ライフサイエンスコーパス: tooth surface

1 trol site (i.e., natural colonization of the tooth surface).

2 supragingival plaque (PI > or =2 on >40% of tooth surfaces).

3 luid-derived protein layer that forms on the tooth surface.

4 mutans attachment to and accumulation on the tooth surface.

5 liva, is a bacterial receptor that coats the tooth surface.

6 ortant role in microbial colonization of the tooth surface.

7 sease induced by a biofilm that forms on the tooth surface.

8 uw ) distribution caused by the drops on the tooth surface.

9 dhesion of this gram-positive species to the tooth surface.

10 droxyapatite (sHA), an in vitro model of the tooth surface.

11 hanisms to colonize and form biofilms on the tooth surface.

12 oped that correlate with mineral loss of the tooth surface.

13 sting and newly formed alveolar bone and the tooth surface.

14 the salivary pellicle coating supragingival tooth surfaces.

15 r that has a seamless interface with natural tooth surfaces.

16 the ability of some streptococci to colonize tooth surfaces.

17 antimicrobial protection to the mucosal and tooth surfaces.

18 ily exert their effects uniformly across all tooth surfaces.

19 odulate the dynamic formation of biofilms on tooth surfaces.

20 on cumulative caries increments on different tooth surfaces.

21 ry (d(2,3)fs) and total (d(2,3)fs+D(2,3)MFS) tooth surfaces.

22 the multispecies environment in biofilms on tooth surfaces.

23 and mediate sucrose-independent adherence to tooth surfaces.

24 rganisms, such as Streptococcus gordonii, to tooth surfaces.

25 anaerobes that initiate biofilm formation on tooth surfaces.

26 ed separate criteria for smooth and occlusal tooth surfaces.

27 olonizers that initiate biofilm formation on tooth surfaces.

28 ing to be 27% for decayed and 14% for filled tooth surfaces.

29 protection against S. mutans colonization of tooth surfaces.

30 protein layer formed from natural saliva on tooth surfaces, acquired enamel pellicle (AEP), protects

31 outcomes within individual patients, such as tooth surfaces affected by a caries lesion, tended to be

32 hypothesize that such polishing may protect tooth surfaces against the damage caused by cariogenic b

33 the children had at least 1 unfilled decayed tooth surface and 33% had at least 1 filled surface.

34 occus sanguinis is an early colonizer of the tooth surface and competes with oral pathogens such as S

35 sucrose to initiate biofilm formation on the tooth surface and consequently produces lactic acid to d

36 romote accumulation of microorganisms on the tooth surface and further establishment of pathogenic bi

37 are initiated by bacteria that colonize the tooth surface and gingival sulcus, the host response is

38 uced by mutans streptococci demineralize the tooth surface and lead to dental caries.

39 us parasanguis is a primary colonizer of the tooth surface and plays a pivotal role in the formation

40 cocci and actinomyces and their adherence to tooth surface and the associated host cells are key earl

41 s streptococci are primary colonizers of the tooth surface and thus form the foundation for the compl

42 Actinomyces naeslundii to the saliva-coated tooth surface and to each other.

43 ciation between caries of the mid- dentition tooth surfaces and AJAP1 (p value = 2e-8), a gene possib

44 val dental plaque differ substantially among tooth surfaces and children of different caries activiti

45 It mediates attachment of S. mutans to tooth surfaces and has been a focus for immunization stu

46 f filled (p < 0.001) and decayed (p = 0.036) tooth surfaces and negatively associated with number of

47 les that selectively adsorb from saliva onto tooth surfaces and provides a protective interface betwe

48 tial adherence of S. mutans to saliva-coated tooth surfaces and subsequent development of dental cari

49 elies on the bacterium's ability to colonize tooth surfaces and survive a strongly acidic environment

50 er of decayed, missing, and filled permanent tooth surfaces and teeth, and the number of decayed, and

51 nd the number of decayed, and filled primary tooth surfaces and teeth.

52 utcomes (e.g., number of carious and missing tooth surfaces), and oral health-related quality of life

53 yces are the major initial colonizers of the tooth surface, and the interactions between them and the

54 sms are not so effective against biofilms on tooth surfaces, and oral hygiene measures such as brushi

55 tached gingiva, the calculus deposition over tooth surfaces, and the subgingival calculus that enable

56 al pH fluctuations within the biofilm on the tooth surface are a ubiquitous and natural phenomenon, t

57 Biofilms formed on tooth surfaces are comprised of mixed microbiota enmeshe

58 Tooth surfaces are exposed to different proteinaceous mi

59 silica nanoparticles were used to polish the tooth surfaces, as compared with conventional polishing

60 Tooth surfaces at baseline (preparation), after restorat

61 g the permanent dentition into categories of tooth surfaces based on co-occurrence of caries) to nomi

62 al remained over the dentin and at the outer tooth surface, but between these regions were invading f

63 Colonization of the tooth surface by actinomyces and viridans group streptoc

64 ted sequential nature of colonization of the tooth surface by members of different genera can be inve

65 parable to the shear stress generated at the tooth surface by salivary flow ( approximately 0.8 dyn/c

66 gen membrane that readily adapts to bone and tooth surfaces by a gel formation of collagen fibers and

67 Percentages of tooth surfaces carrying dental plaque were 41% and 36% f

68 time, ease, number of strokes, and gingival/tooth surfaces changes were recorded.

69 For instance, tooth surfaces close to the gingival sulcus contact seru

70 g a split-mouth design, with half the buccal tooth surfaces coated with serum and the other half with

71 S. sanguinis, which yields understanding of tooth surfaces colonization and contributions to dental

72 Our null hypothesis is that tooth-surface defect dimensions, resulting from instrume

73 led teeth (DMF-Teeth) and decayed and filled tooth surfaces (DF-Surfaces).

74 tion time points, the microbiomes of healthy tooth surfaces differed substantially from those found d

75 d INT impact on decayed, missing, and filled tooth surfaces (dmfs) increment compared with UC.

76 umber of decayed, missing and filled primary tooth surfaces (dmfs) were used as a repeated outcome me

77 tococcus mutans uses adhesin P1 to adhere to tooth surfaces, extracellular matrix components, and oth

78 ed significantly more cellular attachment to tooth surfaces formerly covered by subgingival calculus

79 ary colonizer of the multispecies biofilm on tooth surfaces forming dental plaque and a potential age

80 ctinomyces that initiate colonization of the tooth surface frequently coaggregate with each other as

81 ibronectin in the attachment of cells to the tooth surface has been of considerable interest.

82 arasanguis FW213, a primary colonizer of the tooth surface, has been purified from the culture medium

83 ccus parasanguis, a primary colonizer of the tooth surface, has long, peritrichous fimbriae.

84 , clusters 1-5, groups of similarly behaving tooth surfaces identified through hierarchical clusterin

85 , clusters 1-5, groups of similarly behaving tooth surfaces identified through hierarchical clusterin

86 human dental caries, lives primarily on the tooth surface in biofilms.

87 the number of untreated carious and missing tooth surfaces in adulthood.

88 5-2.9) and filled (OR, 1.4; 95% CI, 1.1-1.8) tooth surfaces in deciduous but not in permanent teeth.

89 of intact dental plaque biofilms on natural tooth surfaces in human subjects.

90 cus gordonii to the acquired pellicle of the tooth surface involves specific interactions between bac

91 rasanguis FW213, a primary colonizer, to the tooth surface is mediated mainly by peritrichous long fi

92 Attachment of S. mutans to the tooth surface is required for the development of caries

93 n is initiated by bacteria that colonize the tooth surface, leading to inflammation and bone resorpti

94 herefore, we used hierarchical clustering on tooth surface-level caries data for 1,068 Appalachian ad

95 Children had a mean of 15 tooth surfaces (median, 14) restored during the 5-year p

96 ar trial period, children had a mean of 18.7 tooth surfaces (median, 16) restored in the amalgam grou

97 ion in its ability to adhere in the in vitro tooth surface model, saliva-coated hydroxylapatite (SHA)

98 restorations placed in previously unrestored tooth surfaces of 4,672 patients by 222 clinicians in th

99 genes may exert differential effects across tooth surfaces of the dentition.

100 teeth (OR = 0.86; 95% CI, 0.60 to 1.22) and tooth surfaces (OR = 0.85; 95% CI, 0.59 to 1.21) were ex

101 09) and a positive trend toward more decayed tooth surfaces (p = 0.027).

102 Plaque samples from caries-free tooth surfaces (PF) and from enamel carious lesions (PE)

103 C-terminal AgI/II fragments to the putative tooth surface receptor salivary agglutinin (SAG), as mon

104 ment loss can vary depending on the external tooth-surface reference points used.

105 Although repeated tooth-surface-specific information is commonly collected

106 was the sum of weighted counts of changes in tooth surface status over 13 months.

107 three coaggregating early colonizers of the tooth surface (Streptococcus gordonii DL1, Streptococcus

108 t dentition can be subdivided into groups of tooth surfaces that are useful for understanding the fac

109 sis yielded evidence of 5 distinct groups of tooth surfaces that differ with respect to caries: (C1)

110 pragingival dental plaque was collected from tooth surfaces that were caries-lesion-free (PF) and fro

111 can be applied to at least three levels: the tooth surface, the individual, or the group/population.

112 MGS adhere rapidly to saliva-coated tooth surfaces, thereby providing an attachment substrat

113 proteins can promote cell attachment to the tooth surface; therefore, attempts have been made to uti

114 osteoclastic bone resorption on the coronal tooth surface to form an eruption pathway.

115 The time needed to clean each tooth/surface was recorded.

116 essary first step toward colonization of the tooth surface, we sought to determine what effect deleti

117 The number of untreated carious and missing tooth surfaces were associated with oral health-related

118 Resulting tooth surfaces were evaluated with a laser metrology sys

119 Images of the tooth surfaces were obtained by video camera and convert

120 ccus mutans proliferates as a biofilm on the tooth surface, where it obtains nutrients and metabolize

121 , initiate the formation of oral biofilms on tooth surfaces, which requires differential expression o

122 e that 23.8 million persons have one or more tooth surfaces with > or = 3 mm gingival recession; 53.2

123 during oral surgery should saturate exposed tooth surfaces with amounts of fibronectin adequate for

124 ales had a lower number of untreated coronal tooth surfaces with caries (1.5), but a higher mean numb

125 on using a dataset with 3 levels of nesting: tooth surfaces within an interproximal (IP) region, IP r

126 es outcomes defined as the number of carious tooth surfaces within each cluster.