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1 nuated in a guinea pig model of diphtheritic toxemia.
2 importance of beta toxin for type C-induced toxemia.
3 ugh a combination of bacterial infection and toxemia.
4 ent to counteract the detrimental effects of toxemia.
5 nhanced by sustained hypoxemia, acidosis and toxemia.
6 found only in case patients with measurable toxemia.
7 entify shared and unique consequences of the toxemias.
8 ibute to a cytokine storm formerly known as "toxemia." A more precise understanding of disease pathog
9 al role as a research tool for understanding toxemia and effects of medical countermeasures for anthr
12 he existence of a strong correlation between toxemia and the occurrence of systemic disease, supporti
13 ic infection is characterized by septicemia, toxemia, and meningitis, the main neurological complicat
15 -arginine amide delays anthrax toxin-induced toxemia both in cells and in live animals, suggesting th
16 aboratory-confirmed case of adult intestinal toxemia botulism in an allogeneic hematopoietic stem cel
17 itor, as revealed herein, can ameliorate the toxemia characteristic of an active B. anthracis infecti
18 e pregnancy-related complications, including toxemia, congestive heart failure, endocarditis and myoc
21 era and body fluids of infected animals, and toxemia is significantly correlated with the development
23 ssay, (ii) 100% protection in a lethal mouse toxemia model against recombinant LF and protective anti
24 as playing a causal role in the etiology of toxemia, mortality from this disease has not varied over
25 xhibited enhanced sensitivity to LPS-induced toxemia, notable for an increase in vascular permeabilit
29 pregnancies in mothers with KTx experienced toxemia of pregnancy, and one mother with SPKTx develope
31 experimental nonhuman primate models for Stx toxemias provides a reproducible platform for testing an
34 thrax and the first to associate acute stage toxemia with subsequent antitoxin antibody responses.
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