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1 irway epithelia (HAE) derived from nasal and tracheobronchial airway regions, we generated recombinan
2 were induced by mechanically stimulating the tracheobronchial airway.
3 sion intensified and was confined around the tracheobronchial airways, while it lessened during the p
4                Despite its localized nature, tracheobronchial amyloid deposition may be asymptomatic
5           Three of 4 patients with localized tracheobronchial amyloidosis required Nd:YAG (neodymium:
6                               Recognition of tracheobronchial anatomy and familiarity with the use of
7                    This review is focused on tracheobronchial anatomy and the use of flexible fiberop
8                      A complete knowledge of tracheobronchial anatomy is a key factor in determining
9                In addition, changes occur in tracheobronchial anatomy with age; therefore, it is very
10 previous bronchial resection, recognition of tracheobronchial anatomy with the fiberoptic bronchoscop
11 a doses and dose relative intensities in the tracheobronchial and alveolar regions of lungs were calc
12 essential for goblet cell differentiation in tracheobronchial and gastrointestinal epithelium of mice
13 osition of nanomaterial(s) will occur in the tracheobronchial and head airways--not in the alveolar r
14                                          The tracheobronchial ASL hypotonicity was hypothesized to re
15 alizes in adults to a discrete population of tracheobronchial basal cells.
16     CBF of basolaterally permeabilized human tracheobronchial cells, re-differentiated at the air-liq
17 d with a significantly greater difference in tracheobronchial damage at the carina and main bronchus.
18 th antiviral properties are present in human tracheobronchial epithelial (HTBE) cell culture secretio
19 duced mucosecretory differentiation in human tracheobronchial epithelial (HTBE) cells and compared wi
20  drug-activated gene (NAG-1) in normal human tracheobronchial epithelial (HTBE) cells and several lun
21                                        Human tracheobronchial epithelial (HTBE) cells from donors wit
22 d trigger beta-defensin (hBD2) mRNA in human tracheobronchial epithelial (hTBE) cells through a CD14-
23 erentiated squamous metaplastic normal human tracheobronchial epithelial (NHTBE) and mucous NHTBE cel
24 uction in normal, well differentiated, human tracheobronchial epithelial (NHTBE) cell cultures, witho
25 EB) in a nonclassical manner in normal human tracheobronchial epithelial (NHTBE) cells.
26  mucous cell differentiation of normal human tracheobronchial epithelial (NHTBE) cells.
27 hly differentiated cultures of primary human tracheobronchial epithelial (TBE) cells with a panel of
28 sfer technique on passage 1 culture of human tracheobronchial epithelial (TBE) cells, the Flag-SPRR1
29 yes were used with well-differentiated human tracheobronchial epithelial cell cultures exhibiting spo
30             We characterized passage 2 human tracheobronchial epithelial cell cultures morphologicall
31   In experiments using primary culture human tracheobronchial epithelial cells (hTBECs) and each of t
32 and tumor specimens than in the normal human tracheobronchial epithelial cells and adjacent normal lu
33 ance regulator (CFTR) complementation and in tracheobronchial epithelial cells from patients with ver
34 n A is able to suppress the proliferation of tracheobronchial epithelial cells in culture.
35                                     Human CF tracheobronchial epithelial cells in primary culture rel
36                  CBF and pHi of single human tracheobronchial epithelial cells in submerged culture w
37 xpression of the novel gene in primary human tracheobronchial epithelial cells in vitro.
38 nstrate that RSV infection of A549 and human tracheobronchial epithelial cells increases the amounts
39 ons were determined in primary culture human tracheobronchial epithelial cells transduced with gene t
40 and growth of selected reassortants in human tracheobronchial epithelial cells.
41 sociated with this effect using normal human tracheobronchial epithelial cells.
42  transcription signaling components in human tracheobronchial epithelial cells.
43 h was developed from a cDNA library of human tracheobronchial epithelial cells.
44 aling pathways that mediate the induction in tracheobronchial epithelial cells.
45 he PMA-inducible expression of the SPRR1B in tracheobronchial epithelial cells.
46                                    Polarized tracheobronchial epithelial cultures from normal and cys
47 's), yet the appearance of mature MCs in the tracheobronchial epithelial surface is a characteristic
48         Furthermore, HBD2 with CD14 in human tracheobronchial epithelium can complex with "toll-like
49 gs demonstrate a novel role for Duox1 in the tracheobronchial epithelium, in addition to its proposed
50  identified NADPH oxidase homolog within the tracheobronchial epithelium, in airway epithelial cell m
51  NAG-1 expression was observed in the normal tracheobronchial epithelium, whereas no expression was f
52 and the Nkx2.8-/- mice exhibited generalized tracheobronchial hyperplasia.
53 9), emergency esophagectomy (P = 0.013), and tracheobronchial injuries (P = 0.0011) were independent
54                                  We compared tracheobronchial injury following short-term intratrache
55                                 The combined tracheobronchial injury scores for all samples were sign
56            Located close to the heart at the tracheobronchial junction, vocal folds or membranes atta
57                           PURPOSE OF REVIEW: Tracheobronchial lesions requiring significant resection
58 ted for removal were partly eroding into the tracheobronchial lumen and 23 were free.
59               Viral RNA was also detected in tracheobronchial lymph node and myocardium, together wit
60  specific lymphoproliferative responses from tracheobronchial lymph node cells, immunoglobulin M (IgM
61 -FLU vaccine induced weaker BAL and stronger tracheobronchial lymph node responses and a lesser reduc
62 body-secreting cells (ASC) in the spleen and tracheobronchial lymph node.
63 nterferon (IFN-gamma)-secreting cells in the tracheobronchial lymph nodes as determined by enzyme-lin
64 ecognized by antibody-secreting B cells from tracheobronchial lymph nodes isolated immediately follow
65 positive (DP) T cells in the mediastinal and tracheobronchial lymph nodes of RALT.
66 ever, relatively high activity levels in the tracheobronchial lymph nodes of the beagles indicated th
67 ions while no alpha activity was seen in the tracheobronchial lymph nodes of this individual.
68 roduction of IFN-gamma by lymphocytes of the tracheobronchial lymph nodes was also detected.
69 at gene expression patterns in the lungs and tracheobronchial lymph nodes would fit into a coherent a
70  in blood, bronchoalveolar lavage (BAL), and tracheobronchial lymph nodes.
71 nhibited the binding activity of CD to human tracheobronchial mucin in a serum concentration-dependen
72              We have recently shown that the tracheobronchial mucin MUC5B is a major component of MG1
73 her lectin was involved in adhesion to human tracheobronchial mucin.
74 as to describe the management and outcome of tracheobronchial necrosis (TBN) after caustic ingestion.
75  day p.i. by TSA-ISH and in retropharyngeal, tracheobronchial, or external iliac lymph nodes and some
76 f human airway tissues derived from nasal or tracheobronchial regions, suggesting that SARS-CoV may i
77                   The lack of other Mesozoic tracheobronchial remains, and the poorly mineralized con
78  strongly express Nkx2.8 seem to function as tracheobronchial stem cells.
79 ful adjuncts for management of select benign tracheobronchial stenoses.
80                                 Attrition of tracheobronchial stent patency is most rapid during the
81                                          The tracheobronchial submucosal glands secrete liquid that i
82 tribution accurately reflected that in human tracheobronchial tissue.
83 cleus); 2) soft palate, pharynx, larynx, and tracheobronchial tree (e.g., dorsal, intermediate, and i
84   Twelve movies of the thoracic aorta (n=3), tracheobronchial tree (n=4), colon (n=3), paranasal sinu
85 air leaks - any extrusion of air outside the tracheobronchial tree - have been attributed to high ven
86 ng evidence suggesting that formation of the tracheobronchial tree and alveoli results from heterogen
87 d within the large conducting airways of the tracheobronchial tree being primarily responsible for oz
88 of partly eroded or free broncholiths in the tracheobronchial tree can be considered safe and effecti
89 m 11.5-day-old LKLF(-/-) embryos show normal tracheobronchial tree formation.
90 of dye placed in the subglottic space to the tracheobronchial tree in a rigid tracheal model and a be
91 l investigation, direct visualization of the tracheobronchial tree might be useful in determining the
92                                   The entire tracheobronchial tree obtained at autopsy was embedded i
93           No invasive tumor was found in the tracheobronchial tree or any other location.
94                   This "colonization" of the tracheobronchial tree, currently believed to be innocuou
95 f systemic and pulmonary vasculature and the tracheobronchial tree.
96 f the ears, nose, peripheral joints, and the tracheobronchial tree.
97  (15.5%) needles did not fully penetrate the tracheobronchial tree.
98 egion, or the more peripheral aspects of the tracheobronchial tree.

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