ライフサイエンスコーパス: toxic action

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1 hemical properties, toxicities, and modes of toxic action.

2 c metabolism, specific and reactive modes of toxic action, activation of adaptive stress response pat

3 philin-based mechanism could account for the toxic actions attributed to certain noncoplanar PCB cong

4 cidal proteins known as colicins exert their toxic action by forming a large, nonselective channel in

5 m dodecylbenzenesulfonate revealed a mode of toxic action consistent with oxidative stress and detrim

6 This toxic action greatly limits the use of NSAIDs in inflamm

7 ty of guanidinocalixarenes in inhibiting LPS toxic action has previously been related to their capaci

8 e curve in accordance with a similar mode of toxic action (i.e., concentration addition).

9 he notion that at least one target of lead's toxic action is the voltage-gated calcium channel.

10 The mode of toxic action (MOA) is recognized as a key determinant of

11 caused developmental lethality, indicating a toxic action not remedied by pharmacochaperoning.

12 d new hypotheses regarding the mechanisms of toxic action of DY7.

13 tive on how Fe deficiency contributes to the toxic action of individual heavy metals.

14 ng NO synthesis, both acting by inhibiting a toxic action of NO that is critical to tissue injury.

15 proteins that protect S. cerevisiae from the toxic action of osmotin.

16 The toxic action of rotenone is attributed to inhibition of

17 l neurodegenerative diseases but the mode of toxic action of Tau is poorly understood.

18 ase domain were isolated which destroyed the toxic action of the colicin.

19 Mechanisms of toxic action of the three compounds were also dependent

20 oves Abeta from the brain and transduces the toxic actions of Abeta.

21 tion with apoE receptors may not mediate the toxic actions of apoE4, because receptor-associated prot

22 There is no approved means to prevent the toxic actions of cocaine.

23 yconitine attenuated both the protective and toxic actions of DMXB, but in temporally distinct manner

24 he mechanisms underlying the therapeutic and toxic actions of general anesthetics helps us reframe th

25 arrays would protect the retina against the toxic actions of methanol-derived formic acid in a roden

26 presence of nAChRs sensitizes neurons to the toxic actions of soluble oligomeric Abeta, perhaps contr

27 We also show that the toxic actions of this biocide are zinc dependent and req

28 Here we show that aldosterone exerts direct toxic actions on myocardium by oxidative activation of C

29 lated via ingestion have a different mode of toxic action than metals taken up from water.

30 DLCs elicit their toxic action through activation of the aryl hydrocarbon

31 etrachlorodibenzo-p-dioxin (TCDD) exerts its toxic action via the aryl hydrocarbon (Ah) receptor, whi

32 he antibody combining site was fast, and its toxic action was unimpeded by this delivery mechanism.

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