C-->U in

54 r and spleen, spleen and cecal tonsil, cecal tonsil and ileum, liver and cecal tonsil, liver and ileu

55 oid cell populations expressing CD155 in the tonsil and intestine, as well as in spinal cord neurons.

56 Droppings, cecal tonsils and 5 internal organs were sampled and cultured

57 hes a latent infection in lymphocytes of the tonsils and adenoids.

58 consists of diffuse lymphoid cells and lacks tonsils and adenoids.

59 ceptor phenotype of the Th17 cell subsets in tonsils and adult blood.

60 influenza virus-specific B-cell responses in tonsils and blood.

61 ry B-cell (MBC) responses were enumerated in tonsils and blood.

62 in oligodendrocyte glycoprotein) in palatine tonsils and cervical lymph nodes of 28 acute stroke pati

63 strated that MHC class II may be detected in tonsils and EBV-negative Hodgkin disease but not in EBV-

64 niquely express lysozyme and can be found in tonsils and in tumors.

65 e mature than other CD56(bright) NK cells in tonsils and less mature than other NK cells in blood, sh

66 ner in single-cell suspensions of both human tonsils and NALT.

67 re swabbed and that swab was used to rub the tonsils and nasal and ocular mucosa of naive pigs.

68 The tonsils and nasal mucosa of each positive-control pig we

69 Virus was, however, isolated from the tonsils and nasal swabs of the asymptomatic T15 pigs at

70 allergen-specific T-cell tolerance in human tonsils and peripheral blood through a mechanism depende

71 responses of allergen-specific T cells from tonsils and peripheral blood were measured by using trit

72 tinal tract) and lymphoid/monocytic cells in tonsils and Peyer's patches (explaining viremia), extend

73 tinal tract) and lymphoid/monocytic cells in tonsils and Peyer's patches (explaining viremia), thereb

74 mucosa-associated lymphoid tissues, such as tonsils and Peyer's patches, which is hard-wired to secr

75 in children and adolescents and colonize the tonsils and pharynx of up to 20% of healthy children.

76 rimary Tfh and non-Tfh T effector cells from tonsils and prepared genome-wide maps of active, interme

77 uely report the mucosal humoral responses in tonsils and saliva after intranasal live attenuated infl

78 Tonsillectomy removes only the palatine tonsils and sometimes adenoids.

79 CNS invasion, prions spread centrifugally to tonsils and spleen at an advanced stage of the incubatio

80 aive patients with FL compared with reactive tonsils and the peripheral blood of healthy donors.

81 sh stage III immature NK cells isolated from tonsils and these cells expressed CXCL8 protein on PMA s

82 red with those of healthy TILs from reactive tonsils and this can be induced on healthy T cells by FL

83 stages in a human secondary lymphoid organ (tonsils) and adult, but not in neonatal, blood.

84 7% in lymphoma biopsies, 12% in inflammatory tonsil, and 6% in tumor-free lymph nodes; P = .001).

85 ific) mBCs in peripheral blood (PB), spleen, tonsil, and bone marrow.

86 l LNs analyzed, as well as in the spleen and tonsil, and correspond to the two known blood DC subtype

87 DNA in B lymphocytes from peripheral blood, tonsil, and spleen and it has been hypothesized that lym

88 By day 4, vDNA was detected in the throat, tonsil, and spleen, and monkeypox antigen was detected i

89 of Fabricius, gastrointestinal tract, cecal tonsil, and trachea.

90 e contact to NK cells in T-cell areas of the tonsils, and a subpopulation of the pDCs expressed GITRL

91 airways usually occur in the upper airways, tonsils, and adenoid structures that make up the Waldeye

92 id tissues (O-MALT) such as Peyer's patches, tonsils, and adenoids.

93 other regions, such as the salivary glands, tonsils, and bone marrow.

94 sity, and ontogeny in paired donor blood and tonsils, and in blood after vaccination.

95 ly observed in the palatine tonsils, lingual tonsils, and soft palate, whereas uptake in the major sa

96 Germinal centers and plasma cells in tonsils appeared normal, as were serum immunoglobulin le

97 (-)) CD127(+)RORC(+) LTi-like cells in human tonsil are precursors to CD56(+)CD127(+)RORC(+)NKp46(+)

98 s with isolated infarction of the cerebellar tonsil are unknown.

99 We find that IPCs in tonsils are in close contact with CD8+ T cells and demon

100 going a germinal center (GC) reaction in the tonsils are limited to the follicles and proliferate ext

101 show that EBV-specific CTL responses in the tonsils are of comparable specificity and breadth but of

102 Tonsils are strategically located in the gateway of both

103 Human tonsils are the primary reservoirs for GAS, maintaining

104 an tissue-resident macrophages isolated from tonsil as a tractable cell model.

105 n tonsillar follicles and support the use of tonsils as lymphoid sites for the study of germinal cent

106 ephalosynapsis correlates with fusion of the tonsils, as well as midbrain abnormalities including aqu

107 ed with the percentage of eGC B cells in the tonsil at the outset of the culture.

108 noted between radiotracer uptake in spleen, tonsil, axillary lymph nodes, and peripheral blood CD4 T

109 lation between the phenotypic composition of tonsil B cells and the CSR to IgE ex vivo.

110 When purified tonsil B cells are incubated with IL-4 and anti-CD40 to

111 his study shows that the maturation state of tonsil B cells determines their capacity to undergo clas

112 Total tonsil B cells give rise to IgE(+) PCs by direct and seq

113 Human tonsil B cells were analyzed by flow cytometry (FACS) an

114 inal center (GC), early GC (eGC), and memory tonsil B cells were isolated by FACS, and their capaciti

115 To generate human IgE(+) cells, we cultured tonsil B cells with IL-4 and anti-CD40.

116 Cox-2 is also highly expressed by human tonsil B cells, as shown by immunohistochemistry.

117 SphK1 inhibition in human tonsil B cells, as well as inhibition or deletion of Sph

118 e of generated IgE(+) cells, the capacity of tonsil B-cell subsets to generate IgE(+) PCs and the cla

119 LC differentiation was not observed from tonsil BDCA-1(+) and BDCA-3(+) subsets.

120 sues close to the viral entry site, with the tonsil being the primary site of virus replication and i

121 racterized by displacement of the cerebellar tonsils below the base of the skull, resulting in signif

122 have circumvented these limitations by using tonsil biopsies to study autoreactive B cells (9G4 B cel

123 Tonsil biopsies were analyzed by confocal microscopy.

124 ected antemortem as an adjunct to testing of tonsil biopsy specimens and surveillance by necropsy for

125 functionally immature transitional B cells, tonsil biopsy tissues revealed active germinal center (G

126 linical diagnosis of probable vCJD was made; tonsil biopsy was not done.

127 Tonsil biopsy will allow early and pre-symptomatic diagn

128 monstrate that macrophages residing in human tonsil blocks can be productively infected ex vivo by pr

129 at the spleen harbors most mBCs, followed by tonsils, BM, and PB, and we detected no major difference

130 es but not in nonmalignant plasma cells from tonsils, bone marrow from healthy individuals, or patien

131 We found that tonsil but not blood leukocytes were responsive to S1P g

132 e expression of EphA4 was detected in EBV(-) tonsils but not in EBV(+) PTLD.

133 lated from four major chicken tissues: cecal tonsil (C), ileum (I), liver (L), and spleen (S) were us

134 e driven predominantly by base-of-tongue and tonsil cancers in men.

135 In tonsils, CD138(+) plasma cells (PCs) are surrounded by C

136 d between liver and spleen, spleen and cecal tonsil, cecal tonsil and ileum, liver and cecal tonsil,

137 Using tonsil cell lysates to repair a G.U mismatch, A/T and G/

138 HIV-1 ex vivo by spinoculating and culturing tonsil cells with HIV-1 GFP reporter viruses.

139 munodeficiency virus type 1 (HIV-1), such as tonsil, cervical, or rectal tissue.

140 ified squamous epithelia of human esophagus, tonsil, cervix, larynx, and cornea.

141 de, nasopharyngeal lymph node and pharyngeal tonsil collected at the peak of clinical disease from be

142 en processing were increasingly expressed in tonsil compared with the epithelium of another oropharyn

143 FOXP3(+) Treg cells, are identified in human tonsils compared with peripheral blood.

144 These data reveal that human tonsil contains long-lived plasma cells, the majority of

145 Little is known about how tonsils contribute to the local immune response after in

146 of expression is detectable in lymph nodes, tonsil, cultured lymphoblasts, and the ovary.

147 Functionally, tonsil DC also only stimulated low levels of antigen-spe

148 We show that tonsil DC are able to sample their antigenic environment

149 n and brain activity in the right cerebellar tonsil, declive, culmen, lingual gyrus and cuneus.

150 These CD8-positive, CD103-positive, tonsil-derived CTL were largely CCR7- and CD45RA- negati

151 immunity, by studying the responses of human tonsil-derived DC to Neisseria meningitidis as a model o

152 fore, examined the susceptibility of a human tonsil-derived follicular dendritic cell-like cell line

153 als, we found BSE in 50% and 12% of gut- and tonsil-derived samples, respectively.

154 ulating TFR are phenotypically distinct from tonsil-derived TFR in humans.

155 ation status of blood TFR is comparable with tonsil-derived TFR.

156 e we report that LMP2A expression in primary tonsil epithelial cells causes them to become migratory

157 irmed the increased presence of CXCR4 in the tonsil epithelium compared with multiple oral epithelial

158 Tonsil epithelium has been implicated in human immunodef

159 Importantly, tonsil epithelium highly expressed genes associated with

160 en together, these findings suggest that the tonsil epithelium of asymptomatic virus carriers is able

161 e with anti-HIV activity, was minimal in the tonsil epithelium, in contrast to oral mucosa.

162 HIV, we performed microarray analysis of the tonsil epithelium.

163 KG2A(+)CD94(+)CD54(+)CD62L(-), is present in tonsils ex vivo and is more mature than other CD56(brigh

164 B cells from different tonsils exhibited varying capacities for CSR to IgE ex v

165 isolated from peripheral blood and palatine tonsils, expanded, and cocultured with naive B cells.

166 ary epithelial-cell cultures, generated from tonsil explants, contained a heterogeneous mixture of ce

167 that B lymphocytes from peripheral blood and tonsils express DC-SIGN and that this expression increas

168 (five male, 12 female; aged 4-43 years) with tonsils extending more than 5 mm below the foramen magnu

169 asion by GAS was reproduced in primary human tonsil fibroblasts, which could be a source of TGF-beta1

170 was stimulated in GAS-infected primary human tonsil fibroblasts.

171 lly characterize, from human lymph nodes and tonsils, four NK cell developmental intermediates spanni

172 nal centers and plasma cells were studied in tonsils from 4 additional children with Down syndrome.

173 Tfh cells in tonsils from control individuals displayed the active fo

174 gene BZLF1 were more frequently detected in tonsils from EBV carriers colonized with GAS than from E

175 FOXP3(+) Treg cells and pDCs is observed in tonsils from nonatopic individuals.

176 Tonsils from patients (n = 617) undergoing tonsillectomy

177 the first time to our knowledge in children, tonsils from seasonal influenza-vaccinated children.

178 Generation of IgE(+) PCs from tonsil GC B cells occurs mainly via sequential switching

179 rted by the presence of CCR10(+)IgA(+)PBs in tonsil GCs.

180 ct on chronic psoriasis because the palatine tonsils generate effector T cells that recognize keratin

181 Human tonsils grafted into immunodeficient mice were therefore

182 asional viremia, and virus was isolated from tonsils, gut mucosa, and draining lymph nodes.

183 ree cell types, and viral replication in the tonsil/gut was associated with histopathologic destructi

184 The developing Th17 cells in tonsils highly expressed both Th1- (CCR2, CXCR3, CCR5, a

185 l subsets include a novel in vivo-stimulated tonsil IL17+ T cell subset detected without any artifici

186 a number of tissue-selective genes for cecal tonsil, ileum, liver, and spleen identified (95, 71, 535

187 we provide evidence of a role for the human tonsil in a comprehensive program of extrathymic T cell

188 ee tonsils (2.54 + or - 0.88; P < .0001) and tonsils in control subjects (2.98 + or - 1.08; P < .0001

189 the liver, spleen, jejunum, ileum, and cecal tonsils in newly hatched chickens 6, 12, 24, and 48 h af

190 The mean SUV(max) ratio between tonsils in patients with carcinoma was 3.79 + or - 1.69,

191 e found in close proximity to ILC3s in human tonsils in situ.

192 fferentiation, which takes place in palatine tonsils in vivo.

193 tivated phenotypes, expressed TSLPR in human tonsils in vivo.

194 her acute or persistent infection except for tonsil, in which the number of responding cells was extr

195 er--specifically of the lingual and palatine tonsils--in white men younger than age 50 years who have

196 orted with unilateral flocculus and anterior tonsil infarction.

197 ys to address the mucosal immune dynamics in tonsils investigating the spatial positioning, frequency

198 c evidence, suggest that EBV infection in IM tonsils involves extrafollicular B cells expressing CD38

199 Lingual tonsil is anatomically big and remains lifelong intact.

200 uptake was usually observed in the palatine tonsils, lingual tonsils, and soft palate, whereas uptak

201 sil, cecal tonsil and ileum, liver and cecal tonsil, liver and ileum, spleen and ileum (P < 10-7), re

202 mately 3-7%, in spleen was 10%, and in cecal tonsil, lung, and bone marrow was approximately 15%.

203 tal details for setting up cultures of human tonsils, lymph nodes and cervicovaginal and rectosigmoid

204 sistently observed, with occasional viremia; tonsil, mesentery lymph nodes, and intestinal mucosa ser

205 5 tissue samples including freshly collected tonsils (n = 101) and archived frozen samples representi

206 Tonsil naive T cells were readily chemoattracted by S1P

207 Moreover, baseline tonsil obstruction detected by either DISE or mean snori

208 Tonsil obstruction was significantly, inversely correlat

209 of the LR mutant virus were detected in the tonsils of acutely infected calves.

210 olonged cytokine stimulation, accumulates in tonsils of EBV carriers, and is able to potently restric

211 se cells are present in peripheral blood and tonsils of healthy subjects and display a degree of hype

212 re, we identified GAS-specific Th17 cells in tonsils of humans naturally exposed to GAS, prompting us

213 R gene (the LR mutant strain) to grow in the tonsils of infected calves and reactivate from latency.

214 DNA was consistently detected by PCR in the tonsils of latently infected calves, suggesting that the

215 1 latency, viral genomes are detected in the tonsils of latently infected calves.

216 level and duration of IL-6 production in the tonsils of pigs intranasally inoculated with NS4B.VGIv w

217 tion of high-dose Zika virus directly to the tonsils of three rhesus macaques results in detectable p

218 hat LAIV elicits humoral B-cell responses in tonsils of young children.

219 (RLNs), and sections of tonsil (sections of tonsil only from captive animals were tested).

220 initially thought to represent SCCUP is the tonsil or base of the tongue, and an increasing percenta

221 We show here that human Ms (isolated from tonsils or generated from monocytes in vitro) drive acti

222 We analysed B cells from NP or tonsil, or after ILC2 coculture, by flow cytometry.

223 xposed to donor saliva via the conjunctivae, tonsils, or nostrils did not become infected.

224 Tonsil pDCs have the ability to generate functional CD4(

225 E pathology samples with known CN, including tonsil, placentae, and FFPE melanoma cell lines.

226 Tonsils play a key role in eliciting immune responses ag

227 late (PRP, 96%; mean SUV, 3.13), and lingual tonsils (PRP, 96%; mean SUV, 3.11).

228 acer uptake was usually seen in the palatine tonsils (PRP, 98%; mean SUV, 3.48), soft palate (PRP, 96

229 siologic FDG uptake was seen in the palatine tonsils (r=-0.51, P <.001) and sublingual glands (r=-0.7

230 at CXCR5(lo)ICOS(lo) CD4(+) T cells in human tonsils represent Tfh lineage-committed cells that provi

231 allergic patients the immune profile of the tonsils represents the atopic status of patients, with l

232 Here, we show that freshly isolated tonsil-resident BDCA1(+) DCs, BDCA3(+) DCs, and pDCs all

233 EBV-specific, tonsil-resident, but not PBMC-derived, T cells expressed

234 ow that in vivo, EBV-infected B cells in the tonsils retain expression of functional and phenotypic m

235 AAV-cellular junction was identified from a tonsil sample and it mapped to a highly repetitive satel

236 AAV DNA was detected in 7 of 101 (7%) tonsil samples and two of 74 other tissues (one spleen a

237 Palatine tonsil samples were obtained from 143 elective tonsillec

238 Blood, saliva, and tonsils samples were collected from 39 children before a

239 formed in 2 h or less, detected viral DNA in tonsil scraping samples 2 to 4 days prior to onset of cl

240 Immunohistochemistry of human tonsil sections demonstrates that tonsillar interdigitat

241 aryngeal lymph nodes (RLNs), and sections of tonsil (sections of tonsil only from captive animals wer

242 Human tonsils show very low levels of allergen-induced T-cell

243 f the incubation period, thus explaining why tonsil specimens were not reliable for detection of simi

244 iased analysis of human DC subsets in blood, tonsil, spleen, and skin.

245 ve immunostaining, were present in the lung, tonsil, spleen, lymph nodes, and colon.

246 roximately 5% of the clones from the thymus, tonsil, spleen, peripheral blood, and bone marrow of new

247 Tonsil standardized uptake values (SUVs) were measured b

248 with the primary sites of persistence being tonsil, sternal lymph node, and inguinal lymph node.

249 and CCR4 to separate foci in T cell zones of tonsil, suggesting that the chemokine-receptor(+) subset

250 with altered memory B cell subpopulations in tonsils, suggests that peripheral blood memory cell reco

251 SAT serotypes from field buffaloes, palatine tonsil swabs were the sample of choice for recovering in

252 Subsequently, serum samples, tonsil swabs, and feces were collected from sows (n = 22

253 Tonsil T cells did not proliferate to common food and ae

254 ombinants in melanoma cells or primary human tonsil T cells in vitro.

255 ate exhibited by the majority of resting CD4 tonsil T cells leads to accumulation of incomplete rever

256 s phenomenon was not tumor specific, because tonsil T cells were similar to FL TILs.

257 nables the allergen-induced proliferation of tonsil T cells, indicating an active role of Treg cells

258 POKA in infectivity levels for primary human tonsil T cells.

259 In lymph nodes and tonsils, T-follicular helper cells have been identified

260 In fact, purified circulating and tonsil Tfh cells increased IgG secretion by blood Ag-ind

261 ture human B-cell subpopulation in the human tonsil that has characteristics of both naive B cells an

262 erize a distinct human NK cell population in tonsils that produces high amounts of the immunomodulato

263 s that express CD11c and/or CD14, whereas in tonsils the same gated population contained primarily MH

264 m plays a gatekeeper role, as lesions of the tonsil, the lobus semilunaris inferior, and parts of the

265 R was clearly expressed on PC from the human tonsil, the lymph node, and the spleen (secondary lympho

266 ar structure in the oral mucosa and palatine tonsils, the high rate of oral blood flow, and innate fa

267 cell carcinomas (HPV-HNSCC) originate in the tonsils, the major lymphoid organ that orchestrates immu

268 prevent colonization and infection of human tonsils, thereby eliminating potential reservoirs that m

269 teeth; mucosal swab samples from the tongue, tonsils, throat, and buccal mucosa; and stimulated and u

270 inized gingiva, hard palate; saliva, tongue, tonsils, throat; sub- and supra-gingival plaques; and st

271 lymphoid hyperplasia, extensive hyperplastic tonsils, thymus hyperplasia, autoimmune lymphocytic thyr

272 gh pneumococcal-specific T-cell responses in tonsil tissue and peripheral blood.

273 r was also robust, approaching that of human tonsil tissue and the human germinal center B cell line,

274 hat the LR gene promotes virus shedding from tonsil tissue during acute infection and reactivation fr

275 in vivo after DEX treatment, explantation of tonsil tissue from calves latently infected with the LR

276 e infection and reactivation from latency in tonsil tissue in vivo.

277 ILC3 frequencies were measured in tonsil tissue of allergic and nonallergic patients and i

278 ILC3 frequency was reduced in tonsil tissue of allergic patients and in peripheral blo

279 ly developed a primary epithelial model from tonsil tissue to study EBV infection in epithelial cells

280 When the patient died, brain and tonsil tissue were obtained at autopsy and assessed for

281 In tonsil tissue, although EBV reactivities outnumbered the

282 In ex vivo cultures of human tonsil tissue, CD4 T cells undergo a pronounced cytopath

283 77-positive centroblasts isolated from human tonsil tissue.

284 , cecal, jejunal, ileal, duodenal, and cecal tonsil tissues.

285 eristic of habitat groupings such as throat, tonsils, tongue dorsum, hard palate, and saliva.

286 The mean maximum SUV (SUV(max)) of tonsil tumors was 9.36 + or - 4.54, which was significan

287 cer, the mean difference in SUV(max) between tonsils was 10.43 + or - 7.07, which was significantly g

288 Uptake in bone marrow, parotid glands, and tonsils was slightly but statistically significantly hig

289 es (a B cell line, a monocyte line and human tonsils) was reactive with HECA-452, a mAb that recogniz

290 a patient with acute infarction of the right tonsil, we found (1) nearly completely abolished ipsilat

291 B cells from peripheral blood and tonsils were assessed using multicolor flow cytometry, a

292 h head and neck carcinomas not involving the tonsils were included as control subjects.

293 sting IgM(+)IgD(+)CD27(-) B cells from human tonsils were labeled with CFSE and stimulated in vitro w

294 We found that 51% of tonsils were positive for Hi, and in 95% of cases analyz

295 ture in secondary lymphoid organs, including tonsils, where common pathogens, such as EBV, enter the

296 , tongue, cheeks, hard and soft palates, and tonsils, which are colonized by bacteria.

297 eloped a mucosal model, using human palatine tonsil with intact external epithelium, to study events

298 could initiate infection either through the tonsil, with spread to respiratory tissues, or through i

299 o evaluate association of FDG uptake between tonsils within control subjects.

300 ose that arise from the lingual and palatine tonsils within the oropharynx.