ライフサイエンスコーパス: tooth eruption

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1 ntoblastic processes until the completion of tooth eruption.

2 HrP-PPR system during root morphogenesis and tooth eruption.

3 tic mechanisms underlying root formation and tooth eruption.

4 n tooth tissue growth, but on the process of tooth eruption.

5 n opaque enamel that fails prematurely after tooth eruption.

6 re observed in the periostin-null mice after tooth eruption.

7 null mice dramatically deteriorate following tooth eruption.

8 ates the alveolar bone resorption needed for tooth eruption.

9 on osteoclastogenesis, root resorption, and tooth eruption.

10 cause the enamel secreting cells are lost at tooth eruption.

11 , and in the junctional epithelium following tooth eruption.

12 esorption and restores the normal program of tooth eruption.

13 ng cranial chondrodystrophy and a failure of tooth eruption.

14 llicle and bone remodeling are essential for tooth eruption.

15 have been associated with root formation and tooth eruption.

16 that the critical initial cellular event of tooth eruption, an influx of mononuclear cells into the

17 port the discovery of an agent that inhibits tooth eruption and also show that tooth eruption require

18 nsgenic mice cause osteopetrosis with normal tooth eruption and bone elongation and inhibit the devel

19 Also, PTHLH, a hormone involved in tooth eruption and invasive growth, was one of the most

20 "Rescued" animals lacked tooth eruption and showed premature epiphyseal closure,

21 nes and axial skeleton but apparently normal tooth eruption and skull plate development, indicating a

22 The reproductive, hematopoietic tooth eruption and tissue macrophage defects of CSF-1-de

23 iodontium, play a role in physiologic (e.g., tooth eruption) and pathologic (e.g., periodontitis) eve

24 ce, including growth retardation, failure of tooth eruption, and abnormal male and female reproductiv

25 osis, with no osteoclasts, marrow spaces, or tooth eruption, and exhibited profound growth retardatio

26 strategy for detecting variants involved in tooth eruption, and potentially craniofacial growth and

27 Root development and tooth eruption are very important topics in dentistry.

28 Root development accompanies rapid tooth eruption, but roots are required for the movement

29 ently corrected Csf1(op)/Csf1(op) defects of tooth eruption, eyelid opening, macrophage morphology, a

30 Experiments in vivo have established that tooth eruption fails in the absence of parathyroid hormo

31 ony-stimulating factor-1 (CSF-1) accelerates tooth eruption in rats and is localized in the dental fo

32 Tooth eruption is defined as the movement of a tooth fro

33 studies indicate that the timing of primary tooth eruption is highly heritable, with estimates typic

34 s that the down-regulation of OPG needed for tooth eruption is mediated by CSF-1.

35 A critical cellular event in tooth eruption is the formation of osteoclasts that are

36 severe enamel hypoplasia, delayed and failed tooth eruption, misshapen teeth, intrapulpal calcificati

37 inhibit the gene expression of the putative tooth eruption molecules, colony-stimulating factor-1 an

38 nd if its secretion is enhanced by potential tooth eruption molecules.

39 hat this bisphosphonate inhibits the time of tooth eruption of both rat molars and incisors.

40 bone, whether through the normal process of tooth eruption or by strains generated by orthodontic ap

41 teum of long bones, nor were they present in tooth eruption pathways.

42 t inhibits tooth eruption and also show that tooth eruption requires alveolar bone resorption.

43 cted and coincides better with estimates for tooth eruption times in Homo erectus.

44 To identify variants involved in primary tooth eruption, we performed a population-based genome-w

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