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1 , patients develop a syndrome of relative T3 toxicosis.
2 nvestigate the phenotypic effects of cadmium toxicosis.
3 pivotal role in the protection from cadmium toxicosis.
4 sed role of the MURR1 gene in hepatic copper toxicosis.
5 gation of survival in a murine model of Stx1 toxicosis.
6 aversion induced by lithium chloride (LiCl) toxicosis.
7 h status of Wilson disease and canine copper toxicosis.
8 ne when paired with lithium chloride-induced toxicosis.
9 TA), a behavioral response characteristic of toxicosis.
10 tein associated with a form of canine copper toxicosis.
11 which reproduce Wilson's disease with copper toxicosis, and their normal counterparts, Long-Evans Ago
13 ulation in Wilson's disease and other copper toxicosis disorders and in cells cultured under high cop
16 cently, we established linkage of the copper toxicosis gene to a microsatellite marker, which has mad
20 potential therapeutic approach against Shiga toxicosis, is a model for the study of metal-regulated p
23 prived rats initially received a single odor-toxicosis or odor/taste-toxicosis pairing and were subse
24 d cirrhosis (which we term idiopathic copper toxicosis, or ICT) is copper accumulating to excess in t
25 ceived a single odor-toxicosis or odor/taste-toxicosis pairing and were subsequently tested, in separ
28 n animals diagnosed with chronic domoic acid toxicosis, the fornix showed signs of altered diffusion
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