ライフサイエンスコーパス: trabecular bone

1 ells and often, but not always, located near trabecular bone.

2 genetic knockdown of Nox4 mitigated loss of trabecular bone.

3 ereas in A. robustus it is a column of dense trabecular bone.

4 l size and perfection in remnant metaphyseal trabecular bone.

5 B number, serum human osteocalcin level, and trabecular bone.

6 completely blocked the tumor-induced loss of trabecular bone.

7 face was equivalent to that of the untreated trabecular bone.

8 tau(Gamma) depends only on the structure of trabecular bone.

9 ions of adipocytes in the marrow cavities of trabecular bone.

10 gions are present in the disordered phase of trabecular bone.

11 astic bone consisting of mature cortical and trabecular bone.

12 and no statistically significant changes in trabecular bone.

13 imals demonstrated a significant increase in trabecular bone.

14 ulation occurred in the epiphyseal plates of trabecular bones.

15 d wide long bones, reduced bone collars, and trabecular bones.

16 exhibited severe osteopenia in cortical and trabecular bones.

17 omography measurements were taken at the 4% (trabecular bone), 20% (cortical bone), and 66% (for meas

18 d the opposite bone phenotype, with 14% less trabecular bone, 22% fewer osteoblasts, and 10% thinner

19 KO mice compared WT controls, with 14% more trabecular bone, 35% more osteoblasts, 73% fewer osteocl

20 sity (+62%), accelerated age-related loss of trabecular bone (-61%), and markedly reduced B-cell numb

21 Tibial trabecular bone among participants completing 70% or mor

22 becular bone mass and attenuated PTH-induced trabecular bone anabolism, supporting the positive funct

23 d with marrow from KO kindred showed loss of trabecular bone analogous to KO mice, consistent with in

24 dge width, including a greater percentage of trabecular bone and a higher bone density compared to co

25 However, loss of trabecular bone and bone strength were most severe at th

26 ates with histological evidence of decreased trabecular bone and CFU-osteoblast colonies at 4 and 8 m

27 mputed tomography confirmed the reduction in trabecular bone and connective tissue densities in GPR10

28 d with control knees, accompanied by loss of trabecular bone and erosion of subchondral bone surface.

29 at any anatomical site or alter cortical and trabecular bone and geometry.

30 the antibody does increase both cortical and trabecular bone and improves bone mechanical properties

31 rogate-spongiosa volume (combined tissues of trabecular bone and marrow)-can be used to accurately sc

32 and signaling were increased in Gja1(Jrt)/+ trabecular bone and osteogenic stromal cell cultures, wh

33 Finally, Wnt10b-/- mice have decreased trabecular bone and serum osteocalcin, confirming that W

34 Increased trabecular bone and sinusoidal space and decreased arter

35 cular surface and is required for sustaining trabecular bone and skeletal growth.

36 The material behavior of the trabecular bone and teeth was always treated as isotropi

37 inclusion of other features like thinning of trabecular bone and the anisotropy of the network.

38 th Pyle's disease, have increased amounts of trabecular bone and unusually thin cortical bone, as a r

39 de that up-regulation of BMP2/4 signaling in trabecular bone and/or stromal cells increases osteoblas

40 animals show a marked reduction in calcified trabecular bone, and an abnormal persistence of hypertro

41 s showed a decreased density of cortical and trabecular bone, and there was biochemical evidence for

42 Decreases in cortical bone, trabecular bone, and whole-bone failure strength were 7.

43 cro-finite-element models for cortical bone, trabecular bone, and whole-bone section were generated f

44 Decreases in cortical bone, trabecular bone, and whole-bone stiffness were 3.7% (P =

45 ensity, lower femoral bone mass, and altered trabecular bone architecture were observed in F508del Cf

46 d risedronate prevented the deterioration of trabecular bone architecture, reduced the degree of mine

47 dels constructed from samples of iliac crest trabecular bone are shown to be in agreement with this a

48 ficiency on ligature-induced bone loss (BL), trabecular bone area (TBA), and postextraction bone heal

49 e showed that Smad1C significantly increases trabecular bone area and length of trabecular surface co

50 +) HSCs were more frequently detected in the trabecular bone area compared with compact bone area, an

51 SBP (subchondral bone plate) and B.Ar/T.Ar (trabecular bone area to total tissue area).

52 Trabecular bone, as compared with cortical bone, appeare

53 h DEX and Scl-ABI, a significant increase in trabecular bone at the femoral metaphysis (bone volume/t

54 cy appeared to be protective against loss of trabecular bone at the lumbar spine.

55 The volumetric density of the trabecular bone at the spine increased substantially in

56 icularly evident for bone mineral density in trabecular bone at the spine on quantitative CT (an incr

57 nuation values (in Hounsfield units [HU]) of trabecular bone between the T12 and L5 vertebral levels,

58 In this case-control study, trabecular bone biopsies from iliac crest were collected

59 TG mice had progressively increased trabecular bone but decreased HSC function.

60 nges in the proximal tibia) was abolished in trabecular bone but not in the cortical component.

61 lds were generated from fully decellularized trabecular bone by using digitized clinical images, seed

62 Cellular isolates from trabecular bone cavities contained approximately 65-fold

63 hat CD45(low)CD271+ MSCs are abundant in the trabecular bone cavity and indistinguishable from aspira

64 type, with different effects on cortical and trabecular bone compartments.

65 s well as reduction of mineral bone density, trabecular bone content, and subcutaneous fat.

66 Assessment of microdamage within the trabecular bone core was performed using synchrotron X-r

67 es of aluminum and polycarbonate to simulate trabecular bone, cortical bone, and cartilage.

68 ne in humans may significantly underestimate trabecular bone damage sustained by ART.

69 ls, whereas the relationship between PTH and trabecular bone decreases was bimodal.

70 Besides increasing osteoblast number in the trabecular bone, deletion of Bmpr1a by Dmp1-Cre also not

71 We measured trabecular bone densities, cortical bone densities, VAT

72 ed with progressive declines in cortical and trabecular bone density at the peripheral skeleton.

73 The low trabecular bone density found in hemophilia is attribute

74 ing for changes in serum biochemical values, trabecular bone density using micro-computed tomography,

75 A have decreased skeletal size, muscle mass, trabecular bone density, cortical bone geometry, and str

76 All JRA patients had decreased tibia trabecular bone density, cortical bone size and strength

77 proliferation in the growth plate and lower trabecular bone density.

78 ling, in TGF-beta1-induced chondrogenesis of trabecular bone-derived MPCs.

79 nctions to couple growth plate maturation to trabecular bone development in growing mice.

80 cy affected development of both cortical and trabecular bone differently, effects apparently dependen

81 The newly formed trabecular bone displayed similar biomechanical properti

82 conducted in 4 different 3D voxel models of trabecular bone for sources localized in the active marr

83 , delayed vascular recruitment and defective trabecular bone formation (resulting in drastically shor

84 intravenous injection of LLP2A-Ale increased trabecular bone formation and bone mass in both xenotran

85 conclusion, ERalpha in osteocytes regulates trabecular bone formation and thereby trabecular bone vo

86 parathyroidism, and simultaneously increased trabecular bone formation and trabecular connectivity, a

87 rox20 expression in osteoblasts, and because trabecular bone formation is arrested in Egr2/Krox20-kno

88 Trabecular bone formation is dramatically diminished in

89 and showed a dose dependent increase in the trabecular bone formation rate in ovariectomized rats fo

90 (Rs1) to address how the massive increase in trabecular bone formation resulting from increased G(s)

91 indicating that Bmpr1a signaling suppresses trabecular bone formation through effectors beyond Smad4

92 d locally over murine calvaria, and enhanced trabecular bone formation when administered systemically

93 increased osteoclastic activity and reduced trabecular bone formation.

94 simultaneously results in massively elevated trabecular bone formation.

95 The inner porous (or trabecular) bone from these sites, which resemble disord

96 Measurements included cortical and trabecular bone geometry, density, and strength at the d

97 deficiency of sFRP4, that cortical-bone and trabecular-bone homeostasis were governed by different m

98 Skeletal analysis revealed that trabecular bone in adult homozygous skeletons was sparse

99 e in male mice but it is dispensable for the trabecular bone in female mice and the cortical bone in

100 of muscle mass and strength, and the loss of trabecular bone in femurs and vertebrae following Folfir

101 Newly formed immature delicate trabecular bone in fibrovascular marrow filled the space

102 radiographic trabecular patterns by removing trabecular bone in four sequential steps from six cadave

103 strogen response in NOER mice), cortical and trabecular bone in long bones, as well as uterus and thy

104 that ERalpha in osteocytes is important for trabecular bone in male mice and for cortical bone in bo

105 trogen in females, but it is dispensable for trabecular bone in male mice and for cortical bone in bo

106 results demonstrated an increased amount of trabecular bone in MULTI calluses at 21 days post-injury

107 n response was highly tissue-dependent, with trabecular bone in the axial skeleton being strongly dep

108 with hypercholesterolemia lose cortical and trabecular bone in the femurs and vertebrae (bone minera

109 Trabecular bone in the resulting digital images was meas

110 f the Hh effector Smoothened (Smo) increased trabecular bone in vivo and inhibited osteoclastogenesis

111 in vitro, suggesting that the restoration of trabecular bone in vivo was due to decreased bone resorp

112 s) that suppress bone remodeling will change trabecular bone in ways such that the size of the failur

113 ts that suppress bone remodeling will change trabecular bone in ways such that the size of the failur

114 es several types of architectural changes in trabecular bone including thinning, increased levels of

115 med quantitative differences in cortical and trabecular bone, including decreased cortical thickness

116 of osteoclastogenesis, completely inhibited trabecular bone loss (-2.2+/-11.9%, P<0.01).

117 Additionally, LLP2A-Ale prevented trabecular bone loss after peak bone acquisition was ach

118 he field of osteoimmunology help explain the trabecular bone loss and generalized osteoporosis linked

119 -out mice, which are resistant to Pb-induced trabecular bone loss and maintain their mechanical bone

120 e protected against PTH-induced cortical and trabecular bone loss as well as from increases in serum

121 CD8+ T cells may further contribute to trabecular bone loss in some patients with advanced AIDS

122 last activity and an overall TMJ subchondral trabecular bone loss in the UAC-treated rats.

123 Osteoporosis is characterised by trabecular bone loss resulting from increased osteoclast

124 After ovariectomy, cortical and trabecular bone loss was reduced by 50% in HDC(-/-) mice

125 uction, stimulate bone resorption, and cause trabecular bone loss, demonstrating that the gut microbi

126 nstrate that Sh3bp2 "cherubism" mice exhibit trabecular bone loss, TNF-alpha-dependent systemic infla

127 formation, bone resorption, and cortical and trabecular bone loss.

128 tion of ART, causes significant cortical and trabecular bone loss.

129 is central in sex steroid deficiency-induced trabecular bone loss.

130 caused by sex steroid deficiency, leading to trabecular bone loss.

131 lar joints (TMJs), displaying as subchondral trabecular bone loss.

132 ht be useful for treatment of postmenopausal trabecular bone loss.

133 c BMD and bone microstructure indicated that trabecular bone mainly contributed to the positive assoc

134 s (MPCs), such as those from bone marrow and trabecular bone, makes them a useful model to investigat

135 cumulation of a hyaluronan matrix within the trabecular bone marrow, and adipocytes and macrophages e

136 n the endosteal surface in the calvarial and trabecular bone marrow.

137 eficient line, which included a reduction in trabecular bone mass and a functional deficit in bone st

138 is (MAFIA) mouse model] reduced cortical and trabecular bone mass and attenuated PTH-induced trabecul

139 entiated osteoblasts substantially increases trabecular bone mass and bone mineral density without af

140 an opposite effect was found with increased trabecular bone mass and increased PTH-induced anabolism

141 It acted as an ERalpha agonist on trabecular bone mass and uterine weight, whereas no effe

142 ow that the F508del mutation in CFTR impacts trabecular bone mass by reducing bone formation.

143 atase epsilon (PTPepsilon) exhibit increased trabecular bone mass due to cell-specific defects in ost

144 b skeletogenic mesenchyme markedly increased trabecular bone mass in adolescent mice.

145 Dmp1-Cre, we observed a dramatic increase in trabecular bone mass in postnatal mice, which was due to

146 t in OC differentiation culminated in a high trabecular bone mass pathology.

147 omy (ovx) reduced the total body BMD and the trabecular bone mass to the same degree in Obl-Wnt16 mic

148 er, suppressed marrow adipogenesis, restored trabecular bone mass, and reduced bone resorption.

149 GC treatment reduced trabecular bone mass, microarchitecture, and the degree

150 y caused mainly by a substantial increase in trabecular bone mass, resulting in improved bone strengt

151 hes the osteoclastogenic deficit and reduces trabecular bone mass.

152 whole body bone mineral density and reduced trabecular bone mass.

153 n wild type animals causes a 50% decrease in trabecular bone mass.

154 lls specifically in postnatal mice increased trabecular bone mass.

155 ssion of WNT16 surprisingly increases mainly trabecular bone mass.

156 iously shown that MGUS patients have altered trabecular bone microarchitecture compared with controls

157 nt LT showed severe deficits in cortical and trabecular bone microarchitecture.

158 odifications of the aBMD and of cortical and trabecular bone microstructures.

159 d reduced bone volume and area, cortical and trabecular bone mineral content, and density.

160 Trabecular bone mineral density (BMD) was determined in

161 ne mineral content and density, cortical and trabecular bone mineral density (BMD), BMC, and bone are

162 independent associations between volumetric trabecular bone mineral density (vBMD) of the lumbar spi

163 Trabecular bone mineral density of the lumbar spine was

164 Total and trabecular bone mineral density were significantly lower

165 diabetic rat model, there is a large loss of trabecular bone mineral density without apparent proport

166 rs by intramembranous ossification forming a trabecular bone network that replaces the amputated cort

167 ese included fracture sites with predominant trabecular bone, not previously reported as being associ

168 tes and osteoblasts showed that increases in trabecular bone occur independently of the improper cart

169 antitative CT and microtomography to measure trabecular bone of limb epiphyses (long bone articular e

170 or phantomless in vivo BMD assessment of the trabecular bone of lumbar vertebrae and enables freely r

171 postprocessing dual-energy CT software, the trabecular bone of lumbar vertebrae L1-L4 were analyzed

172 .Dn), and by increased separation (Tb.Sp) in trabecular bone of the femur and vertebra.

173 mid-L5 level, the mean CT attenuation of the trabecular bone of the L5 vertebral body (L5HU) was meas

174 the total body, total hip, femoral neck, and trabecular bone of the lumbar spine also differed signif

175 in the total body and femur neck but not in trabecular bone of the lumbar vertebra.

176 st activity in the tissue of TMJ subchondral trabecular bone of these UAC-treated rats was also enhan

177 anced osteoclast activity in TMJ subchondral trabecular bone of UAC-treated rats.

178 in the growth plate concomitant with radial trabecular bone orientation.

179 ese mutant mice exhibited continuous loss of trabecular bone over time, which was accompanied by redu

180 habitual tool manufacture, have a human-like trabecular bone pattern in the metacarpals consistent wi

181 ro-computed tomography scans showed that the trabecular bone proximal to the femoral head growth plat

182 Pb and HFD each reduced trabecular bone quality and together had a further detri

183 distal femur cortical bone region but not at trabecular bone region at the 1.4 and 2.8 mg/kg/d GC dos

184 by tumor in beta3-/- mice, but no associated trabecular bone resorption as seen inbeta3+/+ mice.

185 esidual osteopetrosis, significantly delayed trabecular bone resorption in the subepiphyseal region o

186 oth PPR*Tg and control mice, suggesting that trabecular bone resorption was comparably suppressed by

187 pha(-/-) female mice displayed an attenuated trabecular bone response to supraphysiological E2 treatm

188 Further, muCT analysis of trabecular bone revealed that, compared with the vehicle

189 pha antagonist all protect cortical, but not trabecular bone, revealing complex effects of T-cell rec

190 micro-computed tomography of a human femoral trabecular bone sample, allowing full 3D reconstruction

191 Trabecular bone score and in vivo microindentation are n

192 Trabecular bone score was borderline lower (1.21 +/- 0.1

193 Trabecular bone score was measured by specific software

194 ed by the development of methods such as the trabecular bone score, which helps assess bone microarch

195 udy the association between KTR and BMD/BMSi/trabecular bone score.

196 total area ratios, thicker cortical bone and trabecular bone, significantly higher bone mineral densi

197 ERalpha in osteoclasts is crucial for the trabecular bone-sparing effect of estrogen in females, b

198 We propose that the physiological trabecular bone-sparing effect of estrogen is mediated v

199 In a rodent sepsis model, trabecular bone strength is functionally reduced within

200 ion of bortezomib (Bzb) reversed the loss of trabecular bone structure and strength in mice at 4 wk a

201 le of extant primates to assess variation in trabecular bone structure in the human hip joint.

202 at more highly mobile human populations have trabecular bone structure similar to what would be expec

203 ng at 3.0 T provided a better measure of the trabecular bone structure than did MR imaging at 1.5 T.

204 Proximal femur trabecular bone structure was quantified from microCT da

205 Skinner and colleagues, based on metacarpal trabecular bone structure, argue that Australopithecus a

206 ystemic bone loss in CIA mice by maintaining trabecular bone structure.

207 essfully used in practice, especially in the trabecular bone studies because of high contrast between

208 logical analysis showed reduced cortical and trabecular bone, suggesting cell-autonomous functions of

209 ity in a subset of cells associated with the trabecular bone surface of long bones.

210 TRAP-positive osteoclast-covered trabecular bone surfaces also increased in microgravity

211 of TRAP(+) osteoclasts on distal metaphyseal trabecular bone surfaces were significantly decreased.

212 cells can be observed on most endosteal and trabecular bone surfaces.

213 g architectural changes in a given sample of trabecular bone; techniques to study such changes using

214 ull mice displayed significantly less tibial trabecular bone than wild-type mice.

215 red a distinct cell type associated with the trabecular bone that appears to possess osteogenic poten

216 utant PTH1R exhibited a dramatic increase in trabecular bone that was dependent upon expression of Gs

217 ency of Crebbp included a marked decrease in trabecular bone that was predominantly caused by increas

218 Changes in the volumetric density of trabecular bone, the cortical volume at the hip, and lev

219 In trabecular bone, the porosity of the mineralized matrix

220 her, MAGP1Delta mice have significantly less trabecular bone, the trabecular microarchitecture is mor

221 density and content, increased cortical and trabecular bone thickness, and greater net bone formatio

222 rrow, but likely resulted from contaminating trabecular bone; this was supported by reverse transcrip

223 hen vascular mesenchymal cells organize into trabecular bone tissue within the artery wall.

224 Increased subchondral trabecular bone turnover due to imbalanced bone-resorbin

225 Analyses of mass-corrected trabecular bone variables reveal that the forager popula

226 -) mice displayed a substantial reduction in trabecular bone volume (-20%, P < 0.01) compared with co

227 Femoral bone mineral density (12 vs. 27%), trabecular bone volume (32 vs. 48%), and cortical thickn

228 e mice, 6-week-old Runx2-II(+/-) had reduced trabecular bone volume (BV/TV%), cortical thickness (Ct.

229 of the femur are associated with changes in trabecular bone volume (BV/TV) with altered estrogen sta

230 matically recognized compact bone volume and trabecular bone volume (IBV) in CT slices.

231 mineral density (P=0.0074) and percentage of trabecular bone volume (P=0.007), and decreases in numbe

232 hic analyses demonstrated markedly increased trabecular bone volume and bone mineral density in femor

233 th CLP for 2 weeks had significantly reduced trabecular bone volume and cortical bone thickness, asso

234 mice also have small skeletons with reduced trabecular bone volume and cortical thickness and that c

235 Zol treatment increased trabecular bone volume and decreased osteoclast paramete

236 ice, M-PTH(1-34) induced larger increases in trabecular bone volume and greater increases in cortical

237 ion activities, contributing to the enhanced trabecular bone volume and mineral density in these TG m

238 ultinucleated osteoclast-like cells and more trabecular bone volume and number in 26-wk-old male IL-2

239 ll (knockout, KO) mice, we observed that the trabecular bone volume and number of trabeculae were sig

240 Their prevalence correlated with decreased trabecular bone volume and osteoid and osteoblast surfac

241 S3 has been ablated in osteocytes, have high trabecular bone volume and poorly defined metaphyseal co

242 ing female mice, IgGs significantly improved trabecular bone volume and structure and increased both

243 e mass, with SRC-2 KO mice having 80% higher trabecular bone volume as compared with wild type mice.

244 g ACVR2A had significantly increased femoral trabecular bone volume at 6 weeks of age.

245 female mice had no significant reduction in trabecular bone volume but ovariectomized Dmp1-ERalpha(-

246 tomography revealed robust deterioration of trabecular bone volume by both subsets, while CD4+ T cel

247 in signaling in vivo and completely restored trabecular bone volume by increasing bone formation and

248 ale and female mice exhibited an increase in trabecular bone volume due to an increase in osteoblasts

249 -);Rosa(Notch) mice exhibited an increase in trabecular bone volume due to decreased bone resorption

250 grem1 null male mice displayed increased trabecular bone volume due to enhanced osteoblastic acti

251 T of the distal radius to determine apparent trabecular bone volume fraction (BV/TV), apparent trabec

252 me CT of distal radius to determine apparent trabecular bone volume fraction (BV/TV), apparent trabec

253 egative effects on bone, as shown by reduced trabecular bone volume fraction (BV/TV), thickness (Tb.T

254 Structural parameters (trabecular bone volume fraction [BV/TV, bone volume to t

255 All three agents similarly increased trabecular bone volume in both PPR*Tg and control mice,

256 ss phenotype with an approximate doubling of trabecular bone volume in both the tibia and femur.

257 ulates trabecular bone formation and thereby trabecular bone volume in male mice but it is dispensabl

258 The Lef1DeltaN transgenic mice had higher trabecular bone volume in the proximal tibias and L5 ver

259 ondral bone thickness and increased relative trabecular bone volume in the tibial epiphysis.

260 Trabecular bone volume increased in 3-month-old Osx-Cre(

261 olic effect of intermittent PTH treatment on trabecular bone volume is blunted by deletion of Gsalpha

262 In contrast, trabecular bone volume is not altered in these mice.

263 Trabecular bone volume of the distal femoral metaphysis

264 0 mug/kg/day) for 4 weeks failed to increase trabecular bone volume or cortical thickness in male and

265 showed a significantly decreased bone volume/trabecular bone volume ratio, decreased trabecular numbe

266 d increased osteoclast numbers and decreased trabecular bone volume that was apparent at 10 weeks but

267 gnificant reduction in bone mineral density, trabecular bone volume, and cortical bone thickness comp

268 dependent reduction in bone mineral density, trabecular bone volume, and cortical thickness.

269 Cortical bone size, trabecular bone volume, bone mineralizing surfaces, and

270 creased bone size, bone mineral density, and trabecular bone volume, caused by impairment in osterix

271 rmation defect, fail to display increases in trabecular bone volume, cortical thickness, and bone for

272 t acid phosphatase staining revealed reduced trabecular bone volume, decreased cortical bone, and inc

273 ed bone mineral density, reduced mineralized trabecular bone volume, lower flexural strength, and his

274 Hes1 inactivation in osteoblasts increased trabecular bone volume, number, and connectivity due to

275 cts on bone as shown by higher bone mass and trabecular bone volume, number, and thickness and lower

276 d ACVR2B demonstrated sustained increases in trabecular bone volume, similar to those in ACVR2A singl

277 marrow fibrosis, despite similar effects on trabecular bone volume, suggests that marrow fibrosis wa

278 ed bone mineral density, cortical thickness, trabecular bone volume, thickness and number, and decrea

279 graphy analysis of femurs revealed increased trabecular bone volume, thickness, number, and connectiv

280 d thoracic spine, with an associated loss of trabecular bone volume.

281 ase bone turnover, bone mineral density, and trabecular bone volume.

282 on) and overall length, which led to reduced trabecular bone volume.

283 from mild to severe, as evidenced by reduced trabecular bone volume.

284 and an increase in trabecular thickness and trabecular bone volume/tissue volume in U/NP and U/HP gr

285 In this study, PEDF delivery increased trabecular bone volume/total volume by 52% in 6-mo-old P

286 nt osteoporotic deficit, including decreased trabecular bone volumes and reduced trabecular thickness

287 ortical bone was -0.250, and between BMI and trabecular bone was -0.143 (all P < 0.001).

288 Trabecular bone was expanded in ACLL patients and occupi

289 ac bone sample from individual 2 showed that trabecular bone was hypermineralized on the material lev

290 Trabecular bone was removed from each section in four st

291 d densities of psoas muscle and cortical and trabecular bone were -0.460, -0.407, and -0.434, respect

292 raphy (muCT), and changes in subchondral and trabecular bone were assessed by standard muCT.

293 Osteoblasts, osteoclasts, and trabecular bone were increased in TG mice without change

294 doubling in the number of osteoblasts lining trabecular bone, whereas osteoblast numbers in similarly

295 sed bone mineral density in the cortical and trabecular bone, whereas the bone mineral density of Fgf

296 Mmp13-null mice had increased trabecular bone, which persisted for months.

297 d smaller bodies with shorter limbs, reduced trabecular bone with thinner cortices, and decreased ost

298 that accompany prostate cancer metastasis to trabecular bone, with potential implications to therapeu

299 s paralysis and profound loss of ipsalateral trabecular bone within days.

300 lly migrated and colonized tenascin-C-coated trabecular bone xenografts in a novel system that employ