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1 who were provided nutritional support using total parenteral nutrition.
2 transpyloric feeding tubes, and 7% received total parenteral nutrition.
3 ng bone marrow transplantation and receiving total parenteral nutrition.
4 tion included short-bowel syndrome requiring total parenteral nutrition.
5 tically ill neonates, and patients receiving total parenteral nutrition.
6 mia during intensive insulin therapy than is total parenteral nutrition.
7 ne dose typically administered with standard total parenteral nutrition.
8 ial barrier function that is associated with total parenteral nutrition.
9 icantly increased with the administration of total parenteral nutrition.
10 antly lower than in wild-type mice receiving total parenteral nutrition.
11 t-bowel syndrome, which required him to have total parenteral nutrition.
12 surviving graft recipients are weaned off of total parenteral nutrition.
13 asis remain complex problems for patients on total parenteral nutrition.
14 ation predicts the duration of dependence on total parenteral nutrition.
15 with (odds ratio [95% confidence interval]): total parenteral nutrition (2.79 [1.26-6.17]), dialysis
16 9418], P<0.001), and the incidence of use of total parenteral nutrition (31 percent vs. 55 percent, P
17 dence interval, 1.8 to 148.1) and receipt of total parenteral nutrition (adjusted odds ratio, 9.2; 95
19 ion, alcohol use, use of medications, recent total parenteral nutrition, age at symptom onset, recent
21 studies indicate that copper requirements in total parenteral nutrition amount to 0.3 mg/day in the a
22 o distinct physiological differences between total parenteral nutrition and enteral nutrition that ar
23 thers do not, and why some patients tolerate total parenteral nutrition and others develop liver dysf
25 ediate care unit, chronic care requirements (total parenteral nutrition and tracheostomy), specific d
26 l of villus atrophy by the administration of total parenteral nutrition, and a model of villus hypert
27 Nonimmune animals were randomized to chow or total parenteral nutrition, and after 5 days of diet wer
32 tohepatitis, primary sclerosing cholangitis, total parenteral nutrition-associated liver disease, and
33 Patients with short-gut syndrome may develop total parenteral nutrition-associated liver disease, whi
35 d glucose into the systemic circulation with total parenteral nutrition at rates that approximate usu
36 e premature infants, patients with long-term total parenteral nutrition, Crohn's disease, cystic fibr
37 lbumin to hypoalbuminemic patients receiving total parenteral nutrition does not improve morbidity or
38 tter define the parameters that best predict total parenteral nutrition failure and the unique mechan
40 following an active infection, seven of nine total parenteral nutrition-fed animals continued to have
43 by carbachol and glucose were higher in the total parenteral nutrition group compared with the contr
46 atio, 5.8 [95% CI, 3.0 to 11.3]), receipt of total parenteral nutrition (hazard ratio, 4.1 [CI, 2.3 t
49 osing cholangitis, cholestasis of pregnancy, total parenteral nutrition-induced cholestasis, and drug
50 o assess the role of interferon-gamma on the total parenteral nutrition-induced loss of epithelial ba
51 y injured patients compared with intravenous total parenteral nutrition (IV TPN) or no nutritional su
54 al permeability in interferon-gamma knockout total parenteral nutrition mice was significantly lower
55 Although there is evidence to suggest that total parenteral nutrition more effectively spares prote
57 rition: odds ratio, 2.65; 95% CI, 1.93-3.63; total parenteral nutrition: odds ratio, 3.27; 95% CI, 2.
60 ointestinal complications (P = 0.03), use of total parenteral nutrition (P < 0.001), and lung transpl
65 en are noted with stones in association with total parenteral nutrition, prolonged fasting, or ileal
66 comparable with patients given conventional total parenteral nutrition regimens, even when criticall
69 de shortage of injectable zinc available for total parenteral nutrition supplementation over the last
71 uding much higher serum insulin responses to total parenteral nutrition than with enteral nutrition t
72 400 IU of vitamin D(3) and was dependent on total parenteral nutrition that contained 200 IU of vita
74 d male rats (approximately 235 g) were given total parenteral nutrition (TPN) and treated with recomb
75 ntrations in hospitalized patients beginning total parenteral nutrition (TPN) and whether a 3-d regim
76 medium-chain triacylglycerols (MCTs) during total parenteral nutrition (TPN) enhanced macrophage res
78 40% to 60% of infants who require long-term total parenteral nutrition (TPN) for intestinal failure
79 cadaver donors have become an alternative to total parenteral nutrition (TPN) for the treatment of ir
84 showed that enteral nutrient deprivation or total parenteral nutrition (TPN) led to a loss of intest
85 t bowel syndrome are maintained on long-term total parenteral nutrition (TPN) or more frequently cons
88 ich involved removal of amino acids from the total parenteral nutrition (TPN) solution for 8 h before
90 ed to receive for > or = 5 d tube feeding or total parenteral nutrition (TPN) that had identical ener
91 aper we show that feeding chemically defined total parenteral nutrition (TPN) to genetically normal,
92 birth weight (VLBW) infants are dependent on total parenteral nutrition (TPN) to prevent hypoglycemia
93 e enteroplasty [STEP]) in terms of survival, total parenteral nutrition (TPN) weaning, and complicati
94 nit and receiving mechanical ventilation and total parenteral nutrition (TPN) were measured for > or
96 nts were females with a history of long-term total parenteral nutrition (TPN) with TPN-related choles
97 jejunal early enteral nutrition (NJEEN) with total parenteral nutrition (TPN), after pancreaticoduode
98 essional interest, with special reference to total parenteral nutrition (TPN), an area in which I hav
99 occurrence of significant infection, days of total parenteral nutrition (TPN), and days of injectable
100 ics and antifungals, and ICU factors such as total parenteral nutrition (TPN), blood product transfus
101 In several models, including rats given total parenteral nutrition (TPN), IGF-I more potently st
103 a model of enteral nutrient deprivation, or total parenteral nutrition (TPN), resulting in intestina
105 inal failure patients do well with long-term total parenteral nutrition (TPN), while others develop l
108 liver disease (one), A-1-A deficiency (one), Total Parenteral Nutrition (TPN)-related (one), cryptoge
114 port was provided to rats for 7 days by oral total parenteral nutrition (TPN; elemental diet) 307 kca
115 , and judicious jejunostomy tube feeding, or total parenteral nutrition usage may reduce morbidity.
118 The percentages of infants who depended on total parenteral nutrition were 17 of 36 (47.2 percent)
119 , tumor size larger than 10 cm, and need for total parenteral nutrition were shown to further define
120 standard therapy for short bowel syndrome is total parenteral nutrition, which is expensive and assoc
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