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1 al epiretinal membranes and/or vitreomacular traction.
2 ially in those without obvious vitreomacular traction.
3 ernal rounding forces and cell-intercalation traction.
4 ibroblasts were predominant in vitreomacular traction.
5 ascular vitreous veil with localized retinal traction.
6 ular hole after vitrectomy for vitreomacular traction.
7 ular hole after vitrectomy for vitreomacular traction.
8 hopsia in the right eye due to vitreomacular traction.
9 ive trend in identifying focal vitreomacular traction.
10 they are still small and have vitreomacular traction.
11 actile groups within clusters for generating traction.
12 ity and release of symptomatic vitreomacular traction.
13 s closure after vitrectomy for vitreomacular traction.
14 bstrate junctions with no apparent effect on traction.
15 ts to discover inhibitors have gained little traction.
16 ithout the need to explicitly determine cell tractions.
17 of variations in matrix stiffness with cell tractions.
18 s (57.1%), vasculitis (57.1%), vitreoretinal traction (57.1%), and chronic macular edema (ME) (71.4%)
20 roduced by actin polymerization can generate traction across the plasma membrane by transmission thro
21 l motility, whereas the differences in their traction adhesion dynamics suggest that these two strain
23 step-wise fashion, mainly forming stationary traction adhesions along their anterior-posterior axes a
25 hanced iOCT imaging revealed strong vitreous traction and adhesion above the macula and optic disc.
27 is verified experimentally by comparing cell traction and F-actin retrograde flow for two cell types
30 ography showed the release of vitreo-macular traction and multifocal electroretinogram responses show
32 c retinopathy, vitreous hemorrhage, combined traction and rhegmatogenous retinal detachment, or lens
33 hat NAs transmit a distinguishable amount of traction and that NA maturation depends on traction grow
39 Patient-funded trials (PFTs) are gaining traction as a means of accelerating clinical translation
41 ptical coherence tomography (OCT) has gained traction as an important adjunct for clinical decision m
42 ime correlated random walks" are now gaining traction as models of scale-finite animal movement patte
43 Ca(2+) oscillations induced by laser-tweezer-traction at the plasma membrane, providing a model to st
44 M) revealed that cells produced the greatest tractions at the cell periphery, where distinct types of
45 forces in motile tissues and show that such traction-based stresses match those calculated from inst
46 clinical practice who had possible UIP with traction bronchiectasis on HRCT and had not undergone su
47 f nintedanib if they had honeycombing and/or traction bronchiectasis plus reticulation, without atypi
49 ore susceptible to damage from vitreomacular traction by rotational and/or acceleration-deceleration
50 thick stage 3 membranes with anteroposterior traction concerning for progression to stage 4 ROP (3 ey
53 s with small macular holes and vitreomacular traction during vitrectomy after intravitreal ocriplasmi
54 this study was to evaluate the extension and traction effects of posterior vitreous detachment (PVD)
60 al cells on stiff substrates decreased their traction force (from 300 nN to 100 nN) and spread area (
61 ls with inhibited myosin II motors increased traction force (from 41 nN to 63 nN) and slightly reorie
62 , to our knowledge, a novel method to assess traction force after long-term (24 h) uniaxial or biaxia
63 stretch, the cells had similar decreases in traction force and area and reoriented perpendicular to
65 study, we examined the relationship between traction force and vinculin-paxillin localization to sin
66 However, the study of how cell-generated traction force changes in response to stretch is general
67 le-particle tracking (MPT) microrheology and traction force cytometry to probe how genetic induction
70 hanism by which FN fibril assembly regulates traction force dynamics in response to mechanical stimul
72 model that predicts the dynamics of cellular traction force generation and subsequent assembly of fib
73 inding, vinculin was necessary for efficient traction force generation in 3D collagen without affecti
74 he talin rod R3 subdomain decreases cellular traction force generation, which affects talin and vincu
79 imental approaches, we quantified retrograde traction force in Aplysia californica neuronal growth co
80 HspB1 is recruited to sites of increased traction force in cells geometrically constrained on mic
81 combined results indicate that the change in traction force in response to external cyclic stretch is
82 to determine how cells actively alter their traction force in response to long-term physiological cy
83 dies introduce a new model for regulation of traction force in which local actin assembly forces buff
84 eover, our results suggest that the level of traction force is directly correlated to the stiffness o
85 and paxillin FA area did not correlate with traction force magnitudes at single FAs, and this was co
87 trate adhesion strength and myosin activity, traction force measurements, and mathematical modeling t
95 e whole-mount imaging, genetic ablation, and traction force microscopy and atomic force microscopy, w
96 igh nuclear tension that matches trends from traction force microscopy and from increased lamin-A,C.
99 ey are increasingly used in combination with traction force microscopy on soft elastic substrates.
102 y, a robust finite-element-method-based cell traction force microscopy technique is developed to esti
104 netic control of RhoA, live-cell imaging and traction force microscopy to investigate the dynamics of
109 vinculin localization at the cell membrane, traction force microscopy, and phosphorylated myosin lig
115 l, we describe stimulated emission depletion traction force microscopy-STED-TFM (STFM), which allows
119 these observations, we propose that loss of traction force on ligand-bound integrin-beta3 causes rec
120 ntal data for shape, spreading dynamics, and traction force patterns of cells on micropatterned subst
123 cells cultured on soft gels increased their traction force significantly, from 15 nN to 45 nN, doubl
126 of NM-II into actin stress fiber provides a traction force to promote actin retrograde flow and foca
127 ess (1100 nN) and exhibited a larger drop in traction force with uniaxial stretch, but the percentage
128 nce cell migration and for the modulation of traction force, spreading, and migration by ECM stiffnes
129 reduces the ability of PSCs to generate high traction forces and adapt to extracellular mechanical cu
130 t myosin inhibition led to a decrease in the traction forces and an increase in arc radius, indicatin
133 h monolayers exhibit oscillatory patterns of traction forces and intercellular stresses that tend to
134 eton strain energy states and cell-generated traction forces and used a Forster resonance energy tran
139 cellular biophysical state of cells generate traction forces at the basal side of the cells that are
140 e tumor cells exert higher integrin-mediated traction forces at the bulk and molecular levels, consis
143 ids and invaded the matrix while maintaining traction forces at the tips of persistent and newly form
144 zation, greater myosin activity and stronger traction forces compared to cells in the interior of the
148 knowledge, microfluidic technique to measure traction forces exerted by confluent vascular endothelia
149 twork stiffness, which in turn augmented the traction forces generated by human adipose stem cells (h
151 n of the E413K mutant desmin also alters the traction forces generation of single myoblasts lacking o
153 tribution of cellular stresses from measured traction forces in motile tissues and show that such tra
154 We mapped the orientation of integrin-based traction forces in mouse fibroblasts and human platelets
155 showed that Aplysia growth cones can develop traction forces in the 10(0)-10(2) nN range during adhes
156 iderable variability in measurements of cell-traction forces indicates that they may not be the optim
158 ges in length and spatiotemporal dynamics of traction forces measured in chemotaxing unicellular amoe
160 d molecular tension probes (TPs) that report traction forces of adherent cells with high spatial reso
161 ermined the three-dimensional spatiotemporal traction forces of motile neutrophils at unprecedented r
162 their actin cytoskeletons in order to exert traction forces on and move directionally over the dermi
163 Mouse cells expressing the 5C.C7 TCR exerted traction forces on pillars presenting peptide-loaded MHC
164 ing wild-type tandem pairs, each cell exerts traction forces on stationary sites ( approximately 80%
165 matrix through adhesive sites, and can exert traction forces on the local matrix, causing its spatial
167 lts suggest the profound impacts of cellular traction forces on their host ECM during development and
171 scopy technique is developed to estimate the traction forces produced by multiple isolated cells as w
172 tment of actin and myosin but also increased traction forces that rapidly propagate across the cell v
175 d on a surface or to crawl, cells must apply traction forces to the underlying substrate via adhesion
179 ovel method, to our knowledge, for measuring traction forces using an atomic force microscope (AFM) w
180 Combining measurements of cell-scale normal traction forces with FA monitoring, we show that the cel
181 lity of this method to correlatively overlap traction forces with spatial localization of proteins re
182 behaviors and parameters (e.g., adhesion and traction forces) to the collective migration of small gr
183 symmetric distribution of basal protrusions, traction forces, and apical aspect ratios that decreased
184 antly and irreversibly remodeled by cellular traction forces, as well as by macroscopic strains.
185 eases cytoskeletal remodeling, intracellular traction forces, cell migration and invasion, and anchor
186 n over a substrate by generating alternating traction forces, of up to 1.4 kPa, at each flank of the
187 ayers on stiffer substrates showed increased traction forces, vinculin at the cell membrane, and vinc
188 or receptor alpha-mediated contractility and traction forces, which are transduced to Fn through alph
189 in driving polarized motility and generating traction forces, yet little is known about how tension b
197 owever, most ice-sheet models estimate basal traction from satellite-derived surface velocity, withou
198 ns still maintain a regulatory role for cell traction generation and cell locomotion under the physic
199 rectional migration directly correlates with traction generation and is mediated by transforming grow
201 n (HFR) as well as movement trajectories and traction generation of individual HPCs, we find that the
204 Twitching bacterial groups also produce traction hotspots, but with forces around 100 pN that fl
205 ution, these technologies are likely to gain traction in cutaneous oncology research and practice.
206 pigenetic alterations has gained significant traction in overcoming cancer cell resistance to various
207 gamification" of science has gained a lot of traction in recent years, and games that convey scientif
208 ch to decreasing cancer mortality has gained traction in recent years, evidenced by its inclusion in
209 n, we find that individual cells exert local traction in small hotspots with forces on the order of 5
212 mpact of microbes on plants have gained much traction in the research literature, supporting diverse
213 an patient aged 67 years with vitreo-macular traction in the right eye, treated with Ocriplasmin, at
214 onstruction algorithm that resolves cellular tractions in diffraction-limited nascent adhesions (NAs)
215 c vitreomacular adhesion (VMA)/vitreomacular traction, including full-thickness macular hole (FTMH).
216 remained at the VMT stage, the mean angle of traction increased by only 1 degree throughout follow-up
217 auses a rapid and local increase in cellular traction, intercellular tension and tissue compaction.
219 oncept that has recently gained considerable traction is that micronutrients modulate gene expression
220 ased applications in food safety are gaining traction, it is crucial that we consider the effect the
221 s MH (82 scans), 25-line raster missed focal traction (<1500 mum) and MH in 5 scans (P=.07) and 7 sca
222 of foveal detachment in patients with myopic traction maculopathy without posterior vitreous detachme
223 mality associated with retinoschisis, myopic traction maculopathy, epiretinal membrane, vitreoretinal
224 d with PVD can occur in cases of high myopic traction maculopathy, especially in those without obviou
227 Removal of the anterio-posterior vitreous traction may play the main role and may help the spontan
228 istance or remodeled fibers at a distance by traction-mediated reorientation or aligned deposition ga
230 cystoid retinal edema (n = 6; 13%), retinal traction (n = 11; 23%), intralesional cavities (n = 28;
234 hyaloid membrane creates anterior-posterior traction on the fovea, and, during detachment, retinal l
240 ultrasonography and OCT revealed persistent traction on the retinal tear flap in 19 and 15 eyes, res
241 ulopathy, epiretinal membrane, vitreoretinal traction, optic or scleral pit, or advanced glaucomatous
242 eal ocriplasmin injection without release of traction or closure of macular holes during follow-up.
243 reatment of retinoblastoma and may result in traction or rhegmatogenous retinal detachment along with
244 sistent despite CS or in case of threatening traction or visually significant epimacular membrane (28
247 growth and maturation thus culminate with FA traction oscillation to drive efficient FA mechanosensin
252 le performing a vitrectomy for vitreomacular traction posterior hyaloid membrane creates anterior-pos
253 pathic Epiretinal Membrane and Vitreomacular Traction Preferred Practice Pattern(R) (PPP) guidelines,
254 PATHIC EPIRETINAL MEMBRANE AND VITREOMACULAR TRACTION PREFERRED PRACTICE PATTERN(R) GUIDELINES: New e
255 pontaneous resolution (defined by release of traction), progression to full-thickness macular hole, a
257 VMT stage was significantly associated with traction resolution (nasally P = .001, temporally P < .0
261 le outcomes, but eyes undergoing surgery for traction/rhegmatogenous RD carry a more guarded prognosi
262 itize safety and strive to minimize vitreous traction, stabilize anterior chamber volume, maintain ca
264 dent of matrix stiffness, suggesting that 3D traction stress is a key factor in triggering protease-m
266 strate that the cytoskeletal stiffness, cell traction stress, and focal adhesion area were significan
268 ed the spatiotemporal evolution of shape and traction stresses and constructed traction tension kymog
269 microscopy (TFM) was used to establish that traction stresses are limited primarily to leading edge
270 influences the mode of cell invasion is the traction stresses generated by the cells in response to
271 the spatial and temporal evolution of the 3D traction stresses generated by the leukocytes and VECs t
272 ) with antisense morpholinos results in high traction stresses in follower row cells, misdirected pro
273 ment, consequently orchestrating anisotropic traction stresses that drive cell orientation and direct
274 cell motility is to govern the alignment of traction stresses that permit single-cell migration.
275 regulates the distribution and magnitude of traction stresses to maintain a constant strain energy.
279 developed by the International Vitreomacular Traction Study Group by 2 independent masked observers.
280 examine the natural history of vitreomacular traction syndrome (VMTS) in the absence of other ocular
282 shape and traction stresses and constructed traction tension kymographs to analyze cell motility as
285 with 20/20 visual acuity and vitreo-macular traction treated with Ocriplasmin, and, for the first ti
286 itreomacular traction (VMT), vitreopapillary traction, vitreo-fold traction, vitreo-laser scar adhesi
287 (VMT), vitreopapillary traction, vitreo-fold traction, vitreo-laser scar adhesion, diminished foveal
289 ent the management options for vitreomacular traction (VMT) and to recommend an individualized approa
290 at baseline and follow-up for vitreomacular traction (VMT) and vitreomacular adhesion (VMA), fluid,
291 al function in symptomatic VMA/vitreomacular traction (VMT) has not yet been documented, to our knowl
292 terior hyaloidal organization, vitreomacular traction (VMT), vitreopapillary traction, vitreo-fold tr
293 us studies have suggested that vitreoretinal traction (VRT) may contribute to the progression of neov
294 vitreoretinal interface (VRI), in particular traction (VRT), and the characteristics and progression
295 ical recovery with release of vitreo-macular traction was associated with a full functional recovery.
298 ear-old woman with symptomatic vitreomacular traction who received intravitreal ocriplasmin and exper
299 e of MGS is caused primarily by the vitreous traction with further possible formation of the retinal
300 hanges (9 eyes, 69.2%), including tangential traction with temporal vessel straightening concerning f
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