ライフサイエンスコーパス: tracheal tube

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1 c induction and mechanical ventilation via a tracheal tube.

2 ration values necessitating uncapping of the tracheal tube.

3 ide full barrier function and produce normal tracheal tubes.

4 cent of tracheal intubation were with cuffed tracheal tubes.

5 o asphyxiation brought on by fluid-congested tracheal tubes.

6 of the lumen matrix during maturation of the tracheal tubes.

7 e pressure ventilation by an endotracheal or tracheal tube, a PaO2:FiO2 less than 200 mm Hg with at l

8 ed fifty-two adult patients intubated with a tracheal tube allowing subglottic secretion suctioning w

9 ow and pressure measured at the inlet of the tracheal tube and expressed as resistance (Rrs) and reac

10 ted of deflating the cuff on the fenestrated tracheal tube and occluding the tube.

11 ed using calibrated syringes and appropriate tracheal tubes and connections.

12 of mechanical ventilation, the prevalence of tracheal tubes, and behavioral "learned nonuse" may all

13 tory systems, plant vascular systems, insect tracheal tubes, and other distribution networks.

14 n produce some ventilation with or without a tracheal tube, because the straight upper airways of ani

15 In all three mutants, developing tracheal tubes bud and extend normally, but the epitheli

16 After replacing the tracheal tube by a double-lumen one, we initiated latera

17 xchange respiratory gases in their system of tracheal tubes by using either diffusion or changes in i

18 ression appeared in late-stage embryos after tracheal tube formation, with individual PPK genes showi

19 AP kinase, is hyper-activated throughout the tracheal tube in the Pu mutant.

20 Tracheal tubes in mutant larvae are often crushed or twi

21 osis included transplant stricture (n = 13), tracheal tube injury (n = 10), inflammation (n = 6), tra

22 intervention may be precluded by changes in tracheal tube leak during the procedure.

23 the remaining 13 patients, the magnitude of tracheal tube leak increased by > or = 10% after delayed

24 claudins act in the same pathway controlling tracheal tube length.

25 r the Mucus Slurper (study group) or a Hi-Lo Tracheal Tube (Mallinckrodt, St.

26 thway in promoting cell intercalation during tracheal tube morphogenesis in Drosophila embryogenesis,

27 Here we show that three genes required for tracheal tube morphogenesis in Drosophila melanogaster e

28 A tracheal tube occlusion protocol can predict clinically

29 The tracheal tube occlusion protocol consisted of deflating

30 All patients underwent the tracheal tube occlusion protocol followed by bronchoscop

31 r, those patients who failed to tolerate the tracheal tube occlusion protocol had clinically importan

32 was undertaken to test the hypothesis that a tracheal tube occlusion protocol predicts clinically imp

33 xty-three (84%) of 75 patients tolerated the tracheal tube occlusion protocol.

34 or without cardiac arrest, with or without a tracheal tube, showed essentially no ventilation by ster

35 o not disrupt several processes required for tracheal tube size control, including septate junction f

36 netic program and cellular events underlying tracheal tube size control.

37 tinct from other known processes involved in tracheal tube size regulation.

38 s support epithelial SJ barrier function and tracheal tube-size control.

39 known septate junctions genes cause the same tracheal tube-size defects as ATPalpha and nrv2 mutation

40 sophila Dorsal Air Sac Primordium (ASP) is a tracheal tube that grows toward Branchless FGF-expressin

41 pgant35A) are recessive lethal and result in tracheal tubes that are irregular in diameter and morpho

42 larval development, which generate the adult tracheal tubes, the spiracle and the epidermis surroundi

43 sinuous have previously been shown to cause tracheal tubes to be elongated and have diameter increas

44 bunits of the Na+/K+ ATPase cause Drosophila tracheal tubes to have increased lengths and expanded di

45 who developed respiratory distress when the tracheal tube was occluded were deemed to have failed th

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