ライフサイエンスコーパス: tooth movement

1 herapies for patients undergoing orthodontic tooth movement.

2 stigate the effect of obesity on orthodontic tooth movement.

3 d and analyzed: hypoloading and experimental tooth movement.

4 acclimatization can be exploited to optimize tooth movement.

5 neration, and root resorption in orthodontic tooth movement.

6 and metalloprotease domain; OTM, orthodontic tooth movement.

7 n of certain cytokines decreases the rate of tooth movement.

8 eralized osteoporosity and increased rate of tooth movement.

9 e, increases the rate of bone remodeling and tooth movement.

10 r bone reactions, which can be exploited for tooth movement.

11 es of alveolar bone cells during orthodontic tooth movement.

12 bone remodeling associated with orthodontic tooth movement.

13 Osteoclasts play a vital role in orthodontic tooth movement.

14 ession in a rat model system of experimental tooth movement.

15 tients and associated with observed rates of tooth movement.

16 eful for experimentally limiting orthodontic tooth movement, a process involving perturbations of nor

17 BE reduced the amount of orthodontic tooth movement achieved in rats after 28 days.

18 alone, osteotomy-assisted tooth movement, or tooth movement alone.

19 force as applied to bone during orthodontic tooth movement and bone remodeling.

20 r bone alterations influenced by orthodontic tooth movement and can help determine risk assessment pr

21 t, while secondary outcomes included rate of tooth movement and change in clinical parameters (plaque

22 mineral metabolism contributing to disrupted tooth movement and exfoliation.

23 modeled throughout life, particularly during tooth movement and following tooth loss.

24 tongue, cheeks, and lips are known to cause tooth movement and in some situations can cause PTM.

25 f the alveolus has been an approach to speed tooth movement and is referred to as periodontally accel

26 tudying late stages of tooth development and tooth movement and the lack of good model systems.

27 ar spline regression to construct models for tooth movement and to identify factors associated with d

28 nical stress during mastication, orthodontic tooth movement, and wound healing following periodontal

29 ction osteogenesis in the osteotomy-assisted tooth movement animals.

30 This study shows that orthodontic tooth movement can be inhibited with the use of matrix m

31 patients had a significantly higher rate of tooth movement compared with normal-weight patients (+0.

32 with mastication, eruption, and orthodontic tooth movement-does the tissue increase its rate of cell

33 palatal implants that are placed to control tooth movement during orthodontic treatment and removed

34 reatment approaches in patients where buccal tooth movement (expansion) is planned in the anterior ma

35 e likely biologic mechanism underlying rapid tooth movement following selective alveolar decorticatio

36 inhibitors adjacent to teeth to limit local tooth movement in response to orthodontic forces.

37 the chief complaint and patient awareness o tooth movement in the last 5 years.

38 ry dentofacial therapy involving orthodontic tooth movement in the management of malocclusion with as

39 The probability of recession during tooth movement in thin biotype is high to justify gingiv

40 7 was increased in osteocytes in response to tooth movement in vivo.

41 Corticotomy-assisted and osteotomy-assisted tooth movement involves surgical incisions through the a

42 During orthodontic tooth movement, mechanical forces acting on periodontal

43 ounding natural teeth undergoing orthodontic tooth movement or influenced by orthopedic forces throug

44 n significantly increase the rate of initial tooth movement or reduce the amount of time required to

45 ovement, osteotomy alone, osteotomy-assisted tooth movement, or tooth movement alone.

46 ups: corticotomy alone, corticotomy-assisted tooth movement, osteotomy alone, osteotomy-assisted toot

47 Orthodontic tooth movement (OTM) causes transient pain and changes i

48 We tested whether orthodontic tooth movement (OTM) could be blocked by local administr

49 ition, cases in which there will be a facial tooth movement outside of the alveolar process need to b

50 o the maxillary molars inhibited orthodontic tooth movement (p < 0.01).

51 ry teeth, ankylosis, and/or slow orthodontic tooth movement, suggesting altered mineral metabolism co

52 We used a rat model of orthodontic tooth movement to test the hypothesis that periodontal l

53 Orthodontic tooth movement was inhibited by local delivery of Ilomas