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1 al epiretinal membranes and/or vitreomacular traction.
2 ially in those without obvious vitreomacular traction.
3 ernal rounding forces and cell-intercalation traction.
4 ibroblasts were predominant in vitreomacular traction.
5 ascular vitreous veil with localized retinal traction.
6 ular hole after vitrectomy for vitreomacular traction.
7 ular hole after vitrectomy for vitreomacular traction.
8 hopsia in the right eye due to vitreomacular traction.
9 ive trend in identifying focal vitreomacular traction.
10  they are still small and have vitreomacular traction.
11 actile groups within clusters for generating traction.
12 ity and release of symptomatic vitreomacular traction.
13 s closure after vitrectomy for vitreomacular traction.
14 bstrate junctions with no apparent effect on traction.
15 ts to discover inhibitors have gained little traction.
16 ithout the need to explicitly determine cell tractions.
17  of variations in matrix stiffness with cell tractions.
18 s (57.1%), vasculitis (57.1%), vitreoretinal traction (57.1%), and chronic macular edema (ME) (71.4%)
19          Conversely, for focal vitreomacular traction (70 scans) and full-thickness MH (82 scans), 25
20 roduced by actin polymerization can generate traction across the plasma membrane by transmission thro
21 l motility, whereas the differences in their traction adhesion dynamics suggest that these two strain
22 lyzing the spatiotemporal evolution of their traction adhesions (TAs).
23 step-wise fashion, mainly forming stationary traction adhesions along their anterior-posterior axes a
24 e dynamics of the cell's mechanically active traction adhesions.
25 hanced iOCT imaging revealed strong vitreous traction and adhesion above the macula and optic disc.
26  dynamics, thus increasing cell motility and traction and enabling chemotaxis.
27 is verified experimentally by comparing cell traction and F-actin retrograde flow for two cell types
28 al fluid and, when applicable, vitreomacular traction and MH.
29                                        Using traction and monolayer stress microscopy, we show that S
30 ography showed the release of vitreo-macular traction and multifocal electroretinogram responses show
31 n injections in patients with vitreo-macular traction and reduced central visual acuity.
32 c retinopathy, vitreous hemorrhage, combined traction and rhegmatogenous retinal detachment, or lens
33 hat NAs transmit a distinguishable amount of traction and that NA maturation depends on traction grow
34 rylation and the CSD promotes focal adhesion traction and, thereby, cancer cell motility.
35                  Four of the 8 patients with traction and/or rhegmatogenous RD developed recurrent de
36 ng the magnitude and spatial distribution of tractions and cell speed on confined cells.
37 tral serous chorioretinopathy, vitreomacular traction, and full-thickness MH.
38          We also find that 90% of neutrophil tractions are in the out-of-plane axis, and this may be
39     Patient-funded trials (PFTs) are gaining traction as a means of accelerating clinical translation
40             Exon skipping by ASOs is gaining traction as a therapeutic strategy, and the use of ASOs
41 ptical coherence tomography (OCT) has gained traction as an important adjunct for clinical decision m
42 ime correlated random walks" are now gaining traction as models of scale-finite animal movement patte
43 Ca(2+) oscillations induced by laser-tweezer-traction at the plasma membrane, providing a model to st
44 M) revealed that cells produced the greatest tractions at the cell periphery, where distinct types of
45  forces in motile tissues and show that such traction-based stresses match those calculated from inst
46  clinical practice who had possible UIP with traction bronchiectasis on HRCT and had not undergone su
47 f nintedanib if they had honeycombing and/or traction bronchiectasis plus reticulation, without atypi
48 patterns supporting a potential mechanism of traction by Muller cells in the CB.
49 ore susceptible to damage from vitreomacular traction by rotational and/or acceleration-deceleration
50 thick stage 3 membranes with anteroposterior traction concerning for progression to stage 4 ROP (3 ey
51 acting rheological information directly from traction data.
52                                     Vitreous traction does not seem to play a significant pathogenic
53 s with small macular holes and vitreomacular traction during vitrectomy after intravitreal ocriplasmi
54 this study was to evaluate the extension and traction effects of posterior vitreous detachment (PVD)
55                          The strength of the traction exerted by the vitreous on the fovea seems to b
56              The method allows computing the traction field from the substrate displacements within t
57 quisition of continuous in- and out-of-plane traction fields with high sensitivity.
58 s mediate adhesive interactions that provide traction for cell migration.
59 ented organic electronics from gaining rapid traction for sensing applications.
60 al cells on stiff substrates decreased their traction force (from 300 nN to 100 nN) and spread area (
61 ls with inhibited myosin II motors increased traction force (from 41 nN to 63 nN) and slightly reorie
62 , to our knowledge, a novel method to assess traction force after long-term (24 h) uniaxial or biaxia
63  stretch, the cells had similar decreases in traction force and area and reoriented perpendicular to
64 g network, simulations predict the resulting traction force and FN fibril formation.
65  study, we examined the relationship between traction force and vinculin-paxillin localization to sin
66     However, the study of how cell-generated traction force changes in response to stretch is general
67 le-particle tracking (MPT) microrheology and traction force cytometry to probe how genetic induction
68                             In addition, the traction force developed by cells has to reach a minimal
69         Interestingly, the absolute level of traction force did not correlate with growth cone advanc
70 hanism by which FN fibril assembly regulates traction force dynamics in response to mechanical stimul
71                                          The traction force exerted by the growth cone was measured b
72 model that predicts the dynamics of cellular traction force generation and subsequent assembly of fib
73 inding, vinculin was necessary for efficient traction force generation in 3D collagen without affecti
74 he talin rod R3 subdomain decreases cellular traction force generation, which affects talin and vincu
75 , with reduced YAP/TAZ nuclear shuttling and traction force generation.
76 ronments by increasing cell-ECM adhesion and traction force generation.
77 s may be a fundamental element of neutrophil traction force generation.
78 range over which FAs can accurately adapt to traction force generation.
79 imental approaches, we quantified retrograde traction force in Aplysia californica neuronal growth co
80     HspB1 is recruited to sites of increased traction force in cells geometrically constrained on mic
81 combined results indicate that the change in traction force in response to external cyclic stretch is
82  to determine how cells actively alter their traction force in response to long-term physiological cy
83 dies introduce a new model for regulation of traction force in which local actin assembly forces buff
84 eover, our results suggest that the level of traction force is directly correlated to the stiffness o
85  and paxillin FA area did not correlate with traction force magnitudes at single FAs, and this was co
86      In this article, we present single-cell traction force measurements using breast tumor cells emb
87 trate adhesion strength and myosin activity, traction force measurements, and mathematical modeling t
88 ments provided results consistent with total traction force measurements.
89      Conventional force-to-strain based cell traction force microscopies have low resolution which is
90                   Cell traction recorded via traction force microscopy (TFM) commonly takes place on
91                                              Traction force microscopy (TFM) enables determination of
92                                              Traction force microscopy (TFM) revealed that cells prod
93                                              Traction force microscopy (TFM) was used to establish th
94                      Using three-dimensional traction force microscopy and a double hydrogel sandwich
95 e whole-mount imaging, genetic ablation, and traction force microscopy and atomic force microscopy, w
96 igh nuclear tension that matches trends from traction force microscopy and from increased lamin-A,C.
97                                        Using traction force microscopy and microfluidic invasion devi
98                                              Traction force microscopy and time-lapse imaging reveal
99 ey are increasingly used in combination with traction force microscopy on soft elastic substrates.
100                                              Traction force microscopy revealed that tumor-associated
101                                              Traction force microscopy shows that lowering cholestero
102 y, a robust finite-element-method-based cell traction force microscopy technique is developed to esti
103                   We introduce a new type of traction force microscopy that in contrast to traditiona
104 netic control of RhoA, live-cell imaging and traction force microscopy to investigate the dynamics of
105                                  Here we use traction force microscopy to measure the forces exerted
106                                              Traction force microscopy was carried out on primary hum
107       These cell forces can be measured with traction force microscopy which inverts the equations of
108 mp recording, phase contrast microscopy, and traction force microscopy).
109  vinculin localization at the cell membrane, traction force microscopy, and phosphorylated myosin lig
110              However, many experiments, e.g. traction force microscopy, rely on fluorescence microsco
111                                Using Fourier traction force microscopy, we measured the spatiotempora
112                                        Using traction force microscopy, we show that cells exert sign
113                                        Using traction force microscopy, we show that increased bindin
114 d larger than was previously estimated using traction force microscopy-based methods.
115 l, we describe stimulated emission depletion traction force microscopy-STED-TFM (STFM), which allows
116  force and spatial resolution limitations of traction force microscopy.
117        This question is addressed here using traction force microscopy.
118 ctroscopy techniques may be smaller than the traction force of the unfoldase motor.
119  these observations, we propose that loss of traction force on ligand-bound integrin-beta3 causes rec
120 ntal data for shape, spreading dynamics, and traction force patterns of cells on micropatterned subst
121                       Our model captures the traction force patterns of small clusters of nonmotile c
122 at may be responsible for the variability in traction force production.
123  cells cultured on soft gels increased their traction force significantly, from 15 nN to 45 nN, doubl
124 ation, increased focal adhesions, and higher traction force than controls.
125 icity of the FA structure and the associated traction force to accurately sense ECM stiffness.
126  of NM-II into actin stress fiber provides a traction force to promote actin retrograde flow and foca
127 ess (1100 nN) and exhibited a larger drop in traction force with uniaxial stretch, but the percentage
128 nce cell migration and for the modulation of traction force, spreading, and migration by ECM stiffnes
129 reduces the ability of PSCs to generate high traction forces and adapt to extracellular mechanical cu
130 t myosin inhibition led to a decrease in the traction forces and an increase in arc radius, indicatin
131 ntly and irreversibly remodelled by cellular traction forces and by macroscopic strains.
132  flow with rapid and pronounced increases in traction forces and cell-cell stresses.
133 h monolayers exhibit oscillatory patterns of traction forces and intercellular stresses that tend to
134 eton strain energy states and cell-generated traction forces and used a Forster resonance energy tran
135 sions are disordered, and cell spreading and traction forces are decreased.
136            In migrating Xenopus mesendoderm, traction forces are generated by cells through integrin-
137       We applied TPs to reveal that cellular traction forces are heterogeneous within focal adhesions
138                 Engagement of CD28 increases traction forces associated with CD3 through the signalin
139 cellular biophysical state of cells generate traction forces at the basal side of the cells that are
140 e tumor cells exert higher integrin-mediated traction forces at the bulk and molecular levels, consis
141 und circular obstacles, and CIL accounts for traction forces at the edge.
142 ed of motion and magnitude of the associated traction forces at the level of a single cell.
143 ids and invaded the matrix while maintaining traction forces at the tips of persistent and newly form
144 zation, greater myosin activity and stronger traction forces compared to cells in the interior of the
145                 Piezo1 activity triggered by traction forces elicited influx of Ca(2+), a known modul
146                                              Traction forces exerted by adherent cells on their micro
147  exhibit stress relaxation, so that cellular traction forces exerted by cells remodel the ECM.
148 knowledge, microfluidic technique to measure traction forces exerted by confluent vascular endothelia
149 twork stiffness, which in turn augmented the traction forces generated by human adipose stem cells (h
150                         Here, we report that traction forces generated by T cells are regulated by dy
151 n of the E413K mutant desmin also alters the traction forces generation of single myoblasts lacking o
152 mosomal protein regulates actin dynamics and traction forces in motile keratinocytes.
153 tribution of cellular stresses from measured traction forces in motile tissues and show that such tra
154  We mapped the orientation of integrin-based traction forces in mouse fibroblasts and human platelets
155 showed that Aplysia growth cones can develop traction forces in the 10(0)-10(2) nN range during adhes
156 iderable variability in measurements of cell-traction forces indicates that they may not be the optim
157 aces, but other factors such as adhesion and traction forces may be equally important.
158 ges in length and spatiotemporal dynamics of traction forces measured in chemotaxing unicellular amoe
159                            T cells generated traction forces of 100 pN on arrays with both antibodies
160 d molecular tension probes (TPs) that report traction forces of adherent cells with high spatial reso
161 ermined the three-dimensional spatiotemporal traction forces of motile neutrophils at unprecedented r
162  their actin cytoskeletons in order to exert traction forces on and move directionally over the dermi
163 Mouse cells expressing the 5C.C7 TCR exerted traction forces on pillars presenting peptide-loaded MHC
164 ing wild-type tandem pairs, each cell exerts traction forces on stationary sites ( approximately 80%
165 matrix through adhesive sites, and can exert traction forces on the local matrix, causing its spatial
166                Indeed, the magnitude of cell traction forces on the underlying extracellular matrix i
167 lts suggest the profound impacts of cellular traction forces on their host ECM during development and
168 s using a method that does not depend on the traction forces or material properties.
169                               Strikingly, FA traction forces oscillate in time and space, and govern
170                    When actomyosin-dependent traction forces overcome substrate resistance, platelets
171 scopy technique is developed to estimate the traction forces produced by multiple isolated cells as w
172 tment of actin and myosin but also increased traction forces that rapidly propagate across the cell v
173 ECM) elasticity by gauging resistance to the traction forces they exert on the ECM.
174 hich cells exert actin cytoskeleton-mediated traction forces to sense the ECM stiffness.
175 d on a surface or to crawl, cells must apply traction forces to the underlying substrate via adhesion
176 ansion and contraction and apply coordinated traction forces to their environment.
177                                         Cell traction forces transmitted by FAs and integrin tensions
178 rcellular stress in monolayers from measured traction forces upon the substrate.
179 ovel method, to our knowledge, for measuring traction forces using an atomic force microscope (AFM) w
180  Combining measurements of cell-scale normal traction forces with FA monitoring, we show that the cel
181 lity of this method to correlatively overlap traction forces with spatial localization of proteins re
182 behaviors and parameters (e.g., adhesion and traction forces) to the collective migration of small gr
183 symmetric distribution of basal protrusions, traction forces, and apical aspect ratios that decreased
184 antly and irreversibly remodeled by cellular traction forces, as well as by macroscopic strains.
185 eases cytoskeletal remodeling, intracellular traction forces, cell migration and invasion, and anchor
186 n over a substrate by generating alternating traction forces, of up to 1.4 kPa, at each flank of the
187 ayers on stiffer substrates showed increased traction forces, vinculin at the cell membrane, and vinc
188 or receptor alpha-mediated contractility and traction forces, which are transduced to Fn through alph
189 in driving polarized motility and generating traction forces, yet little is known about how tension b
190 ate characterized by a broad distribution of traction forces.
191 d Fn fibres from being stretched by cellular traction forces.
192 cytoplasmic mobility, cellular movement, and traction forces.
193 ening coupled with increases in cell-exerted traction forces.
194 ns independent of cell spread area and total traction forces.
195 tor the dynamics of peripheral arc radii and traction forces.
196 hereby constraining the transmission of cell traction forces.
197 owever, most ice-sheet models estimate basal traction from satellite-derived surface velocity, withou
198 ns still maintain a regulatory role for cell traction generation and cell locomotion under the physic
199 rectional migration directly correlates with traction generation and is mediated by transforming grow
200 s the elongation, directional migration, and traction generation of breast cancer cells.
201 n (HFR) as well as movement trajectories and traction generation of individual HPCs, we find that the
202                                The resulting traction gradient within the growing FA favors SF format
203 f traction and that NA maturation depends on traction growth rate.
204      Twitching bacterial groups also produce traction hotspots, but with forces around 100 pN that fl
205 ution, these technologies are likely to gain traction in cutaneous oncology research and practice.
206 pigenetic alterations has gained significant traction in overcoming cancer cell resistance to various
207 gamification" of science has gained a lot of traction in recent years, and games that convey scientif
208 ch to decreasing cancer mortality has gained traction in recent years, evidenced by its inclusion in
209 n, we find that individual cells exert local traction in small hotspots with forces on the order of 5
210                       Amyloid PET is gaining traction in the clinical arena, but validity and cost-ef
211              This pursuit has recently found traction in the field of optomechanics in which a mechan
212 mpact of microbes on plants have gained much traction in the research literature, supporting diverse
213 an patient aged 67 years with vitreo-macular traction in the right eye, treated with Ocriplasmin, at
214 onstruction algorithm that resolves cellular tractions in diffraction-limited nascent adhesions (NAs)
215 c vitreomacular adhesion (VMA)/vitreomacular traction, including full-thickness macular hole (FTMH).
216 remained at the VMT stage, the mean angle of traction increased by only 1 degree throughout follow-up
217 auses a rapid and local increase in cellular traction, intercellular tension and tissue compaction.
218                                     However, traction is amplified approximately fivefold in groups.
219 oncept that has recently gained considerable traction is that micronutrients modulate gene expression
220 ased applications in food safety are gaining traction, it is crucial that we consider the effect the
221 s MH (82 scans), 25-line raster missed focal traction (<1500 mum) and MH in 5 scans (P=.07) and 7 sca
222 of foveal detachment in patients with myopic traction maculopathy without posterior vitreous detachme
223 mality associated with retinoschisis, myopic traction maculopathy, epiretinal membrane, vitreoretinal
224 d with PVD can occur in cases of high myopic traction maculopathy, especially in those without obviou
225 ich suppresses noise without underestimating traction magnitude.
226                                              Traction maps reveal that local cell responses to slow s
227    Removal of the anterio-posterior vitreous traction may play the main role and may help the spontan
228 istance or remodeled fibers at a distance by traction-mediated reorientation or aligned deposition ga
229  resonance (MR) imaging (MR arthrography and traction MR arthrography).
230  cystoid retinal edema (n = 6; 13%), retinal traction (n = 11; 23%), intralesional cavities (n = 28;
231                   None of the cases involved traction of the vitreomacular interface or posterior vit
232 retinal break (P <0.005) or visible vitreous traction on a retinal break (P = 0.04).
233         Inferior breaks and visible vitreous traction on a tear predicted failure.
234  hyaloid membrane creates anterior-posterior traction on the fovea, and, during detachment, retinal l
235         These actuators have recently gained traction on the one hand due to the technology push from
236        No preoperative evidence for vitreous traction on the optic disc or macula was seen in any eye
237 s are isolated, gliding produces low average traction on the order of 1 Pa.
238 of white vascularized tissue with associated traction on the retina and sometimes hemorrhage.
239         Retinal tear location and persistent traction on the retinal flap was evaluated with B-scan u
240  ultrasonography and OCT revealed persistent traction on the retinal tear flap in 19 and 15 eyes, res
241 ulopathy, epiretinal membrane, vitreoretinal traction, optic or scleral pit, or advanced glaucomatous
242 eal ocriplasmin injection without release of traction or closure of macular holes during follow-up.
243 reatment of retinoblastoma and may result in traction or rhegmatogenous retinal detachment along with
244 sistent despite CS or in case of threatening traction or visually significant epimacular membrane (28
245        However, precisely how and why the FA traction oscillates is unknown.
246               This underpins the observed FA traction oscillation and, importantly, broadens the ECM
247 growth and maturation thus culminate with FA traction oscillation to drive efficient FA mechanosensin
248 her stabilizes the FA and generates a second traction peak near the center of the FA.
249 oximal growth of the FA and contributes to a traction peak near the FA's distal tip.
250 tomyosin contractility, resulting in central traction peak oscillation.
251  motion of membrane components and acting as traction points for cell motility.
252 le performing a vitrectomy for vitreomacular traction posterior hyaloid membrane creates anterior-pos
253 pathic Epiretinal Membrane and Vitreomacular Traction Preferred Practice Pattern(R) (PPP) guidelines,
254 PATHIC EPIRETINAL MEMBRANE AND VITREOMACULAR TRACTION PREFERRED PRACTICE PATTERN(R) GUIDELINES: New e
255 pontaneous resolution (defined by release of traction), progression to full-thickness macular hole, a
256                                         Cell traction recorded via traction force microscopy (TFM) co
257  VMT stage was significantly associated with traction resolution (nasally P = .001, temporally P < .0
258                                      Besides traction retinal detachment, vision loss in IP can occur
259 acterized by fibrotic membrane formation and traction retinal detachment.
260 retinal surface and prevented development of traction retinal detachment.
261 le outcomes, but eyes undergoing surgery for traction/rhegmatogenous RD carry a more guarded prognosi
262 itize safety and strive to minimize vitreous traction, stabilize anterior chamber volume, maintain ca
263            The critical value of compressive traction stress at the transition from a protease-indepe
264 dent of matrix stiffness, suggesting that 3D traction stress is a key factor in triggering protease-m
265 fibroblasts impedes enhanced cell spreading, traction stress, and fibronectin fiber formation.
266 strate that the cytoskeletal stiffness, cell traction stress, and focal adhesion area were significan
267 organizing the distribution and size of high-traction-stress regions at the cell periphery.
268 ed the spatiotemporal evolution of shape and traction stresses and constructed traction tension kymog
269  microscopy (TFM) was used to establish that traction stresses are limited primarily to leading edge
270  influences the mode of cell invasion is the traction stresses generated by the cells in response to
271 the spatial and temporal evolution of the 3D traction stresses generated by the leukocytes and VECs t
272 ) with antisense morpholinos results in high traction stresses in follower row cells, misdirected pro
273 ment, consequently orchestrating anisotropic traction stresses that drive cell orientation and direct
274  cell motility is to govern the alignment of traction stresses that permit single-cell migration.
275  regulates the distribution and magnitude of traction stresses to maintain a constant strain energy.
276                        At low 3D compressive traction stresses, cells utilize bleb formation to inden
277 es the spatial distribution and magnitude of traction stresses.
278  required for the anisotropic orientation of traction stresses.
279 developed by the International Vitreomacular Traction Study Group by 2 independent masked observers.
280 examine the natural history of vitreomacular traction syndrome (VMTS) in the absence of other ocular
281                  Patients with vitreomacular traction syndrome, secondary ERM, or both were excluded.
282  shape and traction stresses and constructed traction tension kymographs to analyze cell motility as
283                                          The traction test (inverted grid with mice clinging to the u
284 at includes robust parameterisation of basal traction, the resistance to ice flow at the bed.
285  with 20/20 visual acuity and vitreo-macular traction treated with Ocriplasmin, and, for the first ti
286 itreomacular traction (VMT), vitreopapillary traction, vitreo-fold traction, vitreo-laser scar adhesi
287 (VMT), vitreopapillary traction, vitreo-fold traction, vitreo-laser scar adhesion, diminished foveal
288 y of patients with symptomatic vitreomacular traction (VMT) after Ocriplasmin treatment.
289 ent the management options for vitreomacular traction (VMT) and to recommend an individualized approa
290  at baseline and follow-up for vitreomacular traction (VMT) and vitreomacular adhesion (VMA), fluid,
291 al function in symptomatic VMA/vitreomacular traction (VMT) has not yet been documented, to our knowl
292 terior hyaloidal organization, vitreomacular traction (VMT), vitreopapillary traction, vitreo-fold tr
293 us studies have suggested that vitreoretinal traction (VRT) may contribute to the progression of neov
294 vitreoretinal interface (VRI), in particular traction (VRT), and the characteristics and progression
295 ical recovery with release of vitreo-macular traction was associated with a full functional recovery.
296                                Vitreoretinal traction was found in 39 eyes (40%).
297    Patients with any degree of vitreomacular traction were excluded from the analysis.
298 ear-old woman with symptomatic vitreomacular traction who received intravitreal ocriplasmin and exper
299 e of MGS is caused primarily by the vitreous traction with further possible formation of the retinal
300 hanges (9 eyes, 69.2%), including tangential traction with temporal vessel straightening concerning f

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