ライフサイエンスコーパス: trabecular

1 l-endocardial distributions were as follows: trabecular 26.1% and subendocardial 20.2%, midwall 33.4%

2 of dysregulated RhoA GTPase activity in the trabecular AH outflow pathway increases intraocular pres

3 ctin, collagen-1A, and total collagen in the trabecular AH outflow pathway.

4 els of collagen-1A and total collagen in the trabecular AH outflow pathway.

5 ld coefficient served as cutoff to recognize trabecular and compact bone.

6 ark the Nppa(+) or Hey2(+) cardiomyocytes as trabecular and compact components of the ventricular wal

7 that is composed of cells derived from both trabecular and compact layers.

8 veral-fold elevated Wnt16 expression in both trabecular and cortical bone compared with wild type (WT

9 Muscle attenuation as well as trabecular and cortical bone densities revealed negative

10 ted tomography (HR-pQCT), we demonstrate low trabecular and cortical bone density contributing to low

11 ns, all materials showed positive outcome on trabecular and cortical bone formation in extraction soc

12 layed reduced peak bone mass and age-related trabecular and cortical bone loss.

13 Estradiol increased the trabecular and cortical bone mass as well as the uterine

14 Treatment with estradiol increased the trabecular and cortical bone mass to a similar extent in

15 sitive cell lineage show significantly lower trabecular and cortical bone mass, serum and bone marrow

16 salpha(OsxKO) mice) yielded markedly reduced trabecular and cortical bone mass.

17 Both trabecular and cortical bone measurements by micro-CT di

18 d and evaluated for bone mineral density and trabecular and cortical bone microarchitecture.

19 We show that the trabecular and cortical osteopenia in Cpdm mice is solel

20 -term SPI diet diminished the loss of total, trabecular, and cortical bone mineral density, whereas S

21 r the diagnosis and monitoring of changes in trabecular architecture associated with periodontitis.

22 Tibial trabecular bone among participants completing 70% or mor

23 becular bone mass and attenuated PTH-induced trabecular bone anabolism, supporting the positive funct

24 However, loss of trabecular bone and bone strength were most severe at th

25 at any anatomical site or alter cortical and trabecular bone and geometry.

26 and signaling were increased in Gja1(Jrt)/+ trabecular bone and osteogenic stromal cell cultures, wh

27 Increased trabecular bone and sinusoidal space and decreased arter

28 th Pyle's disease, have increased amounts of trabecular bone and unusually thin cortical bone, as a r

29 de that up-regulation of BMP2/4 signaling in trabecular bone and/or stromal cells increases osteoblas

30 SBP (subchondral bone plate) and B.Ar/T.Ar (trabecular bone area to total tissue area).

31 In this case-control study, trabecular bone biopsies from iliac crest were collected

32 s well as reduction of mineral bone density, trabecular bone content, and subcutaneous fat.

33 Assessment of microdamage within the trabecular bone core was performed using synchrotron X-r

34 ne in humans may significantly underestimate trabecular bone damage sustained by ART.

35 ls, whereas the relationship between PTH and trabecular bone decreases was bimodal.

36 We measured trabecular bone densities, cortical bone densities, VAT

37 ed with progressive declines in cortical and trabecular bone density at the peripheral skeleton.

38 The low trabecular bone density found in hemophilia is attribute

39 The newly formed trabecular bone displayed similar biomechanical properti

40 parathyroidism, and simultaneously increased trabecular bone formation and trabecular connectivity, a

41 indicating that Bmpr1a signaling suppresses trabecular bone formation through effectors beyond Smad4

42 of muscle mass and strength, and the loss of trabecular bone in femurs and vertebrae following Folfir

43 Newly formed immature delicate trabecular bone in fibrovascular marrow filled the space

44 strogen response in NOER mice), cortical and trabecular bone in long bones, as well as uterus and thy

45 that ERalpha in osteocytes is important for trabecular bone in male mice and for cortical bone in bo

46 results demonstrated an increased amount of trabecular bone in MULTI calluses at 21 days post-injury

47 n response was highly tissue-dependent, with trabecular bone in the axial skeleton being strongly dep

48 in vitro, suggesting that the restoration of trabecular bone in vivo was due to decreased bone resorp

49 -out mice, which are resistant to Pb-induced trabecular bone loss and maintain their mechanical bone

50 e protected against PTH-induced cortical and trabecular bone loss as well as from increases in serum

51 CD8+ T cells may further contribute to trabecular bone loss in some patients with advanced AIDS

52 last activity and an overall TMJ subchondral trabecular bone loss in the UAC-treated rats.

53 Osteoporosis is characterised by trabecular bone loss resulting from increased osteoclast

54 uction, stimulate bone resorption, and cause trabecular bone loss, demonstrating that the gut microbi

55 is central in sex steroid deficiency-induced trabecular bone loss.

56 caused by sex steroid deficiency, leading to trabecular bone loss.

57 lar joints (TMJs), displaying as subchondral trabecular bone loss.

58 ht be useful for treatment of postmenopausal trabecular bone loss.

59 formation, bone resorption, and cortical and trabecular bone loss.

60 tion of ART, causes significant cortical and trabecular bone loss.

61 c BMD and bone microstructure indicated that trabecular bone mainly contributed to the positive assoc

62 cumulation of a hyaluronan matrix within the trabecular bone marrow, and adipocytes and macrophages e

63 is (MAFIA) mouse model] reduced cortical and trabecular bone mass and attenuated PTH-induced trabecul

64 an opposite effect was found with increased trabecular bone mass and increased PTH-induced anabolism

65 It acted as an ERalpha agonist on trabecular bone mass and uterine weight, whereas no effe

66 Dmp1-Cre, we observed a dramatic increase in trabecular bone mass in postnatal mice, which was due to

67 omy (ovx) reduced the total body BMD and the trabecular bone mass to the same degree in Obl-Wnt16 mic

68 y caused mainly by a substantial increase in trabecular bone mass, resulting in improved bone strengt

69 lls specifically in postnatal mice increased trabecular bone mass.

70 ssion of WNT16 surprisingly increases mainly trabecular bone mass.

71 hes the osteoclastogenic deficit and reduces trabecular bone mass.

72 n wild type animals causes a 50% decrease in trabecular bone mass.

73 iously shown that MGUS patients have altered trabecular bone microarchitecture compared with controls

74 nt LT showed severe deficits in cortical and trabecular bone microarchitecture.

75 odifications of the aBMD and of cortical and trabecular bone microstructures.

76 Trabecular bone mineral density (BMD) was determined in

77 ne mineral content and density, cortical and trabecular bone mineral density (BMD), BMC, and bone are

78 Total and trabecular bone mineral density were significantly lower

79 diabetic rat model, there is a large loss of trabecular bone mineral density without apparent proport

80 antitative CT and microtomography to measure trabecular bone of limb epiphyses (long bone articular e

81 or phantomless in vivo BMD assessment of the trabecular bone of lumbar vertebrae and enables freely r

82 postprocessing dual-energy CT software, the trabecular bone of lumbar vertebrae L1-L4 were analyzed

83 .Dn), and by increased separation (Tb.Sp) in trabecular bone of the femur and vertebra.

84 mid-L5 level, the mean CT attenuation of the trabecular bone of the L5 vertebral body (L5HU) was meas

85 st activity in the tissue of TMJ subchondral trabecular bone of these UAC-treated rats was also enhan

86 anced osteoclast activity in TMJ subchondral trabecular bone of UAC-treated rats.

87 habitual tool manufacture, have a human-like trabecular bone pattern in the metacarpals consistent wi

88 Pb and HFD each reduced trabecular bone quality and together had a further detri

89 Further, muCT analysis of trabecular bone revealed that, compared with the vehicle

90 micro-computed tomography of a human femoral trabecular bone sample, allowing full 3D reconstruction

91 Trabecular bone score and in vivo microindentation are n

92 Trabecular bone score was borderline lower (1.21 +/- 0.1

93 Trabecular bone score was measured by specific software

94 ed by the development of methods such as the trabecular bone score, which helps assess bone microarch

95 udy the association between KTR and BMD/BMSi/trabecular bone score.

96 In a rodent sepsis model, trabecular bone strength is functionally reduced within

97 ion of bortezomib (Bzb) reversed the loss of trabecular bone structure and strength in mice at 4 wk a

98 le of extant primates to assess variation in trabecular bone structure in the human hip joint.

99 at more highly mobile human populations have trabecular bone structure similar to what would be expec

100 Proximal femur trabecular bone structure was quantified from microCT da

101 Skinner and colleagues, based on metacarpal trabecular bone structure, argue that Australopithecus a

102 ystemic bone loss in CIA mice by maintaining trabecular bone structure.

103 utant PTH1R exhibited a dramatic increase in trabecular bone that was dependent upon expression of Gs

104 Increased subchondral trabecular bone turnover due to imbalanced bone-resorbin

105 Analyses of mass-corrected trabecular bone variables reveal that the forager popula

106 matically recognized compact bone volume and trabecular bone volume (IBV) in CT slices.

107 th CLP for 2 weeks had significantly reduced trabecular bone volume and cortical bone thickness, asso

108 S3 has been ablated in osteocytes, have high trabecular bone volume and poorly defined metaphyseal co

109 g ACVR2A had significantly increased femoral trabecular bone volume at 6 weeks of age.

110 tomography revealed robust deterioration of trabecular bone volume by both subsets, while CD4+ T cel

111 in signaling in vivo and completely restored trabecular bone volume by increasing bone formation and

112 egative effects on bone, as shown by reduced trabecular bone volume fraction (BV/TV), thickness (Tb.T

113 olic effect of intermittent PTH treatment on trabecular bone volume is blunted by deletion of Gsalpha

114 In contrast, trabecular bone volume is not altered in these mice.

115 0 mug/kg/day) for 4 weeks failed to increase trabecular bone volume or cortical thickness in male and

116 gnificant reduction in bone mineral density, trabecular bone volume, and cortical bone thickness comp

117 d ACVR2B demonstrated sustained increases in trabecular bone volume, similar to those in ACVR2A singl

118 graphy analysis of femurs revealed increased trabecular bone volume, thickness, number, and connectiv

119 d thoracic spine, with an associated loss of trabecular bone volume.

120 In this study, PEDF delivery increased trabecular bone volume/total volume by 52% in 6-mo-old P

121 ortical bone was -0.250, and between BMI and trabecular bone was -0.143 (all P < 0.001).

122 Trabecular bone was expanded in ACLL patients and occupi

123 ac bone sample from individual 2 showed that trabecular bone was hypermineralized on the material lev

124 d densities of psoas muscle and cortical and trabecular bone were -0.460, -0.407, and -0.434, respect

125 lly migrated and colonized tenascin-C-coated trabecular bone xenografts in a novel system that employ

126 d the opposite bone phenotype, with 14% less trabecular bone, 22% fewer osteoblasts, and 10% thinner

127 KO mice compared WT controls, with 14% more trabecular bone, 35% more osteoblasts, 73% fewer osteocl

128 Besides increasing osteoblast number in the trabecular bone, deletion of Bmpr1a by Dmp1-Cre also not

129 med quantitative differences in cortical and trabecular bone, including decreased cortical thickness

130 ese included fracture sites with predominant trabecular bone, not previously reported as being associ

131 pha antagonist all protect cortical, but not trabecular bone, revealing complex effects of T-cell rec

132 logical analysis showed reduced cortical and trabecular bone, suggesting cell-autonomous functions of

133 that accompany prostate cancer metastasis to trabecular bone, with potential implications to therapeu

134 gions are present in the disordered phase of trabecular bone.

135 and no statistically significant changes in trabecular bone.

136 imals demonstrated a significant increase in trabecular bone.

137 astic bone consisting of mature cortical and trabecular bone.

138 ells and often, but not always, located near trabecular bone.

139 l size and perfection in remnant metaphyseal trabecular bone.

140 deficiency of sFRP4, that cortical-bone and trabecular-bone homeostasis were governed by different m

141 ulation occurred in the epiphyseal plates of trabecular bones.

142 inant of intraocular pressure and success of trabecular bypass glaucoma surgeries.

143 onic inflammatory process that demineralizes trabecular cancellous bone.

144 version of neonatal endocardial cells during trabecular compaction generates a distinct compartment o

145 The trabecular compartment of the same bones was spared, as

146 Parietal bone trabecular compartment was mildly altered.

147 Increased myocardial trabecular complexity is one of several preclinical abno

148 usly increased trabecular bone formation and trabecular connectivity, and decreased cortical porosity

149 ession with those in epithelial, stromal and trabecular corneal cells, we selected 9 structural or fu

150 2.1%, and 3.1%, respectively; all P<0.001), trabecular density (4.4%; P<0.001), and stiffness and fa

151 other primate taxa and (ii) the reduction in trabecular density first occurred in early Homo erectus,

152 Thus, the low trabecular density of the recent modern human skeleton e

153 show that only recent modern humans have low trabecular density throughout the limb joints.

154 heses that (i) recent modern humans have low trabecular density throughout the upper and lower limbs

155 n humans today appear to have relatively low trabecular density, but little is known about how that d

156 Moreover, deletion of PKD1 in vivo reduced trabecular development and activity of osteoblast develo

157 sms underlying appropriate downregulation of trabecular ERBB2 signaling are little understood.

158 o calculate the texture parameters along the trabecular fibers in the lower neck area for all subject

159 analysis can quantitatively discriminate the trabecular integrity alterations induced by periodontiti

160 angle opening distance (AOD500, AOD750), and trabecular iris space area (TISA500, TISA750) were measu

161 t (LV), and angle opening distance (AOD750), trabecular iris space area (TISA750), and iris thickness

162 increases in AOD750, angle recess area, and trabecular iris surface area (P < 0.05 for all).

163 Trabecular-iris angle (TIA) and angle opening distance 5

164 The iridocorneal angle measurements: trabecular-iris angle (TIA), angle opening distance (AOD

165 Trabecular-iris angle (TIA), angle opening distance 500

166 ce (AOD), trabecular-iris space area (TISA), trabecular-iris circumference volume (TICV), length of i

167 Trabecular-iris contact (TIC) was observed in 8 eyes of

168 ACA parameters angle opening distance (AOD), trabecular-iris space area (TISA), trabecular-iris circu

169 e (TIA), angle opening distance (AOD500) and trabecular-iris space area (TISA500) 500 mum from the sc

170 and 750 mum (AOD750) from the scleral spur; trabecular-iris space area at 500 mum (TISA500) and 750

171 and 750 mum anterior from scleral spur), the trabecular-iris-space area (TISA, measured 500 and 750 m

172 Nkx2-5(+/R52G) mice demonstrated a prominent trabecular layer in the ventricular wall, so called nonc

173 These protrusions can interact within the trabecular layer to form new cell-cell contacts.

174 l surface, moving their cell bodies into the trabecular layer while elaborating more protrusions.

175 any myocardial cell bodies have entered the trabecular layer, cardiomyocytes extend protrusions that

176 oncentrically encircles the ridge-like inner trabecular layer.

177 id over a premineralized area, and an inner, trabecular-like, endochondral bone, generated mainly by

178 that suppress PTH will prevent cortical and trabecular losses and post-transplant fractures.

179 Microtomography of D2J mice showed decreased trabecular mass, compared to that in wild-type mice [DBA

180 ered in the ciliary muscle (46 +/- 5.6%) and trabecular meshwork (37 +/- 8.3%) of treated eyes relati

181 adly expressed in most tissues including the trabecular meshwork (TM) and heterozygous Sod2 knockout

182 conventional outflow pathway comprising the trabecular meshwork (TM) and Schlemm's canal (SC).

183 The IOP is maintained by the trabecular meshwork (TM) and the elevation of IOP in ope

184 Primary cultures of human trabecular meshwork (TM) cell monolayers were treated wi

185 We have previously shown that trabecular meshwork (TM) cells might detect aqueous humo

186 took advantage of the phagocytic property of trabecular meshwork (TM) cells, and developed a novel ma

187 GC-induced myocilin expression in the trabecular meshwork (TM) has been suggested to play an i

188 To determine if trabecular meshwork (TM) height differs between primary

189 However, their potential role in the trabecular meshwork (TM) in the eye, which regulates int

190 f extracellular matrix (ECM) proteins in the trabecular meshwork (TM) is associated with TM dysfuncti

191 ctional changes in the unlasered portions of trabecular meshwork (TM) of laser-treated primate eyes a

192 Because the trabecular meshwork (TM) plays an important role in aque

193 Given that the trabecular meshwork (TM) provides most of aqueous humor

194 The trabecular meshwork (TM) tissue controls drainage of aqu

195 Trabecular meshwork (TM) tissue in the eye is under cons

196 to endoplasmic reticulum (ER) stress in the trabecular meshwork (TM), the tissue that regulates IOP.

197 was associated with chronic ER stress of the trabecular meshwork (TM).

198 liary body (CB) and its drainage through the trabecular meshwork (TM).

199 50]) and angle opening (all 4 quadrants with trabecular meshwork [TM] visible on gonioscopy after LPI

200 expressing rats was associated with fibrotic trabecular meshwork and increased levels of F-actin, pho

201 pressed within human retina, optic nerve and trabecular meshwork and that ABCA1 and AFAP1 are also ex

202 comatous subjects were analyzed to determine trabecular meshwork anteroposterior length and 3 anterio

203 d a significant positive association between trabecular meshwork anteroposterior length and all anter

204 d a significant positive association between trabecular meshwork anteroposterior length and all anter

205 ere used to evaluate the association between trabecular meshwork anteroposterior length and anterior

206 Mean trabecular meshwork anteroposterior length was 824.86 +/

207 ior chamber angle is associated with greater trabecular meshwork anteroposterior length.

208 sured as the perpendicular distance from the trabecular meshwork at 500 mum and 750 mum anterior to t

209 nd/or vehicle, or mechanical blockade of the trabecular meshwork by protein aggregates or contaminant

210 function in vitro by infecting primary human trabecular meshwork cells and in vivo by injecting livin

211 cular meshwork, which is a scenario toxic to trabecular meshwork cells and leads to early onset of oc

212 By visual inspection, human trabecular meshwork cells infected with scAAV2.dnRhoA sh

213 bolites may increase oxidative damage to the trabecular meshwork cells, resulting in elevation of int

214 ed mutant myocilin toxicity in primary human trabecular meshwork cells.

215 In this clinic-based population, trabecular meshwork height is shorter in PACG patients c

216 Trabecular meshwork height was measured from the scleral

217 creased resistance to AH outflow through the trabecular meshwork in ocular hypertension patients.

218 ression is enriched in the smooth muscle and trabecular meshwork in the eye.

219 t well understood but has been attributed to trabecular meshwork injury from repeated injections, a p

220 ce to aqueous humor (AH) outflow through the trabecular meshwork is a primary risk factor for open-an

221 ance in the aqueous drainage tract distal to trabecular meshwork is potentially an important determin

222 h more advanced glaucoma suggesting that the trabecular meshwork is the primary impediment to outflow

223 and a resulting fibrotic activity within the trabecular meshwork may result in ocular hypertension, w

224 orylation of ERK1/2 was also observed in the trabecular meshwork of transgenic mice expressing 6-fold

225 ened at least 180 degrees (visible posterior trabecular meshwork on gonioscopy) after laser iridotomy

226 m for this complication may be injury to the trabecular meshwork resulting from rapid elevations in I

227 Trabecular meshwork size may play a role in ethnic diffe

228 he juxtacanalicular connective tissue of the trabecular meshwork together with inner wall endothelium

229 eyes were classified as narrow if posterior trabecular meshwork was not visible and open if the angl

230 ced damage to the anterior chamber angle and trabecular meshwork, and reduced postoperative use of st

231 of severely hypomorphic Schlemm's canal and trabecular meshwork, as well as elevated IOP, demonstrat

232 Both ABCA1 and PMM2 are expressed in the trabecular meshwork, optic nerve and other ocular tissue

233 acellularly instead of being secreted to the trabecular meshwork, which is a scenario toxic to trabec

234 of caveolae in the Schlemm's canal (SC) and trabecular meshwork.

235 n of SLT was performed to 360 degrees of the trabecular meshwork.

236 low structures, Schlemm's canal (SC) and the trabecular meshwork.

237 the posterior cornea initiating beneath the trabecular meshwork.

238 lt rhesus macaques by laser treatment to the trabecular meshwork.

239 IL-27 treatment prevented the loss of trabecular micro-architecture and preserved cortical bon

240 We assessed outflow enhancement by trabecular micro-bypass (TMB) implantation or by ab inte

241 Endoscopic cyclophotocoagulation, trabecular micro-bypass stent, ab interno trabeculectomy

242 n are novel techniques that directly measure trabecular microarchitecture and mechanical properties o

243 Despite persistent decrease in BMD, trabecular microarchitecture and tissue quality remain n

244 e, which is mainly related to changes in the trabecular microstructure.

245 hECTs resembled trabecular muscle and beat spontaneously (1.18 +/- 0.48

246 mal formation of the ventricular walls.Fetal trabecular muscles in the heart undergo a poorly describ

247 Failure of trabecular myocytes to undergo appropriate cell cycle wi

248 Trabecular separation and trabecular network heterogeneity were higher (+24.3% and

249 LT recipients had lower trabecular number (-9.7%; P = .004) and lower trabecular

250 histomorphometry showed consistently reduced trabecular number and connectivity.

251 lia is attributed to significantly decreased trabecular number and increased separation; the lower co

252 This effect is due to a higher trabecular number in these mice.

253 becular thickness, trabecular separation and trabecular number of femur and lumbar, serum osteocalcin

254 e femur metaphysis was associated with lower trabecular number, whereas trabecular thickness and numb

255 amax) was proportionate to a 38% increase in trabecular number.

256 WT mice had reduced trabecular thickness and trabecular number.

257 ower bone resorption with either maintained (trabecular) or higher (cortical) bone formation as compa

258 intraoperative gauge of patency through the trabecular outflow pathway.

259 d across species or how and when the present trabecular patterns emerged over the course of human evo

260 helps to differentiate normal from abnormal trabecular patterns in healthy versus diseased hearts.

261 , which induces a thinning of trabeculae and trabecular perforations.

262 rimordium and subsequent persistence of deep trabecular recesses in the myocardial wall.

263 ITS revealed predominant change in trabecular rods, and EDX confirmed less mineralization.

264 Trabecular separation and trabecular network heterogenei

265 mineral density (BMD), trabecular thickness, trabecular separation and trabecular number of femur and

266 Group EP presented greater bone porosity, trabecular separation, and connective tissue attachment

267 , collagen I, and osteocalcin; but increased trabecular separation, osteoclast number and surface, an

268 of trabecular thickness (Tb.Th) (-34.1%) and trabecular spacing (Tb.Sp) (-41.0%).

269 and connective tissue density with decreased trabecular spacing compared with WT.

270 icant change in bone volume/total volume and trabecular spacing, but it simultaneously damaged the bo

271 agriculturalist and forager populations for trabecular spacing, number, or degree of anisotropy.

272 and trabecular thickness, with a decrease in trabecular spacing.

273 l-mediated RV stiffness was determined in RV trabecular strips.

274 analysis to discriminate the changes in the trabecular structure of interdental bone between individ

275 ith no differences in femur length, cortical/trabecular structure or mineral density, or mechanical p

276 expressed in adult tibiae, including at the trabecular surfaces and in cortical osteocytes, epiphyse

277 These findings suggest that trabecular texture analysis might contribute information

278 integrity by using computed tomographic (CT) trabecular texture analysis of the lumbar spine in patie

279 Trabecular texture analysis was performed by using softw

280 performed to determine associations between trabecular texture and body composition.

281 to determine body composition predictors of trabecular texture.

282 rabecular number (-9.7%; P = .004) and lower trabecular thickness (-8.1%; P = .025).

283 cture model index (+119%), and a decrease of trabecular thickness (Tb.Th) (-34.1%) and trabecular spa

284 ciated with lower trabecular number, whereas trabecular thickness and number were lower in the epiphy

285 d group, the PTH-treated WT mice had reduced trabecular thickness and trabecular number.

286 This study reveals that NFPs regulate trabecular thickness by inhibiting Notch1 signaling, con

287 [BV] divided by trabecular volume [TV]) and trabecular thickness was noted in the ABBM group at both

288 At 24 months, bone formation rate, trabecular thickness, and bone volume were higher in the

289 ight/obese hip OA patients exhibited reduced trabecular thickness, increased bone surface/bone volume

290 l density, spinal bone volume/tissue volume, trabecular thickness, mechanical strength, and material

291 ing weight loss, bone mineral density (BMD), trabecular thickness, trabecular separation and trabecul

292 sed bone volume fraction, tissue density and trabecular thickness, with a decrease in trabecular spac

293 defects in p57 expression, proliferation and trabecular thickness.

294 o previous reports, the present study showed trabecular thinning, higher numbers of apoptotic osteocy

295 ng to median value, patients with a ratio of trabecular to skeletal bone volume of more than 37.3% sh

296 ive bone volume (bone volume [BV] divided by trabecular volume [TV]) and trabecular thickness was not

297 ted participants had lower mean z scores for trabecular volumetric BMD (-0.85), cortical volumetric B

298 Trabecular volumetric BMD was lower in coinfected than i

299 4 liver fibrosis had lower mean z scores for trabecular volumetric BMD, cortical thickness, and total

300 IV/HCV-coinfected women had decreased tibial trabecular volumetric BMD, diminished cortical dimension